WEEK 10 UPPER GI Flashcards

(46 cards)

1
Q

Main functions

OF THE GI

A

Ingestion
Digestion
Absorption
Elimination

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2
Q

GI ASSESSMENT

A

Subjective –
Past health history
Medications
Surgery, treatments

Objective –
Physical exam

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3
Q

GI Diagnostic Studies

A

Signed consent

Educate patient

Ensure appropriate prep

Check for allergies or drugs, or contrast media

GI tract often must be cleansed prior
Often uses a ‘tracer’, or contrast
Watch for dehydration during preps

Main types of studies:
Radiological 
Endoscopy
Biopsies
Lab testing (LFT)
Paracentesis
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4
Q

Malnutrition

A

Consider determinants of health and healthy living

Poor nutrition is a key preventable risk factor for major chronic diseases

Nurses can have a strong influence on the nutritional practices of our patients/families

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5
Q

Over-nutrition

A

Ingestion of more food than is required (obesity)

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6
Q

Under-nutrition (focus for PNH401)

A

Poor nourishment, r/t inadequate diet, diseases interfering with appetite and/or assimilation of ingested food

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7
Q

Protein – Calorie Malnutrition

A

High risk of delayed wound healing, susceptiblility to infection, decreased immune function

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8
Q

Lab – FOR MALNURTITION

A

 pre albumin indicates severity; transferrin (protein)

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9
Q

Anthropometric assessments –

A

skinfold thickness, midarm muscle

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10
Q

ASSESSMENT FOR MALNUTRITION

A

Key Areas: skin, eyes, mouth, muscles, CNS
Muscles – wasted, flabby
Delayed wound healing – d/t  protein & WBC’s
Anemia –  folic acid, & iron

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11
Q

Main Goals of Care:

FOR MALNUTRITION

A

Achieve weight gain
Consume appropriate calories per day
Have no adverse consequences

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12
Q

Wounds –

A

require increased protein

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13
Q

Fever –

A

require increased protein and calories

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14
Q

Dysphagia –

A

Difficulty swallowing

Consider type of diet (puree, soft, thickened)

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15
Q

Oral thrush –

A

candida albicans, yeast like fungus

noted in debilitated patients, those with ongoing antibiotics, or steroid therapy

pearly bluish white milk curd on tongue, mucosa of mouth, painful, yeasty breath

Treat – nystatin oral rinses; good oral hygiene

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16
Q

GERD

A

Acid related disorder of the esophagus
Main complaint – Heart burn

Any clinically significant symptomatic condition presumed to be 20 reflux of gastric contents into lower esophagus

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17
Q

Multiple factors contribute and/or cause

GERD

A

Can also include incompetent sphincter, hiatal hernia, decreased clearance of esophagus, decreased gastric emptying

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18
Q

Complications/Concerns

OF GERD

A

Esophagitis

Repeat irritation can cause scar tissue stricture

Barrett’s esophagus – pre cancerous lesion

Aspiration pneumonia

Bronchospasm, laryngospasm

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19
Q

Manage via lifestyle modifications +/- drugs

FOR GERD

A

Elevate HOB
High protein, low fat diet
Avoid foods that decrease LES sphincter (Table 44-7)
Antacids
Quit smoking
Avoid foods that increase acid secretion (milk)

20
Q

GERD PHARM

A

– Antisecretory agents, H2 Receptor Blockers, Proton pump inhibitors, Prokinetic drugs, Cholinergic drugs

Pharm – often used in a ‘step up’ or ‘step down’ approach
(start with antacids and move up; or start with PPI Rx and over time titrate down to OTC)

Surgery is a late option –

21
Q

GERD ONGOING HEALTH TEACHING

A

Weight loss
Small frequent meals
Avoidance of smoking, alcohol, caffeine
Remain upright for 2 -3 hours post eating
HOB elevated
Meds with magnesium  diarrhea; aluminum constipation
Observe for drug effectiveness
If surgery – usual post op care, usually a high incision; DB&C

22
Q

Hiatal Hernia

A

Herniation of a portion of stomach into the esophagus through an opening (hiatus) – usually with no symptoms
AKA – diaphragmatic hernia, or esophageal hernia

More often in women than men
Possible r/t weakening muscles of diaphragm, obesity (increased intra abdominal pressure, pregnancy, ascites, tumours, intense activity, heavy lifting on ongoing basis)

23
Q

Hiatal Hernia

A

May be asymptomatic and found on routine CXR

Similar presentation if symptomatic as GERD

Therefore similar care as in GERD

Surgical care is to enhance the integrity of the LES

24
Q

Gastritis

A

Inflammation of the gastric mucosa

Acute or chronic
Diffuse or localized

Likely some connection to H. Pylori infection and gastric cancer

25
Gastritis - Assessment | ACUTE
``` Anorexia N & V Epigastric tenderness Feeling of fullness Hemmorhage (single symptom; associated with ETOH abuse) ```
26
Gastritis - Assessment | CHRONIC
As above + Anemia (cobalamin deficiency) Note: gastritis is self limiting, and the mucosa heals well
27
Gastritis – Care | ACUTE
Eliminate the cause Supportive care for symptoms Antiemetics Observe for dehydration if vomiting, nausea Start with clear fluids and increase to bland solid food If hemmorage likely – monitor VS carefully and follow care for UGI bleed Pharma – reducing irritation of the mucosa; symptomatic relief
28
Gastritis -- Care | CHRONIC
Evaluate and eliminate cause (ETOH, medications, H. Pylori) Antibiotics for H. Pylori Anemia – Cobalamin injections Small frequent meals Antacids after meals No SMOKING
29
LOOK FOR THE TABLE FOR THE Pharmacology for Gastritis, GERD, HH
.
30
Peptic Ulcer Disease (PUD)
Erosion of the GI mucosa from digestive action of HCL and pepsin Any section of the GI tract that comes in contact with gastric secretions is susceptible
31
PUD – Assessment
``` Inquire about ETOH abuse, smoking, caffeine Any chronic diseases? Medication use – NSAIDs, ASA Recent weight loss N & V? Stool changes to black tarry? Burning, when does it occur, where? Epigastric tenderness on palpation Anemia? ```
32
PUD GOAL
Reduce degree of gastric acidity | Enhance mucosal defense mechanisms
33
PUD TX
``` Consists of: Adequate rest Dietary modification Drug therapy Elimination of smoking and alcohol Long-term follow-up care Stress management ```
34
PUD Generally treated in ambulatory care clinics
May require upper GI scope, barium swallow CBC, LFT’s, Lytes Ulcer healing requires many weeks of therapy. Pain disappears after 3 to 6 days. Aspirin and nonselective NSAIDs may be stopped Smoking cessation Similar pharm as for GERD, HH, Gastritis Avoid foods/beverages irritating to stomach 6 small meals per day Bland food
35
Three major complications include | PUD
Hemorrhage (most common) Perforation Gastric outlet obstruction
36
PUD Develops from erosion of
Granulation tissue found at base of ulcer during healing | Ulcer through a major blood vessel
37
Upper GI Bleed (UGI Bleed)
High association with morbidity and mortality Mortality rate 6 to 10% Often in women, older adults, r/t use of NSAID’s Can be sudden onset, or occult bleeding – both major and significant Severity of bleed depends on type of vessel Can be an emergency, depending on volume of blood lost, and if continuous Often will stop on own Cause must be identified and corrected
38
Esophageal | BLEED
Chronic esophagitis Mallory-Weiss tear (from severe retching, vomiting) Esophageal varices (cirrhosis of liver)
39
Stomach and Duodenal | BLEED
Erosion of blood vessel from an ulcer Ingestion of drugs, ETOH Tumours, vascular lesions that erode
40
BLEEDS NURSING ASSESSMENT
Close monitoring of VS – BP and HR Q 15 mins Peripheral perfusion Observe for shock Once stable, do full assessment
41
UGI Bleeds – Initial Care
Obtain patient history – think of source, cause Lab studies – CBC, BUN, lytes, BS, PTT Prepare for blood transfusions (previous reactions?) Large bore IV lines, more than one Foley catheter IV fluids – RL – volume Start IV PPI’s
42
Endoscopy
Ability to sclerose for hemostasis, clips to clamp bleeder, heat probes to coagulate blood
43
Surgery
If bleeding not being controlled | High incidence of recurrent bleed within 5 years
44
Obtain detailed history – what precipitated bleed?
``` All medications -- N, V, weight loss Stools – characteristic Weakness, fainting Epigastric pain, abdominal pain ```
45
BLEEDS CARE
Maintain IV line Monitor U/O If older, or CVD, monitor for fluid overload Avoid ‘red’ foods Be aware of stool colour – black is a slow bleed; bright red is acute and from the lower GI If bloody vomitus, and no tarry stools, bleed is of short duration Introduce fluids slowly, monitor for bleeding recurrence ETOH abuse – observe for DT’s, withdrawal
46
BLEEDS EXTRA NOTES
Always consider which patients are at high risk (chronic gastritis, PUD, previous bleed, bleeding tendencies) If requires medications likely to precipitate bleed, ensure it is EC if possible, or changed to less toxic drug, and/or is administered with meds for ulcer preventers