week 11- nutrition in older adults Flashcards
(44 cards)
changes with aging
physical, financial, lifestyle, health, mobility, social schedule
physiological changes with aging
- changes in taste and smell
- xerostomia (dec saliva) and dentures
- reduced energy expenditure and metabolic rate
social changes with aging
- may lose significant other or friends
- potential for chronic illness, reduced mobility, depression or dementia
- often consume a tea and toast type of diet
challenges with maintaining good nutrition
- 34% of canadians 65+ are at nutritional risk
- 22% had weight change >10lbs in previous 6 months
- 15% skipped meals every day
- 18% ate <2 servings of F&V daily
- 23% never or rarely ate with someone
- 18% found cooking a chore
healthy adult BMI
18.5-25
healthy older adult BMI
25-32, more reserves if they got sick
protein requirements older adults
- needs are similar to younger adulthood
- adequate daily intake needed to support losses, be mindful of renal function
- consider sources that are appropriate for older adult
fat requirements older adults
increase in FA and dec saturated fats
carb requirements older adults
- necessary for optimal brain function
- fibre is important in preventing constipation
- excess carbs are stored as fat
GI system of older adults
- reduced gastric acid leads to a dec in B12 and iron absorption
- slowed gastric motility, so water and fibre become increasingly important
consequences of decreased gastric acid secretion
low B12 = pernicious anemia (change in RBC shape), iron deficiency anemia
changes to musculoskeletal system OA
- need to support bone density through weight bearing exercises
- vitamin D synthesis decreases with age, 400IU supplement recommended for 50+
- calcium supplements maybe required if intake is inadequate
calcium supplements
- older adults RDA is 1200mg/d
- several studies have linked calcium supplements to CVD risk
- recommendation that calcium come from dietary sources where possible
CVD
- second leading cause of death in canada
- includes ischemic HD, cerebrovascular disease, peripheral vascular disease, heart failure, congenital HD, rheumatic HD
- death rate of CVD has decreased by 21% in the last 20 years
heart disease progression
- people with HT, diabetes, obesity, smoking and high cholesterol are at risk of developing CVD
- we see atherosclerosis (build up of plaque, narrowing of vessels)
- leads to structural heart disease (ischemic HD, MI, arrhythmia)
- eventually they develop symptoms of heart failure (SOB, fatigue, decreased exercise tolerance)
non-modifiable CVD risk factors
age, sex (men have higher risk until postmenopausal age), family history
modifiable CVD risk factors
HT, cholesterol, diabetes, overweight, diet, alcohol, physical inactivity, smoking, stress
LDL
- carries cholesterol to cells, increases risk of atherosclerosis (bad cholesterol)
- LDL should be <3.5mmol/L
- LDL level <2.6mmol/L is optimal
HDL
- retrieves cholesterol and returns it to the liver, decreases risk of heart disease
- HDL >1mmol/L for men, >1.3mmol/L for women
oxidized LDL (VLDL)
transports cholesterol into arterial lining
foam cells
hold oxidized LDL within the arterial wall, contribute to plaque formation and inflammation
development of atherosclerosis
- inflammatory changes to endothelium attract monocytes
- they migrate through endothelial layer, leading to inflammatory changes and becoming macrophages
- macrophages engulf oxidized LDLs, becoming foam cells
- SM cells mix with foam cells and mineralize with calcium, leading to a hardened plaque
- inflammation weakens the fibrous cap of plaque, increasing the risk of a thrombus
atherosclerosis
plaque build-up on arterial wall
formation of a blood clot
- plaque ruptures and forms a thrombus (partial occlusion)
- may turn into a thrombosis (total occlusion)
- if a part of the thrombosis breaks off, it’s called an embolism
- blockage of a BV leads to oxygen deprivation and necrosis of surrounding tissues