week 2 Flashcards

1
Q

Hydrostatic Pressure

A

Force within a fluid compartment

Major force that pushes water out of vascular system at capillary level

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2
Q

Oncotic Pressure

A

Osmotic pressure exerted by colloids in solution

Protein is a major colloid.

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3
Q

Fluid Shifts AND EDEMA

A

Plasma-to-interstitial fluid shift results in edema.

Elevation of hydrostatic pressure
Decrease in plasma oncotic pressure
Elevation of interstitial oncotic pressure

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4
Q

Effects of Edema (cont’d.)

A

-Functional impairment:
Restricts range of joint movement
Reduced vital capacity
Impaired diastole

-Pain:
Edema exerts pressure on nerves locally.
Headache with cerebral edema
Stretching of capsule in organs (kidney, liver)

-Dental practice:
Difficult to take accurate impressions
Dentures do not fit well

-Edema in skin:
Susceptible to tissue breakdown from pressure
Impaired arterial circulation
Ischemia leading to tissue breakdown

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5
Q

Fluid Movement Between ECF and ICF

A

-Water deficit (increased ECF)
Associated with symptoms that result from cell shrinkage as water is pulled into vascular system

-Water excess (decreased ECF)
Develops from gain or retention of excess water

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6
Q

Fluid Spacing

A

First spacing
Normal distribution of fluid in ICF and ECF

Second spacing
Abnormal accumulation of interstitial fluid (edema)

Third spacing
Fluid accumulation in part of body where it is not easily exchanged with ECF

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7
Q

Hypothalamic Regulation

A
  • Osmoreceptors in hypothalamus sense fluid deficit or increase.
  • -Stimulates thirst and antidiuretic hormone (ADH) release
  • -Result in increased free water and decreased plasma osmolarity
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8
Q

Pituitary Regulation

A

Under control of hypothalamus, posterior pituitary releases ADH.
Stress, nausea, nicotine, and morphine also stimulate ADH release.

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9
Q

Adrenal Cortical Regulation

A

Releases hormones to regulate water and electrolytes
Glucocorticoids: Cortisol
Mineralocorticoids: Aldosterone

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10
Q

Renal Regulation

A

Primary organs for regulating fluid and electrolyte balance

-Adjusting urine volume: Selective reabsorption of water and electrolytes. Renal tubules are sites of action of ADH and aldosterone.

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11
Q

Cardiac Regulation

A

Natriuretic peptides are antagonists to the RAAS.

  • -Produced by cardiomyocytes in response to increased atrial pressure
  • -Suppress secretion of aldosterone, renin, and ADH to decrease blood volume and pressure
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12
Q

Gastrointestinal Regulation

A

Oral intake accounts for most water.

Small amounts of water are eliminated by gastrointestinal tract in feces.

Diarrhea and vomiting can lead to significant fluid and electrolyte loss.

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13
Q

Insensible Water Loss

A

Invisible vaporization from lungs and skin to regulate body temperature

  • -Approximately 600 to 900 mL/day is lost.
  • -No electrolytes are lost.
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14
Q

Age-Related Considerations

A

Structural changes in kidneys decrease ability to conserve water.

Hormonal changes lead to decrease in ADH and ANP.

Loss of subcutaneous tissue leads to increased loss of moisture.

Reduced thirst mechanism results in decreased fluid intake.

Nurse must assess for these changes and implement treatment accordingly.

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15
Q

Age-Related Considerations

A

Structural changes in kidneys decrease ability to conserve water.

Hormonal changes lead to decrease in ADH and ANP.

Loss of subcutaneous tissue leads to increased loss of moisture.

Reduced thirst mechanism results in decreased fluid intake.

Nurse must assess for these changes and implement treatment accordingly.

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16
Q

Fluid and Electrolyte Imbalances

A

Common in most patients with major illness or injury

  • -Directly caused by illness or disease (burns or heart failure)
  • -Result of therapeutic measures (IV fluid replacement or diuretics)
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17
Q

Extracellular Fluid Volume Imbalances

A

ECF volume deficit (hypovolemia)

  • -Abnormal loss of normal body fluids (diarrhea, fistula drainage, hemorrhage), inadequate intake, or plasma-to-interstitial fluid shift
  • -Treatment: Replace water and electrolytes with balanced IV solutions.

Fluid volume excess (hypervolemia)

  • -Excessive intake of fluids, abnormal retention of fluids (HF), or interstitial-to-plasma fluid shift
  • -Treatment: Remove fluid without changing electrolyte composition or osmolality of ECF.
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18
Q

Nursing Management Nursing Diagnoses

A
-Hypovolemia
Deficient fluid volume
Decreased cardiac output
Risk for deficient fluid volume
Potential complication: Hypovolemic shock
-Hypervolemia
Excess fluid volume
Risk for imbalanced fluid volume
Ineffective airway clearance
Risk for impaired skin integrity
Disturbed body image
Potential complications: Pulmonary edema, ascites
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19
Q

Fluid Deficit―Dehydration

A

-Insufficient body fluid
Inadequate intake
Excessive loss
Both

-Fluid loss often measured by change in body weight

-Dehydration more serious in infants and older adults
Water loss may be accompanied by loss of electrolytes and proteins (e.g., diarrhea).

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20
Q

Causes of Dehydration

A
  • Vomiting and diarrhea
  • Excessive sweating with loss of sodium and water
  • Diabetic ketoacidosis (Loss of fluid, electrolytes, and glucose in the urine)
  • Insufficient water intake in older adults or unconscious persons
  • Use of concentrated formula in infants
21
Q

Effects of Dehydration

A
  • Dry mucous membranes in the mouth
  • Decreased skin turgor or elasticity
  • Lower blood pressure, weak pulse, and fatigue
  • Decreased mental function, confusion, loss of consciousness
22
Q

Manifestations of Dehydration

A
Decreased skin turgor and dry mucous membranes
Sunken eyes
Sunken fontanelles in infant 
Lower blood pressure, rapid weak pulse
Increased hematocrit
Increased temperature
Decreasing level of consciousness
Urine―low volume & high specific gravity
23
Q

Nursing Management Nursing Implementation

A
I & O
Monitor cardiovascular changes.
Assess respiratory changes.
Daily weights
Skin assessment

Neurological function:
LOC,PERLA, Voluntary movement of extremities, Muscle strength, Reflexes

24
Q

Calcium (Ca)

A
Excess:
Hypercalcemia
Thirst
CNS deterioration
Increased interstitial fluid
Deficit:
Hypocalcemia
Tetany
Chvostek’s, Trousseau’s signs 
Muscle twitching
CNS changes
ECG changes
25
Magnesium (Mg)
``` EXCESS: Hypermagnesemia Loss of deep tendon reflexes (DTRs) Depression of CNS Depression of neuromuscular function ``` DEFICIT: Hypomagnesemia Hyperactive DTRs CNS changes
26
Sodium (Na)
``` EXCESS: Hypernatremia Thirst CNS deterioration Increased interstitial fluid ``` DEFICIT: Hyponatremia CNS deterioration
27
Potassium (K)
``` EXCESS: Hyperkalemia Ventricular fibrillation ECG changes CNS changes ``` ``` DEFICIT: Hypokalemia Bradycardia ECG changes CNS changes ```
28
Sodium
-Imbalances typically associated with parallel changes in osmolality - Plays a major role in: - -ECF volume and concentration - -Generation and transmission of nerve impulses - -Acid-base balance
29
Hypernatremia
Elevated serum sodium occurring with water loss or sodium gain Causes hyperosmolality leading to cellular dehydration Primary protection is thirst from hypothalamus. Manifestations Thirst, lethargy, agitation, seizures, and coma Impaired LOC Produced by clinical states Central or nephrogenic diabetes insipidus
30
Hyponatremia
Results from loss of sodium-containing fluids or from water excess. Manifestations Confusion, nausea, vomiting, seizures, and coma
31
Potassium
Major ICF cation ``` Necessary for Transmission and conduction of nerve and muscle impulses Cellular growth Maintenance of cardiac rhythms Acid-base balance ``` ``` Sources Fruits and vegetables (bananas and oranges) Salt substitutes Potassium medications (PO, IV) Stored blood ```
32
Hyperkalemia
High serum potassium caused by Massive intake Impaired renal excretion Shift from ICF to ECF Most common in renal failure ``` Manifestations: Cramping leg pain Weak or paralyzed skeletal muscles Ventricular fibrillation or cardiac standstill Abdominal cramping or diarrhea ```
33
Hypokalemia
Low serum potassium caused by: - Abnormal losses of K+ via the kidneys or gastrointestinal tract - Magnesium deficiency - Metabolic alkalosis ``` Manifestations Most serious are cardiac. Skeletal muscle weakness (legs) Weakness of respiratory muscles Decreased gastrointestinal motility Impaired regulation of arteriolar blood flow ```
34
Calcium
Obtained from ingested foods More than 99% combined with phosphorus and concentrated in skeletal system Inverse relationship with phosphorus Bones are readily available store ``` Functions:: Transmission of nerve impulses Myocardial contractions Blood clotting Formation of teeth and bone Muscle contractions ```
35
Hypercalcemia
``` High serum calcium levels caused by: Hyperparathyroidism (two-thirds of cases) Malignancy Vitamin D overdose Prolonged immobilization ``` ``` Manifestations: Decreased memory Confusion Disorientation Fatigue Constipation ```
36
Hypocalcemia
Low serum Ca levels caused by: ``` Decreased production of PTH Acute pancreatitis Multiple blood transfusions Alkalosis Decreased intake ``` Manifestations: Positive Trousseau’s or Chvostek’s sign Laryngeal stridor Dysphagia Tingling around the mouth or in the extremities
37
Magnesium
50% to 60% contained in bone. Coenzyme in metabolism of protein and carbohydrates Factors that regulate calcium balance appear to influence magnesium balance. Acts directly on myoneural junction Important for normal cardiac function
38
Hypermagnesemia
High serum Mg caused by: Increased intake or ingestion of products containing magnesium when renal insufficiency or failure is present ``` Manifestations: Lethargy or drowsiness Nausea/vomiting Impaired reflexes Somnolence Respiratory and cardiac arrest ```
39
Hypomagnesemia
``` Low serum Mg caused by:: Prolonged fasting or starvation Chronic alcoholism Fluid loss from gastrointestinal tract Prolonged parenteral nutrition without supplementation Diuretics ``` ``` Manifestations:: Confusion Hyperactive deep tendon reflexes Tremors Seizures Cardiac dysrhythmias ```
40
Normal Saline (NS)
``` Isotonic No calories More NaCl than ECF 30% stays in IV (most) 70% moves out of IV ``` ``` Expands IV volume --Preferred fluid for immediate response --Risk for fluid overload higher Does not change ICF volume Blood products Compatible with most medications -- ```
41
Acid-Base Imbalance
Acidosis Excess hydrogen ions Decrease in serum pH Alkalosis Deficit of hydrogen ions Increase in serum pH
42
Respiratory Acidosis –
lungs can not get enough 02 Acute problems Pneumonia, airway obstruction, chest injuries Drugs that depress the respiratory control center Chronic respiratory acidosis Common with COPD
43
Respiratory Alkalosis –
Blowing off too much C02 Hyperventilating Caused by anxiety, high fever, overdose of aspirin Head injuries Brainstem tumor
44
Metabolic Acidosis – 3 D’s
1. Excessive loss of bicarbonate (HCO3) ions to buffer hydrogen - -Diarrhea―loss of HCO3 from intestines 2.Diabetic acidosis 3.Renal disease or failure Dialysis Decreased excretion of acids Decreased production of HCO3 ions
45
Metabolic Alkalosis
Increase in serum bicarbonate ion - Loss of hydrochloric acid from stomach - Hypokalemia - Excessive ingestion of antacids
46
Effects of Acidosis
``` -Impaired nervous system function Headache Lethargy Weakness Confusion Coma and death ``` -Compensation Deep rapid breathing Secretion of urine with a low pH
47
Effects of Alkalosis
Increased irritability of the nervous system causes: ``` Restlessness Muscle twitching Tingling and numbness of the fingers Tetany Seizures Coma ```
48
Treatment of Imbalances
Treatment of underlying cause Immediate corrective measures to include fluid and electrolyte replacement or removal Caution is required when adjusting fluid levels to ensure appropriate electrolyte balance. Addition of bicarbonate to the blood to reverse acidosis Modification of diet to maintain better electrolyte balance
49
Hydrogen Ion and pH Scale
6. 8= DEATH 7. 35=ACIDOSIS AND INCREASE H+ 7. 4= NORMAL 7. 45= ALKALOSIS AND DECREASE H+ 7. 8= DEATH