Week 2 - Respiratory, ABGs Flashcards
(156 cards)
Acute Respiratory Failure Overview
Occurs when oxygenation, ventilation, or both are ___________
- Insufficient O2 transferred to blood
- ___________
- Decreased PaO2 and SaO2
- Inadequate CO2 removal
- ___________
- Increased PaCO2
- ABGs assess pH, PaO2, PaCO2, bicarbonate, SaO2
- Pulse oximetry assesses arterial O2 saturation(SpO2)
Occurs when oxygenation, ventilation, or both are inadequate
- Insufficient O2 transferred to blood
- Hypoxemia
- Decreased PaO2 and SaO2
- Inadequate CO2 removal
- Hypercapnia
- Increased PaCO2
- ABGs assess pH, PaO2, PaCO2, bicarbonate, SaO2
- Pulse oximetry assesses arterial O2 saturation(SpO2)
Hypoxemic (Oxygenation) or Hypercapnic (Ventilatory)
Hypoxemic
PaO2 is less than or equal to 60 mm Hg on 60% or more oxygen
Hypercapnic
PaCO2 is over 50 mm hg and pH is under 7.35
Common Causes of ARF
Hypoxemic:
* ARDS
* Asthma
* Chronic Bronchitis
* Pulmonary Edema
* Emphysema
* Pneumonia
* PE
* Pneumothorax
Hypercapnic:
* ARDS
* Asthma
* Chronic Bronchitis
* Pulmonary Edema
* Emphysema
* COPD
* Cystic Fibrosis
* Hypoventilation syndrome
* Brainstem injury
* Guillain-Barre Syndrome
* Muscular dystrophy
Hypoxemic:
* ARDS
* Asthma
* Chronic Bronchitis
* Pulmonary Edema
* Emphysema
* Pneumonia
* PE
* Pneumothorax
Hypercapnic:
* ARDS
* Asthma
* Chronic Bronchitis
* Pulmonary Edema
* Emphysema
* COPD
* Cystic Fibrosis
* Hypoventilation syndrome
* Brainstem injury
* Guillain-Barre Syndrome
* Muscular dystrophy
Hypoxemic Resp. Failure
- Mismatch between Ventilation (V) and perfusion (Q)
—. V/Q mismatch - Shunt
- Diffusion limitation
- Alveolar hypoventilation
- Mismatch between Ventilation (V) and perfusion (Q)
—. V/Q mismatch - Shunt
- Diffusion limitation
- Alveolar hypoventilation
pH – the measure of ________ balance found in arterial blood
______ - _______
pH – the measure of acid/base balance found in arterial blood
7.35 – 7.45
PaO2 – the partial pressure of O2 found in _______________
_____ - _______ mmHg
PaO2 – the partial pressure of O2 found in arterial blood
80 – 100 mmHg
PaCO2 – the partial pressure of CO2 found in arterial blood
_____ - _____ mmHg
PaCO2 – the partial pressure of CO2 found in arterial blood
35 – 45 mmHg
HCO3 – the measure of __________ found in arterial blood (buffer that helps balance pH)
_____ - _____ MeQ/L
HCO3 – the measure of bicarbonate found in arterial blood (buffer that helps balance pH)
22-26 MeQ/L
SaO2 – O2 saturation in the arterial blood _______ %
95-100
SpO2 – O2 saturation in the peripheral capillaries; estimate of the amount of O2 that is bound to Hbg in the blood versus the total amount of Hbg _______%
95-100
Hypoxemic Respiratory Failure
Etiology and Pathophysiology
Diffusion Impairment
* Gas exchange across alveolarcapillary membrane is compromised by a process that destroys the alveolar membrane or affects blood flow through the pulmonary capillaries
Alveolar-capillary membrane is thicker (fibrotic) and slows gas exchange
Pulmonary fibrosis, interstitial lung disease, ARDS
Pulmonary edema – accumulation of fluid, WBCs or protein in alveoli
Diffusion Impairment
* Gas exchange across alveolarcapillary membrane is compromised by a process that destroys the alveolar membrane or affects blood flow through the pulmonary capillaries
Alveolar-capillary membrane is thicker (fibrotic) and slows gas exchange
Pulmonary fibrosis, interstitial lung disease, ARDS
Pulmonary edema – accumulation of fluid, WBCs or protein in alveoli
Clinical Manifestations of Hypoxemia and Hypercapnia
Signs of respiratory failure are related to
A sudden decrease in PaO2 or rapid increase in PaCO 2 implies a serious condition or lifethreatening emergency
Signs of respiratory failure are related to Extent of changes in PaO2 or PaCO2
Speed of change (acute versus chronic)
Ability for compensation to occur
Failure of compensatory mechanisms leads to resp. failure
Signs of respiratory failure are related to
A sudden decrease in PaO2 or rapid increase in PaCO 2 implies a serious condition or lifethreatening emergency
Signs of respiratory failure are related to Extent of changes in PaO2 or PaCO2
Speed of change (acute versus chronic)
Ability for compensation to occur
Failure of compensatory mechanisms leads to resp. failure
Clinical Manifestations of Hypoxemia & Hypercapnia cont.
Lack of O2 affects all body systems
* Manifestations may be specific or nonspecific
* First sign of hypoxemic ARF is a change in ________________
Decreased O2
_________, _________, agitation
Increased CO2
morning headache, decreased RR, decreased LOC
- Early signs of ARF – tachycardia, tachypnea, pallor, mild increase in work of breathing (WOB)
Heart and lung compensation for decreased O2 and rising CO2 - Cyanosis is an unreliable indicator of hypoxemia
Clinical Manifestations of Hypoxemia & Hypercapnia cont.
Lack of O2 affects all body systems
* Manifestations may be specific or nonspecific
* First sign of hypoxemic ARF is a change in mental status
Decreased O2
restlessness, confusion, agitation
Increased CO2
morning headache, decreased RR, decreased LOC
- Early signs of ARF – tachycardia, tachypnea, pallor, mild increase in work of breathing (WOB)
Heart and lung compensation for decreased O2 and rising CO2 - Cyanosis is an unreliable indicator of hypoxemia
Hypoxemia and Hypercapnia
Observe
* Position: lie down, sit upright, or tripod
* Work of breathing (WOB); effort needed by respiratory muscles to inhale air into the
lungs
- Breathing patterns
_______, shallow (hypoxemia); monitor for fatigue
_____ RR (hypercapnia)
Change from rapid to slow RR indicating severe muscle fatigue and leading to respiratory arrest
Observe
* Position: lie down, sit upright, or tripod
* Work of breathing (WOB); effort needed by respiratory muscles to inhale air into the
lungs
- Breathing patterns
Rapid, shallow (hypoxemia); monitor for fatigue
Slow RR (hypercapnia)
Change from rapid to slow RR indicating severe muscle fatigue and leading to respiratory arrest
Hypoxemia and Hypercapnia
Observe
* Ability to speak
2 to 3 word _______
* _________ breathing
Increased expiratory time; prevents small bronchial collapse
* _________ of intercostal spaces or supraclavicular area; use of accessory muscles
* Paradoxical breathing
Abdomen and chest move outward with exhalation and inward with inhalation (opposite of normal)
May be diaphoretic from increased WOB
Observe
* Ability to speak
2 to 3 word dyspnea
* Pursed-lip breathing
Increased expiratory time; prevents small bronchial collapse
* Retraction of intercostal spaces or supraclavicular area; use of accessory muscles
* Paradoxical breathing
Abdomen and chest move outward with exhalation and inward with inhalation (opposite of normal)
May be diaphoretic from increased WOB
Hypoxemia and Hypercapnia S&S
Auscultate breath sounds
* Fine crackles: ________ edema
* Coarse crackles: ______ in airways
* Absent or diminished: atelectasis, pneumonia, or hypoventilation
* Bronchial: consolidation
* Pleural friction rub: ____________ involving pleura
Auscultate breath sounds
* Fine crackles: pulmonary edema
* Coarse crackles: fluid in airways
* Absent or diminished: atelectasis, pneumonia, or hypoventilation
* Bronchial: consolidation
* Pleural friction rub: pneumonia involving pleura
Manifestations of Hypoxemia - Specific
- Respiratory: ________, tachypnea, prolonged expiration, nasal flaring, intercostal muscle retraction, use of accessory muscles, decreased SpO2 (less than 80%), paradoxic chest or abdominal wall movement with respiratory cycle (late), cyanosis (late)
- Respiratory: dyspnea, tachypnea, prolonged expiration, nasal flaring, intercostal muscle retraction, use of accessory muscles, decreased SpO2 (less than 80%), paradoxic chest or abdominal wall movement with respiratory cycle (late), cyanosis (late)
Manifestations of Hypoxemia - Nonspecific
- CNS: agitation; _________; disorientation; restless, combative behavior; delirium; decreased level of consciousness; coma (late)
- Cardiovascular: ___________; hypertension; skin cool, clammy, and diaphoretic; dysrhythmias (late); hypotension (late)
- Other: ________, inability to speak in complete sentences without pausing to breathe
- CNS: agitation; confusion; disorientation; restless, combative behavior; delirium; decreased level of consciousness; coma (late)
- Cardiovascular: tachycardia; hypertension; skin cool, clammy, and diaphoretic; dysrhythmias (late); hypotension (late)
- Other: fatigue, inability to speak in complete sentences without pausing to breathe
Manifestations of Hypercapnia - Specific
- Respiratory: ________, ________ position, pursed-lip breathing, __________ RR or rapid rate with shallow respirations, decreased tidal volume, decreased minute ventilation
- Respiratory: dyspnea, tripod position, pursed-lip breathing, decreased RR or rapid rate with shallow respirations, decreased tidal volume, decreased minute ventilation
Manifestations of Hypercapnia - Nonspecific
- CNS: morning __________, disorientation, confusion, progressive somnolence, increased intracranial pressure, coma (late)
- Cardiovascular: __________, HTN, dysrhythmias, bounding pulse
- Neuromuscular: muscle _________, decreased deep tendon reflexes, Tremors, seizures (late)
- CNS: morning headache, disorientation, confusion, progressive somnolence, increased intracranial pressure, coma (late)
- Cardiovascular: tachycardia, HTN, dysrhythmias, bounding pulse
- Neuromuscular: muscle weakness, decreased deep tendon reflexes, Tremors, seizures (late)
Nursing and Interprofessional Management: ARF
Management and care will vary; factors to consider:
Age
Severity of onset
Underlying comorbidities
Suspected or most likely cause
Acute care requires collaboration between nursing, physicians, respiratory therapists,
pharmacists, and others is essential
Management and care will vary; factors to consider:
Age
Severity of onset
Underlying comorbidities
Suspected or most likely cause
Acute care requires collaboration between nursing, physicians, respiratory therapists,
pharmacists, and others is essential
Mild to moderate ARF
* __________ O2
* Non-invasive ventilation (Bi-PAP) for patients who are awake, alert, able to maintain a patent airway, able to clear own secretions
Mild to moderate ARF
* High-flow O2
* Non-invasive ventilation (Bi-PAP) for patients who are awake, alert, able to maintain a patent airway, able to clear own secretions
Severe ARF - ICU care
_________ ventilation
Continuous pulse oximetry
_________ blood pressure (ABP) monitoring
Frequent ABGs
______________________ (CVP) monitoring
Advanced hemodynamic monitoring
Mechanical ventilation
Continuous pulse oximetry
Arterial blood pressure (ABP) monitoring
Frequent ABGs
Central venous pressure (CVP) monitoring
Advanced hemodynamic monitoring
Clinical Problems (ARF)
Impaired ___________ system function
Inadequate tissue _________
__________ imbalance
Impaired respiratory system function
Inadequate tissue perfusion
Acid-base imbalance