Week 5: ENT 2 (common conditions of the nose, mouth and throat) Flashcards

1
Q

Nasal polyps Background

A
  • Fleshy, benign swelling of the nasal mucosa
  • Usually bilateral: common (>40 years)
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2
Q

causes of nasal polyps

A
  • They result from chronic inflammation and are associated with:
    • Asthma
    • recurring infection,
    • allergies
    • drug sensitivity
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3
Q

presentation of nasal polyps

A
  • Polyps look slightly lighter
  • In this pic: Emerge out of the middle meatus (between middle and inferior turbinate’s)
  • Pale or yellow in appearance/ fleshy and reddened
  • Symptoms
    • Blocked nose and water rhinorrhoea
    • Post-nasal drip
      • Drip goes into the pharynx and larynx- irritation and cough
      • Decrease smell and reduced taste
      • Sinusitis- blockage of the sinus air cavities
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4
Q

Unilateral poly +/- blood tinged secretion may

A

suggest tumour – cancer

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5
Q

management of nasal polyps

A

Medical management with topical (nasal drops) and possibly systemic corticosteroids is usually considered the initial treatment of choice, with endoscopic sinus surgery reserved for those patients who fail to improve

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6
Q

rhinitis

A
  • Inflammation of the nasal mucosa lining
  • Entire nasal cavity affected- bilateral
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7
Q

causes of rhinitis

A
  • Simple acute infective rhinitis (viral- common cold)
  • Allergic rhinitis- similar symptoms to infective rhinitis
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8
Q

presentation of rhinitis

A
  • Nasal congestion
  • Rhinorrhoea – runny nose
  • Sneezing
  • Nasal irritation
  • Postnasal drip
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9
Q

management of rhinitis

A
  • Topical/ oral nasal antihistamines
  • Topical intranasal steroids
  • Nasal saline wash
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10
Q

septal haematoma

A
  • Potential complication from nasal injury
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11
Q

septal haematoma causes

A
  • Buckling(bending) of cartilage due to trauma
    • Tears/shears blood vessel
    • Accumulation of blood
    • Strips perichondrium away from cartilage (nasal septum)
    • Starving cartilage of blood supply
    • Cartilage dies fibrosis and affects shape
    • Infection can be an issue
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12
Q

diagnosis of septal haematoma

A

must look up the nostrils for swelling

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13
Q

management of septal haematoma

A

must be incised and drain and a tamponade placed to stick perichondrium back onto cartilage

But if you don’t treat septal haematoma- Saddle nose deformity

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14
Q

acute sinusitis

A

<3 weeks

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15
Q

background to acute sinusitis

A
  • Inflammation of the mucous membrane of the paranasal sinuses
  • Paranasal sinuses are air filled spaces lined with resp mucosa and therefor have cilia and goblets cells – extensions of the nasal cavity

Sinuses drain into nasal cavities via ostia’s into a meatus  most commonly the middle meatus

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16
Q

pathophysiology of acute sinusitis

A
  • Infection leads to reduced ciliary function, oedema of nasal mucosa and sinus ostia and increased nasal secretions → that cant drain
  • Maxillary most commonly affected due to gravity
  • Stagnant secretions- breeding ground for bacterial infection
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17
Q

causes of acute sinusitis

A
  • Causes
    • Usually viral infection
      • Rhinovirus
      • Parainfluenza virus
    • Only 3% require antibiotics
      • Streptococcus pneumonia
      • Haemophilus influenzae
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18
Q

presentation of acute sinusitis

A
  • Facial pain- esp when looking down
  • Headache
  • Nasal discharge
  • Loss of smell
  • Nasal obstruction
  • Coryzal symptoms- yellow sputum
  • Vertigo if mucus builds up in eustachian tube
  • Ear pain, tiredsness
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19
Q

management of acute sinusitis

A
  • Analgesia
  • Intranasal decongestants and nasal saline
  • Don’t give abx if symptoms have been present for 10 days or less
  • Intranasal corticosteroids for 14 days if symptoms present for more than 10 days
  • Oral abx e.g. phenoxymethylpenicillin if severe presentation
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20
Q

chronic sinusitis

A

>3 months

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21
Q

causes of chronic sinusitis

A
  • Allergies esp hay fever and environment allergies
  • Nasal polyps/ Deviated septum
  • Resp tract infection
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22
Q

management of chronic sinusitis

A
  • Avoid triggers, stop smoking
  • Nasal irrigation with saline solution to relieve congestion and nasal discharge
  • Intranasal corticosteroids for up to 3 months
  • Specialist referral if unilateral symptoms
  • Recurrent otitis media/pneumonia in child
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23
Q

deviated nasal septum

A
  • A deviated septum occurs when the thin wall (nasal septum) between your nasal passages is displaced to one side. In many people, the nasal septum is off-center — or deviated — making one nasal passage smaller
24
Q

causes of deviated nasal septum

A
  • Present at birth
  • Injury to the nose
25
presentation of deviated nasal septum
difficulty breathing, crusting + bleeding, recurrent sinus infections, may also have no symptoms
26
investigations for deviated nasal septum
CT scan and nasal endoscopy
27
management of deviated nasal septum
* Nasal sprays including decongestants, antihistamine and corticosteroids * **Surgery:** may need a septoplasty operation to correct the septum deviation if causing significant problems
28
nasal fracture causes
* Trauma to the nose
29
presentation of nasal fracture
deformity to the nose, swelling, skin laceration, ecchymosis, epistaxis and CSF rhinorrhea
30
investigations for nasal fracture
X-ray would only be needed with more serious injuries needing facial and skull x-ray
31
management of nasal fracture
* non-displaced fractures can be managed conservatively * if displaced- manipulation under either local or general anaesthetic, , if not it may need surgery 12 months after the operation * must also exclude complications like septal haematoma which would need draining * Refer to ENT if required
32
salivary stones (sialothiaiss)
* Most stones are located in submandibular glands * Dehydration, reduced salivary flow * Most stoned less than 1cm
33
presentation of sialothiasis
* Pain in gland * Swelling * Infection
34
diagnosis of sialothiasis
* History * X-ray * Sialogram- contrast dye injected into gland
35
**Peritonsillar abscess ‘quinsy’** *
* Diff to tonsilitis * Affects tissue around the tonsil * If its unilateral (quinsy)→ will deviate the uvula towards the swelling
36
**Pharyngeal tonsil (adenoid)- clinical correlates**
* Enlarged pharyngeal tonsils * Block eustachian tube (recurrent/persistent middle ear infection * Snoring/sleep apnoea * Sleeping with mouth open * Chronic sinusitis * Sore throat * Nasal tone to voice
37
dysphagia signs and symptoms
* Coughing and chocking * Sialorrhea (drooling) * Recurrent pneumonia * Change in voice/speech (wet voice) * Nasal regurgitation
38
causes of dysphagia
* **Stroke** * 30% of post stroke death are due to pneumonia * E.g. aspiration pneumonia * Progressive neurological disease * Parkinson’s/MS * COPD * Dementia * MALIGNANCY e.g. oesophageal cancer
39
interventions of dysphagia
* Fluids are thickened
40
false diverticulum
Caused by a posteromedial (false diverticulum)→ arises in weakness between the 2 parts of the inferior constrictor (Killians dehiscence) * Probably due to * Failure of UOS to relax * Abnormal timing of swallowing * Essentially there is a higher pressure in laryngopharynx * Weakness in inferior constrictor muscle produces outpouching
41
presentation of false diverticulum
bad breath regurg of food occasional choking on fluids general difficulty swallowing
42
**Tonsilitis**
**inflammation of the palatine tonsils**
43
presentation of tonsillitis
* Fever * Sore throat * Pain/difficulty swallowing * Cervical lymph nodes * Bad breath * Viral causes (most common) * Bacterial causes (up to 40% of cases) * Strep pyogenes * White spots * Can be bacterial secondary to viral tonsilitis
44
**injury to either the IX (glossopharyngeal) and X (vagus) can cause**
* Obvious things * Absent gag * Uvula deviated away from lesion (Lower Motor Neurone lesion) * More subtle * Dysphagia * Taste impairment (posterior tongue IX) * Loss of sensation oropharynx
45
**Injury to IX/V caused by**
Medullary infarct, jugular foramen issues (fracture)
46
injury to XII- hypoglossal
* Wasted tongue * Stick tongue out- tongue may deviated * Damage to nerve itself (LMN)- points to side of the lesion (tongue never lies) * Muscle wasting * Fasciculations
47
thyroid nodules differentials
* Common head and neck presentation with patient presenting with thyroid masses or nodules * Can be benign or malignant
48
benign causes of thyroid nodules
* Colloid nodules * Hyperplastic nodules * Thyroid adenoma * Thyroid cyst * Viral thyroiditis * Graves disease
49
malignant causes of thyroid nodules
50
investigations for thyroid nodules
* **Swallow and stick out tongue** * Any lump related to thyroid will move on swallowing * If the lump moves when tongue being stuck out- thyroglossal * TSH and T4 levels * US * Fine needle aspiration if malignancy suspected * Calcitonin levels, calcium and PTH in suspected PT pathology * Radioactive iodine uptake- graves and multinodular goitre and thyroiditis differentiation
51
presentation of thyroid nodule
* **Presentation** * Mass effect symptoms of thyroid neck lump * SOB due to tracheal compression * Dysphagia * Hoarseness- irritation of recurrent laryngeal nerve * Hyperthyroid symptoms * Systemic malignant features e.g. weight loss, night sweats and lymphadenopathy * Hypothyroid symptoms
52
cervical lymphadenopathy
* Enlargement of cervical lymph nodes in the neck region * Important clinical indicator of underlying condition of infection
53
causes of cervical lymphadenopathy
* Throat infection * Dental decay * Ear infection * Salivary glands * Cancer * HIV, HEP
54
presentation of cervical lymphadenopathy
* Cervical lymph nodes are usually very small cand cannot be felt upon touch until underlying infection or malignancy has triggered increase in size * Symptoms * \>6 weeks * Firm, hard * Lymph nodes \>2cm * Unintentional weight loss, night sweats, appetite loss * Exposure to HIV or hep * Unexplained fever * Any associated facial swelling
55
diagnosis of cervical lymphadenopathy
* Physical exam * US, CT and MRI * Further test dependent on findings of examination * Biopsy needed if malignancy is being considered * Persistent cervical lymphadenopathy- FBC, LFT, CRP