Week 7 - HEENT (Head, Ears, Eyes, Nose, and Teeth) Flashcards

(55 cards)

1
Q

Show me the eyeball

A
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2
Q

Show me retenal detachment

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3
Q

Show me a corneal Ulcer

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4
Q

Show me the types of glacoma

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5
Q

Show me the structures involved with glacoma

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6
Q

Show me the Nose and the possible fracture sites

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7
Q

Show my what peritonsillar abscess looks like

Puss filled tissue

A
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8
Q

Show me the Ear

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9
Q

Show me the Inner Ear

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10
Q

Dizziness Differentials

Peripheral?

Central?

A

Meniere’s Disease

Acute Otitis Media

Perilymphatic Fistular CNS Trauma

Cholesteatoma

Viral Labyrinthitis

Bacterial Labyrinthitis

Vestibular Neuronitis Motion sickness

Ototoxicity

Otologic Surgery

Otologic Injury/Trauma

Otosyphilis

Benign ParoxysPPV

Acoustic Neuroma

Brainstem CVA

Carotid Stenosis

CNS Neoplams

Multiple Sclerosis

Vertebrobasilar Insufficiency

Medication Overdose

Presbystasis

Psychogenic Disorder

Arnold-Chiari Malformation

CNS Infection

Seizure Disorder

Migraine

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11
Q

Dizziness Differentials

Systemic?

A

Cardiac Arrhythmia

Cardiac Valvular Disease

Orthostatic Hypertension

Alcohol Intoxication

Sleep Deprivation

Toxin Exposure

Hypoglycemia

Autonomic Dysfunction

Hyperventilation

Pain/Anxiety

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12
Q

What is Vertigo?

What is Vestibular Neuritis?

A

an abnormal sensation that is described by a person as a feeling that they are spinning, or that the world is spinning around them, and may be accompanies by intense nausea and vomiting. This feeling may be associated with loss of balance to the point that the person walks unsteadily of falls. Vertigo itself is a symptom or indicator of an underlying balance problem, either involving the labyrinth of the inner ear, or the cerebellum of the brain

thought to be the result of inflammation of the vestibular portion of the eighth cranial nerve and classically presents with vertigo, nausea, and gait imbalance

The seventh cranial nerve sends information between the brain and the muscles used in facial expression (such as smiling and frowning), some muscles in the jaw and the muscles of a small bone in the middle ear

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13
Q

What is Bell’s Palsy?

What is BPPV? (Benign Paroxysmal Positional Vertigo)

A

the most common cause of facial weakness, whereas vestibular neuritis ranks second or third as the most frequent cause of sudden onset of dizziness and vertigo. The two conditions can occur either singly or in combination to cause facial weakness and debilitating dizziness

a brief, intense episode of vertigo that occurs because of a specific change in the position of the head. A person also may experience BPPV when he or she rolls over in bed. BPPV sometimes may result from a head injury or aging

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14
Q

What are the top 9 differentials for HEENT?

What are ototoxic agents?

A

Tinnitus

Ototoxic Agents

Mastitis

Tonsilitis

Otitis Media

Auricular Hematomas/Cauliflower (rugby, MMA, Soccer)

Perichondritis – Trauma/Piercings

Otic Barotrauma – TM Preformation (many tubes)

Ear drainage tubes – Tympanostomy tubes

more than 600 prescriptions and over the counter drugs that can trigger tinnitus, make existing tinnitus worse, or cause a new tinnitus-causing drugs sprinkled throughout.

For example, antibiotics, painkillers, anti-anxiety, and anti-depression drugs, antimalarial medications, anti-cancer drugs, and blood pressure controlling medications – to name a few – can trigger tinnitus

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15
Q

Special Considerations

Eye Trauma?

A

Suspect head injury

Loss og vision is traumatic

Great Anxiety

Contact Lenses

Transient S+S

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16
Q

What are the Priority diagnosis for eyes?

A

Sudden vision loss

chemical injury

vision - threatening trauma

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17
Q

What will you do for a focused assessment for the eye?

A

Visual acuity

External inspection: lids, lashes, conjunctiva, and cornea, symmetry of eyes, eye movement

Paplate orbital rim

pupils

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18
Q

What’s the eyes external examination?

What’s the physical exam?

A

Penlight examination

Eyelids

Conjunctiva

Corneal Clarity

Pupil Size (oculomotor)

Pupil Symmetry

Pupillary light reactions (consensual)

Accommodation

Visual Acuity

Cranial Nerve II – Optic Nerve

Palpate the orbital rim

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19
Q

What are the extraocular movements?

What’s the eye assessment?

A

Cranial Nerve III – Oculomotor

Cranial Nerve IV – Trochlear

Cranial Nerve VI – Abducens

Current History – Mechanism of Injury, New/Recurrent problem, Loss/Change of
vision

How does the eye feel?

How does the eye look?

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20
Q

What’s the differentials for red painful eye(s)?

Eyes - traumatic emergencies?

A

Glaucoma

Corneal Abrasion

Foreign body

Corneal ulcer

Conjunctivitis

Iritis

Scleritis

Episcleritis

Eyes: Traumatic Emergencies

Corneal Abrasions and lacerations

Corneal foreign bodies

Penetrating eye injuries

Retinal detachment (traumatic)

Burns

Acute vitreous hemorrhage & hyphema

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21
Q

Tell me about corneal abrasions and lacerations?

A

Common

Causes partial or complete removal of corneal epithelium

Prognosis – Depends on depth or injury

C/O severe pain, tearing and blepharospasm (lid spasm)

Management: Irrigate with normal saline, dry light pressure dressing over both
eyes with eyelids closed (do not tape eyelids closed)

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22
Q

Corneal Foreign Bodies

What are the clinical features?

What’s the physical exam?

A

Something in my eye

History of object being propelled into the eye

Tearing, conjunctival reddening blepharospasm

Dull non-localizing ocular ache and decreased vision

Complete Inspection of the eye

Note presence of absence of material

Note presence of rust ring

Visual Acuity

Multiple foreign bodies

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23
Q

Corneal Foreign Bodies

What’s the management?

What do you need to do for penetrating eye injuries?

A

ABCs

Avoid external pressure on the globe

Superficial conjunctival or corneal foreign bodies may be irrigated

Cover both eyes if injury/ pain is severe

Be prepared for emesis

Massive trauma only a brief look should be attempted

May have penetrated the globe

Control bleeding using minimal pressure

Do NOT manipulate, palpate, irrigate or apply cold packs

24
Q

Acute Iritis/ Anterior Uveitis

Tell me about it

A

Anterior Uveitis – Inflammation of the anterior segment of the eye

Acute iritis – Mild inflammatory reaction of the iris – seen after blunt trauma

Clinical Features: Pain (Deep ache), Headache above eyebrow, photophobia,
excessive tearing

25
# Retinal Detachment (traumatic) Tell me about it ## Footnote What's the physical exam?
Blunt trauma may caused violent shifting of the vitreous body – causing renal tears Tears may ‘dissect’ the retina for the choroid (vascular layer) as blood enters this space and hematoma develops ## Footnote In acute injury, if patient reports seeing a flash of light, consider detachment May be a latent period between injury and detachment (up to months) Not painful – only symptom is VISUAL Perceived as a curtain descending over the visual field (progressive loss of vision) Loss of vision may also be sudden Reports of light flashes and cloudy vision Nothing visible (blood) on inspection
26
# Retinal Detachment (non-traumatic) Tell me about it
Spontaneous retinal detachment occurs most frequently in the elderly Results from fluid or blood leakage from vessels within the retina Causes: Hypertension, toxemia of pregnancy, papilledema (optic disc swelling) Retinal damage can progress slowly or enlarge quickly resulting in complete detachment
27
# Burns to the Eyes Tell me about akali burns ## Footnote Tell me about acid burns
True ocular emergency Alkaline exposure dissolves tissues until it is removed Immediate Irrigation – minimum 20 minutes Remove any particles Severity can be judged by corneal cloudiness and scleral whitening ## Footnote Tend to be less devastating The body is better able to deal with acids Treatment is same as for alkali exposure
28
# Burns to the Eyes Tell me about thermal burns ## Footnote Tell me about radiation/ultraviolet burns
Affect the eye lids more than the globe Determine if vision is decreased/blurred Limit assessment to inspection Cover affected eyes with moist dressing Hot liquid splashes and cigarette ashes to the cornea usually results in a superficial corneal epithealial injury – treat as corneal abrasion ## Footnote Results in direct corneal epithelial damage Latent period of 6-10 hours Patient develops foreign body sensation, intense pain, tearing, photophobia and blepharospasms Cover the affected eyes for p
29
# Hyphema and Acute Vitreous Hemorrhage What is that? ## Footnote What are the clinical features and physical exam?
Hyphema is blood in the anterior chamber resulting from rupture of one or more iris vessels Extent varies from microscopic to ‘8 ball’ hyphemas in which the entire anterior chamber fills with blood ## Footnote **Clinical** **Features** Principle cause is traumatic, retinopathies in diabetic patients may also be causative **Physical** **Exam** C/O pain and cloudy vision (floaters/spots) Visible reddish tint in anterior chamber
30
What's Enucleation?
Surgical management of malignancy Severe Infection Trauma A ball implant is then inserted
31
What is the physical exam for enucleation?
Orbital edema and ecchymosis Localized facial swelling Pain Tingling/Loss of sensation Diplopia Inability to elevate eye/restricted extraocular movements Subconjunctival hemorrhage Enophthalmos or exophthalmos Subcutaneous emphysema
32
Eyes - what are the medical emergencies?
Central retinal artery occlusion keratitis Corneal ulcers Acute glaucoma Acute iritis Conjunctivitis (bacterial/viral)
33
Tell me about central retinal artery occlusion? ## Footnote What are the causes?
Blood supply to the retina is obstructed producing a painless, total or near total black out of vision Re-establishment of retinal circulation MUST be accomplished within 90 minutes to regain vision ## Footnote Predisposing factors Atherosclerosis of carotid artery (clot formation) Atrial Fibrillation Prolonged pressure on eye with swelling/tight dressing
34
# Central Retinal Artery Occlusion What's the physical exams? | What's the management? ## Footnote What's the clinical progression?
Patient presents without pain Sudden loss of unilateral vision No history of trauma Associated with history of hypertension or hypercoagulable states | Immediate recognition Emotional Support Transport ## Footnote In 90-120 minutes, retina becomes necrotic and edematous – irreversible blindness
35
What is Keratitis/what does it look like? ## Footnote What are corneal ulcers?
Inflammation of the cornea Caused by infections, hypersensitivity reactions, ischemia, defects in tears, trauma Presents as moderate to severe pain depending on the amount of epithelial disruption present Scar tissue formation is the leading cause of blindness Divided into non-ulcerative and ulcerative ## Footnote Integrity of the corneal epithelium is disrupted Symptoms include pain, increased tearing, photophobia, ocular irritation
36
What is glaucoma? ## Footnote What are cataracts?
Defined as a group of conditions characterized by increased intraocular pressure Caused by alterations in the circulation and resorption of aqueous humor Classified as open angle or closed angle ## Footnote Opacity of the lens of the eye or the capsule or both leading to a painless loss of vision Due to aging process, but can be caused by infection, injury, exposure to radiation or chemicals, UV radiation or metabolic disorders Presents with a distortion of the visual image especially at night or in bright light Management is surgical The iris may appear distorted in post-op surgery patients
37
What is Conjunctivitis (Viral)? ## Footnote What is Conjunctivitis (Bacterial)?
Infections and accounts for the majority of “pink eye” Frequently occurs in conjunction with an URI Redness & tearing are common Treatment is symptomatic ## Footnote Presents with a mucopurulent discharge of varying colors (gray, yellow, green). Typically there is matting of the lashes in the morning, but minimal, if any pain. Treatment consists of topical antibiotic drops for one week Warm soaks should be used to keep the lids/lashes free
38
What is Macular Degeneration?
Degeneration of the macular area of the retina in the eye (the macula is in the center of the retina) Causes: Age related unknown, injury, inflammation, nutritional and hereditary factors implicated S/S: loss of central vision, visual distortion
39
# Orbital Cellulitis Periorbital? | The S+S ## Footnote Orbital?
Fever Periorbital edema Eye tenderness No proptosis (bulging eye) Visual Changes ## Footnote Pain with EOM Decreased visual acuity Ophthalmoplegias Spreads from adjacent infections Can extend into dural sinuses and meninges
40
What are the top 3 emergencies for traumatic ear injuries? ## Footnote What are potential ear trauma injuries?
Ear trauma Foreign body in ear Ruptured tympanic membrane ## Footnote Lacerations & contusions Thermal Injuries Chemical Injuries Traumatic perforations Barotitis (ears ‘pop’ from flying at high altitudes, scuba)
41
Foreign Body in Ear clinical features?
Patient usually state something is in ear Quite often an insect (adult), bead (peds) Patient may present with pain and purulent drainage and not be aware of foreign body Important: Is there a history of tympanic membrane rupture
42
What is a ruptered typmpanic membrane? ## Footnote What are more Ear medical emergencies?
Perforations of TM result from: acute changes in air or water pressure (blast injury), direct trauma (foreign body, Q-tips), caustics, lightning strike, otitis media, or associated temporal bone fractures. The patient may complain of slight hearing loss or pain The presence of concurrent vertigo or acute deafness suggests associated injuries to the semicircular canals ## Footnote Otitis media & otitis externa Meniere’s disease Tinnitus Labyrinthitis Vertigo
43
What is Otitis Externa? ## Footnote What is the etiology?
Defined as an inflammation of the outer ear The limited form of otitis externa is common to swimmers Physical findings may be limited to erythema ## Footnote Water-facilitates infection Bacteria – usually pseudomonas Fungal – aspergillosis and candida (immuno-compromised, chronic users of steroid drops) Malignant otitis externa: Osteomyelitis of the skull, begins as typical OE, non￾resolving, OE defaults to this for diabetic, HIV, Ca pts
44
What is Otitis Media? ## Footnote What is Menere's disease?
Otitis media includes the initial acute infection, otitis media with effusion, and chronic otitis media Acute Otitis Media (AOM) presents with the rapid onset of ear discomfort Typically follows URI Pain associated is not aggravated by movement ## Footnote Defined as a chronic disorder of the inner ear. Triad of symptoms: vertigo, tinnitus and sensorineural hearing loss Overaccumulation of fluid in the membranous labyrinth of the inner ear Onset of symptoms may be gradual or sudden
45
What are the S+S of Meniere's disease? ## Footnote What is tinitus?
Severe rotary vertigo (may be associated with N/V) Tinnitus Nystagmus during acute attacks ## Footnote Most common complaint with otologic conditions Ranges from mild ringing to a loud roaring in the ear Causes include: presbycusis, otosclerosis, Meniere's, loud noise, certain drugs and a wide variety of pathological disorders Treatment is resolving underlying disorder
45
What is Labyrinthitis? ## Footnote What is vertigo and dizziness?
Infection of the labyrinth Clinical Manifestations: hearing loss, tinnitus and spontaneous nystagmus to the affected side Causes include: nearby infections of ear, nose or throat. Otitis media or meningitis; toxic effect of certain drugs ## Footnote Common clinical manifestation of many ear disorders Dizziness is described as a disturbed sense of the proper relationship to space Three systems combine to give input regarding balance: visual, vestibular and the proprioceptive system Associated symptoms of vertigo: Nausea/Vomiting, falling, nystagmus, hearing loss and tinnitus
46
What are the nose traumatic emergencies? ## Footnote What is Epistaxis?
Epistaxis Fractured nose Foreign Body in nose ## Footnote Highly vascular Bleeding may originate in the anterior or posterior nose Causes include medications, particularly antiplatelets or anticoagulants and: * Anterior: drying, infection, blunt, trauma, manipulation, cocaine use and local infection * Posterior: Blood dyscrasias, hypertension, diabetes and trauma
47
# Fractured Nose What are the mechanisms? ## Footnote What's the physical exam?
Trauma that produces a fracture of the nasal bones may come from frontal, superior to inferior or lateral direction Of these – laterally directed trauma most likely to fracture since no cartilage to absorb or dissipate the force Suspected in all patients with significant facial trauma ## Footnote Swelling Tenderness/Pain Crepitus Ecchymosis (bruising) Deformity Epistaxis
48
Tell me about foreign body in the nose? ## Footnote What are the clinical features?
Most common for insertion of foreign bodies by children ## Footnote Patient was seen inserting the object Or presents with a purulent unilateral malodourous nasal discharge Body Odor
49
What is sinusitis? ## Footnote What are the acute symptoms?
Inflammation with resulting infection of the mucous membranes of one of more paranasal sinuses Commonly follows URTI or viral rhinitis Causes include URI, nasal polyps, deviated septum, tooth abscess, abuse of nasal decongestants, swimming and diving, frequent changes in barometric pressure ## Footnote Constant, often severe pain and tenderness Maxillary sinus involvement may lead to teeth pain Headache Fever Purulent nasal discharge Sore Inflamed throat
50
# Sinusitis  What are the subacute OR chronic symptoms?
Dull, intermittent or constant pain  Purulent nasal discharge   Chronic cough   Recurrent episodes   Loss of sense of smell
51
# Throat Traumatic Fractured Larynx - tell me about it ## Footnote What's the physical exam?
Blunt or penetrating trauma to the anterior neck can cause fracture or dislocation of laryngeal or tracheal cartilages  Need a high degree of suspicion for associated vascular injury   Injuries associated include fractures of hyoid bone, fractures of thyroid cartilage, fracture/dislocation of cricothyroid and fractures of trachea   ## Footnote History/Mechanism   Hoarseness or aphonia   Edema and bleeding (hemoptysis)
52
# Throat Medical Peritonsillar Abscess  - tell me about it | What are the potential complicaitons? ## Footnote What's the physical exam?
Bacterial – streptococcus/staphylococcus   Infection spreads into tissue between the tonsils and underlying muscle and creates an abscess   Untreated – infection can spread deep into neck with the potential for eroding a large vessel or aspiration of purulent drainage if abscess ruptures when patient is asleep   | Edema may threaten patency of the airway Erosion into the carotid artery ## Footnote Physical Exam/History   Sore throat x days with clinical progression to malaise and fever   Unilateral pain and difficulty swallowing   Patient may drool  Red edematous area above the tonsil between the soft palate and the tonsil often displacing the uvular  
53
# Throat Medical Retropharyngeal Abscess  - tell me about it
Retropharyngeal space extends from base of skull to tracheal bifurcation   Fever, dysphagia, neck pain, sore throat, muffled voice, respiratory distress, stridor in kids   Immediate ENT consult is required   Fatal complication: airway compromised
54
Dental trauma - tell me about it ## Footnote How do you care for an avulsed tooth?
Normal adult mouth has 32 teeth  3 Sections – crown, neck, root   Can involve fractures alone or fractures in combination of jaw (mandibular #) or facial bones   With fractured teeth, oral cavity should be examined for tooth fragments  ## Footnote Cold, fresh milk or saline for tooth transport   Do not rinse tooth   Take care NOT to handle by the root