Week 9 - MSK Injuries + Burns Flashcards
(61 cards)
What is the old/traditional way we used to classify burns? Why do we no longer use this method to classify burns?
Stage stage 1 - 4 method! - it does not directly describe how much of the epidermis and/or dermis is damaged
-We use the layer of skin method now!!!
What are burn injuries in “lower status/class countries correlated with? Why?
In all countries, lower SE status” is correlated with burns. Less access to 911, no smoke detectors in the home, poor equipment/no safety mechanisms
How do we treat joint injuries?
Treatment:
-Relocate joint (procedural sedation in ER or OR)
-Need to relocate the joint—procedural sedation in ER or possibly to OR (knee dislocation needs Immediate intervention—almost guaranteed nerve involvement)
S+S for pelvic fracture?
Signs and Symptoms:
-pain, hypovolemic shock, hematuria, urethral/vaginal/rectal bleeding
shortened or rotated leg
-Large vessel injuries (shearing), Abdominal organ injuries
Instability/Crepitus
Aortic injuries, other large vessel injuries, abdominal organ injuries causing hemorrhage
Pain
Evidence of hypovolemic shock
Shortening or abnormal rotation of the affected leg, other lower abdominal physical deformity
Genitourinary or intra-abdominal injury
Bruising
hematuria
Urethral, vaginal, or rectal bleeding
What are 5 interventions we can do for the Ongoing care + treatment of burn injures ( these are done AFTER eth Early Care + Treatments of Burn injuries!!)
Ongoing Care + Treatment of Burns:
1.) Patient Moved to Specialized Unit
-Burns ICU, plastics unit, etc.
2.) Skin Grafting + Dressings
-Grafts can be from both the patient and/or donors
3.) Escharotomies
-Procedure where burn tissue is removed to allow for pressure release
4.) Nutrition
-Insertion of NG tube or J tube, patient may receive TPN
5.) Reverse Isolation:
-To protect the patient from us!, Antibiotics may be administered, more likely that antimicrobial dressings will be used if positive cultures come back
How do we diagnose Rhabdomyolysis?
-Myoglobin levels are less reliable because the body breaks it down within 6 hrs – more likely to test urine for blood
-Diagnosis made by clinical history + elevated CK which peak at 24-36hrs (can be in the tens of thousands), which is released by the damaged cells.
-Creatinine will also rise as the body converts creatine to creatinine (plus AKI).
-Blood for myoglobin less reliable because it breaks down within 6 hours. More likely to test the urine for blood but even that has 50% false negatives
What kind of mortality rate does/do pelvic fractures have? (what does it depend on?)
Up to 70% mortality depending on fracture type and promptness of treatment
What are fractures associated with? Why?
Associated with large blood loss because or disruption of arteries or veins in close proximity to bones
-Tibia/Humerus Fracture: 250 - 2000mL blood loss
-Femur Fracture: 500 - 3000mL of blood loss
-Pelvic Fractures: varies but may range from 750 mL – 6000mL of blood loss
What is compartment syndrome? What does/can it result in?
Occurs as pressure increases inside a fascial compartment
Results in impaired capillary blood flow and cellular ischemia
What is the Rule of nines? (what is it commonly used for?/what does it do/give)
Rule of Nines:
-Most common method used in the ER, though not as accurate as some (like the Lund-Brower chart)
-Rule of nines is a rough estimate when there is nothing more accurate available—useful for a first guess of extent of the injury
What specific S+S do pelvic fractures have that are unique to them?
Pelvic fractures: unstable to palpation, shock
What is the initial treatment for fractures? (7 different interventions we can do)
Initial treatment:
-Immobilization and stabilization (splints, slings, pelvic binders, backboard for femurs)
-Cool packs
-Analgesia, muscle relaxants
-Sedation for reduction, then cast and sling
-Backslab and sling for support (non-displaced and stable)
-Surgery if unstable
-External fixation (multiple bone fragments) (cage)
What can electrical injuries cause that makes them kind of unique?
Electrical injuries cause severe internal injuries and cardiac arrest but very little visible external
S+S for Rhabdomyolysis?
Signs and Symptoms:
-Dark/Red urine, reduced urine output, severe muscle aching, arrhythmias/arrest
Impaired renal function from the massive release of myoglobin—kidneys hate dealing with the molecule in large amounts.
Damages the nephron—ATN. Urine becomes darker (“rhabdo”)
Release of potassium can cause massive increases in vascular space—arrhythmias and arrest (happens in the first few hours)
What is the MOI/what are fractures usually caused by?
Blunt force Trauma
-Typically caused by MVAs, assaults, falls, sports
MOI: motor vehicle crashes, assaults, falls, sports, leisure, or home activities
Treatment for pelvic injuries? (what do we avoid) (think of 5 things)
Treatment:
-avoid catheters if there is urethral trauma
-Stabilization device (Pelvic binder) – slow blood loss/encourage -clotting
-Surgery: internal/external fixation
-Angiography with embolization for bleeding vessels
-Blood/IV Fluid admin
-Tight stabilizing device that can slow blood loss and encourage clotting by direct pressure – they can hurt!
-Surgery to internally or externally fixate unstable fracture
-Angiography with embolization of bleeding vessels
-Blood or iv fluid admin
How do we diagnose pelvic fractures?
Diagnostic: Xrays, FAST to detect bleeding, CT
When diagnosing a joint injury, what non-mechanical/picture taking diagnostic method/tool will help us differentiate a dislocation from a fracture?
Usually clinical history will help us differentiate a dislocation from a fracture (sound of a crack or pop, for example)
How do crush injuries work/what is the patho behind them?
When muscles are crushed (especially for long periods), they become ischemic, then necrotic, then release potassium, myoglobin (protein found in muscles), and creatine kinase (enzyme found in muscles) into the blood:
-This release of potassium can lead to arrhythmias and cardiac arrest (usually within the first few hours)
-Release of myoglobin and creatine kinase (indicator for myoglobin) can cause acute kidney injury or rhabdomyolysis
Why can patient with crush injuries go into shock?
When muscles are crushed (especially for long periods), they become ischemic, then necrotic, then release potassium, myoglobin (protein found in muscles), and creatine kinase (enzyme found in muscles) into the blood:
Fluids leak into third spaces, causing edema and possibly increased compartment pressure
Patients can go into shock from these massive fluid shifts (distributive shock), or from hemorrhage (hypovolemic shock)
What type of injury has a high morbidity in the elder population?
Fractures!!
Elderly at high risk of being hospitalized for an extremity injury (rate of injury in pop)
What do we need to remember with pelvic injuries? (think about extent of injuries)
remember, A LOT of force to cause this, so there might/will be injuries around the pelvis/abdomen too
For fractures what is the average blood loss associated with?
Average blood loss: Associated with large blood loss due to proximity of vessels to bones
Tibia/Humerus Fracture: 250 - 2000mL
Femur Fracture: 500 - 3000mL
Pelvic Fractures: varies bu