WHAT DIS ECG BIZ Flashcards

(44 cards)

1
Q

Irregularly irregular

A

AF, atrial flutter

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2
Q

Regularly Irregular

A

2nd degrees heart block

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3
Q

Left Axis deviation

A

Left ventricular hypertrophy, LBBB

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4
Q

Right Axis deviation

A

Right Ventricular hypertrophy, RBBB, PE

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5
Q

P mitrale

A

MS, left atrial enlargement

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6
Q

P Pulmonale

A

PE, right atrial enlargement

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7
Q

Fixed increased PR (>0.2s/5 small squares)

A

1st degree AV HB. Also narrow QRS.

–> inferior wall MI, hyperkal, beta blockers/CCBs

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8
Q

Fixed interval increase but dropped QRS

A

Mobitx T1 2nd degree AV HB
–> inferior wall MI, RhF, beta blockers/CCBs
Narrow QRS

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9
Q

Slowly increased PR irregularly then drop

A

Mobitz T2 2nd degree AV HB
Broad QRS
—> RhD, BBs

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10
Q

Delta slope

A

WPW/AVRT

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11
Q

Broad QRS (>0.12s/3squares)

A

Ventricular tachy, BBB, WPW S

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12
Q

Tall complex

A

Left ventricular hypertrophy

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13
Q

Pathological Q wave

A

Old infarct. (a bit of a deeper Q)

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14
Q

QT syndrome

A
(Prolonged QT, >0.44s)
1. Congenital 
2. Hypomagnesemia 
3. Hypokalaemia 
4. Drug 
(MI, myocarditis) 
Increases risk of torsades de pointes
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15
Q

ST elevation

A

MI, pericarditis (saddle shaped), variant angina

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16
Q

ST depression

A

AVRT, Hypokal, posterior MI

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17
Q

T wave inversion

A

R ventricular strain, previous MI, BBB, PE

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18
Q

Tall T waves

A

Hyperkal,

Hyperacute STEMI

19
Q

Biphasic T wave

20
Q

Flattened T waves

21
Q

U waves

A

(small deflections after t waves)

Electrolyte imbalance

22
Q

LBBB - ECG, causes

A

2 deep S waves in V1, 2 tall R waves in V6
LAD
–> AS, IHD, hypertension, ant MI, Dilated cardiomyopathy

23
Q

RBBB - ECG, causes

A

2 tall R waves in V1, 2 deep S waves in V6

–> Cor pulmonale, PE (IDH, RhF)

24
Q

P and QRS asynchonised

A

3rd degree heart block
A cannon wave in JVP
Due to ant/inf MI, CHD, myocarditis, BBs

25
AF
Irregularly irregular, no p waves | IHD, PE, thyrotox, caridomyopathy
26
Atrial Flutter
300bpm no t wave, saaw tooth patten CAUSES: Coronary heart disease, cardiomyopathy, inflammation, blood pressure. EFFECTS: Stroke, dyspnoea, dizziness, angina pectoris, palpitations
27
Ventricular fibrillation
o Chaotic irregular deflections of varying amplitude o No identifiable p waves, QRSs or T waves o Rate 150-500bpm o Amplitude decreases with duration o Coronay heart disease, cardiomyopathy, echo
28
Ventricular flutter
o Monomorphic sine wave o >200bpm o ECG identical when turned upside down
29
AVNRT
 140-180bpm tachycardia  QRS is narrow (unless pre-existing BBB)  ST depression  P waves may be buried in the QRS complex (it starts at the same time)
30
AVRT
 200-300bpm  Can be orthodromic (signal travels down via AV node) or antidromic (signal travels up via AV node)  P waves may not be buried, but can be. May be retrograde  Narrow QRS complexes (<120ms; unless pre-existing BBB)  T wave inversion common  ST segment depression
31
Wolf Parkinson White Syndrome
* Irregular rhythm, axis stable * PR interval shorter  Delta wave * Broad QRS (>120ms) * Leads to AVRT
32
Torades de Pointes
 A type of polymorphic ventricular tachycardia when there is QT prolongation  QRS “twists” around the isoelectric line.  >220bpm  VF
33
ST elevation: | V1-V4
Left Anterior Descending Anterior MI Also axis deviation Pathological Q may be seen
34
ST eelvation I, V5, V6
Left Circumflex, Lateral MI Also axis deviation Pathological Q may be seen
35
ST elevation | I, V1-V6
Left main Stem Antriolatral MI Also axis deviation Pathological Q may be seen
36
ST elevation | II, III, avF
Right Coronary Artery Inferior MI Also axis deviation Pathological Q may be seen
37
ST depression | V1-V4
Posterior Decending artery Posterior MI Also axis deviation Pathological Q may be seen
38
Pericarditis
 Widespread ST elevation on all ECG leads. Saddle shaped.  PR depression on limb leads (except aVR, elevation)  Sinus tachycardia is common (pain or pericardial effusion)  After days/months, it is replaced by T Wave inversion
39
vasospastic angina
ST elevation
40
PE
o Sinus tachycardia & atria tachyarrhythmia o Complete/incomplete RBBB o ST elevation or depression o Right ventricular strain (T wave inversion on V1-4, and inferior leads II, III, a) o RAD o P Pulmonale – Peaked p wave in Lead II o S1Q3T3 (Deep S wave in I, Q wave in III and inverted T in III)
41
Hypokal
``` o Increased amplitude o PR prolongation o T wave flattening and inversion o ST Depression o Prominent U waves o (long QT interval) ``` Tx: potassium
42
Hyperkal
``` o Peaked T waves (K>5.5) o Prolonged PR segment o Loss/flattening of P waves (K>6.5) o Weird QRS complexes (broad and bradycardia) (K>7.5) o Sine wave if extreme ``` Tx: 10% 10ml calcium gluconate to stabilise the myocardium, then dextrose to prevent hypoglycaemia, and insulin to help the cells take up potassium (salbutamol may also be used instead of insulin)
43
J waves
Hypothermia
44
Causes of ventricular tachy? (2)
Hypomagnesium, and hypokal