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Uveitis > White Dot Syndromes > Flashcards

White Dot Syndromes Flashcards

(61 cards)

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1
Q

Big Dots

A

Birdshot, APMPPE, Serpiginous

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2
Q

Small Dots

A

MCP, PIC (MEWDS, AZOOR)

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3
Q

Which are bilateral?

A

All except MEWDS and AZOOR

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4
Q

Which are more common in women?

A

Birdshot, MCP/PIC, MEWDS/AZOOR

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5
Q

Goes better without treatment

A

APMPPE, MEWDS

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6
Q

Which require long-term immunosupression?

A

Birdshot, Serpiginous (MCP, AZOOR)

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7
Q

Which cause CNV?

A

Serpiginous, MCP, PIC

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8
Q

In older patients

A

Birdshot and serpiginous

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9
Q

WD at deep choroid

A

Birdshot

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10
Q

WD at RPE/choriocapillaris

A

MCP, PIC, APMPPE, Serpiginous

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11
Q

WD at deep retina/RPE

A

MEWDS, AZOOR

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12
Q

Make scars

A

MCP, PIC, APMPPE, Serpiginous

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13
Q

nyctalopia, decreased color vision

A

Birdshot

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14
Q

classically quiet anterior segment

A

Birdshot

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15
Q

throughout the fundus

A

Birdshot

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16
Q

F=M

A

APMPPE, Serpiginous

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17
Q

flu-like prodrome

A

APMPPE, MEWDS

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18
Q

cerebral vasculitis

A

APMPPE

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19
Q

erythema nodosum

A

APMPPE

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20
Q

geographic pattern

A

Serpiginous

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21
Q

chronic, recurrent

A

Serpiginous, MCP

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22
Q

moderately myopic women

A

PIC

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23
Q

FA mild hyperfluorescence early with increasing hyperfluorescence late

A

PIC

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24
Q

New lesions usually do not appear

A

PIC

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25
Unilateral
MEWDS, AZOOR
26
Often have vitreous cells, venous sheathing and RAPD
MEWDS
27
Enlarged blind spot
MEWDS
28
FA-early hyperflu in wreath-like configuration. Late staining of lesions. Windows defects after resolution
MEWDS
29
reduced a-wave in ERG
MEWDS
30
may start unilateral but tends to be bilateral asymmetric
AZOOR
31
persistent and stabilizes by 6 months in most cases
AZOOR
32
Anterior uveitis (50%)
MCP
33
ERG remains normal until there is advanced retinal atrophy
MCP
34
involvement is predominantly macular
PIC
35
in 50% stabilization occurs within 6 months but recovery is infrequent
AZOOR
36
EOG shows absence or severe reduction of the light rise
AZOOR
37
ERG - a-wave and b-wave amplitude reduction
AZOOR
38
cones tend to be affected more than rods
AZOOR
39
often temporal
AZOOR
40
FAF. Hyperautofluorescent spots corresponding to the macular lesions are visible during active inflammation
MEWDS
41
Headache and other neurological symptoms are common and can commence many months after ocular disease onset
APMPPE
42
initially at the posterior pole
APMPPE
43
HLA-B7 and HLA-DR2
APMPPE
44
ICGA demonstrates non-perfusion of the choriocapillaris
APMPPE
45
M>F
Serpiginous
46
HLA-B7
APMPPE (also HLA-DR2), Serpiginous
47
typically starts around the optic disc and extends gradually
Serpiginous
48
Recurrence is usually contiguous with or adjacent to existing areas, eventually resulting in extensive chorioretinal atrophy
Serpiginous
49
Relentless placoid chorioretinitis (RPC)
features of both APMPPE and serpiginous choroiditis
50
Persistent placoid maculopathy (PPM)
similar to those of the macular variant of serpiginous choroidopathy, but which generally behave in a more benign fashion unless complicated by CNV
51
Vitiliginous Chorioretinitis
Birdshot
52
varying degree of vitritis is commonly found when the disease is active
Birdshot
53
ERG and VF are the most useful diagnostic tools
Birdshot
54
most numerous nasal to the optic disc
Birdshot
55
shimmering photopsias
MEWDS
56
100-200 um concentrated around the macula
MEWDS
57
typically does not present with vitritis
serpiginous, presumed ocular histoplasmosis syndrome
58
hypopigmented choroidal lesions 1/4 to 1/2 optic disc diameter, clustered around the optic nerve, radiating towards the periphery
Birdshot
59
ICG multiple hypofluorescent spots, which are typically more numerous than apparent on slit lamp
Birdshot
60
ERG prolonged 30 Hz flicker implicit times
Birdshot
61
diminished b waves compared to a wave
Birdshot

Uveitis (7 decks)

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