Wound Care Flashcards

(53 cards)

1
Q

Partial thickness wound healing

A
  • steam burn, abrasion
  • Re-epithelialization and regeneration
  • nl skin function returns
  • No scar
  • Epithelial cells come from hair follicle or edge of wound
  • 10-14 days
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2
Q

Full thickness wound healing

A
  • heal by scar tissue formation
  • lose nl tissue function
  • four phases:
    1. Inflammation
    2. Granulation/proliferation
    3. Epithelialization
    4. Maturation/remodeling
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3
Q

Inflammation stage of full thickness wound healing

A
  • First 7 days
  • Provide hemostasis and clear bacteria, foreign material, dead tissue
  • Can be confused with infection
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4
Q

Granulation/proliferation stage of full thickness wound healing

A
  • Fibroblasts synthesize collagen
  • Angiogenesis infiltrates collagen
  • O2 and nutrition demands are high
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5
Q

Epithelialization stage of full thickness wound healing

A
  • Overlaps with proliferative phase

- Continues from edges like partial thickness wounds

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6
Q

Maturation/remodeling stage of full thickness wound healing

A
  • May last up to 2 years
  • Overlaps proliferation and epithelialization
  • Reorganizes matrix collagen along lines of stress
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7
Q

Intrinsic factors that affect healing

A
  • General health
  • Age
  • Chronic disease
  • Immunosuppression
  • Sensory impairment (DM)
  • Tissue perfusion (DM)
  • Presence of necrotic tissue of foreign body (sutures)
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8
Q

Extrinsic factors that affect healing

A
  • Medication
  • Nutrition
  • Chemo/radiation
  • Stress
  • Infection
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9
Q

Iatrogenic factors that affect healing

A
  • Local ischemia
  • Treatment choices (irrigation material)
  • Trauma
  • Extent of injury
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10
Q

Partial thickness burns

- describe

A
  • epidermis is burned
  • Red/pink, mildly swollen
  • Skin feels raw and tender
  • Sunburn is MC example
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11
Q

Deep partial thickness burns

- describe

A
  • epidermis and dermis burned
  • Blistered, swollen, moist
  • Very painful
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12
Q

Full thickness burn

- describe

A
  • completely through dermis
  • Destroy fat cells, nerve tissue, muscle
  • Dry, leathery, appear dark brown, black, or dry white
  • May feel no pain if nerves are burned
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13
Q

Burn treatment

- overview

A
  • Depends on depth and extent of damage
  • Immediate care is important
  • Improper care → infection, slower healing, shock
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14
Q

What topical agent is usually used to treat burns

A
  • silver!
  • Lots of silver products
  • Good option for pts with sulfa allergy
  • Newer options allow fewer dressing changes
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15
Q

How to treat blisters

  • adults
  • children
A
  • Adults intact: protect, leave intact bc provide moist dressing for wound
  • Adult broken: debride, easily infected with bacteria
  • Peds: break intact blisters and debride, will pop anyways cause kids are crazy
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16
Q

Burn treatment

  • cleansing
  • dressing
  • chemicals
A
  • Cleansing options: Shower, pressurized saline, pulsed lavage
  • Dressing: Non-adherent that allows drainage
  • Chemicals: Only one chemical debridement treatment remains: Santyl. Hastens the removal of slough and eschar
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17
Q

How are facial burns treated differently

A
  • do not use silvadene (silver): can permanently stain

- Bacitracin ointment is best

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18
Q

Circumferential wounds

A
  • risk for compartment syndrome, ischemic limb

- check pulses often

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19
Q

Nutrition for burn care

A
  • protein
  • clear fluids
  • multivitamin
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20
Q

Burn aftercare

A
  • wil be itchy!!
  • Benadryl
  • Cold pack
  • Protect at night from scratching
  • Moisturize frequently
  • Liberal sunscreen!
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21
Q

What is most common cause of leg ulcer

A

venous insufficiency

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22
Q

Location of ulcers due to

  • venous insufficiency
  • arterial
A
  • venous: distal 1/3 of medial leg

- arterial: Lower leg, dorsal surface, foot, malleolus, toe joints, lateral border of foot

23
Q

Venous insufficiency ulcer

- characteristics

A
  • Irregular shape
  • Shallow
  • Mild pain (worse with standing, relieved with elevation)
  • Pink/red base
  • Edema
  • Hemosiderin stain around edge of wound (dark)
24
Q

Venous insufficiency ulcer

- RF

A
  • Prev trauma in the area
  • DVT hx
  • Pregnant
  • Obese
  • Clotting disorder
  • Fam hx
  • Occupation – stand a lot
25
Venous insufficiency ulcer | - 4 steps to manage
- Clean - Debride - Absorb drainage - Manage edema
26
Venous insufficiency ulcer | - clean
* Normal saline (warmed to body temp) | * If slough: wound cleanser with surfactant
27
Venous insufficiency ulcer | - Debride
- Chemical - Sharps (forceps, scissors, scalpel, curette) - Ultrasonic (new)
28
Venous insufficiency ulcer | - Absorb drainage
* 7 categories of dressing, gauze, alginates, hydrocolloids, carboxymethlycellulose, foams, ABD pads * Drainage is caustic to surrounding, healthy tissue
29
Venous insufficiency ulcer | - Manage edema
• Must check ABI prior to applying compression: >1.0, verify with transcutaneous O2 before applying compression - Vasopneumatic pump to move fluid caudally - External bandages - Multi-layer compression dressings - Circumferential measurements before and after to assess effectiveness
30
Because healing a wound doesn't treat venous insufficiency, what must occur to prevent recurrence of wound
maintain reduction of edema
31
How to reduce edema for venous insufficiency
* Forever wear support hose or circaids. * Leg elevation 2 hr a day, 20-30 min at a time, leg higher than heart, NOT bed rest * Exercise that activates calf muscles (use compression) * NO smoking * Don’t’ cross legs * Feet and leg hygiene * Avoid trauma * Nutrition, low sodium * Inspect legs/feet daily
32
Arterial ulcers | - characteristics
* Pain, esp at rest * Foot is cold or cool * Weak, absent pulses * Absence of hair growth * Skin is shiny, dry, pale * Thickened toenails * Necrotic ulcers with min drainage * ABI <0.5 * Pallor with leg elevation * Rubor of dependency (white when elevated, red when down) * Hx: HTN, smoking, claudication
33
Arterial ulcers | - risk factors
* Card hx * HTN * High cholesterol * Smoking * DM * Fam Hx
34
Arterial ulcers | - eval of arterial flow
* Pulses: dorsalis pedis, posterior tibial, popliteal, femoral * ABI: <0.8 → arterial compromise * Transcutaneous O2 <30 mmhg → no healing potential * Refer to vascular surgeon or cardiologist
35
What should NEVER be done to an arterial ulcer
debridement, will lead to further necrosis
36
Arterial ulcer | - treatment
``` - PT to increase circulation • Hot pack to femoral triangle • Rooke boots, bed cradles, and other protective gear and positioning • Antimicrobials: • Electric stimulation - STOP smoking!! - If gangrene keep dry - Wound on leg, encourage exercise to increase blood flow ```
37
Negative pressure therapy
- Wound vac - Can be used on all kinds of wounds - Wound must be fairly clean - Cut foam slightly smaller than wound, do not overpack
38
Biological wound treatment
- Oasis - Porcine small intestinal submucosa. Provides matrix for collagen to use as a framework. - Do not remove, keep adding to it if it works - Might produce inflammatory response but that is ok (it's not infected)
39
Cultures for wound management
- Punch biopsy is best but swab is adequate | - Used to base topical treatment options
40
Hyperbaric oxygen
is another type of wound treatment
41
Topical commonly used
* Clobetasol * Cyclosporin or dexamethasone * Gentamycin
42
Why are pressure injuries important (business aspect)
* Stage III and IV are hospital acquired, won’t be paid to treat * Inspect FULL body of patient when admitted to ensure none present. If present, document size and location
43
What can be confused with pressure injuries
* DM foot ulcers * Skin tears and lacerations * Maceration dt diarrhea or infection * Arterial or venous leg ulcers
44
Pressure injuries | - risk factors
* Pressure * Moisture * Friction * Shear * Hypotension (poor perfusion) * Lengthy immobilization * Poor nutrition
45
Pressure injuries | - Common locations
``` bony prominences: • Occiput • Sacrum • Ischium • Heels • Trochanter • Knee • Ankle ```
46
Describe stage 1 pressure injury
Non-blanchable erythema of intact skin
47
Describe stage 2 pressure injury
* Partial-thickness loss of skin with exposed dermis * Wound bed is viable, moist, pink/red * May be intact or ruptured serum-filled blister
48
Describe stage 3 pressure injury
* Full-thickness loss of skin * Adipose is visible, granulation and rolled wound edges are often present * Slough or eschar may be visible * Depth depends on location * No exposure of fascia, muscle, tendon, ligament, cartilage, bone
49
Describe stage 4 pressure injury
* Full-thickness skin and tissue loss * Exposed or directly palpable fascia, muscle, tendon, ligament, cartilage, bone * Slough and eschar may be visible
50
Describe unstageable pressure injury
* Can’t evaluate dt presence of eschar or slough | * If can’t see base of wound, is unstageable
51
Describe deep tissue pressure injury
Often seen in patients who have “been down” for a long time, injury is still evolving, not sure how bad it will be
52
Describe pressure injury d/t medical device
* New category | * Tubes, drains, cannula, etc.
53
Describe facility acquired pressure injury
* Usually sacrum (also heels and occiput) * Can occur in children * Many pts have scI, spinal orthotics * Perineal dermatitis or excoriation dt diarrhea is NOT a pressure injury but does place pt at increased risk for one