Wound Management Flashcards
(36 cards)
Lipodermatosclerosis
Inverted Champagne Bottle Legs
70% of Chronic Wound is caused by?
Chronic Venous Insufficiency
Signs of Chronic Venous Insufficiency
- Pitting Oedema2. Haemosiderin Staining3. Venous ulcer - painless, irregular, copious exudate4. Atrophie Blanche5. Lipodermatosclerosis
RF of Chronic Venous Insufficiency
Obesity, DVT, Poor mobility
Tx of Chronic Venous Insufficiency
- Graduated compression - toe to knee (ankle 30mmHg)2. Address factors that delay healing3. Must exclude arterial involvement
4 Principles of Wound management
- Define Aetiology - Vascular/Mechanical/Neuropathic/Infective2. Control wound healing factors3. Select appropriate dressing4. Plan for management
Signs of lymphoedema
- Scale and keratin build-up2. Skin thickening3. Hard to pick up skin4. Non-pitting oedema
Secondary causes of Lymphoedema?
- Cancer2. Infection - Filariasis (Elephantiasis)
4 Si/Sx of Peripheral Arterial Disease (PAD)
- Claudication to rest pain2. Lower ABI - nml is 1.03. Weak pulse, poor refill4. Arterial ulcer - regular, punched out, below ankles
Tx of Arterial ulcers
- Improve flow - angioplasty/stent/bypass (essentially angina of leg)2. Amputation of digit or limb
Difference between Ischaemic or neuropathic ulcers?
Neuropathic is:1. Painless2. Bony prominence/area of pressure3. Good circulation for healingWhich are all opposite in ischaemic
RF and Mx for Pressure ulcer
- Neuropathy 2. Immobility 3. Malnutrition Mx: manage RF, risk assessment, Foam
Friction vs Shear
Friction - epidermis worn away by rubbing external surfaceShear - skin is restrained from sliding while tissue are forced to move
4 Stages of Pressure Ulcer
- Erythema remains after light pressure2. Skin loss involving epidermis/dermis3. Skin loss involving subcutaneous tissue/fascia4. Skin loss with necrosis to underlying structures (muscle/bone/joint etc)
Complication of leg ulcers
- Infection2. Gangrene3. Calcification4. Neoplastic development
Risk assessment in pressure ulcers
Norton scale Less than 16/20 is at risk4 marks of each:Physical condition, mental condition, activity, mobility, incontinent
Intrinsic Factors affecting healing? (4)
- Age and build (old or obese?)2. Immune function/DM (immune status)3. Nutrition4. General health status
Extrinsic Factors affecting healing? (4)
- Drugs (NSAIDs, steroids, Bblockers impairs healing)2. Smoking3. Wound factors: Debris, infection, dryness4. Wound location: Mechanical (movement) /Chemical stress
Skin Tear Mx Plan (First aid, dressing, FU)
- Stop bleeding - Alginate2. Cleansing and pat dry(NS/Tap water)3. Steri-strips if skin flap present (mark skin flap direction, avoid tension, max 1cm apart, avoid adhesive tapes)4. Dressing (Hydrogel +/- foam, Mepitel if major skin loss as it is protein and remain in place for 10d)5. Analgesia and tetanus6. RV 24hrs
Why does Skin tear occur in old age?
Skin changes with age, thus trauma easily tears skin:Epidermis: less turnover, langerhansDermis: loses 80% thickness, vessels diminish by 40% and fragile, less collagen, skin less elastic
Burn conversion
Partial thickness thermal burn convert to full thickness from residual energy
Burn first aid
- Cold (not ice cold) running water for 30min2. Check for hypothermia, wrap pt in towel to keep warm
Burn Body surface area estimation
Face/Arms: 9% eachThorax/Abdomen/Legs: 18% each
Major burns must obtain the following information (5)
- Type of burn - thermal/chemical2. Depth and Area%3. Presence of inhalation injury4. Associate injuries5. Medical status of the patient