Yr 2 management of common orthopaedic conditions (main lecture) Flashcards

(68 cards)

1
Q

Session plan

A
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2
Q

General principles in Trauma and orthopaedics

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  • These are pegs to hang your hat/scarf on
  • With these principles you should be able to take a history and examine any part of the limbs and hold your own.

We divide MSK into trauma, orthopaedics and rheumatology (will cover later on)

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3
Q

Bone cells

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-Osteoclasts are phagocytic cells

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4
Q

Organization of compact/cortical bone: osteons

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In cortex where osteons exists and these consist of a haversian canal which has blood vessels nerves and lymphatics.

These canals have concentric lamellae of bone tissue and these contain osteocytes. The osteoblasts and clasts are mainly in the lining of bone so endosteum and periosteum

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5
Q

Structure of long bones

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Centre is medulla-spongey part of bone

Outer bit is cortex

Physis one proximally and one distally

When you have a physis, the bit closest to the joint is the epiphysis, just beneath the physis away from the joint is the metaphysis and long aspect of bone is diaphysis.

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6
Q

Trauma/fractures

Mechanisms of Bone Fracture

A

How can bones break?-trauma eg injury –low-fall from standing height

Abnormal on normal eg marathon

Normal on abnormal-pathological fracture

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7
Q

Pathological or insufficiency – Normal stresses on abnormal Bone: local or general

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Pathological fractures can be caused by abnormal density, abnormal mineralization or malignancy,

Pagets disease is an abnormal turnover of the bone

Insufficiency-where bone is weak due to metabolic diseases eg osteoporosis or osteomalacia

Pathological-anything where bone is abnormal

Osteoporosis can be both

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8
Q

Fracture Patterns

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Is skin covered over fracture?

Is it just 2 fragments or is it comminuted ie many)

Is it displaced?

Are the Soft Tissues Intact?

  • Yes – Closed
  • No – Open

Is the break complete?

  • Yes – How many pieces?
  • 2 – simple
  • >2 – comminuted
  • No – Greenstick

Are the bony ends aligned?

  • Yes – Displaced
  • No - Displaced
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9
Q

Fracture Healing

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When fractures happen you get bleeding, haematoma formed, cells and cytokines start producing collagen and then then turns into callous, initially type 2 collagen and then callous harden with type 1 collagen

Then osteoclasts and blasts remodel bone based on stresses put on it.

Step 1: Bleeding/Haematoma – prostaglandins/cytokines released; growth factors increase local blood flow – Periosteal supply takes over

Step 2: Granulation Tissue/connective tissue/Fibrous tissue laid down / Soft Callus

Step3: Once fracture is bridged with soft callus- hard callus is formed ( laying down of Osteoid/ bone)

Step4 : Bone is remodelled via endochondral ossification lamellar bone in its place.

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10
Q

Trauma/fractures

What are the clinical signs of a fracture?

A

What could be damaged?

What is damaged and what is around structure that energy could have damaged

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11
Q

Investigations of fracture

A

MRI for soft tissue (also getting good at architecture of bone)

CT-architecture of bone

Bone scan-invasive (need dye) and a lot of radiation, so do these rarely, only in bone disease

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12
Q

Describing a fracture radiograph

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4th picture of leg is diaphyseal fracture

Middle-between diaphysis and metaphysis so metaphyseal diaphyseal junction

An avulsion fracture is an injury to the bone in a location where a tendon or ligament attaches to the bone

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13
Q

Displacement

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Translation-movement occurs in staright line

Or rotation

Injury and healing lecture has more detail on this

Here we have left femur, say fracture is midshaft and distal fragment has moved In straight line away from midline so lateral translation

With angulation again have 3 planes

Coronal plane-from front

Varus-towards midline

Axial plane-from foot upwards, can have internal/extrenal rotation

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14
Q

Describe this radiograph

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15
Q

Describe this radiograph 2

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16
Q

Management of fractures

General principles

A

May get a question –tibial fracture-what are principles of management and what are order?

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17
Q

Reduction:

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18
Q

Hold

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Might hold with plaster or stick pin in bone-traction, don’t do this often as lots of time in bed, better to use fixation

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19
Q

Fixation

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Internal-under skin

External-through the skin

Extramedullary-plate on surface of bone

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20
Q

Rehabilitate

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Is lower limb-get patient to weight bare

Strengthen muscles and helps remodelling

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21
Q

What types of fixation are these (internal/external; medullary; mono/multiplanar)?

A

Different types of holding fracture

Plate is on surface of bone so extramedullary fixation but its under the skin so is internal

Pin going through bone is intramedullary internal fixation

External can be in different planes so monoplanar or multiplanar

Reduction-may involve pulling on it, or may have to do an open fixation if doesn’t go back to correct location.

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22
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23
Q

Fracture Complications

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Divide into general-affecting whole body, or local-at the fracture site

General include:

  • Fat embolus-fat from bone marrow can enter blood and travel to lung etc.
  • Can have blood clot in that limb
  • Or blood clot can travel from limb, so thromboembolic pulmonary pathology
  • Systemic infection in form of sepsis
  • Traction-have to lie in bed for 2 months so chest infections as not breathing as deeply, pressure sores as one position, hence this isn’t preferred
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24
Q

Local Complications of fractures

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Urgent-immediate-think muscles, veins, nerves, tendons, plaster can be on too tight and can trap nerve

Early-within 30 days

Late-after 30 days

algodystrophy, also known as complex regional pain syndrome (CRPS),-stiffness of muscles and abnormal pain response due to long term bone problem

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25
Fractured neck of femur
Need to know this! Common due to osteoporosis leading to insufficiency fractures Want to know their risk factors and cormorbidities for doing operation
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Nof Anatomy: label these parts
Intertrochanteric line inbetween Capsule attaches to Intertrochanteric line line at front and half way up the neck on the back
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Types of NoF fractures by location: can you label? Intra or extracapsular?
Subcapital and transcervical fractures are considered intracapsular fractures.
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=transcerviacal fracture Hard to say if extra or intra 2-completely out of position so displaced, but 1 is not
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If extracapullar fracture-risk of bone dying is low If intra-risk of head of femur dying is higher so might want to chuck head away and replace it, except for younger placements as they will have these replacements for years
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Neck of femur management:
Extra-fix Intra- if displaced-depends on age of patient
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How do we classify joints?
Articular surface of hip joint is bits that move against itself, so even an intracapsular fracture can be extraarticular as may not directly affect the joint surface Fibrous-in skull and in tubia and fibula in ankle Cartilaginous-where true bit of cartilage between joints eg between vertebra in spine
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Components of a synovial joint
Has an articular surface which is made up of hyaline cartilage They also have a capsule which is fibrous structure made up of collagen Synovium produces synovial fluid
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Cartilage:
Articular cartilage is trying to reduce friction so has specialized cells called chondrocytes, it has arrangement of cells more horizontal Extracellular matrix
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Joint diseases - overview
Osteoarthritis-mainly disease of articular cartilage Rhematoid is inflammation of synovial membrane
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Radiographic changes in Rheumatoid Arthritis vs.
In both-joint space can be narrowed Sclerosis is extra bone formation Osteoarthritis has mainly thick bone at surface but in RA it is mainly thin here
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Pathophysiology
Can result in inflammation but this is a late bit of the disease and there are lots of inflammatory mediators of the disease. Inflammation occurs after articular surface has been damaged Can result due to obesity or after trauma where surface is no longer smooth so will wear out fater Analgesics, injections and sometimes surgery
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Risk factors:
Weight and obesity and estrogen deficiency are modifiable, some are not modifiable eg gender or age or trauma
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Osteoarthritis How do patients present?
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Assessment for any joint
* Look * Feel * Move * Special tests
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Look
Valgus deformity as distal bit going away from midline Valgus is a term for outward angulation of the distal segment of a bone or joint. (Think you move away from valgor things!) Coronal plane Knee replacement scar
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Feel
Palpation there is a small effusion
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Move
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Special tests
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Special tests
Think would be valgus ie distal limb going out as it was the medial cruciate lig at knee attaching it.
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In summary
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Plan
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More wear produces more bone and as get more wear get more fluid entering joint space
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Patient lying down vs standing up Important to stress the limb by weight bearing
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OA management
In management of anything say conservative or operative
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Normal ankle of right and on left arthritic ankle joint and they put screws through so joint not moving so no pain, although have stiff joint can still weight bear better than replacing joint which can’t take much weight.
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Bone infection
Bacteria seeded on metal from surgery so could be secondary or direct bacteria into blood If chronic infection can get weight loss Bacteria in joint start eating away and ligaments, synvium, lots of pus, red hot painful, hard to move
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Septic arthritis
Can come from cut, insect bite etc Emergency as bugs eating joint surface
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Investigations
The indium white blood cell scan, is a nuclear medicine procedure in which white blood cells are removed from the patient, tagged with the radioisotope Indium-111, and then injected intravenously into the patient. The tagged leukocytes subsequently localize to areas of relatively new infection.
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Treatment
Bone infection-takes a lot of antibiotics to clear it as bone doesn’t have great blood supply
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Some joint examination tips Shoulder
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Shoulder conditions
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Hip
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Hip Conditions:
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Knee
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Knee conditions
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Spine:
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Bone infection
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Investigations
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Treatment:
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Synovial Joints: functional types Classified according to shape and how they move Ankle hinge-as moves in one direction Knee-modified hinge joint as there are some other forms of movement too How are synovial joints stabilized?
How much of these contribute in each joint vary eg in hip-bone congruity big, tendons and lug small Shoulder-bony-small factor but tendons and lig big factor
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Session Plan: common conditions, exam questions, principles of management