CAMHS Flashcards

1
Q

Differences in childhood/adolescent presentation of common psychiatric disorders: depression

A

equal sex ratio before puberty
somatic sx e.g. headache, tummyache
irritability, reduced school performance

mx:
1st medication = fluoxetine (prescribed by specialist after MDT discussion)
1st tx (mild, 4 weeks) = CBT
antidepressant medication only offered in combination with psych therapy
prognosis generally good, severe episodes likely to occur

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2
Q

Differences in childhood/adolescent presentation of common psychiatric disorders: anxiety disorders

A

9-32% period prevalence

equal sex ration

many commence in adolescence

may present with somatic sx

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3
Q

Differences in childhood/adolescent presentation of common psychiatric disorders: self-harm

A

all <16 y/o who self-harm must be reviewed by a CAMHS specialist before discharge and admitted to a paediatric warm to facilitate this if necessary

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4
Q

Differences in childhood/adolescent presentation of common psychiatric disorders: psychosis

A

very rare in children before puberty

poor prognosis, disrupted social development

important to exclude ASD and organic causes (e.g. autoimmune disorders)

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5
Q

Differences in childhood/adolescent presentation of common psychiatric disorders: eating disorders

A

may present with faltering growth/delayed puberty

expect body weight calculations consider sex, age, and height on centile charts

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6
Q

Normal development

A

developmental delays can be referred to CAMHS

further assessment is always needed when developmental milestone attainment is delayed beyond the upper ;limit, with consideration of risk factors

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7
Q

Separation anxiety disorder

A

excessive fear of separation from specific attachment figures → significant distress/functional impairment

sx = thoughts of harm coming to their parent, reluctance to attend school/sleep apart, marked distress at separation, and nightmares about separation

threatened/unmourned loss

mx = family support, child’s anxiety with behavioural therapy, gradually increased separation periods

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8
Q

School refusal

A

Unconcealed school absence

common during transition e.g. new school/sibling

bullying, fear of failure, unsympathetic teacher

may occur in families with so-called precious children (following difficulty conceiving/sibling’s death) or vulnerable parents (experiencing life-threatening illness or agoraphobia)

tummy aches before school (never on weekends or holidays)

mx = family support, school support, rapid return to full attendance is best prognosis, check for parental depression and separation anxiety, get young person to talk separate from parents

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9
Q

Enuresis

A

repeating voiding of urine into clothing/bed by day/night above age of expected urinary continence (5 y/o) in the absence of organic causes

FHx

primary = toilet training not mastered

secondary = dryness is lost after at least a year’s continence (usually stress-related)

nocturnal enuresis = more common in boys

diurnal enuresis = more common in girls

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10
Q

Enuresis mx

A

reassure (common and nobody’s fault)

refer organic causes to a paediatrician e.g. epilepsy, UTI, constipation and diabetes

address stressors and review toilet training received so far

restrict fluids before bed

use star charts to celebrate each dry night (positive reinforcement)

Bell and pad ‘underpants alarm’: clips onto pyjamas, waking the child if moisture is detected, to retrain voiding

Medication e.g. imipramine (tricyclic antidepressant) combined with desmopressin (synthetic antidiuretic hormone) may be considered when all other ex’s have failed

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11
Q

Encoparesis: definition and causes

A

repeated defecation in inappropriate places above 4 years in absence of organic causes, commoner in boys, primary/secondary

mostly due to overflow incontinence due to:

  • dehydration
  • painful defecation (e.g. anal fissure)
  • fear of punishment
  • toilet fears (e.g. monsters in the toilet)
  • Hirschsprung disease (rare: bowel obstruction due to aganglionic section of the colon)

When constipation absent, incontinence due to:

  • diarrhoea
  • disorders of intellectual development
  • hostility (e.g. angrily defecating in a parents shoe)

Punitive toilet training → strsss → trigger secondary incontinence

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12
Q

Encoparesis: mx

A

laxatives and stool softeners for constipation

treat physical causes

reassure, address stress, and review toiler training

star charts to reinforce continence

GOOD prognosis 60-90% become continent within a year

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13
Q

Selective mutism: definition, symptoms, tx

A

consistent selective speech in specific social situations but not others, lasting at least a month, not limited to the first month of school, to the extent of disrupting education

talkative at home but painfully shy and silent elsewhere, tx involves reassurance; stress and behavioural mx

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14
Q

Autism spectrum disorder: epidemiology

A

masked by imitating socially expected behaviours

  • strong genetic basis
  • older parental age
  • maternal infections in pregnancy
  • obstetric complications leading to hypoxia

highly comorbid with other conditions

  • disorders of intellectual development
  • epilepsy
  • tuberous sclerosis = rare genetic condition that causes mainly non-cancerous (benign) tumours to develop in different parts of the body.
  • Down syndrome
  • Rett syndrome = affects brain development
  • fragile X syndrome
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15
Q

ASD: clinical presentation

A

struggle to initiate and sustain reciprocal social interaction and social communication

restricted, repetitive, inflexible interests and behavioural patterns

sx often identified first 3 years of life

severe enough to impact educational/occupational and other functioning

associated with a range of intellectual and language abilities and its functional impact varies across the spectrum

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16
Q

ASD: reciprocal social interaction

A

struggle to express emotions, ‘read’ other people, understand their feelings and intentions

→ may be interpreted as insensitive/socially awkward, influence ability to make friends

prefer to make their own company, especially when surrounded by people

17
Q

ASD: communication

A

difficulty interpreting verbal and non-verbal communication e.g. gestures (pointing), facial expressions, tone of voice

interpret language literally (concrete thinking) including jokes and sarcasm

speech onset often delayed

echolalia = repeat sentences verbatim (echolalia) or speak continuously about their own interests without pausing to hear from others

speech can be monotonous with limited prosody and pronoun reversal (saying I/me to mean you/she)

18
Q

ASD: repetitive behaviour

A

world confusing and unpredictable

daily routines, rigid food preferences, school/transport habits but lead to distress or tantrums when can’t be accommodated

play games repetitively/order toys by abstract properties rather than play imaginatively

may develop intense, focused interests from a young age

19
Q

ASD: associated symptoms

A

Sensitivity to sound, touch, taste, smell, light colour, temperature → anxiety and discomfort

self-stimulating (e.g. hand flapping) and self-injuring behaviour (e.g. head-banging) are common

20
Q

ASD: DDx

A

untreated deafness impairs language acquisition

developmental language disorder → persistent difficulties in language acquisition, understanding, production, or use, disproportionate to intellectual development

disorders of intellectual development → significantly below-average (2 or 3 SD below mean), intellectual functioning and adaptive behaviours, often associated with impairments of complex language acquisition and comprehension, academic skills, self-care, and domestic and practical activities

development syndromes which cause ASD e.g. Rett’s syndrome

neglect can impair language acquisition and socialisation (reversibly, unless it’s particularly severe)

21
Q

ASD: Ix

A
  • hearing tests for deafness
  • SAL assessment
  • neuropsychological cognitiv assessment
  • specialist ASD assessment incoroporating nursery/school reports, detailed developmental and FHx, observation in several settings, physical examination, and tailored assessment of the child’s cognition, communication, behavioural, and mental state e.g. Autism Diagnostic Interview (ADI), Autism Diagnostic Observation Schedule (ADOS)
  • Genetic tests if dysmorphic features, congenital abnormalities, FHx, or comorbid disorder of intellectual development
  • EEG if epilepsy is suspected
22
Q

ASD: Mx

A

Carer support and advice e.g. National Autistic Society

Behavioural therapy = reinforce positive behaviours and discourages challenging ones

SALT

school support via education, health and care plan (EHC) plan

tx for comorbid physical (e.g. epilepsy) and mental health problems (e.g. depression, anxiety disorders)

antipsychotics and mood stabilisers are occasionally prescribed for severe aggression or hyperactivity not responding to behavioural interactions

23
Q

ASD: prognosis

A

lifelong

need skills and strategies for children and families to manage symptoms into adulthood

24
Q

ADHD: what is it? epidemiology

A

persistent pattern (at least 6 months) of inattention and/or hyperactivity-impulsivity

early to mid-childhood, outside normal variation, significantly interferes with functioning, in more than one setting

3:1 m:f

75% heritable

unknown cause

comorbid disorders = oppositional defiant disorder (ODD), and conduct/dissocial disorder (CDD), disorders of intellectual development, Tourette syndrome

25
Q

ADHD: clinical presentation and associated features

A

hyperactivity = excessive movement/difficulty keeping still in situations requiring behavioural self-control

  • boisterous, excessive energy, constantly on the move

Inattention = distractibility, disorganisation, difficulty concentrating on tasks that aren’t exceptionally stimulating, without frequent rewards

  • struggle to focus in class, flitting between activities, leaving tasks unfinished

Impulsivity = act IRT immediate stimuli without deliberating or considering risks and consequences

  • dangerous (e.g. road safety) and can cause parental concern

Associated features = accident-prone or disobedient (through impulsivity rather than defiance), pay little attention to social conventions

26
Q

ADHD: Ddx

A

Organic disorders e.g. hearing impairment, epilepsy

CDD

Agitation in depression or anxiety

Mania (rare in children)

27
Q

ADHD: Ix

A

Questionnaires e.g. Conners Rating Scales, completed by the child, parents, and teacher

Clinician observation in a classroom setting

  • collateral from teacher

Educational psychologist assessment

28
Q

ADHD: Mx

A

Parent training and education programmes

Educational psychologist assessment of child’s classroom needs

Group CBT and social skills training

Social support and self-help for families e.g. the national attention deficit disorder information and support service (ADDISS)

Stimulant medications (improves concentration, facilitate learning) = methylphenidate, dexamftamine, lisdexamfetamine

  • not addictive for ADHD
  • side effects: nausea, diarrhoea, HTN, tachycardia, appetite suppression, insomnia
  • → drug-free weekends and school holidays limit growth restriction s/e

Non-stimulant medication = atomoxetine, guanfacine

29
Q

ADHD: prognosis

A

children may experience low self-esteem, peer rejection, educational underachievement, and harsh parenting

sx often improve in adolescents

untreated ADHD is a RF for later dissociality in personality disorder, criminal behaviour, and substance use

30
Q

Conduct/dissocial disorder CDD: what is it, epidemiology, RFs

A

Repetitive and persistent pattern (1 year or more) of behaviour violating either basic rights of others, or major age-appropriate societal norms, rules, or laws

  • aggression towards people/animals
  • destruction of propertu
  • deceitfulness or they
  • serious rule violations

of sufficient severity to significantly impact the child’s personal, family, social, educational, or occupational functioning

4:1 m:f

RFs: urban upbringing, deprivation, parental criminal activity, harsh and inconsistent parenting, maternal depression, FHx of substance use

Dissocial (antisocial) behaviur is often learned from parental or environmental exposure, and may be reinforced e.g. increased attention

31
Q

CDD: clinical presentation

A

persistently dissocial e.g. bullring, stealing, fighting, fire-setting, truancy, cruelty to animals or people

socialised CDD = dissocial behaviour done in a peer group

unsocialised CDD = rejected by other children, making them isolated and hostile

32
Q

CDD: Ddx

A

ODD = persistent pattern (6 months or more) of markedly defiant, disobedient, provocative, or spiteful behaviour. Prevailing angry or irritable mood, severe temper outbursts or headstrong, argumentative and defiant behaviour sufficiently severe to impact the child’s functioning

ADHD

Depression

33
Q

CDD Mx

A

Support to understand CDD and limit potentially reinforcing responses

Parent management training teaches caregiver to reward good behaviour and respond constructively to undesired behaviour

Education support: close working with teachers is crucial, as children with CDD are at risk of exclusion

Anger management for the child or young person

Treatment of comorbid disorders e.g. ADHD

34
Q

CDD: prognosis

A

50% children with CDD develop substance use or dissociality in real life

35
Q

Psychiatric hospital admission for children reserved in…

A

Eating diorders

Affective disorders

Psychosis

Severe/complex neurodevelopment disorders

Presentations with severe self-harm

36
Q

Psychiatric hospital admission for children reserved in…

A

Eating diorders

Affective disorders

Psychosis

Severe/complex neurodevelopment disorders

Presentations with severe self-harm

37
Q

Tic disorders: what is it, features, epidemiology, mx

A

Sudden, rapid, non-rhythmic, involuntary, recurrent movements or vocalisations

may be simple (e.g. blinking, sniffing, tapping, throat-clearing) or complex (e.g. self-hitting, swearing)

recede when distracted

voluntarily suppressed by cost of internal tension → seen on expression

3:1 m:f

OCD/ADHD can be comorbid

stress and stimulant medications worsen tics

Mx = reassurance and stress mx, clonidine (adrenergic agonist) or antipsychotics can help