11/9- Common Pediatric Musculoskeletal Findings Flashcards

(73 cards)

1
Q

Describe the pediatric skeleton vs. adult

  • What is the most metabolically active part
A
  • Bone is more porous and pliable
  • The metaphysis is the most metabolically active part of the bone
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2
Q

What is the weakest part of the pediatric skeleton?

  • When is this visualized
A

The physis (epiphyseal plate or growth plate)

  • Growth plates usually aren’t visualized until 12-24 mo
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3
Q

T/F: Remodeling and healing occurs rapidly in the pediatric skeleton

A

True

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4
Q

Describe developmental dysplasia of the hip in newborns/infants

  • Prevalence
  • Risk factors
A

Abnormal development that causes the proximal femoral head to displace from the acetabulum (but may present anytime during childhood)

  • `1/1000 live births

Risk factors:

  • Female > Male
  • First born
  • Breech
  • Caucasian descent
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5
Q

How do you test for developmental dysplasia of the hip?

A
  • Barlow test: adduct and push posteriorly (to displace)
  • Ortolani test: abduct and push anteriorly (to replace)
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6
Q

What is seen here?

A

Asymmetric skinfolds (in developmental dysplasia of the hip)

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7
Q

What is seen here?

A

Positive Galeazzi (hip displacement)

  • Lower knee is the affected knee (necrosis)
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8
Q

What should be done if there is newborn/infant developmental dysplasia of the hip?

A
  • Fitting with Pavlik harness
  • Outpatient Orthopedic consultation
  • In high risk patients (girls, + family history, breech)
  • Hip ultrasound at 6 weeks of age or frog-legged films at 4 to 6 months of age
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9
Q

What is Congenital Torticollis?

  • Incidence
  • Risk factors
  • Suspected cause
  • Signs/symptoms
A
  • Incidence 1-2 %
  • More often in breech presentation
  • Suspected trauma to SCM and resulting fibrosis

Signs/symptoms:

  • Head tilt to the side of the affected muscle by 2-4 weeks
  • “tumor” or “psudotumor” noted on exam
  • Can present with facial deformity
  • Can result in plageocephaly
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10
Q

What is seen here?

A

The facial deformity common in congenital torticollis

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11
Q

What is plagiocephaly?

A
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12
Q

What is treatment/therapy for congenital torticollis?

A
  • Stretching Exercises with OT/PT
  • Surgical release if persists 12-18 months
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13
Q

What is Congenital Talipes Equinovarus?

  • Incidence
  • Uni or bilateral
  • Gender prevalence
  • Etiologies
A

Excessively plantar flexed, rotated medially, and the sole facing inward (club foot)

  • Incidence is about 1 -3/1000
  • Almost half are bilateral
  • 2x in females
  • Multiple causes
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14
Q

What is treatment/therapy for Congenital Talipes Equinovarus?

A
  • Cereal casting every 1-2 weeks to hold foot in desired position.
  • Surgical correction
  • Combination of both
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15
Q

What is seen here?

A

Arthrogryposis

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16
Q

What are factors leading to arthrogryposis?

A
  • Neurologic deficits
  • Fetal crowding
  • Maternal illness
  • Connective tissue/skeletal defects
  • Vascular compromise
  • Muscle defects

All contribute to limitation of fetal joint mobility and then joint fixations (arthrogryposis)

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17
Q

What is Osteogenesis Imperfecta?

  • Aka
  • Prevalence
  • Signs/symptoms
A
  • It is often called “brittle bone disease.”
  • Occurs in about 1/20,000 births
  • Severely affected patients suffer multiple fractures with minimal or even no trauma
  • Infants with the worst form of OI die in the perinatal period
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18
Q

Describe the different types of Osteogenesis Imperfecta?

  • Signs/symptoms
  • Severity (lethality)
A

Type I

  • Blue sclera

Type II

  • Most lethal type

Type III

  • Most severe non-lethal form
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19
Q

What is Legg-Calve Perthes?

A

Avascular necrosis of the femoral head

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20
Q

Describe Legg-Calve Perthes

  • Gender prevalence
  • Risk factors
  • When is it commonly seen
  • Uni or bilateral
A

(Avascular necrosis of femoral head)

  • 4x in males
  • Common among first-born children
  • Common during periods of rapid growth of epiphyses (peak between 5-7 years)
  • Usually unilateral but up to 12 percent are bilateral
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21
Q

How does Legg-Calve-Perthes commonly present?

A
  • Present with limp of insidious onset
  • Pain is often referred to the antero-medial thigh or knee.
  • On exam, patients may have limited internal rotation and abduction of the hip.
  • Pain may lead to disuse, which may result in atrophy of the thighs and buttock
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22
Q

What is seen here?

A

Legg-Calve-Perthes disease on radiography

  • Left = normal
  • Right = hip with perthes
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23
Q

What radiographs should be taken in Legg-Calve-Perthes evaluation?

  • What are the radiographic findings (early/intermediate/late)?
A

Should take AP and frog-leg views

Findings:

  • Early: effusion of joint and wide joint space
  • Intermediate: decreased bone density and collapse of the femoral head
  • Late: new bone replacing necrotic bone
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24
Q

How to treat Legg-Calve-Perthes disease?

A
  • Pedi ortho consulation
  • Contain femoral head in acetabulum (may require abduction splint

• < 6 yrs: if no significant subluxation and at least 40-45 degrees abduction, observation is warranted.

> 6 yrs: containment with brace/splint until re-ossification or sugery

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25
What is Slipped Capital Femoral Epiphysis?
Displacement of the proximal femoral epiphysis from the femoral neck through the pysis, usually posteriorly and medially _Caused by:_ - Most cases are idiopathic - Weak growth plates - Local trauma
26
Who gets Slipped Capital Femoral Epiphysis?
- Between ages **6-10 yo** - More in **males** - Obesity - Hypothyroidism - Growth hormone administration
27
What are signs/symptoms of Slipped Capital Femoral Epiphysis?
* Limping with or without pain * Pain can radiate to groin or knee * lost flexion and abduction * child holds his/her leg in external rotation at rest
28
How is Slipped Capital Femoral Epiphysis diagnosed?
* AP and frog-leg lateral * Bloomburg’s Sign (widening epiphysis) * Klein’s Line * Always get contra-lateral films
29
What is seen here?
Slipped Capital Femoral Epiphysis
30
What is shown here?
Slipped Capital Femoral Epiphysis
31
What is Osgood-Schlatter? Symptoms?
* Inflammation of the proximal tibial tubercle at the insertion of the patellar tendon secondary to traction * Pain and swelling at the tibial tubercle
32
What populations/demographic are affected by Osgood-Schlatter? - Uni or bilateral
* Children **9 -14 years** of age who have undergone a **rapid growth spurt** * **20% of adolescents** who are active in **sports** compared with 5% of non-athletes * Sports that involve running, cutting, and jumping * These activities place stress on the tibial tubercle through repetitive contraction of the quadriceps muscle • Bilateral in 25-50% of cases
33
What are signs/symptoms of Osgood-Schlatter? - How is the diagnosis made
* Anterior knee pain that increases gradually over time * Pain is exacerbated by direct trauma, kneeling, running, jumping, squatting, climbing stairs, or walking uphill, and is relieved by rest. * The diagnosis of Osgood-Schlatter disease is made by physical examination, radiographs are optional
34
What are exam findings in Osgood-Schlatter?
- Tenderness and soft tissue or bony prominence of the tibial tubercle - Pain may be reproduced by extending the knee against resistance or squatting - Evaluation the hip to make sure knee pain not referred pain from pathology in the hip
35
What is intoeing? - What most commonly causes it
* Commonly known as **“Pigeon-toed”** * More common than outtoeing * Most commonly due to: * Metatarsus adductus * Tibial Torsion * Medial femoral torsion
36
What are the 3 rotational deformities characterized under "intoeing"?
- Metatarsus adductus - Tibial torsion - Medial femoral torsion
37
What is Metatarsus adductus? - What kind of defect is it - Gender prevalence - Correction
* Intoeing originates at the foot * Considered a packing defect * **Girls** \> Boys * Correction * Actively correctable (spontaneously corrects) * Passively correctable (stretching exercises) * Uncorrectable (casting)
38
What is tibial torsion? - Age group seen in - Gender prevalence - Treatment
Intoeing originating between knee and ankle - Most common cause in **children \< 3 yo** * **Male = Female** * Usually **spontaneous correction** over first year of ambulation * Sometimes takes **up to 8 years** to completely correct!
39
What is Medial femoral torsion? - Could be acquired how - Most common in what populations - Correction
Intoeing originates between the knee and the hip * May be acquired from sitting in the **“W” position** * Most common cause in **children \>3 years old** * Usually **corrects spontaneously but slowly** until about 8-10 years
40
What is outtoeing? - More or less common than intoeing? - Improves when
In utero packing defect * Less common than intoeing * Improves during the first year of walking
41
What are angular deformities seen in childhood?
- Genu valgum (knock-kneed) - Genu varum (bow-legged)
42
What is normal alignment at birth? Progression?
**Varus** - Varus condition can worsen as child begins to stand and walk - Around **18-24 mo**, alignment should be **neutral** - After **24 mo**, alignment should progress to **valgus** until **reaches max at 4 yrs** - Valgus decreases toward physiologic **adult** alignment of **slight valgus to neutral** by about **7 yrs**
43
Describe physiologic genu varum? - Uni or bilateral - Stature/gait
- Bilateral and relatively symmetric deformities - Normal stature - Normal gait
44
Describe pathologic genu varum? - Causes - Seen in what diseases/conditions
- Blount disease - Rickets - Skeletal dysplasia - Asymmetric growth: * Unilateral trauma * Infection
45
Describe physiologic genu valgum - Uni or bilateral - Stature/gait - Other signs/symptoms
- Bilateral and relatively symmetric deformities - Normal stature and normal gait - Flat feet and external tibial torsion
46
Describe pathologic genu valgum - Uni or bilateral - Causes/associated conditions
- Unilateral or unequal _Seen with:_ - Fracture of metaphysis - Fracture of physeal with growth plate arrest - Rickets - Skeletal dysplasia
47
What is teratment for angular deformities (genu varum and valgum)?
- **Physiologic**: don't treat; will resolve over time - **Pathologic**: optimize medical management * If not improved, surgical intervention
48
Mnemonics for genu varum/valgum?
- **Varum**: air between the knees (or barrel of rum) - **Valgum**: gum sticking kenes together
49
What is scoliosis? - Gender prevalence - Complication in severe cases
Scoliosis is defined as a **10' curvature** of the spine in the **coronal** plane, and **typically** accompanied by **rotation** - More in **girls** - Severe curvature may -\> impairment of **pulmonary function**
50
How is scoliosis evaluated?
- Feet hip-width apart - Hands joined at midline - Stand directly behind patient - Bend forward 90'
51
What are the 3 classifications of Scoliosis?
- Ideopathic - Neurogenic - Congenital
52
What are signs of scoliosis?
- Uneven shoulders - Curve in spine - Uneven hips
53
Describe ideopathic scoliosis - Etiology - Age ranges/classes
No definite etiology; it is therefore a diagnosis of exclusion _Types:_ - Infantile: **0-3 yrs** - Juvenile: **4-9 yrs** - Adolescent: **10+ yrs**
54
Describe neurogenic scoliosis - Seen with what conditions - Due to what - Do most have rotational component?
_Conditions such as:_ * Cerebral Palsy * Myelomeningocele * Muscular Dystrophy * Neurofibromatosis Due to **muscle imbalance and lack of trunk control** **Most** have **no rotational** component
55
Describe congenital scoliosis - Causes - Manifestation (time)
Asymmetry in the vertebrae secondary to congenital anomalies * Hemivertebrae * Failure of segmentation Congenital scoliosis **usually manifests before adolescence**
56
What is the management for scoliosis with a curve \< 20'?
PT and exercises to strengthen back muscles
57
What is the management for scoliosis with a curve 20-40'?
(Immature skeleton): back brace to prevent further curve progression
58
What is the management for scoliosis with a curve \> 40'?
Spinal fusion to correct deformity
59
What is the concern in management for scoliosis with a curve \> 60'?
Associated with poor pulmonary function
60
Describe periosteum in pediatrics vs. adults
- Osteogenic potential - More metabolically active than adult - Active periosteum may result in: * Callus formation * Union of fractures * Remodeling - Periosteum is thicker and stronger: * Limits fracture displacement * Reduces likelihood of open fractures * Maintains fracture stability
61
What is a buckle (torus) fracture? - Commonly occur where - Management
* Often occur at the **junction between the porous metaphysis and the denser diaphysis** * Commonly in **distal radius** after fall on an outstretched hand (also seen in the distal tibia, fibula, and femur) * Stable fracture that can be managed with **splinting** and a single orthopedic follow-up visit
62
What is seen here?
Buckle (torus) fracture
63
What is a plastic deformation fracture? - What cuases it - Commonly seen where - Management
* Longitudinal force exceeds the bone's ability to recoil * Microscopic fractures dissipate the impact energy * Commonly seen in the **ulna**, the **radius**, and sometimes in the **fibula** * If **\< 20'** or if the deformity occurs **\< 4 yo**, the angulation often **corrects itself**
64
What is seen here?
Plastic deformation fracture
65
What is a greenstick fracture? - Management
Fracture line does not extend completely through the width of the bone • May be the most significant risk factor for repeat fracture * occurring in as many as 84 – 100% of forearm re-fractures • Closed reduction and casting
66
What is seen here?
Greenstick fracture
67
What is a physeal (growth plate) fracture? - What causes it?
* Growth plates are susceptible to fracture and represent a weak point in pediatric bone * Tensile strength of pediatric bone is less than that of the ligaments * Physis will separate or fracture before disruption or "spraining" of an strong and flexible ligament
68
What is seen here?
Physeal (growth plate) fracture
69
What is the Salter-Harris classification for pediatric trauma?
I- Separation through the physis; usually through areas of hypertrophic and degernating cartilage cell columns II- Fracture through a portion fo the physis that extends through the metaphyses III- Fracture through aprotion of hte physis that extends throught he epihpysis and into the joint IV- Fracture across the metaphysis, physis and epiphysis V- Crush injury to the physis
70
What is the Salter-Harris classification for physeal fractures?
S- straight across (type I) A- above (type II) L- lower or beLow (type III) T- two or through (type IV) ER- erasure of growth plate or cRush (type V)
71
Case 1 - You are called to the delivery of a term female with decreased amniotic fluid. - Baby will be delivered via c/s for breech presentation. Which of the following will be an important part of her newborn exam secondary to her risk factors? A. Finding a Klein Line B. Babinski Sign C. Ortolani Maneuver D. Moro Reflex
A. Finding a Klein Line B. Babinski Sign C. Ortolani Maneuver D. Moro Reflex
72
Case 2 - A 9 year old boy is brought by his mother to your office because she noticed he has been limping for the last 2 weeks. - He reports unilateral knee pain after he began an exercise program prescribed to him by you after you calculated his BMI in the 90th percentile. - On exam he holds his leg in external rotation and has pain with flexion of the hip. Knee exam is normal. What is the most-likely diagnosis? A. Osgood Schlatter B. Slipped Capital Femoral Epiphysis (SCFE) C. Subluxation of the patella D. Legg-Calve-Perthes
A. Osgood Schlatter B. Slipped Capital Femoral Epiphysis (SCFE) C. Subluxation of the patella D. Legg-Calve-Perthes
73
Case 3 What type of Salter Harris Fracture is shown? A. Type I B. Type II C. Type III D. Type IV E. Type V
A. Type I B. Type II C. Type III D. Type IV E. Type V