11. Oral Surgery Flashcards

1
Q

Extractions

  1. 3 types of instruments
  2. Function of elevators and 3 techniques
  3. Function of luxators
  4. 4 indications for extractions
A
  1. Forceps, elevators, luxators
  2. Dilate sockets to facilitate extractions, establish effective and logical point of application, remove teeth, remove retained roots. Wheel and axle, wedge, lever
  3. Break PDL, increasing access and mobility
  4. Unrestorable teeth, symptomatic PE teeth, traumatic position, ortho indications, interference with denture construction
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Peri-operative complications 1

  1. 3 causes of access/vision difficulties and 2 Mx options
  2. 4 causes of abnormal resistance
  3. 3 causes of crown fracture
  4. 3 causes of root fracture
  5. 3 causes of dento-alveolar fractures
  6. Management of dento-alveolar fracture
  7. 3 causes of jaw fracture
  8. Jaw fracture management
  9. 3 causes of maxillary tuberosity #
  10. 3 features of Dx
  11. Mx
A
  1. Trismus, produced aperture, crowded/malpositioned teeth, small mouth. Mouth props, other opening devices, XGA
  2. Inadequate initial pressure, thick cortical bone, divergent roots, number of roots, hypercementosis, ankylosis, sclerotic bone
  3. Caries, alignment, size, thick cortical bone
  4. Fused, convergent, divergent, extra roots, abnormal morphology, hypercementosis, ankylosis
  5. Thin bone, inappropriate direction of force, excessive force, inappropriate instrument use
  6. Dissect fractured bone free, suture, stabilise teeth, free mucoperiosteum, smooth edges
  7. Impacted 8s, large cyst, atrophic mandible, application of force in the wrong direction
  8. X-ray (OPT/PA mandible), ensure analgesia, refer to OMFS. If delayed referral - stabilise fracture (tie free ends of bone to to on opposite side of fracture and tie teeth on opposite sides of fracture site together), antibiotics if compound
  9. Extraction of single standing molars, extraction in wrong order, unknown unerupted 8, pathological gemination, inadequate alveolar support
  10. Crack, mucosal tear, mobility, mobile teeth
  11. If small - dissect out bone and close
    If large - reduce and stabilise, ensure tooth attached to tuberosity is occlusion-free, XLA 8wks later
    Antibiotics if compound
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Peri-operative complications 2

  1. How to manage tooth loss
  2. 3 causes of damage to adjacent teeth/restorations
  3. Management
  4. How to manage wrong tooth extractions
  5. 2 causes of broken instruments
  6. Management
  7. 2 causes of jaw dislocation
  8. Management
  9. 5 types of nerve damage
  10. Define neurapraxia
  11. Define axonotmesis
  12. Define neurotmesis
A
  1. Stop and search. If risk of inhalation/ingestion of cannot be accounted for -> CXR in A&E
  2. Inappropriate application point of elevators, large overhang contacting tooth to be extracted, hit opposing teeth with forceps
  3. Pre-XLA warning, temp dressing, arrange definitive
  4. Attempt to replant immediately, splint, tell patient. Never event
  5. Incorrect use, instrument fatigue
  6. Attempt to retrieve or refer
  7. Inadequate jaw support, inappropriate application point, excessive force, open too long
  8. IDB, relocation, post-op advice (soft diet, supported yawning)
  9. Anaesthesia, paraesthesia, dysaesthesia, hyperaesthesia, hypoaesthesia
  10. Mild contusion. Continuity of epineural sheath and axons are maintained. Temp (6-8wks) loss of function due to blockage of nerve conduction. Sunderland grade I
  11. Crush/more severe contusion (moderate). Continuity of axons maintained, continuity of epineural sheath is disrupted. Sunderland grade II-IV. Motor and sensory function distal to point of injury are completely lost over time, leading to Wallerian degeneration due to ischaemia
  12. Transection. Severe. Continuity of epineural sheath and axons disrupted. Complete loss of nerve continuity (complete section of nerve). Sunderland grade V
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Post-operative complications 1

  1. 3 causes of post-op pain
  2. 2 causes of post-op oedema
  3. 2 causes of post-op ecchymosis
    1. causes of post-op trismus
  4. Mx
  5. 3 local reasons for bleeding
  6. 3 systemic reasons for bleeding
  7. Define 3 types of bleeding
  8. Describe Mx for peri- or post-op bleeding
A
  1. Rough tissue handling, laceration/soft tissue tears, exposed bone, incomplete extraction
  2. Rough tissue handling, pull/tear flaps, crushing tissue with forceps
  3. Poor surgical technique, rough tissue handling
  4. Oedema, muscle spasm, mouth open for too long, haematoma into muscle, IDB into MP, damage to TMJ
  5. Soft diet, gentle opening, monitor. May require opening exercises
  6. Mucoperiosteal tears, alveolar bone #, cutting vessels
  7. Clotting abnormalities, liver disease, medications
  8. Immediate/primary - where true haemostasis has not been achieved
    Reactionary/rebound - <48hrs. Due to rise in BP opening up small divided vessels (hot drinks, exertion, alcohol)
    Secondary - >48hrs. Usually due to infection destroying clot or ulcerating local vessels
  9. Reassurance, medical history
    Bite on damp gauze, suction to identify source
    LA with vasoconstrictor (inject/on pledget), haemostatic agents - packing (oxidised regenerated cellulose, fibrin foam, thrombin liquid and powder, gelatine sponge), suturing
    Diathermy, ligatures, bone wax
    Systemic - tranexamic acid
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Post-operative complications 2

  1. Define bony sequestrum
  2. Mx
  3. 2 other names for dry socket
  4. 4 predisposing factors
  5. 4 symptoms
  6. Mx
  7. Infected socket Mx
  8. Pathophysiology of ORN
  9. How to prevent
  10. How to Mx
  11. Definition of osteomyelitis
  12. Pathogenesis
  13. 2 predisposing factors
  14. 3 types
  15. Radiographic appearance
  16. Mx
A
  1. Piece of dead bone tissue formed within diseased/injured bone
  2. Remove
  3. Alveolar/localised osteitis
  4. Posterior tooth, mandible, smoking, female, OCP, excessive post-XLA rinsing, LA vasoconstrictor, traumatic XLA
  5. Moderate/severe dull aching throb, radiates around ear/jaw, continuous, sleep disturbance, exposed bone, bad taste/malodour
  6. LA, ensure no tooth fragments/sequestrum, irrigate and debride, place WHVP, PoI (no smoking, no rinsing today, WSWM tomorrow), review
  7. LA, ensure no tooth fragments/sequestrum, irrigate and debride, ABX if systemically unwell
  8. Radiation therapy induces endarteritis obliterans which leads to progressive fibrosis and capillary loss, leaving bone susceptible to avascular necrosis. Slowed bone turnover, ineffective self-repair. Gradually worsens
  9. Atraumatic technique, primary closure, CHX MW, pre-XLA hyperbaric O2
  10. Irrigate and debride necrotic debris, remove sequestra, resect exposed bone with margin, primary closure, hyperbaric O2
  11. Bone marrow inflammation
  12. Begins in medullary cavity, spreads to cortical bone, then to periosteum. Bacteria invade cancellous bone, leading to soft tissue inflammation and oedema in closed bone marrow spaces. Increase in tissue hydrostatic pressure and compromised blood supply causing tissue ischaemia and necrosis
  13. Immunocompromised, odontogenic infection, mandibular tooth
  14. Early, acute suppurative, chronic ± pus
  15. Moth-eaten/mottled. If long-standing, involucrum (radiopacity surrounding radiolucency)
  16. LT antibiotics ± surgery
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Post-operative complications 3

  1. 3 medications that cause MRONJ
  2. 3 indications for bisphosphonates
  3. 3 MRONJ risk factors
  4. Describe 4 stages of MRONJ
  5. Define low risk MRONJ categories
  6. Define high risk MRONJ categories
  7. MRONJ prevention
  8. MRONJ treatment
  9. 2 bacteria that cause actinomycosis
  10. What is unusual about the spread of infection
  11. How to treat/manage
  12. Define IE
  13. Name 3 groups of patients at high-risk of IE
  14. How to manage high-risk patients
A
  1. Bisphosphonates, antiresorptives, biologics, antiangiogenics, RANK-L inhibitors
  2. Multiple myeloma, osteoporosis, Paget’s (LT steroids)
  3. Extremes of age, bisphosphonates ± steroids, systemic conditions affecting bone turnover, malignancy and invasive dental procedures
  4. Stage 0 - no necrotic bone clinically. Symptomatic, radiographic changes, non-specific clinical findings
    Stage 1 - exposed and necrotic bone/fistula that probes to bone
    Asymptomatic, no clinical infection
    Stage 2 - exposed and necrotic bone/fistula that probes to bone
    Infection ± pus drainage (pain, local erythema)
    Stage 3 - exposed and necrotic bone/fistula that probes to bone
    One or more of exposed/necrotic bone extending beyond alveolus, EO fistula, OAC, osteolysis extending to sinus floor or inferior border of mandible
  5. Oral/IV bisphosphonates for <5yrs without systemic steroids or denosumab without systemic steroids
  6. Oral/IV bisphosphonates for >5yr, oral/IV bisphosphonates or denosumab with systemic steroids, anti-resprotive or anti-angiogenic for cancer, previous MRONJ
  7. Avoid surgery where possible. Atraumatic, primary closure
  8. Limited sequestrectomy and coverage of exposed bone
  9. A. israelli, A, naselundii, A. viscosus
  10. Erodes through tissues rather than following fascial planes and spaces
  11. High-dose ABX and surgery
  12. Inflammation of endocardium, particularly heart valves of CMP caused by bacteria
  13. Prosthetic valve, prev IE, cyanotic CHD
  14. Consider prophylactic ABX - consult with GP/cardiologist
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Post-operative instructions

  1. Name and describe 9 key post-op instructions that should be given
A
  1. Pain - normal, will settle over 1wk. Normal painkillers before anaesthetic wears off (can alternate NSAIDs/paracetamol)
    Bleeding - unlikely, don’t explore socket, rest for today. Bleeding advice (bite on damp gauze, repeat, contact)
    Smoking - avoid
    Infection - rinse from tomorrow with WSMW
    Sensitivity to adjacent teeth
    Jaw stiffness - normal for few days, should settle
    Swelling - variable, max extent 2 days post-Rx. Alternate hot/cold compress
    Bruising - variable, max extent 2-3 days post-Rx. Cold compress
    Sutures - if present, leave and resorb
    Contact details if unusual/pain, etc. (OOH too)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

LA

  1. What are 2 functions of LA
  2. How does LA work
  3. 2 types of LA esters
  4. 4 types of LA amides
  5. Purpose of vasoconstrictor
  6. 2 types
  7. 2 types of topical anaesthetic and function
  8. Describe 2 types of LA injections
  9. How to confirm anaesthesia
  10. 6 complications of LA
  11. Describe 4 advanced LA techniques
  12. Max doses of lidocaine, articaine and prilocaine
  13. Contraindication for felypressin/octapressin
  14. 4 key anatomical landmarks for IDB
A
  1. Prevent pain, reduce bleeding
  2. Blocks voltage-gated Na channels. LA binds to a site in Na channel and blocks it, preventing Na influx. This blocks AP generation and propagation. Block persists so long as sufficient number of channels are blocked. Smaller diameter axons have fewer Na channels and are more susceptible to LA block. Na channels are concentrated at nodes of Ranvier in myelinated axons. To block AP, LA needs to act on several along the axon
  3. Benzocaine, procaine, cocaine
  4. Lignocaine, artisane, prilocaine, bupivicaine, mepivicaine
  5. Act locally to constrict BVs, reduce bleeding and blood flow and help to increase duration of LA and keep/hold LA in tissues (prevents wash out of LA)
  6. Adrenaline, felypressin
  7. 2% lidocaine gel, 20% benzocaine. Superficial soft tissue manipulation and surface anaesthesia
  8. Infiltration - LA deposited around terminal nerve branches (inject distal to apex of tooth into mucogingival fold)
    Nerve block - LA deposited beside nerve trunk, abolishing sensation distal to site. Line up correctly, advance to bony contact, withdraw slightly, aspirate, inject slowly (30s). Final quarter administered during slow withdrawal to anaesthetise lingual nerve. If bony contact too soon, reposition mesially
  9. Ask patient - rubbery, tingly, numb, swollen/fat (IDB - lower lip and chin to midline, BM, gingiva anterior to mental foramen and lingual gingivae)
  10. Failure, prolonged (temporary/permanent), pain during/after, trismus, heamatoma, intra-vascular, blanching, facial palsy (into parotid - CNVII. Reassure patient, ensure not stroke - raise arms, close eyes, cover eye with patch until blink reflex returns), allergy (usually to preservatives), broken needle, interaction with other drugs, toxicity, infection, soft tissue damage
  11. Palatal anaesthesia - chasing anaesthesia (follow blanching), intraligamentary (quicker diffusion), intraosseous (into bone), intrapulpal, Now Gates (open mouth aim for neck of condyle), Varizani-Akinosi (closed mouth; trismus; insert needle buccal to teeth on same site as injection site)
  12. 4.4mg/kg lido, 7mg/kg art, 8mg/kg pril
  13. Pregnancy
14. Coronoid notch
Junction of buccal fat pad/pterygomandibular raphe
Contralateral 4-6s
5-10mm above lower occlusal plane
Bone contact 25-30mm in
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Analgesia

  1. 2 functions of paracetamol
  2. Max dose
  3. 2 cautions
  4. How does aspirin work
  5. 2 functions
  6. 4 contraindications
  7. 2 functions of ibuprofen
  8. Max dose
  9. 2 contraindications
  10. What is diclofenac
  11. Max dose
  12. What is co-codamol
  13. Midazolam dose for anxiety
A
  1. Analgesic, antipyretic
  2. 4g/day
  3. Hepatic/renal impairment, alcohol-dependent
  4. Non-selective COX inhibitor that reduces production of PGs by inhibiting COX-1 and COX-2
  5. Antiplatelet, anti-inflammatory
  6. PUD, <16yrs old (Reye’s), asthmatics, other NSAIDs, bleeding problems, pregnant, steroids
  7. Analgesic, anti-pyretic
  8. 2.4g/day
  9. Peptic ulcers, pregnant, other NSAIDs, LT steroids, renal/cardiac/hepatic impairment
  10. PoM NSAID, more potent
  11. 150mg/day
  12. Codeine and paracetamol
  13. 2mg tablets up to 15mg
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Surgery 1

  1. 8 key stages
  2. 4 methods of preventing site contamination
  3. 4 key aims of surgery
  4. 5 principles of access
  5. What is the ideal type of flap
  6. 2 purposes of soft tissue retraction
  7. 3 instruments used
  8. What instrument used for bone removal and tooth division and why
  9. How is bone removed
  10. Function of irrigation
  11. Definition of debridement
  12. 3 types
  13. 3 instruments used
  14. 4 aims of suturing
  15. 2 principles of suture technique
  16. Function and 2 types of biopsies
  17. 2 indications for cryosurgery
A
  1. Anaesthesia, access, bone removal, tooth division, debridement, suturing, haemostasis, POI and post-op meds
  2. HH/scrubbing, PPE (sterile gloves, gowns, mask), no touch technique, op site prep
  3. Max access with minimal trauma, preserve and protect soft tissues, healing by primary intention (minimise scarring), tension–free wound closure, flap margins and sutures on sound bone
  4. Wide-based crevicular incision using scalpel in one firm continuous motion, full thickness incision to bone (through mucoperiosteum), no sharp angles, adequate size, flap reflection cleanly down to bone, minimise trauma to papillae, no crushing of soft tissues, keep tissues moist
  5. 2-sided (good access, good reflection, better aesthetics)
  6. Improved access to field, protect soft tissues
  7. Howarth’s, Wards, Lack’s, Minnesota, rake retractor
  8. Electric straight handpick with a saline-cooled tungsten carbide bur (usually round or fissure). Air-driven can cause surgical emphysema
  9. Deep narrow buccal gutter (with mesial/distal extension). Allows for correct application of elevators
  10. Prevents heat necrosis of bone, damage to soft tissue, clogging of bur, allows field to be kept clean of debris
  11. Removal of dead, damaged or infected tissue to improve healing potential of remaining healthy tissue
  12. Surgical, mechanical, chemical
  13. Bone file, handpick, Mitchell’s trimmer, Victoria curette, irrigation, suction
  14. Approximate/reposition tissues, compress BVs, achieve haemostasis, cover bone, prevent wound breakdown, encourage healing by primary intention
  15. Tension-free wound closure, evert wound edges in apposition, do not overtighten
  16. Surgical diagnostic method
    Incisional - FNA, punch, true cut
    Excision - for small/obviously benign lesions
  17. Vascular malformations, mucoceles, atypical facial pain, viral warts, superficial BCC, post-enucleation of OKC
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Third Molars 1

  1. How often are they absent
  2. 4 nerves at risk of damage during SR of L8
  3. Define unerupted tooth
  4. Define partially erupted tooth
  5. Define impacted tooth
  6. 2 reasons for impaction
  7. 4 strong indications for L8 removal
  8. 3 relative indications
  9. 4 contraindications
  10. 4 indications for prophylactic extraction
  11. How is angulation of impaction measured and name 5 types
  12. Describe depth of impaction categories
A
  1. Up to 25%
  2. IAN, lingual, nerve to mylohyoid, (long) buccal
  3. Tooth lying within jaws, entirely covered by soft tissue, partially/completely covered by bone
  4. Tooth that has failed to erupt fully into normal position. May/not be seen, but in communication with oral cavity
  5. Tooth prevented from completely erupting into normal functional position
  6. Lack of space, obstruction (other tooth), abnormal eruption path
  7. Recurrent pericoronitis, abscesses, PA pathology, unrestorable, caries in 7 which cannot be adequately treated, cyst/pathology formation, 8 causing resorption of 7
  8. Active/previous significant infection, MH (removal > retention), limited access to dental care (astronaut, etc.)
  9. MH precludes extraction, risk of surgical complications too high, where successful eruption likely and tooth will be functional, deeply impacted asymptomatic tooth
  10. Continual food trapping, stagnation area, risk of future GAs if GA required, MH, denture construction/implants, pre-RTx, cardiac surgery, bisphosphonates
  11. Measured against occlusal curve of Spee
    Vertical, mesial, distal, horizontal, transverse/aberrant
  12. Measured from alveolar crest to max depth of crown
    Superficial - 8 crown related to 7 crown
    Moderate - 8 crown related to 7 crown and root
    Deep - 8 crown related to 7 root
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Third Molars 2

  1. Define pericoronitis
  2. Define operculum
  3. 4 clinical features
  4. 3 Rx options
  5. 4 tissue spaces infection can spread
  6. 8 post-op complications of SR of L8
  7. % risks of temporary and permanent paraesthesia
  8. Define coronectomy
  9. Key indication (and investigation required)
  10. 3 risks
  11. 3 contraindications
  12. 6 signs of close root relationship with IDC
A
  1. Inflammation of soft tissues around crown of tooth. Requires communication between tooth and mouth. Food trapped under operculum
  2. Flap of gingivae overlying tooth
  3. Pain (throbbing), swelling (red, tender operculum), pus, bad taste, ulceration, bad smell, trismus, dysphagia, lymphadenopathy, pyrexia, malaise, fever
  4. Abscess I+D; XLA U8 if traumatising operculum; XLA L8 after acute episode resolution, ABX
  5. Buccal, submasseteric, sublingual, submandibular, parapharyngeal
  6. Pain, swelling, bruising, bleeding, infection, dry socket, altered taste, temporary altered sensation, permanent altered sensation
  7. Temp - 20% (10-20%)
    Perm - 1% (<1%)
  8. Removal of crown of tooth with deliberate retention of roots
  9. L8 roots appear closely involved/related (radiographically) to IDC
  10. Infection, slow healing, pain, roots may migrate and erupt, removal of entire tooth if root mobilised
  11. Mobile tooth/root, non-vital tooth, where sectioning puts nerve at risk (horizontal/disto-angular impaction), predisposition to local infection/immune comp
  12. Diversion/deflection of IDC (white lines not parallel), darkening of root where crossed by canal, interruption of white lines/canal lamina dura (loss of continuity), root deflection, canal narrowing, root narrowing, dark and bifid root, junta-apical area
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Maxillary Sinus 1

  1. Type of epithelium
  2. 2 functions
  3. What is found in posterior wall
  4. Function of cilia
  5. How is an OAC formed
  6. 2 Dx methods
  7. Mx
  8. 4 POI
  9. Define OAF
  10. 3 clinical features
  11. 2 Dx methods
  12. Mx
  13. Root in sinus Dx
  14. 3 Mx options (including approach)
A
  1. Pseudostratified columnar
  2. Voice resonance, reserve chamber for warming air, reduce weight of skull
  3. PSA nerves and vessels
  4. Mobilise trapped particulate matter and foreign material within the sinus and move this towards the ostia for the elimination into the nasal cavity
  5. When a tooth with roots projecting into the sinus is extracted, leaving an opening between the sinus and oral cavity
  6. Direct vision (good suction and lighting), probe (gentle), nose holding/blow test, bubbling of blood at base of socket
  7. Small - encourage clot and suture margins
    Large - close with BAF
  8. Don’t dislodge clot, avoid using straws, playing wind instruments and nose blowing, WSMW from tomorrow, decongestants/steam inhalation, closed sneezing
  9. Formation/creation of a pathological epithelial-lined tract between mouth and maxillary sinus. Chronic and occurs secondary to OAC
  10. Liquid reflux into nose, minor nose bleeds, nasal speech/singing, problems playing brass/wind instruments, bad taste, sinusitis-type symptoms
  11. Direct vision, nose holding/blow test
  12. Remove sinus tract (excision), primary closure/BAF
  13. Radiograph (OPT/occlusal)
  14. Through socket (small curette, narrow-bore suction), OAF-type approach (flap), Caldwell-Luc approach, FESS (endoscopic retrieval)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Maxillary Sinus 2

  1. Define sinusitis
  2. 4 clinical features
  3. Cause of acute sinusitis
  4. Cause of chronic sinusitis
  5. 4 dental pathologies that mimic sinusitis
  6. 3 indictors of sinusitis
  7. Mx options
  8. 4 traumatic/iatrogenic causes of sinusitis
A
  1. Paranasal inflammation and infection
  2. Pain/pressure or altered sensation over cheeks (infraorbital region), nasal dischage/congestion, nasal obstruction, hyposmia, headache, fever, dental pain, fatigue, cough, earache, halitosis
  3. Following URTI bacterial superinfection of cilia, foreign bodies
  4. Foreign bodies, poor drainage
  5. TMD, deep caries, PA abscess, perio infection, atypical facial pain, recent extraction sockets
  6. Tenderness over cheeks, diffuse maxillary tooth pain, pain that worsens with head movements
  7. Decongestants (steam/menthol inhalation, pseudoephedrine), ABX, removal of foreign bodies
  8. Orbital wall #, RCT apical perforation, extraction, sinus lifts/implant placement, deep perio Rx, nasal packing, NG tube, mechanical ventilation, foreign objects in sinus
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

TMD 1

  1. Define TMD and give 3 other names
  2. 4 causes of TMD
  3. 6 clinical features of TMD
  4. 6 differential Dx of TMD
  5. Describe 6 types of conservative advice
  6. Describe 3 other types of reversible Rx
  7. What are 3 types of TMJ surgery that could be used
A
  1. Pain associated with area of face involving MoM and TMJ. Myofascial pain dysfunction, pain dysfunction syndrome, TMJ, TMD, facial arthromyalgia
  2. Myofascial pain, anterior disc displacement ± reduction, degenerative disease (OA, RA), chronic recurrent dislocation, ankylosis, hyperplasia, neoplasia, infection, stress, psychogenic, direct/indirect trauma, parafunctional habits
  3. Intermittent pain, muscle/joint/ear pain particularly on waking, trismus/jaw locking, clicking and popping noises, headaches, crepitus
  4. TMD, dental pain, sinusitis, ear pathology, SG pathology, referred pain (neck), headache, atypical facial pain, TN, angina, condylar fracture, temporal arteritis
  5. No chewing gum, replace missing posterior teeth, supported yawning, soft diet, hot compress, massage, stress Mx (relaxation), reduce opening distance
  6. Physiotherapy, hypnotherapy, jaw exercises (screw, spatula, therabite), meds (NSAIDs, muscle relaxants, TCAs, botox, steroids), splints
  7. Arthrocentesis, arthroscopy, arthrotomy, condylotomy (high condylar shave) TJR
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Muscles of Mastication

Give the origin, insertion and function for the following muscles:

  1. Temporalis
  2. Masseter
  3. Medial pterygoid
  4. Lateral pterygoid
  5. What 2 muscles can be easily palpated
A
  1. Origin - temporal fossa and deep temporal fascia
    Insertion - Coronoid process and anterior border of ramus
    Function - elevation and retrusion
  2. Origin - temporal process of zygomatic bone and zygomatic arch
    Insertion - angle and ramus
    Function - elevation and protrusion
  3. Origin - maxillary tuberosity and medial surface of lateral pterygoid plate
    Insertion - medial surface of ramus and angle
    Function - elevation and protrusion
  4. Origin - infra temporal surface of greater wing of sphenoid and lateral surface of lateral pterygoid plate
    Insertion - neck of mandible (fovea) and capsule/intracapsular disc
    Function - depression and protrusion
  5. Temporalis, masseter
17
Q

Sedation 1

  1. Define conscious sedation
  2. 3 methods/techniques of conscious sedation
  3. 5 indications
  4. 5 contraindications
  5. Describe ASA classification
A
  1. Technique in which the use of a drug or drugs produces a state of depression of the CNS enabling Rx to be carried out, but during which verbal contact with the patient is maintained through the period of sedation.
    The drugs and techniques used to provide conscious sedation for dental Rx should carry a margin of safety wide enough to render unintended LoC unlikely. The level of sedation must be such that the patient remains conscious, retains protective reflexes and is able to understand and respond to verbal commands
  2. IV, IS, oral, trans mucosal
  3. ASA I or II, mild/moderate learning difficulties, moderate/severe anxiety, medical conditions aggravated by stress (epilepsy, HTN, asthma), medical conditions that inhibit excellent cooperation (Parkinson’s, CP), traumatic/difficult/unpleasant procedures (SR L8), excessive gag reflex
  4. Severe/uncontrolled systemic disease, severe mental disability, severe psychogenic problems, unaccompanied, unwilling/uncooperative, narcolepsy, hypothyroidism
  5. I - normally healthy
    II - mild systemic disease
    III - moderate systemic disease (limits activity but not incapacitating)
    IV - severe systemic disease that is a constant threat to life
    V - moribund patient who is not expected to survive >24hrs
    VI - Declared brain-dead patient whose organs are being removed for donor purposes
18
Q

Sedation 2 - Inhalation Sedation

  1. 5 advantages
  2. 5 disadvantages
  3. 5 indications
  4. 5 contraindications
  5. Give 5 key pieces of equipment
  6. Describe technique
  7. 2 complications
  8. 4 signs of adequate sedation
  9. 5 key safety features
A
  1. Flexible duration, rapid onset, rapid recovery, no injection required, few side effects, can be used in kids
  2. Problems with nasal hood staying in place, coordination of nasal breathing with open mouth, not very potent, expensive, difficulty determining actual dose
  3. Mild/moderate anxiety, ASA I or II, enhanced gag reflex, traumatic procedure, medical conditions aggravated by stress, unaccompanied adult requiring sedation
  4. Blocked nose, tonsillitis, severe COPD (ASA III or more), neuromuscular disease (MS), pregnancy (first trimester), adequately trained staff not available
  5. Gas cylinders, pressure reducing valve, flow control metres, reservoir bag, gas delivery hoses, nasal hood, waste has scavenging system
  6. Machine on, mix to 100% O2, flow 5-6l/min. Nasal hood on, patient breathe through nose, check reservoir bag movements, O2 reduced by 10% first min and 5% every other min until patient feels different. Rx finished then O2 increased by 10-20% every min. 2-3mins of 100% O2 to prevent diffusion hypoxia, nasal hood removed, machine off
  7. Over-sedation - nausea, headache, vomiting, unresponsive
    Panic - reduce sedation, reassure patient
  8. Relaxation, warmth, giddiness, paraesthesia, lethargy, lessened pain awareness, slowed response to commands (but still responsive)
  9. Air entrapment valve, oxygen flush button, oxygen monitor, colour coding, reservoir bag, scavenging system, pressure dials, pressure reducing valve
19
Q

Sedation 3 - IV Sedation

  1. 5 advantages
  2. 5 disadvantages
  3. 5 indications
  4. 5 contraindications
  5. What drug is used in what concentration and describe increments used
  6. What is GABA
  7. How is GABA affected by sedation
  8. 3 side effects of sedation
  9. Name reversal agent and concentration and how to use
  10. Risk of flumazenil
  11. 3 vital signs to monitor
  12. 5 complications of IV sedation
  13. 3 signs of adequate sedation
  14. 2 common cannulation sites
A
  1. Good sedation and muscle relaxation, lessened pain awareness, forget after Rx, easy to control/titrate, few side effects if done properly
  2. IV cannula, difficulty assessing sedation level, behaviour during recovery, swallowing efficacy, escort for 24hrs, doesn’t address anxiety
  3. ASA I or II, >12yrs old, mild/moderate anxiety, traumatic procedure, medical conditions aggravated by stress, cerebral palsy, Parkinson’s
  4. ASA III or IV, COPD, <12yrs old, pregnancy, NM diseases (myasthenia gravis), hepatic insufficiency, intracranial pathology
  5. Midazolam. 1mg/ml. 1-2ml bolus then 0.5-1ml increments every 2 mins
  6. Inhibitory neurotransmitter
  7. BNZ act on CNS receptors to enhance the effect of GABA, reducing neuronal excitability and prolonging time for receptor repolarisation
  8. Resp depression, hypotension, tachycardia
  9. Flumazenil. 100mcg/ml. Injected in the same volume as midazolam, but can be given in larger boluses if ME
  10. Shorter half life than midazolam so may wear off and re-sedate patient
  11. Oxygen sats, BP, HR
  12. Venospasm, intra-arterial infection, extravascular injection, haematoma, fainting, hyper-response, hypo-response, paradoxical reaction, allergic reaction, over-sedation, sexual fantasy
  13. Slurring/slowing of speech, delayed response to commands, relaxed, Verrill’s sign (halfway eyelid ptosis), Eve’s sign (can’t touch nose)
  14. Dorsum of hand, antecubital fossa
20
Q

TMJ Surgery 1

  1. 5 indications
  2. Define internal derangement
  3. Describe difference between anterior disc displacement with and without reduction
  4. Name and briefly describe 5 types of TMJ surgery
A
  1. Neoplasia/other pathology, ankylosis, recurrent chronic dislocation, developmental disorders, OA, trauma, TMD, internal derangement, chronic severe limited mouth opening
  2. Painful clicking. Lack of coordinated movement between condyle and articular disc. Condyle has to overcome mechanical obstruction before full point movement can be achieved
  3. With reduction - disc displaced anteriorly during opening until disc reduction occurs. Disc returns to normal on closing. After closing, process repeats. Short-lasting tightness
    Without reduction - condyle cannot translocate as normal. Permanent tightness/locking. Requires disc relocation
  4. Arthrocentesis - lavage of upper joint space (endoscopic) using Hartman’s/hyaluronic acid to break down adhesions and remove inflammatory exudate, allowing disc to reposition
    Arthroscopy - endoscopic lavage of joint space, adhesion removal, removal of damaged tissue, plication to reposition disc
    Arthrotomy - open joint surgery, lavage of space and removal of disc (discectomy)
    Condylotomy - high condylar shave. Condyle repositioned anteriorly and inferiorly beneath disc, improving function
    TJR - where gross destruction of joint architecture and marked reduced function. Condylar head and glenoid fossa replaced
21
Q

Orthognathic Surgery

  1. Purpose
  2. 3 indications
  3. Name and briefly describe 4 common surgical procedures
A
  1. Correct conditions of jaw and face caused by underlying skeletal disharmonies
  2. Gross jaw deficiencies, airway defects, TMJ pathology, acromegaly, trauma, soft tissue discrepancies
  3. LFI - disarticulate maxilla from BoS and reposition
    LFII - midface advancement
    LFIII - move entire mid face and zygoma complex
    SSO - separation of ramus from body (BSSO)
    Vertical subsigmoid osteotomy - mandible posterior movement
    Bimax - LFI and SSO
22
Q

Bone Grafts and Implants

  1. 3 indications
  2. 5 contraindications
  3. Describe osseointegration
  4. Describe 4 types of bone graft
A
  1. Bone preservation and prevention of resorption, restore aesthetics and function, denture retention
  2. RTx to jaw, LT bisphosphonates, poor OH, perio disease, pathology, uncontrolled diabetes, poor bone quality or quantity
  3. Direct abutment of bone to implant surface such that osteoblasts can be seen on electron micrographs to be growing on the implant surface
  4. Autograft - own tissue (chin, ramus, iliac crest, rib)
    Allograft - other (donor) human
    Xenograft - anomal
    Alloplast - synthetic bone substitute
23
Q

Fractures 1

  1. 6 clinical features of mandible #
  2. 2 Ix requested
  3. 3 Mx options
  4. 7 classification methods
  5. 4 factors contributing to displacement
  6. 6 clinical features of maxillary #
  7. 2 Ix requested
  8. 3 Mx options
  9. Describe 3 classification systems
  10. 6 clinical features of ZOC #
  11. 2 Ix requested
  12. 3 Mx options
  13. Describe Henderson’s classification
  14. 3 aetiological reasons for OMF trauma
A
  1. Pain, swelling, bruising
    Limitation of function (trismus), occlusal derangement (disclusion), lower lip/chin numbness, loose/mobile teeth, bleeding, AOB, asymmetry, deviation to opposite side, step deformity, gap between teeth, SL haematoma, battle signs
  2. OPT, PA mandible, CBCT
  3. Control pain and infection; If undisplaced, KUO; closed reduction (IMF); ORIF. POI - soft/liquid diet for 6wks, rest, analgesia
  4. Side, number, site, type (involvement of surrounding tissues), displacement, direct of # line, specific types
  5. Direction of #line, opposing occlusion, magnitude of force, mechanism of injury, intact soft tissues, other associated fractures
  6. Pain, swelling, bruising, mobile teeth, tooth loss, disclusion, trismus, otorrhoea, rhinorrhoea, cracked cup sound, bleeding, infraorbital numbness, mid face mobility, retruded mid face, orbital rim step deformity
  7. CBCT, OPT
  8. If undisplaced, KUO; closed reduction (IMF), ORIF
  9. LFI - horizontal. Detachment of tooth-bearing portion of maxilla via line from anterior margin of ant nasal aperture running laterally and back to lower third of pterygoid plate
    LFII - pyramidal. Detachment of true mid face, involvement of inferior orbital rim
    LFIII - transverse. Detachment of entire facial skeleton from cranial base. Involves z arch and FZ suture
  10. Pain, swelling, bruising (bilateral ecchymosis), subconjunctival haemorrhage, infraorbital numbness, trismus, lacerations or excoriations, facial flattening, orbital rim step deformity, proptosis, diplopia, tethering, enophthalmos, reduced visual acuity
  11. OM 15d and 30d views, CBCT
  12. If undisplaced, KUO; closed reduction (IMF), ORIF (Gillies lift)
13.I – undisplaced
II – zygomatic arch
III – tripod # with F-Z undisplaced
IV – tripod # with F-Z displaced
V – orbital blowout 
VI – orbital rim #
VII – comminuted #
  1. Assaults, RTA, industrial, iatrogenic, falls, war
24
Q

Dentofacial Infections

  1. 2 key microbes
  2. 5 cardinal signs of inflammation/infection
  3. Standard Rx of dentofacial infection
  4. 3 emergency signs for OMFS referral

Describe which fascial space/plane that untreated infections of the following teeth can invade into:

  1. Lower anteriors (2 and why)
  2. Lower posteriors (7 and why)
  3. Upper anteriors (2)
  4. Upper posteriors (5)
  5. 5 indications for ABX with dento-facial infections
  6. 3 signs of Ludwig’s angina
A
  1. S. anginosus, P. intermedia
  2. Heat, redness, swelling, pain, loss of function
  3. I+D, remove source (often XLA), PO analgesia/ABX
  4. Airway compromise, swallowing difficulties, rapid spreading facial infection (over midline), sepsis risk
  5. Submental, sublingual (roots above mylohyoid attachment/line)
  6. Sublingual (roots above mylohyoid attachment) or submandibular (roots below mylohyoid attachment), buccal (roots below buccinator attachment), submasseteric, parapharygneal, retropharyngeal, pterygomandibular
  7. Infraorbital, palate
  8. Buccal (roots above buccinator attachment), superficial temporal, deep temporal, infratemporal, palate
  9. Immunocompromised, extremes of age, FoM, associated systemic symptoms (fever, malaise), lymphadenopathy, dysphagia, airway compromise
  10. Bilateral abscess and cellulitis of SM and SL spaces
    Raised FoM, board-hard neck, FoM and tongue, systemic symptoms, skin hot to touch
25
Q

Cysts 1

  1. Definition
  2. How do they arise
  3. 6 clinically features
  4. 2 key Ix
  5. Describe 2 Rx options
  6. Name 3 odontogenic
  7. Name 3 non-odontogenic
  8. How common is radicular
  9. How does radicular arise
  10. Mx
  11. 2 histological features
  12. 2 other types of radicular cyst
  13. How does an inflammatory paradental cyst present
  14. Define dentigerous
  15. How common
  16. How does it appear
  17. 2 histological features
  18. Name variant associated with anterior teeth and (young) kids
  19. Difference between enlarged follicle and dentigerous cyst
A
  1. Pathological cavities with fluid, semi-fluid or gaseous content, not create by pus accumulation
  2. Odontogenic epithelium (cell rests of Malaise or glands or Serres)
  3. Asymptomatic, pressure build-up, tooth mobility, tooth displacement, tooth discolouration, delayed eruption, ectopic eruption, swelling, discomfort, altered sensation, bone perforation, bad taste, sinus tract
  4. Radiographs (usually unilocular, well corticated, unicentric expansion), aspiration Bx
  5. Enucleation - removal of entire cystic lesion including lining and fibrous CT wall. Single visit, good for path and primary closure. Gold standard unless v large or high risk of recurrence
    Marsupialisation - de-roof and gradual deflation. Surgical window in cyst wall, removal of intracystic contents and suturing cyst wall open. Encourages cyst to decrease in size and usually followed by enucleation. For when enucleation contraindicated (risk of damage to important structure, difficult access, risk of jaw #)
  6. Radicular, residual, dentigerous, OKC
  7. Nasopalatine, simple bone, aneurysmal, Stafne cavity
  8. 60-70%
  9. Tooth pulpitis (caries/trauma) leading to necrosis, PA granuloma (apical bone inflammation), stimulation of rests of Malaise, epithelial proliferation and radicular cyst formation and growth
  10. RCT of tooth
  11. Thin (often incomplete) epithelial lining, fibrous CT wall/capsule with inflammation present. Epithelial hyperplasia/ulceration if infection present
  12. Residual (cyst left after extraction), inflammatory lateral perio
  13. Usually adjacent to crown but doesn’t surround it, related to erupted/PE tooth. Pouch lined by non-keratinised epithelium. Usually 8s, associated with recurrent pericoronitis
  14. Developmental cyst that forms around crown of unerupted tooth
  15. 10-15%
  16. Wall attached to ACJ and entire crown sits in cystic cavity
  17. Thin non-keratinised mucosa, layer of fibrous CT sandwiched between reduced enamel epithelium and oral mucosal epithelium
  18. Eruption cyst
  19. <2.5mm diameter - follucle
    >4.2mm diameter - probable cyst
    >10mm diameter - cyst
26
Q

Cysts 2

  1. Prev name for OKC
  2. 2 unique features
  3. 3 features that increase risk of recurrence
  4. How common
  5. Key radiographic appearance (and 3 other differentials)
  6. 3 histological features
  7. Key FNA feature vs normal cysts
  8. Syndrome associated with multiple OKCs and superficial BCCs
  9. Key difference between radicular and nasopalatine
  10. What is unique about simple bone cysts and how are they managed
A
  1. KCOT
  2. Aggressive growth (bone infiltration), high rate of recurrence
  3. Site, size, residual cells (daughter cells in CT wall)
  4. 5-10%
  5. Multilocular, scalloped, drowns in AP direction usually in mandible ramus/angle
    Ameloblastoma, giant cell lesion, odontogenic myxoma, cherubsim, aneurysmal bone cyst
  6. Thin epithelium, parakeratosis, pallasading basal
    layer, thin capsule, daughter cysts/satellite cysts, low soluble protein content (<4g/dl)
  7. Keratin/cheese-like semi-solid fluid. Normal is clear fluid
  8. Gorlin-Goltz syndrome
  9. NP - associated with vital teeth so PDL space present
  10. Often empty cavities devoid of lining. Resolve spontaneously or after trauma (debridement, exploration)
27
Q

Odontogenic Tumours 1

  1. 4 types of non-odontogenic tumours
  2. 4 types of odontogenic tumours
  3. 2 clinical features of papillomas
  4. 2 clinical features of osteomas and syndrome associated with multiple
  5. 2 clinical features of ossifying fibromas
  6. 2 radiographic features
  7. 2 histological features
  8. 3 types of ameloblastoma
  9. 3 clinical features
  10. 2 Ix
  11. How to reduce recurrence
  12. 3 histological features
A
  1. SC papilloma, fibroma, lipoma, osteoma, osteoblasto, ossifying fibroma
  2. Ameloblastoma, adenomatoid odontogenic tumour, calcifying epithelial odontogenic tumour, myxoma, ameloblastic fibroma, odontome
  3. White/pink, cauliflower appearance
  4. Smooth, hard benign neoplasms of bone. Unilateral, covered by normal mucosa. Gardner syndrome
  5. Slow growing, well demarcated, painless, expansive growth
  6. Well-defined radiolucency, well-corticated
  7. Cellular fibrous tissue, immature bone, acellular calcifications
  8. Unicystic, polycystic, peripheral
  9. Slow growing, expansive growth, locally destructive, rarely metastasise
  10. CBCT, Bx
  11. Clear surgical margins
  12. Islands of follicles, peripheral cells resemble ameloblasts, centre resembles stellate reticulum which may show changes
28
Q

Odontogenic Tumours 2

  1. Radiographic appearance of adenomatoid odontogenic tumours
  2. Why low recurrence rate
  3. Radiographic appearance of calcifying epithelial odontogenic tumours
  4. Radiographic appearance of odontogenic myxomas
  5. 2 histological features
  6. Why high recurrence rate
  7. Describe difference between 2 types of odontomes
A
  1. Patchy calcification
  2. Capsule
  3. Radiolucency with scattered radiopacities
  4. Multilocular ‘soap bubble’ appearance
  5. Very loose CT, sparse cells, high quantity of glycans
  6. Soft/gelatinous tissue which can tear easily
  7. Compound - multiple small teeth (denticles) in fibrous sac. Tissue formed in correct order
    Complex - irregular mixture of dental hard tissues formed/mixed in a random order