old persons 18/10 Flashcards

1
Q

Difference in symptoms before syncope between cardiac cause and vaso-vagal?

A

Cardiac syncope:
-Immediate
-Collapse without warning
-Regain consciousness quickly

Vaso Vagal:
-Theres a build up (patient may complain of nausea, weakness and blurred vision, and appear pale with bradycardia on examination)
-Bp remains low afterwards

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2
Q

Presentation of syncope with fainting. Caused by cardiac syncope secondary to aortic stenosis. What ausculation sign would be found?

A

A soft second heart sound and an ejection systolic murmur radiating to the carotids characterises aortic valvular stenosis.

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3
Q

Presentation of acute bronchitis

A

**Cough **
(May or may not have sputum, wheeze, or breathlessness. Substernal or chest wall pain may be present when coughing. Sometimes mild constitutional symptoms)

Acute bronchitis is temporary inflammation of the airways that causes a cough and mucus. It lasts up to 3 weeks. Acute bronchitis is usually caused by a viral infection.

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4
Q

What is predominant feature in both acute bronchitis and community acquired pneumonia?

A

Cough is the predominant symptom in both acute bronchitis and community-acquired pneumonia

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5
Q

Whats the difference in chest xrays between a community acquired pneumonia and Acute bronchitis?

A

Acute bronchitis chest xray = normal

Cap chest xray =Chest X-ray abnormal (new infiltrate provides definitive diagnosis of pneumonia)

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6
Q

Causes of syncope examples

A

Cardiac (e.g. Aortic stenosis on excertion) (e.g. Supraventricular tachycardia)
Respiratory (e.g: hyperventilating due to anxiety)
Metabolic (e.g. hypoglycaemic)
Neurological (e.g. epilepsy)
Vaso-vagal attack

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7
Q

Presentation of supraventricular tachycardia (SVT) with syncope?

A

Since syncope episode:
Lightheadness
Palpatations
dyspnoea

On ECG Sinus tachycardia have only one P wave for each QRS complex. Whereas SVT mostly have one P wave, but some can have no P waves or more than one.

Research into the ECG stuff to check this is correct

Need to research into the difference between presentations of SVT and sinus tachycardia.

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8
Q

What is syncope?

A

Syncope is a transient loss of consciousness and postural tone followed by spontaneous recovery
Ultimately results from decreased cerebral perfusion.

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9
Q

What is Atrioventricular nodal reentrant tachycardia (AVNRT).

A

A type of supraventricular tachycardia.

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10
Q

Side effects of adenosine?

A

Bronchospasm, dyspnea, facial flushing and the feeling of impending doom

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11
Q

Treatment of SVT syncope

A

Vagal manoevours,
Carotid sinus massage
IV Adenosine

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12
Q

Mechanism of adenosine for treating SVT?

A

The A 1 receptor is responsible for its effect on the AV node. These receptors are linked to the same cardiac potassium channel that is activated by acetylcholine, and adenosine hyperpolarises cardiac conducting tissue and slows the rate of rise of the pacemaker potential accordingly.

**Adenosine [ah-DEN-oh-seen] is a naturally occurring nucleoside, but at high doses, the drug decreases conduction velocity, prolongs the refractory period, and decreases automaticity in the AV node. **

(In cardiac nodal tissue, adenosine binds to type 1 (A1) receptors, which are coupled to Gi-proteins. Activation of this pathway opens potassium channels, which hyperpolarizes the cell. Thus AV node and SA node are hyperpolarised. This decreases HR. Activation of the Gi-protein also decreases cAMP, which inhibits L-type calcium channels and therefore calcium entry into the cell. Reduced calcium currents leads to increased refractory period in AV node.)

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13
Q

Whats the refractory period (e.g. for AV node)?

A

a period immediately following stimulation during which a nerve or muscle is unresponsive to further stimulation.

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14
Q

Presentation of Pericarditis?

A

The most common sign of acute pericarditis is chest pain, usually worsened when taking a deep breath.
Pleuritic chest pain and pericardial friction rub on auscultation of the left lower sternal border. (sharp chest pain)

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15
Q

ECG of pericarditis?

A

Widespread concave ST elevation and PR depression throughout most of the limb leads (I, II, III, aVL, aVF) and precordial leads (V2-6).
Reciprocal ST depression and PR elevation in lead aVR (± V1).
Sinus tachycardia is also common in acute pericarditis due to pain and/or pericardial effusion.

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16
Q

Difference between pericarditis and pericardial effusion?

A

Inflammation of the pericardium secondary to infection, localised injury or systemic disorders producing characteristic chest pain, dyspnoea and serial ECG changes.

A pericardial effusion is an abnormal accumulation of fluid in the pericardial cavity (can lead to cardiac tamponade)

Patients with pericarditis are at risk of developing a pericardial effusion.

infection causes inflammation

https://www.osmosis.org/learn/Pericarditis_and_pericardial_effusion

17
Q

Pericardial effusion can lead to what?

A

adversely affect heart function is called cardiac tamponade.

18
Q

What aggrevates and relieves pericarditis chest pain?

A

Sitting up and leaning forward tends to ease the pain, while lying down and breathing deep worsens it.

19
Q

ECG Findings in Massive Pericardial Effusion?

A

Consecutive, normally-conducted QRS complexes that alternate in height.
Occurs when the heart swings backwards and forwards within a large fluid-filled pericardium

20
Q

Framework for A to E assessment:

A

At end ask about Past medical history, medications and allergies

21
Q

Framework for history taking

A

SQITARS
(ask about associated symptoms e.g. if cough: chest pain, sputum+colour, wheeze, haemoptysis)

Alcohol+smoking

PMx, Socx, Familyx

ICE

22
Q

Symptoms of encephalitis?

A
23
Q

Can type II diabetics get hypos?

A
24
Q

Infective endocarditis presentation

A

Symptoms: Fever, fatigue, dyspnoae, weight loss

Signs: Splinter haemorrhaging, splenomegaly, Vegitations seen on transthoracic echocardiogram, heart murmour due to vegetations, Janeway lesions.

Risk factor: replaced valve

https://www.osmosis.org/learn/Infective_endocarditis:_Clinical_practice

25
Q

Prostate cancer is progression slow or fast?

A

In many cases, prostate cancer is relatively slow-growing, which means that it can take years to become large enough to be detectable, and even longer to metastasize outside the prostate.

26
Q

Is furosemide used in heart failure?

A

Yes! Its a loop diuretic and is the most effective symptomatic treatment for HF.

27
Q

Why mustn’t you ask patient to squeeze fist when taking blood?

A

Pseudohyperkalemia is uncommon, but important. Local release of potassium caused by contraction of the forearm muscles from a tightly clenched fist or repeated fist clenching during phlebotomy is a recognized cause of pseudohyperkalemia

28
Q

Causes of JVP rise?

A

Fluid overload - excessive IV fluids, renal disease, heart failure

Right ventricular systolic failure - cor pulmonale, left ventricular failure

Right ventricular diastolic failure - constrictive pericarditis, tamponade

Pulmonary hypertension.

29
Q

Sky high ALT caused by?

According to speicalty registra

A

1) Ischaemic hepatitis
2) Viral hepatitis
3) Congestive hepatitis

According to speicalty registra