Prematurity And Rubella Flashcards

1
Q

also known as “German Measles

A

CRS

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2
Q

Crs is characterized by:

A

○ Maculopapular rash
○ Lymphadenopathy
○ Fever

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3
Q

CRS Defects are often rare when the infection occurs in the

A

20th week or after the 20th week of gestation.

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4
Q

If infection occurs 0-28 days before conception

A

the infant has a 43% chance of being a ected

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5
Q

If the infection occurs 0-12 weeks after conception

A

the chance increases to 51%

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6
Q

If the infection occurs 13-26 weeks after conception

A

the chance is 23%

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7
Q

Infants are not generally a ected if rubella is contracted during

A

The 3rd trimester

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8
Q

Gestational age: 1-8 weeks

A

Cardiac defect (heart) and hearing impairment, other CRS anomalies (80%)

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9
Q

Gestational age: 9-12 weeks

A

Hearing impairment and features of CRS (50%)

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10
Q

Gestational age: 13-16 weeks

A

CRS anomalies (30%), hearing loss is prominent than other features.

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11
Q

Chances of fetal damage are minimal or none

A

> 20 weeks

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12
Q

Rubella immunoglobulin M (IgM) antibody detected

A

infants < 6 months old

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13
Q

Sustained rubella immunoglobulin G (IgG) antibody level detected in serum;

A

present on at least two occasions between 6 and 12 months of age

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14
Q

Any infant < 12 months with suspicion of CRS. The following clinical manifestation should lead to suspicion of CRS:

A

(1) Congenital heart disease and/or
(2) suspicion of hearing impairment; and/or one or more of the following eye signs: (a) cataract; (b) congenital glaucoma; or (c) pigmentary retinopathy (salt and pepper).

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15
Q

An infant of < 12 months in whom a qualified clinician detects:

A

At least two of the complications listed in group A or one in Group A and one in group B:

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16
Q

Cataract(s), congenital glaucoma, congenital heart disease, hearing impairment, pigmentary retinopathy

A

Group A

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17
Q

Purpura, splenomegaly, microcephaly, meningoencephalitis, radiolucent bone disease, jaundi

A

Group B

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18
Q

An infant who is a suspected case with one condition from Group A and meets laboratory criteria for CRS

A

Lab confirmed CRS

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19
Q

An infant who does not have group A clinical signs of CRS but who meets the laboratory criteria for CRS is classified as having congenital rubella

A

CRI

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20
Q

Auditory defect of CRS

A

sensorineural hearing impairment

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21
Q

Neurologic impairment of CRS

A

○ Microcephaly
○ Cerebral calcifications
○ Meningoencephalitis
○ Behavioral disorders, mental retardation

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22
Q

Neonatal manifestations of CRS

A

○ Low birth weight
○ Interstitial pneumonitis

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23
Q

A birth defect where a baby’s head is smaller than expected when compared to babies of the same sex and age.

A

Microcephaly

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24
Q

There are calcium deposits in the brain and the e ects of that vary depending on the severity and area of the calcification

A

Cerebral calcifications

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25
Q

Very rare, life-threatening condition simply means you have meningitis and encephalitis at the same time.

A

Meningoencephalitis

26
Q

the classic triad of symptoms for CRS are:

A

1) Sensorineural Hearing Loss
2) Eye Abnormalities
3) Pulmonary Stenosis

27
Q

observed approximately around 58% of the patients

A

Sensorineural Hearing Loss

28
Q

are present among 40% of the patients

A

eye abnormalities

29
Q

clouding of the lens in the eye that a ects vision

A

Cataract

30
Q

Also called Microphthalmos
- This is developmental disorder of the eye in which
one or both eyes do not fully develop hence they are small

A

Microphthalmia

31
Q

The narrowing of the valve between the lower right heart chamber, right ventricle, and the lung arteries (pulmonary artery)

A

Pulmonary Stenosis

32
Q

Presents as a characteristic as a “blueberry mu n rash”
- It occurs in organs outside of the bone marrow and it occurs in very diverse condition,

A

Extramedullary Hematopoiesis

33
Q

less than 37 weeks gestation is ___ baby

A

Premature

34
Q

(37 to 42 weeks gestation) is a ___ baby

A

Full term

35
Q

born after 42 weeks gestation is a ___ baby

A

Post term

36
Q

A pregnant woman experiences regular contractions that result in the opening of the cervix after week 20 and before week 37 of pregnancy.

A

Pre term labor

37
Q

less than 28 weeks

A

Extremely preterm

38
Q

28 to 32 weeks

A

Very preterm

39
Q

32 to 37 weeks

A

Moderate to late preterm

40
Q

AKA induced preterm delivery
- Often recommended by obstetrician and
gynecologist when the mother has usually multiple gestations with complications

A

Elective

41
Q

May or may not have an obvious trigger.
Sometimes it could be an infection or placenta abduction.

A

Spontaneous

42
Q

Risk Factors for Premature Birth

A

● Cervical Incompetence/ cervical insu ciency
● Birth defects in the uterus
● History of preterm delivery
● Infection (UTI or infection of the amniotic
membrane)
● Poor nutrition right before during pregnancy ● Pre-eclampsia
● Placenta previa or placental abruption

43
Q

Means that the muscles and cervix are weak and that’s the reason it opens or the woman experiences painful cervical dilation that often results in preterm delivery.

A

Cervical Incompetence/cervical Insu ciency

44
Q

the placenta is located low in the uterus and it may be covering the cervix.

A

placenta previa,

45
Q

placenta detaches from the uterus and with this one it has an unknown cause.

A

Placental abruption

46
Q

Often caused by deficiency by the pulmonary surfactant this would a ect the breath supply of the infant because this would a ect the breath supply of the infant because the pulmonary surfactant plays an important role in maintaining the integrity of the alveoli and is essential for e cient exchange of gasses.

A

Respiratory Distress Syndrome

47
Q

Symptoms: grunting respiration, use of the accessory muscles, nasal flaring

A

Respiratory distress syndrome

48
Q

under-developed lung which is a chronic lung disease and is often used by prolonged use of ventilators.

A

Bronchopulmonary Dysplasia

49
Q

caused by the immaturity of the neurologic or mechanical function of the RS because of the immaturity of the system. They do not allow non stop breathing

A

Apnea

50
Q

High levels of bilirubin in the bloodstream and you will often observe the white part of the eyes or mucous membranes will turn yellow.

A

Jaundice

51
Q

a type of brain damage that can result from high levels of bilirubin in the baby’s blood and it is associated with athetoid CP and HL.

A

kernicterus

52
Q

It means that you have a “hole” in your heart
- There is an undisclosed hole in aorta and it allows
the blood to skip the circulation to the lungs

A

Patent Ductus Arteriosus (PDA)

53
Q

Occurs when the glucose in the blood is too low

A

Hypoglycemia

54
Q

tissue of the intestines are inflamed and
causes that part to die or it will form a hole in the
babyʼs intestines

A

Necrotizing enterocolitis (NEC)

55
Q

A disorder of movement, muscle tone, or posture that can be caused by an injury in the brain due to infection, inadequate blood flow, or injury to a newborn’s developing brain either early during pregnancy or while the baby is still young.

A

CP

56
Q

related to their neurological impairment/cognitive deficits and language impairment,

A

Impaired learning

57
Q

preterm delivery interferes with the vascularization process of the eye or the forming of blood vessels in the eye = causing blindness

A

Retinopathy of Prematurity

58
Q

perform worse that their age-matched peers on their total language, receptive language, expressive language, phonological awareness, and grammar abilities by early school age

A

Children born VPT and have very LBW

59
Q

the associated medical conditions or health
complications amongst premature children that affect the structure and function of the brain.

A

Biological constraints

60
Q

the amount of exploration that the child does during the early stages of his life.

A

Environmental constraints

61
Q
  • While the patient is there, we have to facilitate:
A
  • Facilitates the nutritive sucking process
  • Discusses the maturation levels of the nutritive sucking with bottle and breastfeeding
  • Train parents on how to facilitate age-appropriate feeding, swallowing, language/ communication skills
  • Discuss the red flags in speech-language development and the importance of the early intervention
62
Q

For early intervention or school-based SLPs it is our duty, if the child comes to us and we have already identified that they have a language deficit or they have a persisting language deficit then it is our responsibility to:

A

● Determine the cause of the language deficits
● Identify children who needs speech-language therapy
● Identify children who may not qualify for therapy services through traditional standardized testing
● Train teachers to incorporate the therapy techniques in class discussions to enhance the child’s classroom learning