Prematurity And Rubella Flashcards

(62 cards)

1
Q

also known as “German Measles

A

CRS

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2
Q

Crs is characterized by:

A

○ Maculopapular rash
○ Lymphadenopathy
○ Fever

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3
Q

CRS Defects are often rare when the infection occurs in the

A

20th week or after the 20th week of gestation.

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4
Q

If infection occurs 0-28 days before conception

A

the infant has a 43% chance of being a ected

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5
Q

If the infection occurs 0-12 weeks after conception

A

the chance increases to 51%

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6
Q

If the infection occurs 13-26 weeks after conception

A

the chance is 23%

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7
Q

Infants are not generally a ected if rubella is contracted during

A

The 3rd trimester

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8
Q

Gestational age: 1-8 weeks

A

Cardiac defect (heart) and hearing impairment, other CRS anomalies (80%)

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9
Q

Gestational age: 9-12 weeks

A

Hearing impairment and features of CRS (50%)

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10
Q

Gestational age: 13-16 weeks

A

CRS anomalies (30%), hearing loss is prominent than other features.

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11
Q

Chances of fetal damage are minimal or none

A

> 20 weeks

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12
Q

Rubella immunoglobulin M (IgM) antibody detected

A

infants < 6 months old

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13
Q

Sustained rubella immunoglobulin G (IgG) antibody level detected in serum;

A

present on at least two occasions between 6 and 12 months of age

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14
Q

Any infant < 12 months with suspicion of CRS. The following clinical manifestation should lead to suspicion of CRS:

A

(1) Congenital heart disease and/or
(2) suspicion of hearing impairment; and/or one or more of the following eye signs: (a) cataract; (b) congenital glaucoma; or (c) pigmentary retinopathy (salt and pepper).

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15
Q

An infant of < 12 months in whom a qualified clinician detects:

A

At least two of the complications listed in group A or one in Group A and one in group B:

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16
Q

Cataract(s), congenital glaucoma, congenital heart disease, hearing impairment, pigmentary retinopathy

A

Group A

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17
Q

Purpura, splenomegaly, microcephaly, meningoencephalitis, radiolucent bone disease, jaundi

A

Group B

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18
Q

An infant who is a suspected case with one condition from Group A and meets laboratory criteria for CRS

A

Lab confirmed CRS

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19
Q

An infant who does not have group A clinical signs of CRS but who meets the laboratory criteria for CRS is classified as having congenital rubella

A

CRI

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20
Q

Auditory defect of CRS

A

sensorineural hearing impairment

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21
Q

Neurologic impairment of CRS

A

○ Microcephaly
○ Cerebral calcifications
○ Meningoencephalitis
○ Behavioral disorders, mental retardation

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22
Q

Neonatal manifestations of CRS

A

○ Low birth weight
○ Interstitial pneumonitis

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23
Q

A birth defect where a baby’s head is smaller than expected when compared to babies of the same sex and age.

A

Microcephaly

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24
Q

There are calcium deposits in the brain and the e ects of that vary depending on the severity and area of the calcification

A

Cerebral calcifications

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25
Very rare, life-threatening condition simply means you have meningitis and encephalitis at the same time.
Meningoencephalitis
26
the classic triad of symptoms for CRS are:
1) Sensorineural Hearing Loss 2) Eye Abnormalities 3) Pulmonary Stenosis
27
observed approximately around 58% of the patients
Sensorineural Hearing Loss
28
are present among 40% of the patients
eye abnormalities
29
clouding of the lens in the eye that aects vision
Cataract
30
Also called Microphthalmos - This is developmental disorder of the eye in which one or both eyes do not fully develop hence they are small
Microphthalmia
31
The narrowing of the valve between the lower right heart chamber, right ventricle, and the lung arteries (pulmonary artery)
Pulmonary Stenosis
32
Presents as a characteristic as a “blueberry mun rash” - It occurs in organs outside of the bone marrow and it occurs in very diverse condition,
Extramedullary Hematopoiesis
33
less than 37 weeks gestation is ___ baby
Premature
34
(37 to 42 weeks gestation) is a ___ baby
Full term
35
born after 42 weeks gestation is a ___ baby
Post term
36
A pregnant woman experiences regular contractions that result in the opening of the cervix after week 20 and before week 37 of pregnancy.
Pre term labor
37
less than 28 weeks
Extremely preterm
38
28 to 32 weeks
Very preterm
39
32 to 37 weeks
Moderate to late preterm
40
AKA induced preterm delivery - Often recommended by obstetrician and gynecologist when the mother has usually multiple gestations with complications
Elective
41
May or may not have an obvious trigger. Sometimes it could be an infection or placenta abduction.
Spontaneous
42
Risk Factors for Premature Birth
● Cervical Incompetence/ cervical insuciency ● Birth defects in the uterus ● History of preterm delivery ● Infection (UTI or infection of the amniotic membrane) ● Poor nutrition right before during pregnancy ● Pre-eclampsia ● Placenta previa or placental abruption
43
Means that the muscles and cervix are weak and that’s the reason it opens or the woman experiences painful cervical dilation that often results in preterm delivery.
Cervical Incompetence/cervical Insuciency
44
the placenta is located low in the uterus and it may be covering the cervix.
placenta previa,
45
placenta detaches from the uterus and with this one it has an unknown cause.
Placental abruption
46
Often caused by deficiency by the pulmonary surfactant this would aect the breath supply of the infant because this would aect the breath supply of the infant because the pulmonary surfactant plays an important role in maintaining the integrity of the alveoli and is essential for ecient exchange of gasses.
Respiratory Distress Syndrome
47
Symptoms: grunting respiration, use of the accessory muscles, nasal flaring
Respiratory distress syndrome
48
under-developed lung which is a chronic lung disease and is often used by prolonged use of ventilators.
Bronchopulmonary Dysplasia
49
caused by the immaturity of the neurologic or mechanical function of the RS because of the immaturity of the system. They do not allow non stop breathing
Apnea
50
High levels of bilirubin in the bloodstream and you will often observe the white part of the eyes or mucous membranes will turn yellow.
Jaundice
51
a type of brain damage that can result from high levels of bilirubin in the baby’s blood and it is associated with athetoid CP and HL.
kernicterus
52
It means that you have a “hole” in your heart - There is an undisclosed hole in aorta and it allows the blood to skip the circulation to the lungs
Patent Ductus Arteriosus (PDA)
53
Occurs when the glucose in the blood is too low
Hypoglycemia
54
tissue of the intestines are inflamed and causes that part to die or it will form a hole in the babyʼs intestines
Necrotizing enterocolitis (NEC)
55
A disorder of movement, muscle tone, or posture that can be caused by an injury in the brain due to infection, inadequate blood flow, or injury to a newborn’s developing brain either early during pregnancy or while the baby is still young.
CP
56
related to their neurological impairment/cognitive deficits and language impairment,
Impaired learning
57
preterm delivery interferes with the vascularization process of the eye or the forming of blood vessels in the eye = causing blindness
Retinopathy of Prematurity
58
perform worse that their age-matched peers on their total language, receptive language, expressive language, phonological awareness, and grammar abilities by early school age
Children born VPT and have very LBW
59
the associated medical conditions or health complications amongst premature children that affect the structure and function of the brain.
Biological constraints
60
the amount of exploration that the child does during the early stages of his life.
Environmental constraints
61
- While the patient is there, we have to facilitate:
- Facilitates the nutritive sucking process - Discusses the maturation levels of the nutritive sucking with bottle and breastfeeding - Train parents on how to facilitate age-appropriate feeding, swallowing, language/ communication skills - Discuss the red flags in speech-language development and the importance of the early intervention
62
For early intervention or school-based SLPs it is our duty, if the child comes to us and we have already identified that they have a language deficit or they have a persisting language deficit then it is our responsibility to:
● Determine the cause of the language deficits ● Identify children who needs speech-language therapy ● Identify children who may not qualify for therapy services through traditional standardized testing ● Train teachers to incorporate the therapy techniques in class discussions to enhance the child’s classroom learning