ECG Flashcards

1
Q

ASD

A

Partial RBBB (rsr)
Superior axis (if primum)

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2
Q

Superior axis

A

AVSD
Noonans
ASD (primum)
Ebsteins anomaly
Tricuspid atresia
Dextrocardia (or RAD)

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3
Q

Dextrocardia

A

RAD or superior axis
Poor R wave progression (dominant S waves in all leads)

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4
Q
A
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5
Q

Right ventricular hypertrophy

A

Right axis deviation
Dominant R wave in V1 (or narrow rSR’- incomplete RBBB)
Dominant S wave in V5-V6
May have upright T waves V1 (usually negative 7 days to 7 years)

“Rules” for RVH by age
R waves in V1:
0-6mo V1 > 5 big squares
6mo-1 year > 4 big squares
>1 year > 3 big squares

S waves in V6 :
0-6mo V1 > 3 big squares
6mo-1 year >2 big squares
>1 year > 1 big squares

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6
Q

Left ventricular hypertrophy

A

Deep dominant S wave in V1, tall dominant R wave in V5-V6
S wave depth in V1 + tallest R wave height in V5-V6 > 35 mm
May have LAD for age
May have deep q waves in lateral/inferior leads

LVH “Rules” by age
S waves in V1:
0-6 mo >3 big square
6mo-12 mo >4 big square
>1 year >5 big square

R waves in V6:
0-6 mo >3 big square
6-12 mo >4 big square
>12 mo >5 big square

May have ST depression and T wave inversion in left sided leads AKA LV strain pattern

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7
Q

Causes of Dominant R wave in V1

A

Normal in children and young adults
Right Ventricular Hypertrophy (RVH)
Left to right shunt
Right Bundle Branch Block (RBBB)
Wolff-Parkinson-White (WPW) Type A
Incorrect lead placement (e.g. V1 and V3 reversed)
Dextrocardia
Hypertrophic cardiomyopathy
Myotonic dystrophy
Duchenne Muscular dystrophy

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8
Q

Right bundle branch block

A

QRS duration > 120ms
RSR’ pattern in V1-3 (“M-shaped” QRS complex)
Wide, slurred S wave in lateral leads (I, aVL, V5-6)

Causes of Right Bundle Branch Block:
Pulmunary HTN / cor pulmonale
ASD
Ebsteins anomaly
Post VSD or TOF repair
Pulmonary embolus
Rheumatic heart disease
Myocarditis
Cardiomyopathy

If post op and rbbb always say vsd repair!
Also can’t comment on hypertrophy with Rbbb

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9
Q

Dextrocardia

A

Positive QRS complexes (with upright P and T waves) in aVR
Negative QRS complexes (with inverted P and T waves) in lead I
Marked right axis deviation
Absent R-wave progression in the chest leads (dominant S waves throughout)

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10
Q

HOCM

A

LVH (dominant S waves in V1, dominant R waves V6)
Deep narrow Q waves in inferior/lateral leads

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11
Q

Left atrial enlargement

A

Broad bifid P wave in lead II (p mitrale)

In isolation: mitral stenosis
With LVH: aortic stenosi, HOCM, HTN

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12
Q

Right atrial enlargement

A

Peaked P wave (p pulmunale)

Caused by: pulmunary HTN –> PS, ToF, primary pulmunary hypertension

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13
Q

Tricuspid atresia ECG

A

LAD
LVH
RA + LA enlargement (tall wide p waves )

CXR: reduced pulmunary markings

Rx: univentricular pathway

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14
Q

Partial RBBB

A

w LAD: primum ASD
w RAD: secundum ASD
w RAH + delta waves: Ebsteins anomaly

Complete RBBB- post VSD repair

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15
Q

Ebsteins ECG

A

RAH
RBBB
WPW

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