162. Ped Upper Airway Obs, Infxn Flashcards
(242 cards)
What anatomical/physiologic things make children more predisposed to resp failure? -increased airway R due to small compressible airway<br></br>- low FRC<br></br>- high O2 metabolism –> quicker fatigue
shorter safe apnea time
with precipitous hypoxia
Upper airway obstruction - main 3 categories 1. Infxn<br></br>2. Congenital anomaly<br></br>3. FB - esoph/airway
Clinical features important to dx of upper airway obstruction 1. onset and duration<br></br>2. assoc sx<br></br>3. progression with age<br></br>4. exacerbating factors (ie pos
URI
crying)<br></br>5. Feeding abnorm or dysphagia<br></br>6. Prior airway procedure - ie intub neonate<br></br>7. Choking episode<br></br>8. BL noises
cry quality and voice
Upper airway obstruction: key PE -vitals<br></br>- WOB<br></br>-character/timing of stridor<br></br>-breath sounds
Key components of resp failure extreme distress with hyper or hypoventilation
AMS
pale/mottled/cyanotic skins color
hypotonia
Upper airway obstruction: stridor defn harsh
vibratory sound caused by partial obstruction or collapse
result of turbulent airflow through some portion nose to trachea<br></br>- timing in inspir vs expir both<br></br>- coarse vs high pitched
Inspiratory stridor: where is the lesion? above glottis
Expiratory stridor: where is the lesion? intrathoracic obstruction
Biphasic stridor indicates a __ or … critical or fixed obstruction at any level
Snoring/stertor: defn low pitched inspiratory noise caused by nasal/nasopharyngeal obstruction
Stridor from pharynx: what does this classically sound like? “ex PTA: sonorous
gurgling
coarse<br></br>"”hot potato voice”””
Laryngomalacia and croup have what kind of inspiratory stridor? high pitched inspiratory indicating supraglottic/immed subglotic trachea<br></br>-hoarse/weak voice but can be N
A biphasic stridor indicates a fixed lesion - examples of this? laryngeal webs<br></br>vocal cord paralysis
Examples of stridor from lower part of trachea: (diseases x2) FB<br></br>bact tracheitis
How to take an XR when considering epiglotitis/RPA etc? neck in ext<br></br>film in inspiration
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Supraglottic airway - where is this? nose<br></br>pharynx<br></br>epiglottis<br></br>surrounding structures
Diseases of the pharynx/nose: symptoms? noisy congested breathing and resp distress
Congenital lesions of supraglottic airway disease: choanal atresia<br></br>macroglossia<br></br>micrognathia<br></br>thyroglossal duct cyst<br></br>lingual thyroid
Acquired cuases of supraglottic disease: nasal polyps<br></br>nasal FB<br></br>hypertrophic tonsils and adenoids<br></br>epiglottis<br></br>RPA<br></br>PTA<br></br>pharyngitis<br></br>mono<br></br>Upper airway FB
What is the most common congenital abnormality of the nose? choanal atresia
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urgent referral to surgical repair
2. Glycogen storage disorder
3. Congenital hypothyroidism
*obstrxn worsens when supine
muffled hot potato voice
trismus
bulging asym tonsil
deviation of avula away from abscess
drainage/needle aspirate
uncommon but massive airway enlargement secondary to enlarged tonsil can cause
incr risk TLS
complicates risk stratification
2. hepatosplenomegaly
3. rash
4. abn bl count
suppration LN
contiguous spread of infection
hematogenous seeding
2. mediastinal extension
fever
sore throat
neck stiffness/nuchal rigidity
torticollis
trismus
neck swelling
drooling
stridor
muffled voice
mediastinal extension
mediastinitis
pneumonia
empyema
2. ENT involved early
3. IV abx for some small
4. surgery for other
2. rim enhancement
3. lesions >2cm
ceftriaxone/third gen
+ vanco/linezolid if suspect MRSA
not LN
tender woody induration of sublingual space
trismus
odynophagia
airway support
adm
consult ent/anesth if emergent airway
H influ A
streptococci
s aureus
N. meningidits
steam inhalation
caustic injury
allergic rxn
FB
irritant
Lymphoprolif disorders
tender anterior neck to palpation
unstable: BMV until intub of successfel. if neither success --> needle crich or trach
if time to call for help: anes
what abx? 3-5d for abx therapy to decrease swelling *second or third gen cephalosporin
bronchodil bronchospasm
NO steroids
who has highest rte mortality? allergy to __ peanuts and tree nuts with atopy and asthma
0.3
incomplete devleopment of support cartilage of larynx
lying down
2. resp compromise
3. pulmonary hypertension
4. failure to thrive
2. compression mediastinal structure
feeding diff
aspiration
stridor mild
severe resp distress
blunt trauma to neck
biph stridor and cough within first few wks to mo of life
sx peak at 6mo due to infant growth and hemangioma during first few mo
subglottic tracheal disease
inspir stridor
abn cry by 3-4y
resp distress if large
inflamm from infection
trauma - prolonged intubation
other - RSV
expands where? subglottic
up into airway lumen because the cricoid cartilage forms a complete and nonexpanding ring in that part of the trachea
barky cough ## Footnote hoarse voice high pitched inspiratoy stridor
hoarse
4-7days
stridor with agitation but not rest
mild tachypnea
tachycardia
minimal distress
barky cough
increased wob - retraction ## Footnote tachypnea tachycardia
fussy but alert interactive and comforted by parents
HYPOXIA rare
IV if severe - same dose
2. Stridor at rest or sign resp distress: nebulizedc L epi at 1:1000 solution 0.5ml/kg max 5L OR racemic epi 2.25% solution 1.125mg/kg max 11.25mg/dose --> either diluted 2-3ml NS and given via nebulizer
d/c if no recurrence stridor
but really only benefits first hour ## Footnote not beyond
2. persistence rep distress
3. hx airway disease or recurrent croup
4. <6mo of age
5. diff with feeds
6. poor social supports
hypoxia
hypercapnia
AMS
extreme resp distress
often seen with allergy
r/ airway lesions like subglottic stenosis contribution
DS
Digeorge
symptoms worsen with agitation and age as tracheal diameter is fixed
mediastinal lesions - vascular ring ex
right aortic arch with persistent L ligamentum arteriosum
anomalus innominate artery
anomalous L CCA
L PA
aberrant R subclavian A
barium esophagram traditionally most important if complete
mucosa becomes necrotic and sloughs in the lumen so microabscesses possible in trachea
high fever
rapidly worsening stridor
fails to improve with racemic epi
sx atypical for croup (high fever/cyanosis/severe distress)
poor response to neb epi
inspire and expiratory stridor
serial endoscopy for secretions
vanco or clinda + ceftr
septic shock
renal failure
post intubation pulmonary edema
acute resp distress syndrome with need for reintubation
00> helpful for radioopaque materials ## Footnote mediastinal shift emphysema atelectasis
choking <1y: 5 back blocks then 5 chest thrusts head below trunk
heimlech >1y w/ chest compression to unconscious child
cont cough to clear obstruction
breath spont until op management arranged
AVOID RSI
needle crich <8: 14 angiocath into a 3ml syringe and with 7.5mm ETT adaptor
BVM
a. Airwayforeignbody
b. Bacterial tracheitis
d. Epiglottitis
b. Admit the patient for overnight observation.
c. Allow the patient to go home.
a. Extensionduringinspiration
b. Flexion
d. Neutral
a. Muffled, hot potato voice b. Pain radiating to the ear c. Patient 3 years of age
d. Patient 13 years old
e. Trismus
b. Candida albicans
d. Staphylococcus aureus
e. Streptococcus pneumoniae