1st Test Flashcards

1
Q

IDEA

A

Individuals with Disabilities Education Act

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2
Q

IFSP

A
  • Individualized Family Support Plan
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3
Q

IFSP

A

Identifies the strengths and needs of the child and family an
Sets forth a plan for implementation of needed services
Identifies who, what ,when, and specific goals and outcomes

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4
Q

IFSP environment

A

Free, appropriate, public education in the least restrictive environment

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5
Q

IEP

A

Individualized Educational Plan

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6
Q

IEP

A

Identifies the present level of performance
Sets forth a plan for implementation of needed services
Identifies who, what ,when, and specific goals and outcomes

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7
Q

Gross Motor assessment

A

ing – propping – turning – sitting – crawling – creeping – kneel standing – pull to standing – squatting – walking

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8
Q

Fine

A

Grasping – bilateral manipulation – pinching – placing – inserting

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9
Q

Moro reflex

A

develops 24-28 weeks of gestation. Elicited by suddenly lowering infant with a response of straightening the arms and legs to extension

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10
Q

Extensor reflexes

A

seen after four weeks of age when body tone is greater in the extensor muscles. Influenced by the position of the head and pressure on the soles of the feet.

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11
Q

symmetric tonic neck reflex (STNR)

A

( 1- 4 months) When the face is lifted up, the two upper limbs or arms bend; when the baby looks down the legs straighten.

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12
Q

The asymmetric tonic neck reflex

A

ATNR) rotation of the head will produced arm extension on the ipsilateral side of rotation and flexion on the contralateral side of rotation

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13
Q

Reflex Stepping

A

pressure on plantar surface causes LE extension

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14
Q

Labyrinthine reflexes

A

In supine (lying face-up) extension of the head causes extension of the limbs and hyper-extension of the trunk, in prone flexion of the head causes limb and trunk flexion.

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15
Q

Righting reactions

A

return the body into the anatomical position and are mediated by afferent messages from visual, labyrinthine and neck or spinal muscles as well as touch and pressure receptors in the skin, influencing the position of the head in space, the head and neck relative to the trunk, and of the trunk relative to the limbs

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16
Q

Parachute reactions

A

vestibular and visually mediated. Begins months after birth. The forward parachute is elicited when the infant is sharply lowered head-first: the arms are extended to cushion the fall. This response also occurs laterally and in extension.

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17
Q

Equilibrium reactions

A

Equilibrium reactions are the final stages in the acquisition of balancing skills up to the point of independent walking. Further balancing skills are learned throughout early childhood. Such reactions involved highly coordinated responses of the body as a whole in response to displacing stresses, demanding the brains ability to integrate its widely separated regions

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18
Q

Motor Development (1-4 months)

A

Rooting and sucking reflexes are well developed.
Swallowing reflex and tongue movements are immature;inability to move food to the back of the mouth.
Grasp reflex – automatic grasp when object is placed in palm
Landau reflex appears near the middle of this period; when baby is held in a prone (face down) position, the head is held upright and legs are fully extended.
Grasps with entire hand; strength insufficient to hold items. Holds hands in an open or semi-open position.
Movements are large and jerky.
Raises head and upper body on arms when in a prone position.
Turns head side to side when in a supine (face up) position; can not hold head up and in line with the body.
Upper body parts are more active: clasps hands above face, waves arms about, reaches for objects.

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19
Q

Team Members

A
Parents / child
PT
OT
ST  / SLP
MSW
Child-Life Therapists
Nursing
Physicians
Othotists
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20
Q

Communicating with babies

A

not able to communicate what they feel or want

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21
Q

Benefits of positioning

A
Maintenance or improvement of ROM
Prevention of or minimization of contractures
Maintenance or improvement of strength
Facilitation of developmental skills
To promote social interaction with peers
To promote functional skills
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22
Q

Mats

A

for free floor mobility

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23
Q

Wedges

A

for prone, supine, side-lying or sitting positions & promotes weight-bearing through UEs & LEs

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24
Q

Side-lie

A

– promotes hands to midline for function or positioning purposes, increases visibility of hands to the child

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25
Q

Bolsters

A

good for various forms of therapeutic exercise to stimulates normal postural responces and balance (ie: in a straddle position)

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26
Q

Balls

A

Good for Therapeutic exercise, balance, strengthening, trunk control, stimulates normal postural responces and coordination.

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27
Q

Scooter-board

A

used to promote floor level mobility in prone, strengthening of spinal extensors and UEs, mobility and play

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28
Q

Floor level carts

A

used for floor level seated mobility for young children

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29
Q

Support Components

A

placed strategically to support the trunk or pelvis or provide better alignment of the limbs

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30
Q

Prone stander

A

provides anterior support

Stimulates postural activation of head & trunk extensors

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31
Q

Supine stander

A

provides posterior support (good for head control)

Encourages full weight bearing yet provides full support

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32
Q

Standing frame

A

support is anterior to knees, poster to hips and trunk

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33
Q

Parapodium

A

stander/static support feet on pads- allow shuffle

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34
Q

Dynamic wheeled standers

A

supported standing with ability to mobilize by pushing/pulling the wheels

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35
Q

Pediatric Orthoses

A

Provide external support to maintain or correct alignment of extremities or trunk
Allow for greater mobility and function (due to postural stability
Reduce the effects of spasticity through alignment of joints and muscle

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36
Q

AFO

A

ankle foot orthosis

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37
Q

KAFO

A

knee ankle foot orthosis

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38
Q

HKAFO

A

hip knee ankle foot orthosis

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39
Q

RGO

A

reciprocating gait orthosis

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40
Q

TLSO

A

thoracic lumbar sacral orthosis

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41
Q

SMO

A

supramalleolar orthoses

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42
Q

Neuro- developmental ( NDT) – Bobath

A

A form of sensory input to effect changes in motor output
Responses to sensory input can be affected by
Environment
Health
Emotions

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43
Q

Inhibition

A

To decrease motor output

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44
Q

Inhibition techniques

A
Use gentle handling
Rocking
Firm but gentle touch
Rhythmic  movements
Slow movements
Gentle deep pressure stroking 
Consistent sensory input
Soft singing
Warm water
Wrapping or swaddling
Relaxing soft music
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45
Q

Sensory-motor Integration (SI)

A
Is based on the belief that problems arise when there is faulty integration of sensory input including
Learning difficulties
Attention deficits
Behavioral problems
Visual perceptual problems
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46
Q

SI Treatments

A

Treatment includes provision of systematic sensory input to help the child organize motor output

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47
Q

MOVE

A

mobile opportunities via education

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48
Q

Developmental Dysplasia of the Hip (DDH)

A

Poor alignment of the acetabulum and head of the femur in the developing hip

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49
Q

Developmental Dysplasia of the Hip Signs / symptoms

A
Asymmetrical hip abduction in flexion
Asymmetrical groin or buttock skin folds
Postponing of affected hip
Apparent femoral shortening on affected side
Positive test for hip subluxation
Usually begins with a limp
Mild pain in groin, medial knee or thigh
Decreased ROM (especially hip abduction and IR
Trendelenburg gait
Thigh, calf, or buttock disuse atrophy
Leg length discrepancy
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50
Q

During ambulation in the older child

A

Trendelenburg gait
Decreased hip abduction
Thigh pistoning

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51
Q

Bilateral DDH

A

Lumbar Lordosis

Swaying (waddling) gait typical of a bilateral Trendelenburg

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52
Q

Bracing & splinting to hold hip in flexion and abduction

under 9 mo

A

Usually use a Pavlik harness which allows for active kicking which promotes strength and mobility and decreases the occurrence of avascular necrosis of thee hip

53
Q

Bracing & splinting to hold hip in flexion and abduction

over 9 mo

A

Need abduction orthosis that allows for gait

54
Q

over 18 mo

A

Traction & surgery

55
Q

Legg-Calve’-Perthes Disease (LCPD)

A

Usually self-limiting degeneration of femoral head causing
Pain
Decreased ROM
Gait deviations

More common in boys 4 to 7 years of age

LACK OF BLOOD TO FEMORAL HEAD

56
Q

(LCPD) initial stage

A

failure of femoral head to grow due to decreased blood supply

57
Q

(LCLPD) Fragmentation

A

Epiphysis appears fragmented

Revascularization of the femoral head is occurring

58
Q

(LCPD) Re-ossification

A

bone density returns to normal with changes in shape and structure of femoral head and heck

59
Q

(LCPD) Healed stage

A

Femoral head and neck retain deformity from the repair process

60
Q

LCPD Etiology:

A

Disturbance in blood supply to femoral head
Can be result of:
Genetic pre-dispositIon
Trauma
Anatomical variation
Generalized disorder of epiphyseal cartilage

61
Q

Slipped Capital Femoral Epiphysis (SCFE)

A

Hip deformity related to slippage of femoral epiphysis

62
Q

SCFE Etiology

A

Hormonal influences
Genetic predisposition (weak growth plates)
Occurs in boys 2 to 3 times more than girls
More common in:
Children who are tall with delayed skeletal maturity
Obesity
In children between 9 and 16 years old
Can occur bilaterally (25 to 30%)
Can be precipitated by trauma

63
Q

SCFE Signs/Symptoms

A
Intermittent limp
Pain in:
Groin
Buttock
Knee
Thigh
Antalgic gait
Trendelenburg (weakened abductors on involved side)
LE held in ER
Decreased ROM
64
Q

SCFE Treatment

A

Goals include
Minimize or reduce slippage of femoral head on neck
Maintain hip ROM & function
Minimize possible future degeneration
Surgery usually required to pin the hip & prevent further slippage in mild to moderate cases
Surgery – Varus - Osteotomy
Bed rest / traction may be used to reduce pain & spasm before surgery

65
Q

SCFE Interventions

A

Providing appropriate assistive devices (wheelchair/ crutches
Gait & mobility training
ROM (active & passive)
Strengthening exercises
Assessment of home environment to accommodate mobility and safety
Family training for all of the above
Consultation with teachers for campus mobility

66
Q

Juvenile Rheumatoid Arthritis (JRA)

A

A group of disorders characterized by inflammation of connective tissue including joints and other systems
Classified in children under 16 years of age with condition lasting > 6 weeks

67
Q

JRA Oligo-articular

A

(<5joints)

68
Q

JRA Poly-articular

A

(>5 joints)

69
Q

JRA Etiology

A

unknown (may have several contributing factors
Viral or bacterial infection that triggers the auto-immune response
May be genetically pre-disposed

70
Q

JRA Signs / Symptoms

A
Pain, swelling, stiffness in joints
Fever
Rash (more common in systemic)
Inflammation of the iris of the eye
Myalgia
Involvement of   (more common in systemic)
Lymph nodes
Jeart
Liver
Spleen
Pericardium
Lungs
71
Q

JRA Medications

A
NSAIDS
Slow acting antirheumatic drugs (SAARDS)
Corticosteroids
Immunosuppressive and cytotoxic agents 
Cyclosporing or methotrexate
72
Q

JRA Surgical treatment:

A
Synovectomy
Soft tissue releases
Osteotomy 
Joint fusion 
Total joint replacement
73
Q

Osteogenesis Imperfecta Etiology

A

Genetic
Problems with the amount & quality of collagen in the body
Presents with fragile bones / low bone density

74
Q

Osteogenesis Imperfecta Signs & Symptoms

A

Frequent fractures
Scoliosis/Kyphosis
Short stature
Hearing loss

75
Q

Osteogenesis Imperfecta Goals

A

Minimize fractures through protective measures
Joint protection
Promote bone strength
Maximize activity & weight bearing for increased bone strength
Maximize functional skill

76
Q

Arthrogryposis (Multiples Congenita

A

Non-progressive neuromuscular disorder
Presents with
Multiple contractures (distally > proximally) due to decreased fetal movement
Dislocation at hips or knees
Deformities of joints (fusiform or cylindrical shape)
Joint fusion (in some cases)
Thinning of subcutaneous tissue
Absence or decreased size of muscle groups
Absent skin creases

77
Q

Arthrogryposis (Multiples Congenita) Etiology

A
Unknown
Possible trauma during first trimester
Maternal history of a condition that limits fetal movement 
Fevers during pregnancy
MS
Myasthenia gravis
Myotonic dystrophy
Uterine abnormalities
78
Q

Pediatric Fractures

A

Fracture patterns are different due to bones that are more flexible, more porous, and less dense than adult bones
Due to thicker periosteum than adults, there is better blood supply therefore healing is faster (2-4weeks)

79
Q

Pediatric Fractures Etiology

A

Trauma
Child abuse
MVA
Genetic (Osteogenesis imperfect)

80
Q

Pediatric Fractures Common sites

A

Distal radius
Tibial shaft
Clavicle
Elbow

81
Q

Pediatric Fractures Signs / Symptoms

A
Redness
Swelling
Pain
Heat and deformity of extremity
Muscle spasm
Crying
Not using extremity
82
Q

Ankle / Foot Deformities Etiology

A
Congenital deformities
In-utero positioning
Neuromuscular disorders 
Myelomeningocoele
Arthrogryposis
Genetic disorders
83
Q

Ankle / Foot Deformities Classification

A
Metatarsus adductus
Talipes Equinovarus (clubfoot)
Calcaneovalgus (dorsiflexion of foot with eversion or valgus of hind-foot)
84
Q

Ankle / Foot Deformities Sign/symptoma

A

Obvious deformities
Gait abnormalities
Delay is gross motor and mobility skills

85
Q

Ankle / Foot Deformities Treatment

A

Serial Casting

Surgery

86
Q

ANOXIC ENCEPHALOPATHY

A

Definition - brain damage due to lack of oxygen
aka
CEREBRAL PALSY
Caused by an insult to the developing brain and resulting in permanent and non-progressive damage
Affects tone, posture, and movement

87
Q

CP Prenatal

A

Genetic

Viral Infections

Bacterial infections

Drug exposure

88
Q

CP Peri-natal

A
Prematurity’
Low-birth weight
Severe jaundice
Intra-ventricular hemorrhage
Poor nutrition
Asphyxia
89
Q

CP Post-Natal

A
Infection
Asphyxia
TBI
MVA
Fall
Shaken baby syndrome
Child abuse
CVA
Near drowning 
Brain tumor
90
Q

CP Diplegia

A
Both LEs
Trunk and UEs to a lesser extent may be affected
Standing  Posture (typical)
Crouched with trunk flexion
Adduction, & IR at both hips
Knees are flexed
Ankles are plantar-flexed
91
Q

Diplegia

A

Typically child has
spasticity / high tone
In both LEs

92
Q

Diplegia gait

A

Poor disassociation between trunk and legs
Body rotates during gait
UEs held in the “high guard” position for balance

93
Q

Diplegia assissitive devices

A

Crutches
Walker
wheelchair

94
Q

Hemiplegia Posture

A
Same side UE & LE
Standing Posture (typical)
Shoulders in adduction & IR
Elbow & wrist flexion
IR of hip
Knee extension
 Ankle plantar flexion
95
Q

Hemiplegia Gait (typical)

A

Asymmetrical gait pattern

Circumduction of LE

96
Q

Hemiplegia assissitive devices

A

Crutch or cane for balance

97
Q

Quadriplegia/Tetraplegia

A

All 4 extremities are involved

98
Q

Triplegia

A

3 extremities are involved

Usually both LEs and one UE

99
Q

CP / ABNORMAL TONE - HYPOTONICITY

A

Also referred to as LOW TONE / HYPOTONIA

“Rag Doll” / floppy
Soft mushy feel to muscles
DTRs are weak
Likelihood of obesity due to decreased activity
Usually demonstrate impaired speech due to impaired oral motor control and drooling

100
Q

CP / HYPOTONICITY Gait

A

Wide base of support
Short stride lengths
Impaired balance

101
Q

CEREBRAL PALSY / HYPERTONICITYSigns & Symptoms

A

HIGH TONE / HYPERTONIC / SPASTICITY
Hyperactive DTRs
Hard, stiff, tight feel to muscles
Persistence of primitive reflexes which prevent development of normal movement patterns
ATNR
STNR
TLR
Usually thin due to excessive energy output
Speech usually impaired due to poor oral motor control, drooling

102
Q

Hypertonic gait

A

Impaired muscle control

Impaired balance due to abnormal variations & distribution of muscle tone

103
Q

CEREBRAL PALSY / SIGNS & SYMPTOMS - DYSTONIA

A

Also referred to as
Mixed tone
Fluctuating tone
Athetosis

104
Q

Dystonia Characteristics

A

More than one type of tone
Can be mild to severe
In infancy presents as low tone and changes with maturity
Stringy, elastic muscles to the touch
Child is usually thin due to extreme expenditure of energy
Impaired speech as in hypertonia

105
Q

Dystonia gait

A

Gait is only possible to those who have enough stability to stand or walk
Excessive movements
Impaired balance
Walker or crutches can be used but for the most part these children use a wheelchair
As children grow and become stronger, they can present a challenge with management of transfers etc. to their caregivers / family.

106
Q

Ataxia characterisitcs

A

Poor balance
Wide base of support and “high-guard arm position” during gait
Tremors
Low postural tone
Poor visual tracking
Distributed throughout the body
ROM usually normal or excessive due to low tone
Hyporeflexia of DTRs, with weak primitive reflexes
Muscles have a soft doughy feel to the touch

107
Q

Secondary Conditions / Impairments of Children with CP

A
Mental retardation (50 – 75%)
Seizures (30%
Visual impairment (50
Speech/language deficits or delays (50%)
108
Q

General Principles for PT Interventions / Cerebral Palsy

A

Intervention is individualized

Intervention is optimal through a team approach with child and family involved in setting goals

109
Q

Pediatric Traumatic Brain Injury (TBI)

A

An acquired injury to the brain resulting in a change in consciousness or an anatomical abnormality in the brain

110
Q

Pediatric Traumatic Brain Injury Etiology

A
Trauma
Child abuse
In infants, “Shaken Baby  Syndrome” is most common injury
MVA
Falls
Sports related Injuries
GSW
111
Q

TBI Secondary causes

A

Increased intracranial pressure due to edema

Hematoma due to bleeds in the brain from trauma

112
Q

TBI Signs & Sympt

A

Depends on
Severity
Area of brain involved
Length of time for loss of consciousness (coma)
Other injuries to the body and their severity

113
Q

Sequelae of brain injury can include

A
Motor impairment
Spasticity
 Ataxia 
Weakness
Contractures and resultant deformities
 Cognitive impairment
Language disorders
Sensory impairments
Psychosocial disorders
114
Q

SPINA BIFIDA

A

A group of congenital malformation of the spine, including the vertebrae and the spinal cord

115
Q

SPINA BIFIDA Etiology

A

Genetically predisposed
Nutritional deficiencies (especially maternal folic acid deficiency)
Environmental (IE: alcohol during first 4 weeks of pregnancy when neural tube closes)

116
Q

Occulta

A

(not visible) No disability usually

117
Q

Acculta or cystic

A

visible

118
Q

Meningocele

A

no disability usually
CSF and superficial tissue protrudes from the spine in a sac at the level of the lesion
Neurological tissue is rarely involved

119
Q

Myelomeningocele

A

Meninges and parts of spinal cord protrude in a sac at the level of the lesion
There is an abnormality of the spine
Disability includes paralysis with loss of sensation below the level of the lesion
Extent of the disability depends on the level of the lesion and scope of the neurological involvement

120
Q

SPINA BIFIDA / Hydrocephalus

A

Awareness of potential shunt malfunction ~~~safety issue~~

121
Q

Signs of malfunction: (may require emergency response)

A
Irritability
Headache
Vomiting
Lethargy
Fever
Bulging eyes or fontanel
Change in behavior or level of alertness
Seizure activity
Change in coordination
122
Q

Duchenne Muscular Dystrophy

A

The most common form of congenital, degenerative disease of muscle tissue
Almost always in boys

123
Q

Duchenne Muscular Dystrophy Signs & Symptoms

A

Muscles appear hypertrophied due to fat and connective tissue replacing muscle tissue
Starts at approx. 2 years of age and progresses till death
Some live to early adulthood

124
Q

Gower’s sign –

A

Evident by 4 to 7 years of age
When a child uses his hands in a walking motion up the thighs to assist while attempting to stand (hip & knee extensors

125
Q

Contractures develop in

A

Heel-cords
TFL
Hamstrings
Hip flexors

126
Q

CYSTIC FIBROSIS

A

Chronic suppurative pulmonary disease which causes the greatest mortality
Median age of survival now is approximately 36 years of age which is an improvement due to newer management strategies and techniques

127
Q

HIPPO-THERAPY PRECAUTIONS/POSSIBLE CONTRAINDICATION

A
Abnormal fatigue
Allergies
Arnold Chiari malformation
Cardiac condition
Diabetes
Heterotrophic ossification Hip 
Dislocation 
Subluxation 
Dysplasia with significant restriction of hip abduction 
History of breakdown of grafting over bony/weight –bearing areas
Any child 2-4 yrs of age
Hydrocephalus
128
Q

HIPPO-THERAPY / Contraindications

A
Orthopedic: 
Acute herniated disc, 
Atlanto-axial instability
Coxal arthrosis (degeneration of the hip joint)
Excessive kyphosis or lordosis
Hemivertebrae
Severe osteoporosis
Orthopedic: (cont’d)
Pathologic fractures (osteogenesis imperfecta)
Spondylolisthesis
Structural scoliosis greater then 30 degrees
Unstable spine