Stroke Syndromes Flashcards

1
Q

Supplemental Motor Area (SMA) Location

A
  • Part of motor association cortex (area 6)

- Medial surface of frontal lobe just in front of primary motor area

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2
Q

Supplemental Motor Area is strongly associated with

A

Basal ganglia - coordinates responses across brain regions and switches motor programs

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3
Q

Supplemental Motor Area Function

A
  • Associated with internal initiated movements
  • Associated with movements that were previously learned
  • Assembles subroutines of complex movements
  • Active during mental practice
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4
Q

Premotor Cortex (PMC) Location

A
  • Area 6 - lateral hemisphere just in front of primary motor cortex
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5
Q

Premotor cortex (PMC) is strongly associated with

A
  • Cerebellum - monitors & updates movement based on sensory feedback; error detection; optimizes movement plan
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6
Q

Premotor Cortex (PMC) Function

A
  • Activated when movement initiation is dependent on external cue
  • Associates with specific sensory cues with specific movements
  • More active when movement is visually guided
  • More active in early phases of learning
  • Mirror neurons are found here
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7
Q

SMA v. PMC Activities

A
  • PMC more active with early practice with explicit information
  • SMA more active when sequence is more automatic
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8
Q

Primary Motor Cortex (Area 6) Function

A
  • Movement initiation
  • Fine motor control
  • Novel movements
  • Highly fractionated movements
  • Good control over force and speed of movements
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9
Q

Primary Motor Cortex Afferents

A
  • Supplemental motor cortex

- Premotor cortex

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10
Q

Inferior Parietal Lobule

A
  • Generates spatial map of body and environment
  • Codes parameters for movement between body and environmental targets and informs premotor cortex
  • Mirror neurons located here
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11
Q

Apraxia

A
  • Difficulty or inability in executing appropriate or purposeful movements despite absence of paresis, ataxia, or sensory loss, comprehension, attention, or willingness to perform movement
  • Affects previously learned and new tasks
  • Affects non-paretic side
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12
Q

Apraxia results from damage to

A
  • Parietal lobe damage, especially inferior parietal lobule
  • Sometimes frontal motor association areas
  • Usually dominant (left) hemisphere
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13
Q

Types of apraxia

A
  • Ideomotor apraxia
  • Oral motor apraxia
  • Ideational apraxia
  • Constructional apraxia
  • Dressing apraxia
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14
Q

Ideomotor Apraxia

A
  • Has form of concept but cannot translate to execution
  • Most common form of apraxia
  • Associated with inferior parietal lobule
  • Motor planning deficits
  • Has difficulty performing movements on commands but can perform spontaneously
  • Movement is clumsy and awkward
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15
Q

Oral Motor Apraxia

A
  • Common in children and can happen in adults after stroke
  • Affecting lips and face
  • Affects purposeful movements associated with speaking and facial expression
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16
Q

Ideational Apraxia

A
  • More severe motor planning deficit
  • Associated with dominant parietal lobe
  • Cannot conceptualize the motor task (no idea of what to do)
  • With more severe involvement, cannot perform appropriately either automatically or on command
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17
Q

Constructional Apraxia

A
  • Inability to construct or copy simple design or models
  • Visual association cortex of non-dominant parietal lobe affected
  • Aware of mistakes
  • Inability to comprehend, interpret, and reproduce reciprocal relationships of objects in space
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18
Q

Dressing Apraxia

A
  • Inability to dress oneself properly due to disorder in body schema or spatial relationships
  • Damage to non-dominant occipital or parietal lobe
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19
Q

Clinical Tests of Apraxia

A
  • See if patient can use common objects for ADLs
  • See if patient can imitate gesture like peace, sign, okay sign
  • Is response clumsy?
  • Does pt perseverate?
  • Can pt do things spontaneously that they cannot do on command?
  • Do they know what to do with object?
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20
Q

Treatment principles for patients with apraxia

A
  • Explicit information or not
  • Give short, simple commands
  • Pt should repeat aloud or in their heads
  • Verbal or visual mediation of the task
  • As therapist, adjust or facilitate movement
  • Start and master first component before moving on
  • Task should be done same way each time
  • Repetition
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21
Q

Agnosia

A
  • Inability to recognize
  • Tends to be in non-dominant hemisphere
  • Types: Tactile, visual, auditory, autotopagnosia, anosognosia, prosopagnosia
22
Q

Tactile Agnosia

A
  • Areas 5, 7 - non-dominant parietal lobe

- Cannot recognize objects by touch

23
Q

Visual Agnosia

A
  • Areas 18, 19 - visual association areas for nonsymbolic objects (paperclip, teacup) AND non-dominant inferior parietal lobule for symbols
  • Cannot recognize by sight, “what” visual pathway
24
Q

Auditory agnosia

A
  • Cannot recognize by sound

- Superior part of temporal lobe, bilateral damage

25
Q

Autotopagnosia (a.k.a somatoagnosia)

A
  • Inability to identify body or its parts or to orient them correctly
  • Non-dominant inferior parietal lobule (areas 39, 40)
26
Q

Anosognosia

A
  • Unawareness or denial of illness

- Nondominant inferior parietal lobule (areas 39,40)

27
Q

Prosopagnosia

A
  • Inability to recognize faces

- Damage to non-dominant occipto-temporal area (“what” pathway)

28
Q

Neglect

A
  • Failure to report, orient toward, or respond to stimuli on the contralateral side of space that cannot be attributed to sensory or motor dysfunction
29
Q

Neglect - area of lesion

A
  • Usually non-dominant inferior parietal lobe (areas 39, 40) –> usually involved with a left hemiparesis
30
Q

Features of unilateral neglect

A
  • Fails to respond to stimuli on contralateral side
  • Overattends to stimuli on ipsilateral side
  • Can see patient turn head away from side that they are neglecting
  • Degree of neglect varies much from patient to patient
  • Often occurs with anosognosia
31
Q

Proposed mechanisms of neglect

A
  1. Disorder of attention - attention biased to ipsilateral side
  2. Disorder of coding visual information - cannot put parts together to make the whole
32
Q

Testing for neglect

A
  1. Copy a simple drawing
  2. Line bisection
  3. Cancellation
  4. Reading aloud
  5. Extinction
33
Q

Homonymous Hemianopia v. Neglect

A
  • Homonymous hemianopia is sensory deficit
  • Neglect is perceptual deficit
  • Can teach someone with HH to compensate by scanning but not able to do so with neglect
34
Q

Intervention for unilateral neglect

A
  • Simple and direct commands, avoid subtleties and sarcasm
  • Use perceptual anchor to direct attention to left
  • Direct information to patient’s left side
  • use hemiparetic side limb movement
  • Encourage visual tracking to the left
  • Eliminate distractions
  • Educate patient on compensatory strategies
35
Q

Right Parietal Syndrome

A
  • Damage to the non-dominant (right) hemisphere, specifically parietal lobe, often resulting in severe perceptual deficits that often occur in combination
36
Q

Right Parietal Syndrome Symptoms/Impairments

A
  • Anosognosia
  • Autotopagnosia
  • Constructional apraxia
  • Contralateral neglect
  • Easily disoriented/distracted
  • Be labile/inappropriate with verbal expression
  • Apraxic in dressing
  • Concrete communication style
37
Q

MCA v. ACA Stroke

A

MCA - areas affected - face = hand > arm > UE

ACA LE > UE

38
Q

Brainstem signs

A
  • Cortical stroke - cortico-bulbar tracts affected
  • Brainstem signs with UMN components (+ reflexes)
  • Usually recover well because bilateral innervation
39
Q

Internal Capsule

A
  • Pure motor hemiplegia and/or sensory hemianesthesia

- other impairments depend on size of infarct

40
Q

Hypoperfusion (Borderzone or Watershed)

A
  • Distal most territory of each artery affected
  • Weakness shoulder > arm > face
  • Anomic aphasia & ideational apraxia
41
Q

Left MCA Superior Division Stroke

A
  • Right face & arm weakness
  • Non-fluent/Broca’s aphasia
  • Left gaze preference
  • Can have right face and arm sensory issues
42
Q

Left MCA Inferior Division

A
  • Right visual field deficit
  • Apraxia possible
  • Fluent/Wernicke’s aphasia
  • Maybe right face and arm sensory issues & weakness
43
Q

Left MCA Deep territory

A
  • Right pure motor hemiparesis

- Can have aphasia

44
Q

Left MCA Stem

A
  • Right hemiplegia & hemianesthesia
  • Right homonymous hemianopia
  • Global aphasia
  • Left gaze preference
  • Apraxia possible
45
Q

Right MCA Superior Division

A
  • Left arm and face weakness
  • Left hemineglect
  • Right gaze preference possible
  • Sometimes left sensory loss
46
Q

Right MCA Inferior Division

A
  • Profound left hemi-neglect
  • Left visual field and somatosensory deficits
  • Motor neglect with decreased voluntary and spontaneous initiation of movements on L side
  • Right gaze preference
47
Q

Right MCA Deep territory

A
  • Left pure motor hemiparesis

- Left hemi-neglect

48
Q

Right MCA Stroke

A
  • Left hemiparesis, hemianestheisa
  • Left homonymous hemianopia
  • Left hemi-neglect
  • Right gaze preference
49
Q

Left ACA

A
  • Right leg weakness and sensory loss
  • Grasp reflex, frontal lobe behavioral abnormalities, transcortical aphasia
  • Right hemiplegia
50
Q

Right ACA

A
  • Left leg weakness and sensory loss
  • Grasp reflex, frontal lobe behavioral abnormalities, transcortical aphasia
  • Left hemiplegia
51
Q

Left PCA

A
  • Right homonymous hemianopia

- With thalamus and internal capsule - aphasia, right hemisensory loss, right hemiparesis

52
Q

Right PCA

A
  • Left homonymous hemianopia

- With thalamus and internal capsule - aphasia, left hemisensory loss, left hemiparesis