Diabetes Flashcards

1
Q

Chronic disorder of impaired

A

metabolism with vascular and neurologic complications

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2
Q

Blood glucose level normally regulated by

A

insulin

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3
Q

if you have gestational diabetes you are more likely to have

A

diabetes in the feature

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4
Q

secondary DM occurs by

A

induced by trauma, surgery, pancreatic disease, or medications

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5
Q

Type 1 DM is

A

Absence of endogenous insulin

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6
Q

Type 1 DM is usually Called what

A

Formerly called juvenile-onset diabetes because it most commonly occurs in juveniles and young adults

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7
Q

How does Type 1 DM Occur

A

An autoimmune process, possibly triggered by a viral infection, destroys beta cells, the development of insulin antibodies, and the production of islet cell antibodies (ICAs

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8
Q

in affect type 1 DM people need

A

Affected people require exogenous insulin for the rest of their lives

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9
Q

Type 2 DM is

A

Inadequate endogenous insulin and body’s inability to properly use insulin

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10
Q

Type 2 Occur because

A

Beta cells respond inadequately to hyperglycemia; results in chronically elevated blood glucose
Continuous high glucose level in the blood desensitizes the beta cells; they become less responsive to the elevated glucose

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11
Q

Type 2 is most common in

A

adult

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12
Q

how do you control type 2 Dm

A

Controlled by diet and exercise; may require oral hypoglycemic agents or exogenous insulin

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13
Q

Role of glucose

A

Insulin stimulates active transport of glucose into cells

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14
Q

Role of fatty acids

A

Promotes fatty acid synthesis and conversion of fatty acids into fat, which is stored as adipose tissue

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15
Q

Role of protein

A

Enhances protein synthesis in tissues and inhibits the conversion of protein into glucose

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16
Q

? are identified in more than 80% of all people with type 1 diabetes at the time of diagnosis

A

Islet cell antibodies

17
Q

thought to be prescuror to diabetes

A

metabolic syndrome

18
Q

long term complications

A
retinopathy
nephropathy
atherosclerosis]
CAD
Neuropathy inadequate blood supply
19
Q

hypoglycemia s/s

A

tachycardia, palpitations, tremor, sweating, nervousenes

20
Q

3 p’s

A

polyuria, polydipsia (thirst), polyphagia (hunger)

21
Q

oral glucose test

A

Diet of 150 to 300 g carbohydrate for 3 days before test
Night before test, patient fasts after midnight
Morning of test, blood drawn for fasting serum glucose
Patient then given a drink (Glucola) containing 75 g of carbohydrates and instructed to remain quiet
Blood drawn at 30 minutes and 1 hour after the ingestion of glucose. After these two samples, blood is drawn at hourly intervals until the test is completed

22
Q

all pt with what type of DM will need insulin injections

A

type1, eventually 2 in the future if not controlled will need them

23
Q

rapid acting and short acting are what kind of liquid

A

clear, all others are cloudy

24
Q

all insulins are given by which injection route

A

sub q

25
Q

which insulin is given iV

A

Regular

26
Q

Site rotation helps prevent

A

lipohypertrophy or lipoatrophy

27
Q

Somogyi phenomenon

A

Rebound hyperglycemia in response to hypoglycemia

28
Q

Dawn phenomenon

A

An increase in fasting blood glucose levels between 5 and 9 AM that is not related to hypoglycemia

29
Q

hypoglycemia Develops when the blood glucose level

A

falls to less than 45 to 50 mg/dL

30
Q

cause if hypoglycemia in insulin therapy is

A

Causes include taking too much insulin, not eating enough food or not eating at the right time, an inconsistent pattern of exercise

31
Q

Exogenous hypoglycemia

A

Results from outside factors acting on the body to produce a low blood glucose
Include insulin, oral hypoglycemic agents, alcohol, or exercise

32
Q

Endogenous hypoglycemia

A

Occurs when internal factors cause an excessive secretion of insulin or an increase in glucose metabolism
These conditions may be related to tumors or genetics

33
Q

Functional hypoglycemia

A

From a variety of causes, including gastric surgery, fasting, or malnutrition

34
Q

Life-threatening emergency caused by a relative or absolute deficiency of insulin

A

Diabetic Ketoacidosis (DKA)

35
Q
s/s 
Diabetic Ketoacidosis (DKA)
A

Anorexia, headache, and fatigue

As condition progresses, classic symptoms of polydipsia, polyuria, and polyphagia develop