412 Substance Use Flashcards

1
Q

10 substances in SUDs

A
  • alcohol
  • cannabis
  • opioids
  • hallucinogens
  • inhalants
  • sedatives and hypnotics
  • anxiolytics
  • tobacco
  • stimulants
  • other/unknown
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2
Q

SUDs diathesis-stress model

A
  • risk-seeking tendencies could act as a diathesis (strong reward neural pathway)
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3
Q

correlates of SUDs

A
  • associated with 3 leading causes of death in adolescents: accidents/injuries, suicide/self-harm, interpersonal violence
  • legal and educational problems
  • comorbidity with other disorders
  • earlier use (14 vs. 21) linked with more substance-related impairment later (risk marker, not causal)
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4
Q

prevalence of substance use and SUDs

A
  • a lot of variability in adolescents, gets more common with age
  • experimentation very common (normative)
  • SUDs roughly 11.4% 13-18 years
  • substance use rates decreased during COVID and remained low
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5
Q

Early brief interventions for SUD

A
  • norm-based interventions for college students (helping people realize how much others are actually drinking - shift toward descriptive norms)
  • large discrepancy between perceptions of peer substance use and what actually happens (overestimating)
  • informing about how much you drink (ranked with peers), your perception of others, and how much your peers drink (shift in perceived norms = changing your behaviour according to the norm)
  • help reduce frequency and quantity of drinking, but not many studies of efficacy in long-term
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6
Q

injunctive norms

A
  • how much others approve/disapprove of a behaviour like drinking
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7
Q

descriptive norms

A
  • how much others are actually engaging in a behaviour like drinking
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8
Q

outpatient treatment for SUDs

A
  • family therapy
  • alcoholics anonymous
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9
Q

family therapy

A
  • multidimensional
  • working with caregivers to increase parental monitoring
  • working with teens to improve coping strategies or fix risk-taking behaviour
  • good efficacy (maybe better than CBT)
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10
Q

alcoholics anonymous

A
  • very popular and common worldwide (easily accessible)
  • acknowledge that alcohol is a problem, abstinence is the goal, supported by a peer
  • 12-step program
  • has other equivalents for various drugs (nicotine not as supported)
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11
Q

3 hypotheses in AA

A

(1) attendance itself leads to reduced substance use (meetings help you use less)
(2) lower alcohol use is associated with more AA attendance (less severe = more likely to use AA and more likely to recover)
(3) better prognosis (more motivated, less comorbidity, more protective and less risk factors) = lower alcohol use and benefit from AA

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12
Q

how does evidence support AA

A
  • RCTs show a lack of efficacy (but studies aren’t well done, participants are coerced instead of self-selected into treatment)
  • following Ps who received Tx at a hospital, then attended AA = support for efficacy (lower alcohol use at follow-up)
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13
Q

support for 3 hypotheses of AA

A

(1) AA involvement at 1-year post-treatment predicted lower alcohol use at 2-year post-treatment
(2) alcohol use at year 1 didn’t predict AA involvement at year 2 (people using less don’t attend AA more)
(3) results not explained by AUD severity, comorbidity, or motivation (not a good prognosis that mediates the results)

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14
Q

inpatient treatment for SUDs

A
  • short duration (4-6 weeks)
  • individual counselling, family therapy, treatment for comorbidities
  • often followed by outpatient treatment
  • can get very expensive, especially for teenagers who need multiple stints
  • good rationale (changing the environment and losing access to substances) but very few controlled studies about efficacy
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