5. Adolescence Development Flashcards

(38 cards)

1
Q

What is adolescence?

A

derives from latin
- ‘to grow into adulthood’

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2
Q

What ages do early, middle and late adulthood occur?

A
  • early: 10-13
  • middle: 14-18
  • late: 19-25
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3
Q

What does adolescence begin with?

A

puberty
- period of rapid physical growth and sexual maturation

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4
Q

What are the key characteristics of puberty?

A
  • sequence of changes is universal, but timing varies (typically 3-5yrs)
  • age of onset: 8-15 years
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5
Q

What factors affect the onset age for puberty?

A
  • sex: girls ahead of boys by months/years
  • stress = earlier
  • genes and ethnicity
  • weight: malnutrition delays puberty, obesity is the opposite
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6
Q

When does adolescence end?

A
  • in the 1960s was thought to be 18-10
  • social revolution further lengthened adolescence as uni student lingered in this stage of life
  • contemporary society it is determined by social factors rather than biologically
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7
Q

How did Stanley Hall historically view adolescence?

A
  • period of ‘inevitable turmoil’ taking place during the transition from childhood to adulthood
  • focus on aggression and crime
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8
Q

How does the storm and stress model explain adolescence?

A
  1. conflicting instincts and urges: triggered by puberty
  2. stress and turmoil caused by changes
  3. individual development takes over evolutionary development ‘appropriate environment’
  4. ‘reborn’ into ‘civilised society
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9
Q

What are the key components of the storm and stress model?

A
  • a model that considers evolutionary, hormonal and environmental factors (all linked)
  • Hall saw human development as a reflection of the evolutionary development the species went through (animalistic to civilised)
  • this is mediated by biology
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10
Q

What are key components of the maturational imbalance model (dual system model)?

A
  • suggests there is an imbalance between systems that mature early in development (limbic, affective-motivational system) and systems that are late to mature (cortical control system)
  • this imbalance gives rise to adolescent-specific behaviours: such as greater propensity to take risks
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11
Q

What is the limbic system?

A
  • ‘primitive part of the brain’
  • no universal agreement on the complete list of structures

areas that most agree on:
- cingulate gyrus
- ventral striatum
- hypothalamus
- amygdala
- hippocampus

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12
Q

What areas of the brain are involved in social-emotional processing?

A
  • ventral striatum: responsible for salience of behaviour and motivation (greater activity = predict risk taking behaviours)
  • amygdala: responsible for emotion processing and fear responses
  • prefrontal cortex and anterior cingulate cortex: responsible for executive function, behavioural control and decision making
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13
Q

What is the maturational imbalance model?

A

children:
- prefrontal cortex (PFC) and subcortical regions are equally underdeveloped
- pathways between them are also underdeveloped: don’t communicate well

adolescents:
- imbalance: PFC signalling much weaker than subcortical regions
- pathways are also imbalanced: subcortical regions in charge of PFC
- limbic system leads, leading to display of adolescent behaviours

adults: fully matured brain
- PFCs signalling strengthens
- subcortical region signal lessens
- areas communicate well: PFC mediates limbic system allowing behaviours to even out

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14
Q

What are the key components of the maturation imbalance model?

A
  • model for enhanced ‘affective and incentive-based’ behaviour in adolescence
  • early maturation of the subcortical regions such as the amygdala and ventral striatum
  • late maturation of prefrontal cortical regions
  • predicts non-linear enhancement in affectively-driven behaviour during adolescence
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15
Q

What study did Galvan and McGlennan conduct into adolescents response to taste?

A

tested: 15 adolescents and 15 adults
two stimuli:
- appetitive: sucrose water
- aversive: salt water

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16
Q

What did Galvan and McGlennan find about adolescents response to taste?

A

adolescents rated sucrose more positively and salt more negatively than adults (bigger reaction)
- ventral striatum was more activated by sucrose water in adolescents than adults
- caudate was more activated by salt water in adolescents
- insula was more activated by salt water in adults

17
Q

What study did Hare et al conduct into adolescents response to faces?

A

tested 3 groups on their responses to emotive faces:
- 12 children
- 24 adolescents
- 24 adults

  • PPs completed a no-go task with fearful, happy and calm facial expressions as targets and nontargets
  • PPs told to respond when seeing one facial expression
18
Q

What did Hare et al find about adolescents response to faces?

A
  • exaggerated activity in amygdala for adolescents
19
Q

What study did Chein et al conduct into risk taking in adolescents?

A
  • randomly assigned adolescents, young adults and adults to complete a series of tasks
  • either alone or with a peer (same sex and same age)
  • risk taking propensity was assessed using the stoplight game
20
Q

What did Chein et al find about adolescents risk taking?

A
  • adolescents had significantly more risks with peer (more crashes)
  • they also has had much more activation in the ventral striatum and orbitofrontal cortex with peers and in comparison to the other groups
  • the presence of peers increases adolescent risk taking by heightening sensitivity to the potential reward value of risky decisions
  • risk is viewed as having a higher reward
21
Q

Why is adolescence seen as an age of vulnerability?

A
  • ‘window of vulnerability’ to risky behaviour
  • key period for preserving life-long health (social, mental, physical etc)
  • 75% of lifetime mental health disorders have their onset before 24
  • quality of relationships in this time manifest to those later in life
22
Q

What evidence did Degenhardt et al find for age of onset being inconsistent across substances?

A

used a large sample from 17 countries
- alcohol: 16-19
- cannabis: 18-19
- cocaine: 21-24

  • is the age of onset variation due to ease of access?
23
Q

What did Dick et al show about nature and nurture for alcohol use behaviour?

A
  • both influence behaviour: nature more so
  • steady increase in the influence of genetic factors on alcohol use across adolescence
  • corresponding decrease in the influence of common environmental factors
24
Q

What evidence did Bourque et al find for substance use increasing the risk of psychosis?

A
  • becoming more regular marijuana user during adolescence is associated with increased risk of psychotic symptoms
25
What do rodent studies indicate about substance use and risk?
- exposure to cannabinoids in adolescence results in structural brain changes and impaired synaptic plasticity - this impacts the fronto-limbic system that is involved in higher brain functions - overaction by CB1R agonists during adolescence and the resulting changes in neuroplasticity could alter brain maturation
26
Why can adolescence be seen as an age of opportunity?
- education correlates with success - inequality is challenged - quality long term friendships are made - interventions for positive social and health outcomes
27
What are gonads?
- glands that produce sex dependant hormones
28
What is the status of gonadal function in children?
- testis and ovary are differentiated as are the secondary sex organs - very low levels of sex hormones: T for boys and E for girls - very low levels of GnRH (in hypothalamus) and FSH/LH (in anterior pituitary)
29
What is the biological trigger for puberty?
- in boys and girls: production of GnRH from the hypothalamus - this stimulates the pituitary gland to release LH and FSH - these two hormones signal the gonads
30
Why does puberty onset have gender differences?
- different receptor levels: varying brain maturation
31
What are primary sex characteristics vs secondary sex characteristics?
primary: - parts of the body directly involved in reproduction e.g testicles and ovaries - aren't functional until puberty secondary: - not necessary for reproduction (e.g acne, breast development etc)
32
What 3 reasons allow us to know that the limbic-hypothalamic system is responsible for the immaturity of the reproductive system before puberty?
1. immature gonads transplanted into an adult animal will mature immediately and demonstrate a normal reproductive function 2. the pituitary, taken from a prepubescent animal and transplanted into an adult animal, will regulate a normal reproductive function 3. alterations/lesions/trauma of the hypothalamus and specific areas in the limbic system will prevent the prepubertal animal to develop a normal reproductive function
33
What 5 things did Carnegie Corporation state regarding childhood brain development?
1. brain development that takes place during the prenatal period and in the first year of life is more rapid and extensive than previously realised 2. brain development is much more vulnerable to environmental influence than ever suspected 3. influence of early environment on brain development is long lasting 4. the environment affects not only the number of brain cells and number of connections among them, but also the 'wiring' 5. we have new scientific evidence for the negative impact of early stress on brain function
34
What are the cortical vs subcortical brain areas?
cortex: surface of brain - e.g motor cortex, frontal cortex - structures responsible for the 'higher level functions' e.g decision making - grey matter: no myelin subcortex: inner part of the brain - e.g basal ganglia, amygdala - structures responsible for emotion, memory and other 'basic functions' - white matter
35
How can we study brain changes?
- structural MRI - fMRI - diffusion tenser imaging
36
What NTs are adolescent brains more sensitive to?
dopamine: linked to motivation (risk taking) oxytocin: linked to social reward (peer pressure)
37
What brain changes happen during adolescence?
- grey matter density reduces - helps brain mature - grey matter decreases - white matter increases: myelination of neurons
38