Chapter 2 Flashcards

1
Q

When do the crowns of deciduous teeth develop?

A

~14th week gestation through 12 months of age

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2
Q

When do crowns of permanent dentition develop?

A

~6months to 15 years

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3
Q

What is the term for teeth that have enamel defects seen in the permanent teeth caused by periapical inflammation of the overlying deciduous tooth?

A

Turner’s hypoplasia

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4
Q

When is the critical period for children developing dental fluorosis?

A

2nd and 3rd year of life.

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5
Q

Congenital syphilis can cause what type of odontogenic malformations?

A

Hutchinson’s incisors. The teeth will have a straight-edge screwdriver shape and the molars will be “mulberry molars.”

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6
Q

What is the triad of congenital syphilis?

A

Hutchinson teeth, interstitial keratitis, and 8th nerve deafness.

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7
Q

How would you ID these teeth?

A

Hutchinson Incisors

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8
Q

How would you ID these teeth?

A

Mulberry Molars

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9
Q

What is attrition?

A

Wear of teeth due to tooth to tooth contact.

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10
Q

What is abrasion?

A

Wear of teeth due to pathologic wearing IE tooth brush.

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11
Q

What is demastication?

A

Wear of teeth due to attrition and abrasion. IE chewing tobacco.

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12
Q

What is erosion?

A

Wear of teeth due to a nonbacterial chemical process.

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13
Q

What is perimolysis?

A

Wear of teeth due to gastric secretions.

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14
Q

What is the term when teeth are fused by cementum?

A

Concrescence.

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15
Q

What is anodontia?

A

Lack of tooth development.

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16
Q

What is hypodontia?

A

Lack of one or more teeth.

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17
Q

What is oligodontia?

A

Lack of 6+ teeth.

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18
Q

List the most common teeth affect by hypodontia.

A

3rd molars, 2nd premolars, lateral incisors.

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19
Q

What is a paramolar?

A

Devloping 4th molar.

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20
Q

What is a mesiodens?

A

Supernumerary tooth inbetween maxillary incisors.

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21
Q

What is it called when two teeth switch position during eruption?

A

Transposition.

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22
Q

What are the most commonly impacted teeth?

A

Mand. 3rd molars, Max. 3rd molars, and Max. canine.

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23
Q

What is gemination?

A

Single enlarged tooth. Looks like it is splitting into two. Tooth count will be normal when it is counted as one tooth.

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24
Q

What is fusion?

A

Single enlarged tooth. Tooth count will be low when the tooth is counted as one.

25
Q

What is Riga fede disease?

A

Tramatic ulcers of soft tissue that may occur during breast feeding.

26
Q

What is this?

A

Riga fede disease

27
Q

What is this?

A

Talon Cusp

28
Q

What is dens evaginatus?

A

Elevation of the enamel located in the central groove of a tooth, often in the premolars. Very common in native americans and Inuits.

29
Q

What is this?

A

Dens evaginatus

30
Q

What is dens-in-dente?

A

Known as a tooth within a tooth. Deep surface invagination of the crown or root that is lined by enamel.

31
Q

What is the frequency of dens-in-dente?

A

Lateral incisors, central incisors, premolars, caninies, molars.

32
Q

What is this?

A

Dens-in-dente

33
Q

What is taurodontism?

A

An enlargement of the body and pulp chamber of a multi-rooted tooth.

34
Q

What are the three syndromes associated with taurodontism?

A

Kleinfelters Syndrome. Amelogenesis imperfect. Tricho-dento-osseous syndrome.

35
Q

What are the four aspects to tricho-dento-osseous syndrome?

A

Taurodontism, kinky hair, brittle nails, and osteosclerosis.

36
Q

What is this?

A

Hypercementosis

37
Q

What is hypercementosis?

A

non-neoplastic deposition of excessive cementum that is continuous with the normal radicular cementum.

38
Q

What disease is strongly associated with hypercementosis?

A

Paget’s Disease.

39
Q

What direction will a dilaceration usually present?

A

Distal

40
Q

What are the three forms of amelogenesis imperfecta?

A

Hypoplastic, hypocalcified, and hypomaturation.

41
Q

What are the characteristics of the hypoplastic form of amelogenesis imperfecta?

A

It is the inadequate deposition of enamel matrix. Teeth will typically be more brown/yellow in color due to lack of enamel.

42
Q

What are the characteristics of the hypomaturation form of amelogenesis imperfecta?

A

It is when the enamel matrix is laid down appropriately, but there is a defect in the maturation of the crystal structure. The enamel will be soft and chips away with the “snow capped” teeth.

43
Q

What are the characteristics of the hypocalcified form of amelogenesis imperfecta?

A

This is when the enamel is laid down appropriately, but there is no mineralization.

44
Q

What is causing this?

A

Hypoplastica amelogenesis imperfecta

45
Q

What is causing this?

A

Hypoplastic amelogenesis imperfecta.

46
Q

What is causing this?

A

Hypomaturation amelogenesis imperfecta.

47
Q

What is causing this?

A

Hypomaturation amelogenesis imperfecta

48
Q

What is causing this?

A

Hypocalcified amelogenesis imperfecta

49
Q

What is causing this?

A

Hypocalcified amelogenesis imperfecta

50
Q

What causes dentinogenesis imperfecta?

A

Mutation of the dentin sialophosphoprotein gene.

51
Q

What is dentinogenesis imperfecta called when it occurs with osteogenesis imperfecta?

How will patients eyes appear?

A

Osteogenesis imperfecta with opalescent teeth.

Blue sclera

52
Q

How will the radiographs look with dentinogenesis imperfecta?

A

Bulbous crowns, cervical constrictions, thin roots, and early obliteration of canals and chambers.

53
Q

What is causing this?

A

Dentinogenesis imperfecta

54
Q

What is causing this?

A

Dentinogenesis imperfecta

55
Q

What are the characteristics of dentin dysplasia Type I

A

The crowns will appear normal, but the teeth will be rootless.

56
Q

What are the characteristics of dentin dyplasia type II?

A

It is closely related to dentinogenesis imperfecta(cant tell the difference). Root length is normal. On radiograph, will have thistle shaped chamber and canal anatomy.

57
Q

What is the term for a localized, non-hereditary developmental abnormality of the teeth with extensive adverse effects on the formation of enamel, dentin, and pulp?

A

Regional odontodysplasia.

58
Q

What is causing this? (note: it is only happening in a single quadrant.)

A

Regional odontodysplasia.