9/11 - Movement Considerations II Flashcards

1
Q

Abnormal Muscle Tone

A

hypertonia, hypotonia, spasticity, rigidity

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2
Q

Paresis

A

Motor weakness/abnormal motor control

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3
Q

Dysmetria

A

lack of coordination of movement typified by the undershoot or overshoot of intended position

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4
Q

Hypometria

A

a condition of cerebellar dysfunction in which voluntary muscular movements tend to result in the movement of bodily parts (as the arm and hand) short of the intended goal

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5
Q

Hypermetria

A

a condition of cerebellar dysfunction in which voluntary muscular movements tend to result in the movement of bodily parts (as the arm and hand) beyond the intended goal

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6
Q

Involuntary movements

A

tremor, ballismus

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7
Q

Abnormal cerebellum

A

hypotonia, incoordination, intention tremor, impaired error correction affecting motor learning

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8
Q

co-activation

A

primitive/unrefined; normal in early learning

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9
Q

coordination

A

sequencing, timing, and grading of the activation of multiple muscle groups

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10
Q

incoordination

A

movements are awkward, uneven, inaccurate

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11
Q

Timing problems

A

problems initiating movement; slowed execution time; problems terminating movement

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12
Q

problems initiating movement

A

Time between the persons decision to move and the movement itself; Requires ability to overcome inertia, gravity, and antagonistic restraint; Deficits could be in ROM, force production, motivation, postural control

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13
Q

dysdiadochokinesia

A

problems terminating a movement

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14
Q

dexterity

A

ability to fractionate fine movement; depends on ability to move and respond to environment; most important in manipulative functions

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15
Q

coordination exam: non-equilibrium

A

finger to nose, heel to shin; quality of movement; subjectively graded 1-5

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16
Q

primary impairments of NM system

A

abnormal muscular tone; motor weakness/abnormal motor control; abnormal synergies; co-activation and coordination problems; involuntary movements

17
Q

Abnormal basal ganglia

A

hypokinetic disorders (bradykinesia, akinesia); rigidity; resting tremor; hyperkinetic disorders (choreiform, athetoid movements, dystonia)

18
Q

tremor

A

rhythmic and oscillatory

19
Q

dystonia

A

sustained muscle contractions, often twisting, repetitive, and abnormal

20
Q

associated movements

A

unintentional movements of one limb during the voluntary movement of another limb

21
Q

General interventions: Biomechanical

A

Sensory modalities: icing, vibration, approximation (weak evidence)

22
Q

General interventions: Neurophysiologic

A

Stretching, serial casting, casts, orthoses

23
Q

General interventions: Muscle Reeducation

A

Recruitment and type (fast or slow); Alignment issues; Fast and slow speeds
Accuracy; Intensity

24
Q

Hedman temporal sequence

A

initial conditions preparation initiation execution termination outcome

25
Abnormal muscle tone
hypertonia; hypotonia; spasticity; rigidity
26
Abnormal muscle tone treatment
pharm, surgical, physical
27
Pharma tone treatments - botox
Botulinum toxin Type A Injected directly into the muscle at the motor point Blocks the neuromuscular junction by reducing the release of Ach Initial effects in 3-7 days Wearing off in 4-6 months
28
Surgical tone treatments
``` Selective posterior rhizotomy Selected roots of L2-S2 Variable results Risk of reducing function Tendon lengthening, release or transfer Variable results Generally successful for improving ROM Risk of reducing muscle strength Selective posterior rhizotomy ```
29
PT tone treatments
``` Saladin Videos of Rhythmic rotation in sidelying Rotation, Counter Rotation in sidelying Rhythmic Rotation hooklying Rhythmic Rotation LEs on ball ```
30
Weakness and motor control interventions
Patient, family and entire team education Encourage functional use Consider stationary bicycle with vital sign monitoring Guarded ambulation if appropriate PREs of individual muscle groups Task specific training and strength training CVA ? Winstein, 2004 Bimanual Training FES ? Sullivan and Hedman, 2004
31
CVA secondary effects
Immobilization and shortened position causes atrophy Loss of sarcomeres Accumulation of intramuscular connective tissue Increased skeletal muscle fat content Degenerative changes at myotendinous junction Increase in mechanical spindle stimulation by stretch
32
Dodd systemic review
Systematic review that strength training programs improve strength in those with CP without increased spasticity
33
Ada systemic review
Systematic review in those with stroke Acute, very weak; acute weak; chronic very weak; chronic weak Strength training is effective early after stroke, It is not harmful (no increased spasticity) It may be worthwhile, but adequate strengthening interventions applied to several muscles of a limb and tests need to address this in future research e.g. 10 meter walk or 9 hole peg test
34
Sullivan and Hedman 2004
Sensory Stimulation 2 hours per day FES to wrist extensors during a lifting task 15 minutes, twice a day 18 weeks of home exercise and 6 home PT visits
35
Coordination Interventions
Repetitions of functional movements with increasing demands on accuracy Assist with knowledge of results and/or knowledge of performance Weight-bearing activities for LEs Repetitive non-functional movements
36
Timing problems - interventions
Give a time constraint to a task Metronome Time an activity Verbal, visual, manual feedback
37
Scaling problems - interventions
Practice grading force Faster movements tend to be easier Repetition of functional activity that will carry over
38
Treatment (intervention) of involuntary movements
Reduced effort/increase efficiency Weight bearing and approximation Distal fixation Limb is weighting-controversial