9 - CHF Flashcards

(39 cards)

1
Q

Acute heart failure can manifest in 2 ways:

A

1) Acute pulmonary edema

2) Acute MI–>cardiogenic shock

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2
Q

Increased pressure inside heart chambers cause contractility to increase or decrease? What is this law called?

A

Increase, Frank-Starling

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3
Q

Causes of HF:

A

1) MI
2) htn (this is only rapid in pre-eclampsia)
3) dysrhythmia
4) “itis” (pericard, myocard, endocard)
5) dissection
6) cardiac tamp
7) valve disorder
8) PE
9) cardiomyopathy
10) high output heart failure

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4
Q

What are some examples of high output heart failure?

A

1) anemia
2) Beriberi
3) Paget’s
4) thyrotoxicosis
5) AV fistula

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5
Q

In systolic heart failure, EF will be < _____%

A

40

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6
Q

What will the body do to compensate for low CO?

A

Increase Renin and Angiotensin to increase fluid volume

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7
Q

The heart has an inability to relax. This is called ______heart failure

A

diastolic

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8
Q

What are some causes of diastolic HF?

A

LVH, htn, CAD

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9
Q

Would you want to diurese pts with diastolic HF?

A

NO! The problem is with back up in preload, you need to increase fluid volume and give vasodilators

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10
Q

Patient has SOB, cough, weakness, orthopnea, PND, fatigue, JVD. Do you suspect right or left sided failure?

A

left (mixed b/c it’s also causing right heart failure)

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11
Q

What are the right sided heart failure symptoms

A

peripheral edema
RUQ pain
JVD
hepatojugular reflex

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12
Q

Most common cause of right heart failure

A

left HF

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13
Q

Labs to dx HF in ED:

A

LFT (end organ damage)
BNP
cardiac enzymes

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14
Q

How do you treat hypotensive HR?

A

Inotropes, fluids. worst prognosis

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15
Q

heart sound specific to CHF

A

S3

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16
Q

why can you expect liver tenderness/pain/megaly?

A

backflow and engorgement into liver from backup in vena cava

17
Q

what’s are some fun findings on CXR is common with CHF?

A

Kerley B lines and cephalization (vertical lines), big heart, pleural effusion, big vena cava

18
Q

What rule of thumb are you using for ventilation of CHF patients?

A

Mild-Mod: Conscious and cooperative–non-invasive

Mod-Severe: AMS or unstable–intubation

19
Q

Reduce preload with

A

nitrates and diuretics

20
Q

Reduce afterload with

A

ntg

ACE inhibitors

21
Q

Inoptropes

A

dobutamine, dopamine

22
Q

pain control

A

morphine which will also vasodilate

23
Q

dilation of arterial wall

24
Q

AAA that is concerning will be > ___cm

25
AAA that should be considered for repair is > ____cm
5 (also repair if pt is symptomatic)
26
What is the red flag complaints for AAA?
back pain + abd pain
27
What is the classic triad of AAA?
1) back pain + abd pain 2) pulsatile mass 3) hypotension
28
Cullen's sign is __________ and Grey-Turner sign is ________. Both are extremely alarming for what?
umbilical, flank, AAA rupture
29
Dx AAA
1) CXR is 65% sensitive, but NOT specific (can't r/o) 2) US is 90% sensitive but can't r/o 3) CT w/ is gold standard***
30
Tx for AAA?
1) rupture suspected? emergent repair 2) >5cm? elective repair 3) 3-5cm? close monitoring
31
what lethal problem mimics many other "chest pain" complaints?
dissection
32
what are the 3 layers of the aortic wall?
inside vessel out: intima, media, adventitia
33
Dissection dissects through what layers?
intima and media
34
most dissections are ascending, descending, or localized to the arch?
ascending 60% descending 30% arch 10%
35
Patient c/o back pain, diaphoresis, n/v, syncope, what is leading DDx
dissection, must rule this out first!!
36
Common exam findings in dissection
MI (dissects at coronary vessels), syncope, neuro deficit
37
EKG findings in dissection
normal, or may show inferior MI
38
Dx test of choice for dissection
CTA --will show aortic dilation, false lumen, etc
39
Tx of dissection
1) pain control--Fentanyl (only enough to take edge off, you need to monitor) 2) decrease pressure to 100 3) minimize shear pressure (lower HR <60)*** So #1 is BB--Esmolol that primarily targets HR 4) sx repair