A&P for Cardiac Flashcards

(28 cards)

1
Q

describe the pericardium

A

fibrous outer sac, serous parietal layer that minimizes friction during contraction, and visceral layer aka epicardium

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2
Q

what are the three layers of the heart

A

epi, myo, and endocardium

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3
Q

where is the tricuspid valve

A

between RA and RV

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4
Q

where is the pulmonary valve

A

between RV and Pulm Artery

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5
Q

where is the mitral valve

A

between the LA and LV

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6
Q

Without neural influence, the impulse from the SA node beats at _______ bpm (inherent rhythmicity)

A

100

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7
Q

what is the purpose of the AV node

A

delay A-V impulse until atria have had time to eject their contents into the ventricles

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8
Q

what is the inherent rhythmicity of the AVN

A

40-60 bpm

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9
Q

what is the p wave

A

atrial contraction

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10
Q

what is the QRS wave

A

ventricular contraction

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11
Q

what is the T wave

A

ventricular relaxation

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12
Q

how does the vagus nerve exert its influence over the heart?

A

parasympathetically - decreases HR

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13
Q

how do the upper thoracic nerves and rami of the sympathetic trunk exert their influence over the heart?

A

sympathetically - inc HR

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14
Q

the sympathetic receptors of the heart are primarily _________, and stimulation by ________ increases overall activity of the heart

A

beta-adrenergic; norepinephrine

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15
Q

the sympathetic receptors of the peripheral vasculature are primarily __________, and when activated they can cause systemic _________

A

alpha-adrenergic; vasoconstriction

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16
Q

describe the pressure-resistance relationship of the R side of the heart

A

low pressure system with little resistance from the pulm arteries

17
Q

describe the pressure-resistance relationship of the L side of the heart

A

high pressure system with high resistance from systemic circulation

18
Q

what is the primary determinant of oxygen delivery

A

cardiac output!

CO = SV x HR

19
Q

what are the three factors that influence stroke volume

A

preload (starling effect), afterload, and myocardial contractility

20
Q

how does preload influence stroke volume

A

increased preload = increased SV

21
Q

how does afterload influence stroke volume

A

increased afterload = decreased SV

22
Q

what is the frank-starling effect

A

increase in ventricular volume increases systolic force but excessive stretching in fluid overload impairs actin/myosin interaction thus impairing contractility

23
Q

blood pressure is a product of __________ and ____________

24
Q

what is pulse pressure and what can it tell you about heart disease

A

pulse pressure is the difference between SBP and DBP and increased pulse pressure is a risk factor for heart disease

25
where in the body do we regulate BP?
vasomotor center in the medulla which mediates sympathetic and vagal inputs
26
what is the short term regulator of BP
arterial baroreceptors - if BP increase, CO and TPR decrease
27
what is the long term regulator of BP
renin-angiotensin-aldosterone system - kidneys compensate for BP decrease by activating angiotensin 2 which leads to vasoconstriction
28
water retention in the kidneys is another long term regulatory mechanism in response to low blood volume, but how does it work?
low blood volume stimulates posterior pituitary ADH which leads to vasopressin upregulation