ABNORAMALITY OF AMNIOTIC FLOW Flashcards

(109 cards)

1
Q

Normal AFI

A

10 to 20cm

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2
Q

Low fluid
Increased fluid
AFI

A

5 to 10 Cm
20 to 24 Cm

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3
Q

Oligohydramnios

A

AFI < 5cm
With largest vertical pocket 2 Cm aless

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4
Q

Poly hydramnios

A

AFI > 24 cm
Largest vertical pocket 8cm
Or more
AF volume of > 2000 ml

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5
Q

Polyhydramnios sono orders to rule out

A

Multiple gestation
Molar pregnancy
Fetal size greater Than dates

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6
Q

Polyhydramnios clinical finding

A

Uterus greater than dates

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7
Q

Polyhydramnios associated with

A

Perinatal mortality
Morbidity
Maternal complications

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8
Q

Acute one set of hydramnius may

A

Be painful
Compress other organs and vascular structures
Hydronephrosis

Produce sob

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9
Q

Polyhydramnious associated with

A

CNS disorders
GI problems

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10
Q

Clinical finding fetal in Polyhydramnios

A

Hydrops
Skeletal anomalies
Renal disorders

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11
Q

Maternal condition with Polyhydramnios

A

Diabetes mellitus
Obesity
Rh

Anemia
Congestive cardiac faiure

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12
Q

Polyhydramnios in US

A

Freely floating fetus within swollen amniotic cavity
AFI =20cm or more
Accentuated feral anatomy
Single vertical pocket more than 8cm

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13
Q

When Oligohydramnios has poor prognosis

A

-Second trimester
It maternal AFT level is elevated

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14
Q

Maternal condition associated with

Oligohydramnios

A

Hypertension
pre eclampsia
Renal disease
Connective tissue disorders
Indomethacin
Cardiac disease

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15
Q

Reasons for Oligohydramnios

A

Fetal hypoxemia
→IUGR and oligohydramnios
Placental insufficiency→IUGR

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16
Q

Commoncomplication of posidate pregnancies

A

Oligohydramnios associated with diminished placental function
Redistribution of blood with brain-sparing effect

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17
Q

Cause of Oligohydramnios

A

Nonanamalous conditions
Fetal anomalous conditions

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18
Q

Nananomalous condition of Oligohydramnios

A

IUGR
PROM
Post date pregnancy
42 weeks
Sampling villus

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19
Q

Fetal anomalous

Oligohydramnios

A

Infantile polyclystic kidney disease
Renal agenesis
Pysplastic kidney
Chromosomal abnormality
Posterior urethral valve syndrome

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20
Q

Reason for rupture of chorinomniotic membrane under normal conditions

A

Normal cell death activation of enzymes and mechanical forces

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21
Q

Condition when membrane rupture abnormally resulting gir loss of AF and oLigohydramnious

A

PROM premature
PPROM_ preterm
SPROM _spontaneous

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22
Q

Clinical finding in ROM

A

With sudden gush or leaving of fluid
Nitrazine paper and Fern test
Checked for cervical dilation and leaking of fluid with coughingor or fundal pressure

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23
Q

Prognosis of fetus affected by PROM depends on

A

FeTal GA
Fetal status
Ability to control uterine contractions

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24
Q

PROM associated with

A

Preterm delivery
Fetal death
Neaonatal respiratory distress
Prolapsed umbilical cord
Chorioamnionitis
Placental abruption

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25
PROM is associated with
Placenta abruption
26
Amniotic band syndrome
Associated with fetal membranes abnormality
27
Common nonrecurrent cause fetal of various malformations involving limbs cranial facial region trunk
Amniotic band syndrome
28
Other names for disruption of feral tissue due amniotic bands
ADAM COMPLEX_ amniotic deformities, Adhesion mutilation Ammotic band sequence Aberrant tissue bands Congenital constricting bands
29
Entrapment of feral parts by bands may cause
Lymphedema Amputations Slash defects in none embryologic distributions
30
Protect fetus from contact with the chorion
Amnion
31
Clinical finding of amniotic band syndrome
Represent milder form of limb-body wall complex May predicted by amniotic bands that entangle or amputate fetal parts
32
Common US finding in amniotic band syndrome
Facial cleft Asymmetric encephalóceles Amputation defects of extremities Clubfootdeformities
33
Ammoniotic bands
-siring like bands Fibrous strands
34
Sonographic finding amniotic band
Echogenic band floating in The AF The echogenic band may attach to The wan of uterus or gestational sac Following the band closely with real-time scan can be observed the band is attached to Tue uterin wall and constriction is placed on the fetus
35
Amniotic sheets
Shelves or folds identified as echogenic bands crossing through amniotic cavity Thicker than Bands syndrome
36
Most likely signify uterine synechiae
Amniotic sheets
37
Cause of amniotic sheets
Uterine scars from previous instrumentation used in uterus Cesarean section Episodes of endemetritis
38
Risk factors for uterine scars and sheets
-history of Endometrial DC Intrauterine infection Endometritis Removal fibroids or polyps C section
39
Synechia associated with
Infertility Miscarriage
40
Ashermans' syndrome
Formation of scar tissue in the uterine cavity
41
US in amniotic sheets
Fine echo-dense line in uterine cavity separated from uterine wall by echo-lucent space Membrane may completely surround fetus or be freely mobile in amniotic cavity
42
Amniotic sheets on US
Extending from me side of uterus to other side oblique across uterus Or multiple echogenic lines
43
Hydrops fetalis
A life threatening condition abnormal amount of fluid accumulation in two armore body areas of fetus
44
Most often sides of fluid accumulation in hydros
Abdomen Around the heart Lungs Under skin
45
Other symptoms of hydropes condition include
Polyhydramnioes Thickening of the placenta Placenta edema Enlarged umbilical cord Enlarged liver and spleen
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Accumulation of fluid aredema can represented by
Pleural effusion Cardiac effusion Ascities Skinedtima Anarsarca
47
Types sofhydrops
Immune Nonimmune
48
More common type of hydrops
Non-immune An diseases arcomplicarians that interferes with now the fetal body manages fluid balance.
49
Fetal hydropshighly associated with
mortality
50
US hydros
Presence of abnormal collection of fluid
51
Ascities and psedoascites
Are choir fluid surrounding abdominl, pelvic organs and umbilical cord Mistaken normal hypoechoic abdominal musculature for ascities
52
Skin edema in hydros
Î skin tnickness around the skull, neck, extremities or abdomen
53
Soft tissue thickening for diagnosis hydros
> 5-6mm
54
Anasarca
'skin edema is massive encasing most of the body
55
Pericardial effusion in hydrops
Excessive anechoic fluid in pericardial cavity More than 2 mm
56
Placental edema in hydrops
Thickening of placenta more than 4 -4.5cm in anterior posterior diameter
57
Non immune hydrops reason
Cardiac insufficiency one of the most common causes From cardiac anomalies Tumors or arrhythmias like tachycardia
58
One of the Most common causes of , nonimmune hydros
Cardiac insufficiency Or hyperoteinuria Structural lymphatic obstruction Decreased venous réturn to heart
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Anomalies may present in non immune hydrops
Feral tuners heart or liver Cardiac anomalies Cynic adenomatoid malformation of lung Charinargiama of placenta
60
Which abnormalities should be documented in hydros
Due to relationship with NIHF Trisomy 21 45x Feral infections TORCH
61
Clinical Findings • Fetal hydrops, skeletal anomalies, some renal disorders may be associated with hydramnios. • Maternal conditions associated with polyhydramnios • Diabetes mellitus • Obesity
• Fetal hydrops, skeletal anomalies, some renal disorders may be associated with hydramnios. • Maternal conditions associated with polyhydramnios • Diabetes mellitus • Obesity • Rh incompatibility • Anemia
62
Sonographic Findings
• Freely floating fetus within swollen amniotic cavity • Accentuated fetal anatomy (increased AF improves image resolution) • AFI = 20 cm or greater
63
Sonographic Findings
Fetus presenting at 29 weeks gestation with duodenal atresia. The single vertical pocket measurement of 13.71 cm suggests polyhydramnios. An AFI of 32.36 cm supports the findings. The + indicates fetal head.
64
Etiology
• Development of oligohydramnios may be attributed to: • Congenital anomalies • IUGR • Post term pregnancies • Rupture of membranes (ROM) * latrogenesis
65
Second trimester oligohydramnios often has poor prognosis,
• Second trimester oligohydramnios often has poor prognosis, especially if maternal serum alpha-fetoprotein level also elevated • Maternal conditions associated with oligohydramnios • Hypertension • Preeclampsia • Chronic cardiac or renal disease • Connective tissue disorders • Patients receiving indomethacin
66
Clinical Findings
• Fetal hypoxemia may produce growth restriction and oligohydramnios. • Four-fold increased risk of growth delay when oligohydramnios present • Doppler evaluation of growth-restricted fetus shows abnormal umbilical flow in patients with oligohydramnios.
67
Clinical Findings
• Placental insufficiency may cause IUGR associated with oligohydramnios. • Produces redistribution of fetal blood flow away from kidneys and toward brain to counteract hypoxia • Results in decreased urine output, which decreases fluid volume
68
Clinical Findings
• Post term pregnancy defined as gestational age of 42 weeks or more • Oligohydramnios is common complication of postdate pregnancies. • Is associated with diminished placental function and arterial redistribution of fetal blood flow with brain-sparing effect
69
Post term pregnancy defined
as gestational age of 42 weeks or more
70
• Medications associated with oligohydramnios
• Medications associated with oligohydramnios • Nonsteroidal antiinflammatory drugs • Angiotensin-converting enzyme inhibitors • Calcium channel blockers • Nitrous oxide
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Causes of Oligohydramnios Causes of Oligohydramnios Nonanomalous Conditions
• IUGR • Premature rupture of membranes • Postdate pregnancy (42 weeks) • Chorionic villus sampling
72
Causes of Oligohydramnios Fetal Anomalous Conditions
Fetal Anomalous Conditions • Infantile polycystic kidney disease • Renal agenesis • Posterior urethral valve syndrome • Dysplastic kidneys • Chromosomal abnormalities
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Fetal Membranes
The amnion divides the amniotic and chorionic cavities and at 10 weeks gestation.
74
Ruptured Fetal Membranes
• Membranes normally rupture after onset of labor. * Premature rupture of membranes (PROM), preterm premature rupture of membranes (PROM), and spontaneous rupture of membranes (SPROM) describe conditions in which membranes rupture ("water breaks" abnormally, resulting in loss of AF and/or oligohydramnios. • Multiple underlying pathologic processes associated with abnormal ruptured membranes
75
Ruptured Fetal Membranes
• Clinical findings • Patients suspected to have ROM present clinically with sudden gush or leaking of fluid. Nitrazine paper and fern test used as screening test to determine presence of AF in vaginal secretions • Patient is checked for cervical dilation and for leaking of fluid with coughing or fundal pressure.
76
Patient is checked for cervical dilation and for leaking
Patient is checked for cervical dilation and for leaking of fluid with coughing or fundal pressure.
77
• Abnormal ROM associated with:
• Prognosis of fetus affected by abnormal ruptured membranes depends on fetal GA, fetal status, and ability to control uterine contractions • Abnormal ROM associated with: Preterm delivery Fetal and neonatal death Neonatal respiratory distress Prolapsed umbilical cord Chorioamnionitis* Placental abruption
78
•SONOGRAPHIC FINDING
Role of sonography is to document integrity of placenta, fetal size, AF volume, fetal well-being, to perform fetal Doppler studies. • Common for patients to be evaluated every day assess fetal well-being and fluid volumes
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• Evaluate the integrity of the placenta, fetal growth, and biophysical profile. •........ is associated with ROM.
• Evaluate the integrity of the placenta, fetal growt! and biophysical profile. • Placenta abruption is associated with ROM.
80
Coanechoic fetal cord was not misrepresented as fluid.
Patient presents at 30 weeks gestation with ROM.AFI reveals severe oligohydramnios. Color Doppler was used to ensure that the anechoic fetal cord was not misrepresented as fluid.
81
지/ Amniotic Band Syndrome
• Is associated with abnormality in fetal membranes • Is a common, nonrecurrent cause of various fetal malformations involving limbs, craniofacial region, trunk • Synonyms used to describe disruption of fetal tissu due to the presence of amniotic bands: ADAM complex (amniotic deformities, adhesion, mutilation) • Amniotic band sequence • Aberrant tissue bands Congenital constricting bands
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• ADAM complex) •
(amniotic deformities, adhesion, mutilation
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• Synonyms used to describe disruption of fetal tissue due to the presence of amniotic bands:
• ADAM complex (amniotic deformities, adhesion, mutilation) • Amniotic band sequence • Aberrant tissue bands • Congenital constricting bands
84
Amniotic Band Syndrome • May represent milder form of limb-body wall complex • May be predicted by amniotic bands that entangle or amputate fetal parts • Common findings:
facial clefts, asymmetric encephaloceles, constriction or amputation defects of extremities, clubfoot deformities
85
Sonographic Findings
Echogenic band floating in the AF. The echogenic band was attached to the wall of the gestational sac. Copyright
86
Sonographic Findings
Soft tissue edema is seen in the forearm, where the band is constricting the soft tissue.
87
Amniotic Band Syndrome
• May represent milder form of limb-body wall complex
88
Amniotic Sheets
•Amniotic sheets, shelves, or folds identified as echogenic, nonfloating bands crossing through amniotic cavity • Are thicker than bands associated with amniotic band syndrome • Do not cause fetal malformations • Most likely signify uterine synechiae
89
Amniotic Sheets
Etiology • Visualization of amniotic sheets believed to be caused by: • Uterine scars from previous instrumentation used in uterus • Cesarean section • Episodes of endometritis
90
Amniotic Sheets Clinical findings
Clinical findings • Patients with history of endometrial D&C, intrauterine infections, endometritis, removal of fibroids or endometrial polyps, or prior cesarean section are at risk for developing uterine scars. • Synechiae associated with infertility and miscarriages * Patients who present with uterine synechiae and infertility often diagnosed with Asherman's syndrome (Asherman syndrome is the formation of scar tissue in the uterine cavity).
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Synechiae
Synechiae associated with infertility and miscarriages
92
Amniotic Sheets
Sonographic findings • May show fine echo-dense line in uterine cavity separated from uterine wall by echo-lucent space • Membrane may either completely surround fetus or be freely mobile in amniotic cavity • Can appear anywhere in uterine or cervical cavity • Are seen extending from one side of uterus to other, oblique across uterus or as multiple echogenic lines
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Hydrops
•Other fetal findings identified with hydrops • Enlarged umbilical cord • Polyhydramnios • Placental edema • Enlarged liver and spleen • In many cases, fetal hydrops highly associated with mortality
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• Do not to mistake normal hypochoic abdominal musculature).
for ascites (pseudoascites
95
Hydrops
Skin edema can be seen as increased skin thickening around the skull, neck, extremities, or abdomen. • Measurement of >5 to 6 mm for soft tissue thickness is used for didgnosis in some reports. • When skin edema is massive, encasing most of the body, the term anasarca is used.
96
Normally, a small amount of fluid is noted in this cavity, particularly in the apex. • pericardial effusion considered
If fluid collection measures >2 mm,
97
• Placental edema
can be identified as a thickened placenta measuring >4 to 4.5 cm in true anterior-posterior diameter.
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Oligohydramnios in the third trimester is most likely a result of: a. duodenal atresia b. diaphragmatic hernia c. infantile polycystic renal disease d. cystic adenomatoid malformation
infantile polycystic renal disease
99
Which portion of the biophysical profile study is a chronic marker of fetal hypoxia? a. fetal tone b. fetal movement c. amniotic fluid volume d. maturity of the placenta maturity of the placenta
c. amniotic fluid volume Amniotic fluid volume is a chronic marker of fetal hypoxia. Acute markers of fetal hypoxia in- clude fetal breathing movement, fetal tone, nonstress test, and fetal movement.
100
When measuring amniotic fluid volume,
When measuring amniotic fluid volume, the transducer must re- main perpendicular to the maternal coronal plane and parallel to the ma- ternal sagittal plane.
101
Doppler of the umbilical artery evaluates fetal well-being using the: a. resistive index b. pulsatility index c. peak systolic velocity d. systolic–diastolic ratio
The systolic-to-diastolic ratio of the umbilical artery can evaluate fetal well-being after 30 weeks’ gestation. A ratio greater than 3.0 is abnormal. Absence or reversal of the diastolic component is also abnormal.
102
The single most sensitive indicator of intrauterine growth restriction is: a. femur length b. head circumference c. abdominal circumference d. head circumference-to-abdominal circumference ratio
The abdominal circumference is the single most sensitive indicator
103
Which technique is both valid and reproducible when assessing amniotic fluid volume? a. uterine volume b. amniotic fluid index c. single vertical pocket d. subjective assessment
Of all the techniques to assess amniotic fluid volume, the amniotic fluid index (AFI) is both valid and reproducible. 31. d. Maternal diabetes can resul
104
Intrauterine growth restriction is defined as a fetal weight: a. below the 5th percentile for gestational age b. below the 10th percentile for gestational age c. at or below the 5th percentile for gestational age d. at or below the 10th percentile for gestational age
c. at or below the 5th percentile for gestational age d. at or below the 10th percentile for gestational age
105
A transverse fetal position in the late third trimes- ter of pregnancy is most likely associated with: a. macrosomia b. placenta previa c. polyhydramnios d. intrauterine growth restriction
b. placenta previa
106
Which of the following fetal positions is at most risk for cord prolapse? a. oblique b. transverse c. frank breech d. incomplete breech
Incomplete or footling breech places the fetal foot as the present- ing part and places the greatest risk for cord prolapse.
107
A fetus presents with multicystic dysplastic renal disease. The amniotic fluid volume is expected to appear: a. below normal b. slightly lower than normal c. slightly higher than normal *d. normal
Multicystic dysplastic kidney disease is a unilateral disease. The normal contralateral kidney will continue urinary function, allow
108
To demonstrate the umbilical cord insertion into the fetal abdomen, one would look: A. Superior to the fetal kidneys B. Superior to the fetal bladder C. Posterior to the fetal stomach D. Posterior to the umbilical vein E. At the level of the adrenal glands
B
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138. Which long bone is LEAST likely to be affected by intrauterine growth restriction? A. Femur B. Humerus C. Clavicle D. Tibia E. Radius E 139. You are measuring abdominal circumference. You choose to do so at the level
C