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Cardiopulmonary PT > Abnormal EKG > Flashcards

Abnormal EKG Flashcards

(54 cards)

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1
Q

What leads go with the lateral, anterior, septal, and posterior part of the heart?

A

Lateral: I, aVL, V5, V6
Anterior: V3, V4
Septal: V1, V2
Inferior: II, III, aVF

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2
Q

You see a EKG with a normal looking rhythm, and you notice there is a P wave for every QRS and a T wave is present. What is the rhythm?

A

Normal sinus

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3
Q

What is sinus arrhythmia?

A

THe QRS complexes are not equidistant from each other.

Will have some variability because when we breathe in our HR increases and when we breathe out our HR decreases. But too much variability is a bad thing

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4
Q

Who has sinus bradycardia and what are the symptoms?

A

Normal in athletes, beta blockers cause this, decreased function of the SA node causes this.

Asymptomatic unless pathological condition exists (presence of 2 or 3 degree heart block)

Dizziness, syncope, angina

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5
Q

T/F: low blood flow is a risk factor for dementia or Alzheimer’s?

A

True

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6
Q

What are causes of sinus tachycardia?

A 
Fear
Anxiety
Caffeine
Nicotine
Amphetamines
Demands of O2 are higher (exercise, infection, MI, hemorrhage)
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7
Q

What are causes and symptoms of sinus tach?

A

Generally benign
SA node automaticity increased
Patients asymptomatic

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8
Q

T/F: PAC is a block in the conduction of impulse from SA node to atria?

A

False: sinus exit block

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9
Q

On an EKG you see a normal rate (~70) and you notice that after a T wave there is a pause equal to 2 complete cardiac cycles before the next P wave. What is the rhythm?

A

Sinus exit block

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10
Q

Do patient with sinus exit block have symptoms?

A

These blocks are less than a second so they are usually asymptomatic. BUT if they complain of SOA or other symptoms you may need to contact someone

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11
Q

What are causes of PAC? Symptoms? Possible consequences?

A

Causes: emotional stress, nicotine, caffeine, alcohol, infection, hypoxemia, MI, atrial damage

Symptoms: asymptomatic and if frequency is low no treatment required and pt should be able to participate in activity

If frequency increases a tach or a fib may result.

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12
Q

What is a rhythm where the P wave comes quick after the T wave and is a little more peaked?

A

Premature atrial contraction: ectopic focus in either atria initiates an impulse before the next impulse initiated by the SA node

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13
Q

What are some causes for atrial tachycardia?

A

PAC, pulmonary HTN, altered pH, COPD

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14
Q

What are symptoms of a tach?

A

Q compromised if prolonged: dizziness, fatigue, SOB

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15
Q

What does a tach look like on an EKG?

A

R-R very close together.
HR may be 100-200 bpm
Hard to make out P wave (blends with T)

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16
Q

What are causes of A flutter?

A

Mitral valve disease, CAD, infarction, stress, renal failure, pericarditis, rheumatic heart disease, MI

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17
Q

What are symptoms of A flutter?

A

Unless ventricular rate becomes too high Q is not compromised.
So patients are asymptomatic.
In their chart you may see: uncontrolled, controlled, with symptoms, w/o symptoms (uncontrolled means R waves are coming faster and rate is not as normal)

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18
Q

What are characteristics of A flutter?

A

Saw tooth P waves
R-R interval is normal
What’s happening is there is a rapid firing of ectopic source in atria and the AV node is blocking the signal from getting to the ventricles.

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19
Q

What is A fib?

A

Erratic quivering of atria: multiple ectopic foci, no true depolarization, AV node acts to control ventricles

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20
Q

What are causes of A fib?

A

Advanced age, CHF, ischemia or infarction, cardiomyopathy, digoxin toxicity, drug use, stress/pain, renal failure

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21
Q

WHat are consequences of Afib?

A

puts patient at risk for stroke because blood is sitting in the atria and it gets sticky and clotted. The clot will be at risk for moving and traveling to the brain

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22
Q

What are problems associated with Afib?

Study These Flashcards
A

No atrial kick: accounts for 30% of Q so in a fib Q will be compromised

If rate is 100 bpm at rest: monitor assessed during all activities and engaged with caution (likely to have Q decompensation)

23
Q

T/F: in both a fib and a flutter it’s easy to make out the P wave?

Study These Flashcards
A

False: in a fib it’s hard to make out the P wave

24
Q

You are looking at an EKG and you see that there is an immediate QRS after a T wave and it is missing a P wave. What is this rhythm?

Study These Flashcards
A

Premature junctional contraction

25
You are looking at an EKG you see QRS complexes but no P waves and you notice the rate is very slow. What is the likely rhythm?
Junctional escape rhythm
26
What are causes of junctional escape rhythm and what are symptoms?
Causes: increased vagal tone, digoxin toxicity, infarction, severe ischemia to conduction system (R CAD) Decreased HR= low Q, results in sx of dizziness, fatigue. **will have symptoms with mobilization** Will be candidates for pacemaker or meds to increase HR
27
What happens to SA node in junctional escape rhythm?
Not functioning properly. | AV junction takes over and ventricular rate is between 40-60 bpm
28
What are causes of PVCs?
Caffeine, nicotine, stress, overexertion, electrolyte imbalance, ischemia, cardiac disease, CHF, over distension of ventricle, acute infarction, irritation of myocardium or vessels, chronic lung disease and hypoxemia
29
What are symptoms of PVCs?
May have symptoms if more frequent or more serious in nature. Decreased Q for that heart beat. Patient activity can be compromised. Exercise should be monitored if patient having isolated or couplet PVC
30
What is sequelae if there are increased frequency of PVCs?
``` Filling time is decreased Decreased pre load Decreased SV Decreased Q (dizzy, SOB) Can lead to v tach ```
31
What are the types of PVC?
``` Unifocal: one part of ventricle Multifocal: coming from two different places in ventricle Couplet Run Bigeminy Trigeminy ```
32
What are causes of V tach?
Ischemia, acute infarction, hypertensive heart disease, reaction to meds
33
What are symptoms of V tach?
Q and BP diminished | Symptoms: light headed, syncope, weak thread pulse, disorientation
34
What is V fib?
Sequel to v tach Life threatening Emergency Shockable rhythm
35
What is a idioventricular rhythm?
No p wave wide QRS Ventricle is pacing: 20-40 bpm Will convert to systole because the rate is not sustainable for long due to decreased cardiac output
36
What are causes of first degree heart block?
CAD, infarction, reaction to digoxin
37
What are symptoms of first degree heart block?
asymptomatic unless rate
38
What are causes and symptoms of second degree heart block type 1?
Cause: right CAD or infarction, digoxin toxicity, excessive beta blocker Benign arrhythmia; generally asymptomatic (no sx and sufficient Q)
39
What are causes and symptoms of second degree heart block type 2?
Cause: MI (especially LAD), ischemia/infarction of AV node, digoxin toxicity Symptomatic when HR is low and Q compromised
40
What are causes and symptoms of third degree heart block?
Cause: acute MI, digoxin toxicity, degeneration of conduction system Sx: if slow HR, Q is diminished; pt may complain of dizziness, SOB, possibly angina Medical emergency
41
What does a pacemaker rhythm look like?
A properly functioning pacemaker will show a sharp spike, followed immediately by depolarization of the chamber it is intended to pace. This rhythm strip shows ventricular pacemaker capturing every beat. Note absence of mappable P waves and sharp spikes followed immediately by wide QRS complexes.
42
How do you determine LVH?
Add mm of height of S wave in V1 or V2 with R wave in V5 or V6 If sum is >35 mm OR/AND R wave in aVL is >11 mm you have LVH
43
How do you determine if you have RVH?
If R wave is bigger than S wave in V1 and gets smaller through precordial leads you have RVH Remember that normally the R wave increases from V1-V6
44
T/F: From V1-V6 the S wave gets smaller?
True
45
What signifies LAE?
Broad p wave in lead II (sometimes seen in I and aVL) OR/AND Diphasic p wave in V1 with larger terminal component
46
What signifies RAE?
Peaked pointy p wave in lead II (sometimes III, V1, aVF) which is greater than or equal to 2.5mm
47
what are injury patterns seen in EKG?
Injury: elevated ST segments greater than 1 mm Ischemia: symmetrical T wave inversion >3 mm in two adjacent leads Infarct: Q waves that are at least 0.04 seconds wide or 1/3 of entire QRS in height
48
What are changes in EKG at each stage of MI?
Acute stage: ST elevation Hours: ST elevation, decreased R wave, Q wave begins Day 1-2: T wave inversion, Q wave deeper Days later: ST normalizes, T wave inverted Weeks: ST and T normal, Q wave persists
49
What are locations onf the heart of the leads?
II, III, aVF: inferior LV, RA, LA I, aVL: high lateral LV V1, V2: RV, RA, LA, anteroseptal LV V2-V4: anterior and apical LV V4-V6: anterolateral LV
50
Where will you see the BBB?
Right BBB in V1, V2 Left BB in V5, V6 Right BBB: more M shaped Left BBB: bat man hat
51
Why are there two R's in a BBB?
There is a delay with a signal so one side will fire before another causing two waves. So in left BBB: R is the right ventricle firing and R' is the left ventricle firing (flip this if there was a right BBB)
52
What conditions are seen our acute cardiac patient population?
``` Cardiopulonary insufficiency CP sequelae of surgery CP sequelae of restricted mobility Cardiac conditions: post MI, CABG Deconditioning/bed rest ```
53
What is EBP for cardiac patients and exercise (chronic heart failure and CV disease)?
Guidelines similar to older adults: FITT Study had participants do five 40 min ex sessions per week at 60-70% HRR. They found exercise was well tolerated, they increased distance walked, VO2 peak, and QOL. But they found long term adherence was a barrier
54
What is clinical decision making model for cardiac patients?
1) Identify all factors contributing to deficits in O2 transport 2) Determine whether mobilization and ex are indicated and which specifically address O2 deficits. 3) Match appropriate mobilization/ex to patient's O2 transport capability 4) Set intensity within therapeutic and safe limits of pt O2 transport. 5) Post surgical- combine upright position with thoracic mobility; active or AAROM, breathing control 6) Set duration to pt response vs. time 7) Increase intensity, duration or both as pt tolerates w/o distress 8) Continue to progress until functional status improves or until threat of O2 transport is minimized

Cardiopulmonary PT (19 decks)

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