Abnormal Psych Chapter 4 Flashcards
(198 cards)
What is fear?
Present-oriented
Immediate fight-or-flight response to danger or threat
Strong avoidance/escapist tendencies
Abrupt activation of sympathetic nervous system
How is anxiety different from fear?
Future-oriented
Worry about future danger or misfortune
Expectations of negative events, or that positive events won’t happen
Bodily tension
What are the areas of the brain that regulate the fear system?
Prefrontal cortex - cognitions
Amygdala - emotional reactivity, also fear conditioning
Hippocampus - memory
Anxiety disorders
Pervasive and persistent symptoms of anxiety and fear
Excessive avoidance and escapist tendencies
Symptoms and avoidance cause clinically significant distress and impairment
What is the pattern of activity of different brain networks in anxiety disorders?
Overactivity in salience, underactivity in executive control and intrinsic networks
Generalized Anxiety Disorder (GAD)
Excessive anxiety and worry
Difficulty in controlling the worry
Restlessness or feeling keyed-up or on edge
Easily fatigued
Difficulty concentrating
Irritability
Muscle tension and sleep distrubance
What percent of the general population meet diagnostic criteria for GAD?
4%
What is the gender distribution of GAD?
Females outnumber males approximately 2:1
Psychodynamic GAD Theory
Neurotic anxiety - id based
Realistic anxiety - ego based
Moral anxiety - superego based
Humanistic GAD Theory
Conditions of worth
Lack of authenticity, incongruence
Existential GAD Theory
Realities of the human condition: freedom, responsibility, loneliness, certainty of death
Existential angst
Cognitive GAD Theory
Focus on threat, lack of control
Metacognitive theory (or “metaworry”, when you worry about worrying)
Avoidance theory: worrying reduces bodily arousal
Biological GAD Theory
Reduced GABA activity
Less inhibition of areas involved in fear response
Variety of mechanisms
GABA receptors dense in PFC, amygdala, hippocampus
Antianxiety drugs increase GABA; serotonin also involved
Treatments for GAD
Cognitive-behavioral approaches often most effective
Traditional cognitive approaches
Newer approaches e.g., mindfulness
Biological treatments: anti-anxiety drugs (focus on increasing GABA), relaxation training, biofeedback
What is a panic attack?
Abrupt experience of intense fear or discomfort
Accompanied by several physical symptoms (e.g., breathlessness, chest pain)
Often presents as heart attack
Panic disorder
Experience of unexpected panic attack
Develop anxiety, worry, or fear about having another attack or its implications
Symptoms and concern about another attack persists for 1 month or more
Could also experience agoraphobia
Agoraphobia
Fear or avoidance of situations/events associated with panic
Biological component of panic disorder
Neurotransmitters:
Fight-or-flight response poorly regulated
Dysregulation of norepinephrine, especially in locus ceruleus (part of pons in brainstem)
Affects limbic system, stress response
What percent of the general population meet diagnostic criteria for panic disorder?
3.5%
What is the gender distribution of panic disorder?
2/3 are female
Onset of panic disorder
Onset is often acute, beginning betwen 25 and 29 years of age
Onset of GAD
Onset is often insidious, beginning in early adulthood
Tendency to be anxious runs in families
How does genetics relate to panic disorder?
Runs in families to some extent
Parent/child and twin studies
Agoraphobia also has genetic component
Biological treatments for panic disorder
Tricyclic antidepressants: increase levels of norepinephrine, serotonin, and other neurotransmitters; imipramine preferred
Selective serotonin reuptake inhibitors: increase levels of serotonin; prozac and paxil currently preferred treatment
Benzodiazephines (e.g., valium, xanax): suppress the central nervous system and influence functioning in the GABA, norepinephrine, and serotonin neurotransmitter systems; high relapse rate when drug treatments are discontinued