ACLS Flashcards

1
Q

which rhythms are shockable

A

VF, pVT

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2
Q

which rhythms are not shockable

A

asystole, PEA

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3
Q

high quality CPR

A

rate 100-120 compressions/min, depth at least 2 inches, allow chest recoil, minimize interruptions

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4
Q

what are the ABCs

A

airway, breathing, circulation

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5
Q

which line is preferred

A

central line, meds reach the heart faster

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6
Q

intraosseous

A

used when IV access not available, treat as a central line

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7
Q

endotracheal

A

last line option for vascular access, absorption occurs in alveolar capillaries, doses often 2-2.5x higher than IV

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8
Q

administer _____ after each med administered

A

NS flush 10-20 mL

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9
Q

how does epinephrine work

A

increases coronary perfusion pressure

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10
Q

negative side effects of epinephrine

A

tachycardia, dysrhythmias, increased myocardial oxygen demand

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11
Q

IV/IO dose for epinephrine

A

1 mg q3-5 min

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12
Q

endotracheal dose for epinephrine

A

2-2.5 mg q 3-5 min

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13
Q

how does amiodarone work

A

antidysrhythmic properties through inhibition of sodium, potassium and calcium channels and alpha/beta adrenergic receptors

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14
Q

IV/IO dose for amiodarone administration

A

300 mg push once followed by 150 mg push

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15
Q

endotracheal dose for amiodarone

A

can’t be given via endotracheal tube.

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16
Q

pearl for amiodarone

A

only give IV push if patient is pulseless, IV push can cause hypotension and bradycardia in patients with a pulse

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17
Q

what is one alternative to amiodarone in ACLS algorithm

A

lidocaine

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18
Q

how does lidocaine work

A

antidysrhythmic properties through inhibition of Na channels

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19
Q

IV/IO dose for lidocaine

A

1-1.5 mg/kg once then 0.5-0.75 mg/kg if needed. may repeat for maximum total dose of 3 mg/kg

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20
Q

endotracheal dose for lidocaine

A

2-4 mg/kg once then 1-2 mg/kg if needed

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21
Q

what are the reversible causes

A

H’s and T’s

22
Q

H’s

A

hypovolemia, hypoxia, hydrogen ion (acidosis), hypo/hyperkalemia, hypothermia

23
Q

T’s

A

tension pneumothorax, tamponade (cardiac), toxins, thrombosis (pulmonary), thrombosis (cardiac)

24
Q

magnesium indication

A

cardiac arrest due to torsades or hypomagnesemia

25
magnesium mechanism
inhibits calcium channels, suppressing early abnormal depolarizations responsible for arrhythmias
26
magnesium dose
1-2 g diluted in 10 mL D5W or NS given over 5-20 minutes. rapid administration can cause hypotension in patients with a pulse
27
sodium bicarbonate indication
acidosis or hyperkalemia
28
mechanism of sodium bicarbonate
neutralizes acidosis; pushes K into cells via H/K exchange
29
dose of sodium bicarbonate for acidosis
1 mEq/kg/dose IV/IO; repeat doses as needed guided by arterial blood gas
30
dose of sodium bicarbonate for hyperkalemia
50 mEq once IVP/IO
31
naloxone indication
suspected opioid overdose
32
naloxone mechanism
opioid receptor antagonist
33
naloxone dose IV/IM/SQ
0.4-2 mg
34
naloxone dose intranasal
4-8 mg
35
naloxone dose endotracheal tube
0.8-4 mg
36
when should you give atropine for adult bradycardia
persistent bradyarrhythmia causing hypotension, acutely altered mental status, signs of shock, ischemic chest discomfort, acute heart failure
37
what meds to give for adult bradycardia if atropine is ineffective
dopamine or epinephrine
38
atropine mechanism
inhibits muscarinic acetylcholine receptors, increasing automaticity of SA & AV nodal cells
39
atropine dose
0.5-1 mg q3-5 min, max total dose 3 mg
40
dopamine mechanism
dopaminergic & beta-1 adrenergic receptor agonist
41
dopamine dose
5-20 mcg/kg/min; titrate by 5 mcg/kg/min every 2 minutes
42
epinephrine mechanism of action
beta1 receptor activation results in increased inotropy and chronotropy
43
epinephrine dose
initial dose 2-10 mcg/min, max 40 mcg/min
44
treatments for adult tachycardia
atropine (nonpharm: vagal maneuvers)
45
what to do if persistent tachycardia causing: hypotension, acutely altered mental status, signs of shock, ischemic chest discomfort, acute heart failure
synchronized cardioversion, consider sedation; if regular narrow complex, consider adenosine
46
if persistent tachycardia with no symptoms and wide QRS > 0.12 s
IV access & 12 lead ECG, consider adenosine only if regular & monomorphic, consider antiarrhythmic infusion
47
if persistent tachycardia with no symptoms and no wide QRS
IV access & 12 lead ECG, vagal maneuvers, adenosine if regular, CCB or BB
48
how do vagal maneuvers work
intended to stimulate vagal nerve, resulting in acetylcholine release and slowing of conduction through AV node
49
examples of vagal maneuvers
bear down, blow through a syringe, immersion of face in ice water
50
adenosine mechanism
slows conduction through AV node, 6 mg rapid IVP, can follow with 12 mg IVP if needed... MUST BE RAPID PUSH
51
what to counsel patient before adenosine
may feel impending doom or feeling of dropping on a rollercoaster
52
which line is preferred for adenosine
central line. if peripheral, raise extremity