Acquired Neurogenic Communication Disorders Flashcards

(82 cards)

1
Q

What do SLPs do?

A

diagnose and treat communication, swallowing, and related disorders of the oropharyngeal mechanism

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2
Q

SLP settings:

A

schools
SNF
private practices
hospitals
NICU
acute care

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3
Q

Neurological Processes of Communication:

A

Cognitive – linguistic processes

Motor speech programming

Neuromuscular execution

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4
Q

Aphasia =

A

acquired communication disorder caused by brain damage

characterized by an
impairment of language modalities: speaking,
listening, reading and writing

not the result of a
sensory deficit, a general intellectual deficit, or a
psychiatric disorder

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5
Q

Multimodality Deficit
Language is a Central Process

A

3 levels:
1) word
2) sentence
3) discourse

comprehension:
> listening
> reading

production:
> speaking
> writing

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6
Q

word:

A

comprehension:
> listening: listen to word & point to object/picture

> reading: read work & point to object/picture

production:
> speaking: name objects/pictures

> writing: write names of objects/pictures

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7
Q

sentence:

A

comprehension:
> listening: follow command
> reading: follow instruction

production:
> speaking: describe actions
> writing: describe actions

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8
Q

discourse:

A

comprehension:
> listening: listen to story & answer questions

> reading: read paragraph & answer questions

production:
> speaking: describe complex picture

> writing: write letter/story

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9
Q

Aphasia multimodal language problem

A

Basic pattern of relative intact function / deficit

listening comprehension is the most preserved function

writing is the most impaired

auditory comprehension (listening) is relatively intact compared to expressive language functions (speaking and writing), especially in cases of more severe impairment

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10
Q

Aphasia is an acquired impairment of the cognitive
system specialized for:

A

comprehending and formulating
language, leaving other systems relatively intact

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11
Q

Aphasia Symptoms - Expression

A

anomia
non-fluent aphasia
telegraphic speech
agrammatism
telegraphic speech
paraphasia
neologism
jargon

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12
Q

anomia =

A

Having difficulty finding words

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13
Q

non-fluent aphasia =

A

Speaking haltingly or with effort

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14
Q

telegraphic speech =

A

names of objects

Speaking in single words

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15
Q

agrammatism =

A

Speaking in short, fragmented phrases

Making grammatical error

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16
Q

telegraphic speech =

A

Omitting smaller words like the, of, and was

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17
Q

paraphasia =

A

Substituting sounds or words

semantic word level
paraphasia “table” for bed

phonemic sound level paraphasia
“wishdasher” for dishwasher

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18
Q

neologism =

A

Making up words

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19
Q

jargon =

A

Fluently stringing together nonsense words and real words, but leaving
out or including an insufficient amount of relevant content (e.g., fluent
aphasia with empty speech, paucity of content

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20
Q

Aphasia Symptoms - Comprehension

A

difficulty understanding
spoken utterances

Requiring extra time to
understand spoken messages

Providing unreliable answers to “yes/no” questions

Failing to understand complex grammar

Finding it very hard to follow fast speech (e.g., radio or television news)

Misinterpreting subtleties of
language (e.g., taking the literal meaning of figurative speech such as “It’s raining cats and dogs.”)

Lacking awareness of errors (e.g., anosognosia)

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21
Q

classification of aphasia:

A

nonfluent = speech production is halting and effortful, grammar is impaired, content words may be preserved

fluent = person is able to produce connected speech, sentence structure is relatively intact but lacks meaning

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22
Q

nonfluent aphasia:

A

language comprehension relatively intact -> broca’s aphasia & transcortical motor aphasia

language comprehension impaired -> global aphasia

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23
Q

broca’s aphasia =

A

repetition of words/phrases poor

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24
Q

transcortical motor aphasia =

A

strong repetition skills

may have difficulty spontaneously answering questions

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25
global aphasia =
severe expressive and receptive language impairment may be able to communicate using facial expression, intonation, and gestures
26
fluent aphasia:
language comprehension relatively intact -> conduction aphasia & anomic aphasia language comprehension impaired -> wernicke's aphasia & transcortical sensory aphasia
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conduction aphasia =
word finding difficulties, difficulty repeating phrases
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anomic aphasia =
repetition of words/phrases good word finding difficulties uses generic fillers ("thing") or circumlocution
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wernicke's aphasia =
repetition of words/phrases poor
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transcortical sensory aphasia =
repetition of words/phrases good may repeat questions rather than answering them "echolalia"
31
Characterize the communication output?
Non-fluent – Speech production is halting and effortful *grammar is impaired, content words may be relatively preserved Fluent - produces connected speech *sentence structure is relatively intact, but lacks meaning
32
Characterize the auditory comprehension?
Language comprehension relatively intact Language comprehension relatively impaired
33
If the repetition abilities were good, what type of aphasia do you think this person has? a) Broca’s b) Transcortical motor c) Conduction d) Global
b) Transcortical motor pattern of relatively preserved listening ability, but impaired speaking and writing, along with moderate difficulties in reading, fits with transcortical motor aphasia anomia (difficulty finding words) and agrammatism (telegraphic speech with missing function words) are common Paraphasias (word substitutions) might be minimal or absent argon or neologisms (nonsense words) are typically not prominent
34
If the repetition abilities were poor, what type of aphasia do you think this person has? a) Broca’s b) Transcortical motor c) Conduction d) Global
c) Conduction (listening) is relatively intact Fluent speech but with frequent phonemic paraphasias (word or sound substitutions) Anomia (difficulty finding words) is present, but agrammatism is less common compared to Broca’s or transcortical motor aphasia no jargon or neologisms
35
Broca’s aphasia also involves poor repetition, but the speech is typically ____
non-fluent, and the graph shows moderate to severe impairments across
36
Transcortical motor aphasia is characterized by ___
good repetition
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Global aphasia involves severe deficits across ____
all modalities, including listening including listening, speaking, reading, and writing, with very little preserved ability
38
If the repetition abilities were poor, what type of aphasia do you think this person has? a) Wernicke's b) Transcortical sensory c) Anomic d) Global
a) Wernicke's Listening comprehension is typically significantly impaired in Wernicke's aphasia Fluent speech is common, but it often includes paraphasias (word substitutions), jargon, and neologisms (nonsense words) Anomia (difficulty finding the right words) is also typica
39
Transcortical sensory aphasia would involve good ___ despite poor ___
repetition comprehension
40
Anomic aphasia is primarily characterized by ____
word-finding difficulties (anomia) but good comprehension and good repetition
41
If the repetition abilities were good, what type of aphasia do you think this person has? a) Wernicke's b) Transcortical sensory c) Anomic d) Global
b) Transcortical sensory typically have poor comprehension Fluent speech is common, but often filled with paraphasias (word substitutions) or even neologisms (made-up words) Anomia (difficulty finding words) is present
42
Wernicke's aphasia typically involves:
poor repetition, despite fluent speech and poor comprehension
43
Anomic aphasia involves:
good comprehension and good repetition, but it is primarily characterized by word-finding difficulties
44
Screening tool for aphasia
Language Screening Test
45
Motor Speech Disorders
Dysarthria(s) Communication through vocal symbols = Speech
46
Speech =
embodies language into the physical (acoustic) properties for the purposes of recognition and interpretation
47
Motor Speech Disorders – Dysarthria(s)
group of neurogenic speech disorders characterized by "abnormalities in the strength, speed, range, steadiness, tone, or accuracy of movements required for breathing, phonatory, resonatory, articulatory, or prosodic aspects of speech production
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abnormalities are due to one or more sensorimotor problems—
weakness or paralysis incoordination involuntary movements excessive, reduced, or variable muscle tone
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predominant framework for differentially diagnosing dysarthria is based on ___
a perceptual method of classification
50
perceptual method of classification relies on _____
auditory perceptual attributes of speech that point to the underlying pathophysiology perceptual attributes are used to characterize the dysarthrias and, along with pathophysiological information, can help identify underlying neurologic illness
51
Motor Speech Disorders: Dysarthria types
congenital degenerative diseases demyelinating and inflammatory diseases infectious diseases neoplastic diseases toxic/metabolic diseases trauma vascular diseases
52
congenital =
cerebral palsy, chiari malformation
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degenerative diseases =
54
demyelinating and inflammatory diseases =
multiple sclerosis, encephalitis
55
infectious diseases =
acquired immune deficiency syndrome (AIDS), herpes zoster
56
neoplastic diseases =
CNS tumours, cerebral, cerebellar, or brainstem tumours
57
toxic/metabolic diseases =
botulism, carbon monoxide posioning
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trauma =
traumatic brain injury chronic traumatic encephalopathy
59
vascular diseases =
stroke (hemorrhagic or ischemic)
60
flaccid =
associated with disorders of the lower motor neuron system and/or muscle Weakness, flaccidity, atrophy, fasciculations, hypoactive gag reflex, facial myokymia (involuntary, spontaneous, localized quivering of a few muscles), nasal backflow while swallowing
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Spastic =
associated with bilateral disorders of the upper motor neuron system Pathologic oral reflexes (sucking reflex; snout reflex; jaw jerk reflex), lability of affect, hypertonia, hyperactive gag reflex
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Ataxic =
associated with disorders of the cerebellar control circuit dysmetric jaw, face, and tongue AMRs, head tremor
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Hypokinetic =
associated with disorders of the basal ganglia control circuit Masked facial expression, tremulous jaw, lips, tongue, reduced range of motion on AMR tasks, resting tremor, rigidity
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Hyperkinetic =
associated with disorders of the basal ganglia control circuit Involuntary head, jaw, face, tongue, velar, laryngeal, and respiratory movements, relatively sustained deviation of head position, multiple motor tics, myoclonus of palate, pharynx, larynx, lips, nares, tongue, or respiratory muscles
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Traumatic Brin Injury - Cognitive-Communicative Disorders
form of nondegenerative acquired brain injury, resulting from an external physical force to the head (e.g., fall) or other mechanisms of displacement of the brain within the skull (e.g., blast injuries)
66
Traumatic Brin Injury - Cognitive-Communicative Disorders: symptoms
Changes in levels of consciousness Memory disturbances Changes in cognitive function (e.g., attention, memory, executive function) Disturbances of sensory & motor function Confusion associated with deficits in orientation Neurological signs, such as brain injury observable on neuroimaging, new onset or worsening of seizure disorder, visual field deficits, hemiparesis, etc.
67
TBIs can result in focal damage =
(e.g., gunshot wound) or be more diffuse in nature (e.g., diffuse axonal injury) symptoms can vary depending on the site of lesion and extent of damage to the brain TBI is often associated with polytrauma (injury to the brain in addition to one or more other body systems)
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TBI can be categorized as:
mild, moderate, or severe based on the extent and nature of injury, duration of loss of consciousness, post- traumatic amnesia, and the severity of confusion at initial assessment during the acute phase of the injury
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Potential Consequences/Impact of Cognitive-Communication Impairment Can Include:
Reduced ability to effectively communicate needs Reduced awareness of impairment and its degree Reduced memory, judgment, and ability to initiate and effectively exchange routine information Difficulty performing personal lifestyle management activities (i.e., pay bills) Reduced ability to anticipate potential consequences, lack of judgment and problem solving Reduced social communication skills and/or ability to manage emotions
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Cognitive-Communicative Disorders – Potential Impact on Everyday Function
Difficulty anticipating consequences of own actions Poor organization, with limited problem solving and judgment Difficulty with concepts of time and money Difficulty self-disciplining and self-monitoring to follow rules; exhibits socially inappropriate behavior Difficulty changing routine or schedule; difficulty learning new rules
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Limited communication:
difficulty selecting appropriate words and remembering names limited memory and/or knowledge of current events and/or personal history responses in conversation may be verbose, redundant, or tangential missing or misunderstanding humor difficulty understanding nonverbal communication (i.e., facial expressions and/or body language) difficulty understanding abstract information
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Difficulty managing home or maintaining a job or business due to:
Difficulty following directions Difficulty comprehending or applying abstract written information Difficulty analyzing personal and/or business problems, identifying and applying solutions Difficulty assessing own strengths and weaknesses, developing effective plans to improve weaknesses Difficulty managing multiple responsibilities simultaneously Difficulty managing emotions (especially anxiety, frustration, or anger) related to performance difficulties Difficulty making, following, and modifying plans as needed Difficulty effectively communicating with colleagues and/or customers
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Traumatic Brin Injury – Observations: Cognition
Orientation Attention Learning & Memory Recognition, prosopagnosia, gnosis, etc. Executive/Goal directed behavior Self-Awareness
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Traumatic Brin Injury – Observations: Language & Communication
* Verbose * Tangential * Paucity of thought * Slow processing speed * Word finding * Expressive impairments * Receptive impairments * Dysarthria
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Traumatic Brin Injury – Observations: Behavioral Regulation
* Affect * Agitation * Lability * Sensitivity * Impulsivity
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Traumatic Brin Injury - Neurocognitive Effects
Deficits in shifting attention between tasks Difficulty with selective attention Impaired sustained attention (e.g., for task completion) Increased response latencies Reduced processing speed Impaired goal directed behavior Deficits in short-term memory that negatively affect new learning or planned tasks Post-traumatic amnesia (retrograde) Lack of insight for monitoring one's strengths, weaknesses, functional abilities, problem situations, and so forth Reduced awareness of deficits (anosagnosia) Impaired goal directed behavior Deficits in orientation to self, situation, location, and/or time Impaired spatial cognition that can affect ability to navigate and ambulate
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Traumatic Brin Injury - Language & Communication Effects
* Pragmatic/Social Communication * Spoken Language
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Traumatic Brin Injury - Neurobehavioral Effects
Affective changes, including over-emotional or over-reactive affect or flat (i.e., emotionless) affect Agitation and/or combativeness Anxiety disorder, depression Difficulty identifying emotions in others (alexithymia) Emotional lability and mood changes or mood swings Excessive drowsiness and changes in sleep patterns, including difficulty falling or staying asleep (insomnia), excessive sleepiness (hypersomnia) Feeling of disorientation or fogginess Increased state of sensory sensitivity accompanied by exaggerated response to perceived threats (hypervigilance) Impulsivity, irritability and reduced frustration tolerance Stress disorders
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Pragmatic/Social Communication
Conversational turns marked by verbosity Difficulty initiating conversation and maintaining topic Difficulty inhibiting inappropriate language or behavior Impaired ability to use nonverbal communication effectively (e.g., tone of voice, facial expression, body language) Impaired social cognition skills (e.g., regulating emotion; expressing emotion and perceiving emotion of others; ability to take the perspective of others and to modify language accordingly) Tendency to be tangential
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Spoken Language =
Anomia or word retrieval deficits Decreased ability to formulate organized discourse or conversation Difficulty following directions Difficulty understanding abstract language/concepts Difficulty making inferences Tendency to perseverate in verbal responses Use of incoherent or confabulatory speech
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Difficulty functioning independently due to:
unresponsiveness to all external stimuli may open eyes, suck, and/or yawn does not attend to others no purposeful speech extremely impaired attention and memory with impulsivity extremely limited communication:
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extremely limited communication:
difficulty responding to and/or saying name difficulty expressing basic needs to others using simple words and/or gestures (i.e., yes/no difficulty saying greetings (i.e., "hi" and "bye") on own