Acute Heart Failure Flashcards
(43 cards)
Low perfusion at rest =
cold
in Cold and Wet, diuresis will improve
cardiac output
ionotropic drugs if
cold and dry
cold and wet and normal SVR
most patients iwith acute decomp HF fall into this category
wet and warm
impact on cardiac oxygen consumption milrinone
not significant
risks of increased calcium with ionotropes
arrhythmias
limitations of nitroprusside
cynaide toxicity -nausea, weird (if low hepatic perfusion)
accumulation of thiocynate (esp if impaired renal fxn)
diuresis not possible if
renal perfusion impaired
considered a inodilator because ionotrope and vasodilator (cAMP changes lead to relaxation)
milrinone
diuretic side effects
electrolyte abnml
hypotension
gout exacerbation
hearing loss
Digoxin toxicity
renal insuf
muscle cramps (if too rapid)
impact of hypervolemia on CVP
increases CVP
healthy perfusion at rest =
warm
warm vs cold
cardiac index
cardiac index >2.2 = warm
side effects milrinone
hypotension
arrhythmia
tachycardia
congestion at rest =
wet
challenges of diuretics
braking - long term tolerance of natriretic response
rebound may lead to Na retention
long term tolerance due to tubular hypertrophy
Cl levels cold/wet/dry/warm
Warm/Dry - Cl normal
Warm/Wet - Cl normal
Cold Dry - Cl decreased
Cold/wet - Cl decreased
impact on mortality, diuretics
no improvement
therapuetic effect/mechanism Levosimendan
Troponin c to increase Ca sensitivity
Phosphodiesterase III inhibitor
Decreaseases LVEDP, afterload
increases stroke volume
____ vein direct measure of right atrium
right internal jugular
evidence of low perfusion
narrow pulse p
sleepy
low serum Na
Cool extremities
hypotension with ACE
renal dysfxn
requirements for iontrope use
Advance systolic HF
Low output syndrome
hypotension
Vasodilators inffective/contraindicated
Fluid overload + unresponsive to diuretics (or renal failure)
Cold and dry requires _____
cathetar placement to evaluate filling prossure
goal of therapy > wet and warm
Decrease filling pressures (LVEDP, PCWP)