Acute Kidney Injury Flashcards

(51 cards)

1
Q

Kidney stone diet **

A

look in blue box on page 2 of the kidney stone diet handout

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2
Q

Acute kidney injury

A

almost always recoverable - rapid loss of kidney function
elevated serum creatinine
decreased urine output (oliguria)
potentially reversible

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3
Q

azotemia

A

too much nitrogen in the blood

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4
Q

3 types of kidney injury

A

pre-renal
inter-renal
post-renal

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5
Q

prerenal causes

A
caused by:  Ischemia- whats causing it? 
severe dehydration
heart failure (or anything that decreases cardiac output) 
arythmias/tachycardia/ ect 
anything that decreases CO

ischemia decreases GFR
causes oliguria
autoregulatory mechanisms attempt to prevent blood flow (only as effective as the blood flow)

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6
Q

intrarenal damage

A
tissue damaged by conditions 
prolonged ischemia
NEPHROTOXINS
abx (gentamycin) 
NSAIDS
Hgb released from hemolyzed RBC 
Myoglobin released from necrotic muscle cells
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7
Q

ATN acute tubular necrosis

A

results from ischemia, nephrotoxins, or sepsis

potentially reversible

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8
Q

only need about % of one kidney to survive

A

40%

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9
Q

Post renal causes (5) :

A
BPH
Prostate cancer 
calculi   
trauma (ex bike injury on urethra) 
extrarenal tumors
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10
Q

AKI
RIFLE classification-
how much filtration rate is lost?

A
Risk for injury? 
Injury ( start having scant urine) 
Failure  (lost all but  50% GFR) 
Loss (lost all but 25% GFR)
End stage kidney disease
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11
Q

oliguric phase urine output?

A

urine output <400mL/day
occurs 1-7 days after insult
lasts 10-14 days

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12
Q

oliguric phase UA may show?

A

UA may show casts, RBC WBC

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13
Q

oliguric phase =fluid retention

sx are-

A
neck veins distended 
bounding pulse 
pulmonary edema 
HTN 
anasarca
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14
Q

Kussmaul respiration

A

lungs try to make pt take deeper breaths faster

trying to maintain acid pH balance in body

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15
Q

hyponatremia can lead to

A

cerebral edema

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16
Q

anasarca becomes life threatening when:

A

it spreads to brain tissue-the swelling has no where to go (cerebral edema limits amount of blood that can get into the brain)
33:03

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17
Q

quickest way to die from renal prob?

A

potassium excess

affects electrocardiac activity of heart= EKG changes

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18
Q

what happens to lab value from waste product accumulation?

A

Elevated BUN ans serum creatinine levels

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19
Q

Diuretic phase of AKI-
how much urine?
what should you monitor?

A

daily urine output 1-3L
May reach 5L or more

monitor for hyponatremia, hypokalemia, and dehydration

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20
Q

AKI - diagnostic studies?

A

UA
serum creatinine
kidney ultrasonography- looking for hydronephrosis fluid buildup in kidney
renal scan-
CT of abdomen with contrast dye (risk to kidneys from dye)
renal biopsy 14G needle apply pressure dressing and keep pt on that side to limit bleeding

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21
Q

therapy for hyperkalemia ( biggest life or death)

A

Kayexalate med- very sweet pulls K+ from colon thru pooping
combo of IV insulin/ IV dextrose/ Ca Gluconate
47:50

22
Q

osmotic diuretic

A

mannitol or albumin

23
Q

CRRT continuous renal replacement therapy

A

ICU 1 nurse monitors pt- 1 nurse monitor machine to verify I and O

24
Q

nutrition therapy for AKI

A

adequate caloric intake
restrict Na, K
increase dietary fat ( so they still get amino acids)
enteral nutrition
LIMIT protein= causes higher nitrogenous wastes

25
Assessment for AKI
VS I and O examine urine assess appearance of pt (itching? skin turgor? dehydration?) observe access site for dialysis LOC oral mucosa lung sounds (crackles? sign of pulonary edema) heart rythym (irregular = hyper/hypo kalemia)
26
Nusing DX for AKI
excess fluid volume risk for infection fatique anxiety
27
1KG water = how much water retained?
1L
28
chronic kidney disease ******see table 47-6 memorize the TABLE!
progressive irreversible loss of kidney function | SEE TABLE 47-6 stages of kidney disease++
29
leading cause of ESKD
diabetes HTN leads to atherosclerosis= succession of blood flow glomulonephritis interstitial nephritis polycystic kidney disease (cysts increase size of kidney/decrease function) prolonged obstuction of urinary tract vesicoureter reflux chronic pyelonepritis (infection leads to inflammation and scarring)
30
oliguria
scant urine
31
isothenuria
large qty of very dilute urine
32
anuria
producing almost no urine
33
which dialysis is least extreme?
peritoneal dialysis- aka continuous ambulatory dialysis- peritoneum of body is used as a permeable membrane as pt ambulates
34
peritoneal dialysis is started when-
pt GFR is 15mL or less | begun when pt uremia can no longer be managed conservatively
35
peritoneal dialysis is done by
inserting a catheter thru the abdominal wall= direct access into inner part of body STERILE TECHNIQUE MANDATORY!
36
peritoneal dialysis based on pt
weight and BP
37
Dialysis solutions
1, 2, or 3L bags concentration of 1.5%, 2.5%, or 4.25% dextrose must be warmed slowly to BODY TEMP do not microwave!!!!!!!! Pt takes BP and dry weight (empty) in the AM to determine fluid deficient 1.5%/ neutral2.5% /overload 4.25%
38
dialysate should be:
``` clear (no infection) no strings (fibrin starting to form clotting cascade- dr would give heparin to prevent that from progressing) ```
39
dialysate should be:
``` clear (no infection) no strings (fibrin starting to form clotting cascade- dr would give heparin to prevent that from progressing) allows independence allows travel short training program fewer dietary restrictions greater mobility than with hemodialysis ```
40
benefit of peritoneal dialysis
allows freedom of movement , go about life
41
complications of peritoneal dialysis
``` exit site infection hernia (walking around with 3KG of fluid in abdomen) peritonitis lower back problem (from the weight of the 3KG of fluid) clog in tubing bleeding pulmonary complications protein loss ```
42
hemodialysis complication
``` Complications: Thrombosis Infection Aneurysm Ischemia – “steal syndrome” ```
43
dialyzer
artificial kidney
44
access sight for hemodialysis
arteriovenous fistula and graft, temporary vascular access ** risk for infection, clotting
45
hemodialysis vascular access
fistula - surgically created anastamosis of artery and vein - much less likely to blow like a vein would do (more common- but if poor blood supply or cant use large enough needle) shunt/graft- same process, but using teflon or dacron but deaing with a foreign substance in the body
46
Dialyzer/blood lines are primed with saline solution to eliminate air/blood in the line T or F?
TRUE
47
Fill a thrill means?
turbulance that can be felt at site
48
hemodialysis complications?
``` Hypotension Muscle cramps Loss of blood Hepatitis (getting lower) Disequilibrium syndrome ```
49
nursing considerations for pt with hemodiallysis:
NEVER use HD catheter without a physician order NO….. In affected extremity Blood pressure measurements Insertion of IVs Phlebotomy Palpate for thrills and auscultate for bruits every 4 hours while pt is awake Assess the patient’s distal pulses and circulation in the arm with the access Elevate the affected extremity post operatively Encourage routine range of motion exercises Check for bleeding at needle insertion sites Assess for s/s of infection at the needle insertion site. Instruct pt not to carry heavy loads in the affected arm Instruct pt not to sleep with the body weight on the affected arm
50
more nursing considerations for hemodialysis
Monitor vital signs Monitor laboratory values before, during and after dialysis Assess the client for fluid overload prior to the procedure Assess patency of the blood access device Weigh the client before and after the procedure to determine fluid loss. Know the Patient’s dry weight. Assess the patient’s LOC Assess for headache, nausea or vomiting.
51
MORE nursing considerations for hemodialysis
Hold antihypertensives and other medications that can affect the BP prior to the procedure, as prescribed Hold medications that could be dialyzed off, such as water-soluble vitamins and certain antibiotics Monitor for shock and hypovolemia during the procedure Provide adequate nutrition (client may eat prior to the procedure)