Acute Perio Conditions Flashcards

(33 cards)

1
Q

what can NUG/NUP both be manifestations of?

A

HIV

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2
Q

What is NUP?

A

extends into perio attachment
marker of severe immunosuppression that affects the gums and underlying bone
Is a unique side effect of AIDS

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3
Q

NUG/ANUG are what type of infections?

A

endogenous infections

systemic changes predispose the gingiva to invasion by some bacteria in the oral flora

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4
Q

who is Trench Mouth most common in?

A

16-30 year olds, males more common
5% incidence
seasonal variation

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5
Q

Patient presents with?

A

pain, sudden onset, severe, may affect eating

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6
Q

what do the necrotic ulcers of NUG present like?

A

initially are red swollen ID papillae. Ulceration starts at tip of papillae and spreads laterally along the gingival margin

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7
Q

6 features of NUG

A
  • grey pseudomembranous membrane slough - removed to display raw bleeding surface
  • halitosis - foeter oris
  • spont. bleeding
  • metallic taste
  • ID ulceration
  • localised or generalised
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8
Q

If NUG is severe what may it present with systemically?

A

lymphadenopathy

general malaise

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9
Q

if NUG is not treated what happens?

A

acute symptoms last 2-3 weeks
heals as chronic gingivitis
tends to reoccur with further ID papillae loss - creates stagnation areas

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10
Q

If NUG is not treated in a developing country what can happen?

A

can progress in malnourished or diseased children, can affect the facial tissues - oro facial necrosis
can cause cancrum oris - disfiguring/fatal

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11
Q

what is the microbiology of NUG?

A

a fuso - spirochaetal complex

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12
Q

what are the principle bacteria in NUG?

A

treponema vincentii and denticola
fusobacterium nucleatum
prevotela intermedia
porphromonas gingivalis

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13
Q

what are some risk factors for NUG?

A

poor oh
smoking
stress
immunodeficiency

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14
Q

how can smoking cause NUG?

A
  • causes vasoconstriction - anaerobic conditions
  • suppress serum IgG leveles against supragingival bacteria
  • depresses T helper lymphocytes
  • reduces motility and chemotaxis of PMNs
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15
Q

how can stress cause NUG?

A

changes in behaviour - increased smoking, reduced oh care, reduced salivary flow, vasoconstriction in gingival end arteries, suppress immune response

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16
Q

what is the histopathology of NUG?

A
  • bacterial zone - surface slough rich in bac
  • pmn rich zone - epithelium and supra CT rich in PMNs
  • necrotic zone - mainly spirochaetes
  • deeper tissues - still viable with plasma cells and macrophages
  • some bacteria infiltrate
17
Q

how to treat NUG?

A

OHI and explanation
Gentle USS
antimicrobial metronidazole 200mg for 3 days
review 48 hours

18
Q

subsequent NUG tx?

A
OHI
smoking cessation
supra/sub debridement
review/maintenance
perio surgery to reverse architecture
19
Q

what kind of abscesses can occur?

A

periodontal
endodontic
combined

20
Q

what is an endodontic lesion?

A

toothache
occurs at apex of root, collection of pus under high pressure in small space. Enclosed infection. Very vascular - throbs. Discharging sinus can form through bone.

21
Q

what is a periapical granuloma?

A

benign tumour

sequale of something

22
Q

what is a combined lesion?

A

started in perio pocket as a perio abscess

travels down accessory canal = endodontic abscess - common in furcations

23
Q

what is a perio abscess?

A

needs an established perio pocket
blocked channel of moving bacteria = trapped area of virulent destructive bacteria
caused by scaling - calculus or trauma - inflammation - blocks pocket.
flush/rsd unless recent rsd has caused the abscess

24
Q

what is a gingival abscess?

A

outer layer of gingivae affected
caused by something causing trauma/infection in the gingival sulcus
e.g food/tb trauma = becomes infected

25
what is pericoronitis?
infection of gingivae covering erupting teeth | hard to clean - bact/food stuck causing infection
26
what are non plaque induced lesions?
specific bacterial origin/viral/fungal/genetic origin gingival manifestations of systemic conditions traumatic lesions foreign body reactions and NOS
27
specific bacterial origin lesions caused by?
- neisseria gonorrhea associated lesions - treponema pallidium - syphillus/rare - streptococcal species - viral infection. 2ndry colonises
28
two types of primary herpetic gingivostomatitis?
HSV1 - STI type HSV2 - caught from others - air particles, red gingivae and lips, common in kids, mouth and tongue ulcered, lies in trigeminal ganglia, lasts 1-2 weeks, becomes re active if cold/immunosuppressed etc as coldsores
29
types of viruses causing lesions?
primary herpetic gingivostomatitis recurrent oral herpes varicella zoster infections - primary = chickenpox/secondary - shingles
30
fungal infections causing lesions?
candida species infection general g.candidosis linear g.erythema histoplasmosis - deep seated mycoses/rare/non specific ulceration
31
genetic conditions causing lesions?
hereditary ginival fibromatosis | uncommon/autosmal dominant/general gingival enlargement/sometimes associated with epilepsy and mental retardations
32
what are some gingival manifestations of systemic conditions?
``` lichen planus pemphigoid phemphigus vulgaris erythema multiforme lupus erythematosus drug induced ```
33
where could allergic reactions occur from?
dental restorative materials mercury/nickel/acrylic etc TP/MW/chewing gum additives/food additives