acute stroke Flashcards

(42 cards)

1
Q

what does timely out of bed activities prevent

A
  • DVT
  • pulmonary emboli
  • pneumonia
  • falls
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2
Q

why is orthostatic hypertension so bad for CVA pts

A
  • because it decrease blood perfusion and O2 to the brain
  • do not avocate for high intensity in acute care only rehab
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3
Q

types of stokes

A
  • embolic
  • lacunar
  • athero-thrombotic
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3
Q

what does the lacunal strokes affect

A
  • motor due to lack of O2 during stroke to internal capsule
  • usually involves the posterior limb of the internal capsule
  • no sensory, aphasia, or visual field abnormalities
  • dysarthria might be present
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3
Q

lacunar stroke or lacunar infarct pathology

A
  • most common type of ischemic stroke
  • resulting from the osculation of small penetrating arteries the provide blood to the brains deep structures (internal capsule)
  • etiology of chronic hypertension
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4
Q

signs and symptoms to review prior to out of bed activities

A
  • nausea
  • vomiting
  • dizziness
  • light-headedness
  • loss of alertness
  • confusion
  • headache
  • ringing in ears
  • sweating
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5
Q

location of the internal capsule

A
  • subcortical structures that include: internal capsule, caudate, putamen, globus pallidus, thalamus, brainstem
  • anterior limb separates the caudate nucleus and lenticular nucleus
  • the posterior limb separates the thalamus and lenticular nucleus
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6
Q

type of fibers in the internal capsule

A
  • anterior limb: frontopontine fibers, thalamocortical fibers
  • genu: corticobulbar fibers
  • posterior limb: corticospinal fibers (cortex to spine), sensory fibers
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7
Q

blood supply to the internal capsule

A
  • anterior limb: lenticulostriate branches of MCA and ACA
  • Genu: lenticulostriate branches of MCA
  • posterior limb: lenticulostriate branches of MCA and anterior choroidal artery
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8
Q

clinical findings in internal capsular stroke

A
  • weakness of the face, arm and or leg (test CN 7 and 2)
  • UMN signs: hyperreflexia, babinski sign, hoffman present, clonus, spasticity
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9
Q

watershed infarctions

A
  • infarcts in areas where this is reduced profusion due to blockage of the ACA, MCA, or PCA.
  • occurs with internal carotid artery blockage or a carotid artery blockage
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10
Q

Transient Ischemic Attacks (TIA)

A
  • typically less then 10 minutes
  • longer then 10 minutes tissue cells will start to die
  • TIA lasting longer then an hour will cause small infarcts
  • requires an ER visit
  • Early warning sign for a larger stroke
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11
Q

Etiology of TIA

A
  • thrombus emerging
  • vasospasm
  • small emboli: dissolves
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12
Q

signs and symptoms of a TIA

A
  • high BP
  • weakness on one side of the body
  • vision problems
  • slurred speech
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13
Q

treatment of TIA

A

medication, surgery, and healthy lifestyle changes

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14
Q

cerebral edema

A
  • internal pressure within the cortex from hemorrhage or inflammation
  • fluid can push the brain contents to the other side
  • serious consequence of stroke that can lead to early mortality
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15
Q

cerebral perfusion pressure (CPP)

A

the net pressure gradient that drives oxygen delivery to cerebral tissue

16
Q

intracranial pressure (ICP)

A
  • measured invasively though an intracranial pressure transduction device (intraventricular monitor)
  • mean arterial pressure - ICP = cerebral perfusion pressure
17
Q

normal CPP

18
Q

normal ICP

A

5 to 15
ICP values of 20 to 30 represent intracranial hypertension

19
Q

hemorrhagic stroke

A

leads to increased pressure in teh brain and reduces cerebral perfusion

20
Q

tumors in the brain

A

create cortical pressure and reduce brain perfusion

21
Q

infection of the brain

A

changes in the regulation of cerebral perfusion may occur

22
Q

CARE for individual with CVA

A

stage 1: hospital and acute care
stage 2: inpt rehab or SNF
stage 3: At home health care
stage 4: outpt care
stage 5: after discharge

23
goldilocks : u shaped curve
make sure the ex is an appropriate level of intensity so the BP does not get too high or too low
24
tissue plasminogen activator (tPA)
- clot buster and timely management of high BP - when administered 3 hrs after a sroke it helps to restore blood flow to brain regions affected by a stroke, thereby limited the risk of damage and functional impairment
25
what is tPA associated with
- more frequent independent ambulation at discharge - discharge to home - reduced mortality and symptomatic intracerebreal hemorrhage
26
not a candidate for IV tPA or IAT
BP up to 220/120 may be permitted to allow for maximal perfusion unless otherwise contraindicated
27
candidate for IV tPA or IAT
lower BP to less them 185/110 prior to giving tPA or in preperation for the angiography suite
28
hypotension in the intial setting of stroke care
no formal guidelines exist for a lower limit of BP, hypotension is associated with worse outcomes. ensure that it is not related to volume depletion, cardiac abnormalities or shock
29
during IAT
maintain systolic BP at 140-180
30
incomplete reperfusion after tPA
maintain BP of greater then 180/105 consider induces hypertension
31
successful reperfusion of tPA
- consider a lower threshold of systolic less then 140-160 - monitor closely for changes in neurologic examination which can suggest reperfusion injury
32
mobility after acute ischemic stroke
pt should receive mobility 13-24 hours after receiving tPA SBP should be between 140-160
33
what is the recommendation for out of bed activities
Early lower- dose out of bed activities first day: 10-20 minutes
34
Preparing for out of bed activities
- receive orders from MD - review the chart (look for type of stroke, location and meds)
35
PT area if assessment and decision making
- skin integrity: ER and one floor - Mobility: out of bed with PT - Caregiver availability (education) - spasticity - durable medical equipment - recommendation regarding transition to appropriate level of care
36
rapid large falls in BP can reduce
cerebral blood flow leading to extension of the cerebral infarction or perihematomal ischemia
37
what is the general goal for CVA patients BP
less then 180. 20 point role fro systolic
38
deterioration
change in orientation - symptoms: new headache, acute hypertension, nausea or vomiting, changes in pulse ox or 02 sat, SOB, syncope
39
when should BP start to return back to nomal after tPA
48 hrs after given
40
outcome measures for stroke
- 6 minute walk - TUG - 5 TSTS - 10 MWT