ARDS Part 1 Flashcards

1
Q

ARDS affect what part of the lungs

A

Alveoli

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2
Q

This is a form of acute respiratory failure

A

(ALI) ACUTE LUNG INJURY

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3
Q

this is the most severe subset of ALI

A

ARDS

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4
Q

In ARDS the what happens to the alveolar-capillary

A

disrupted or destroyed

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5
Q

ALI is defined by ______ and presence of ______

A

Hypoxemia and bilateral infiltrates

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6
Q

Ards is commonly affect what type of patient

A

Critically ill or injured

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7
Q

ARDS starts with ______ of the lung tissue

A

Swelling

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8
Q

In ARDS there are ______ in the tiny air sacs impeding gas exchange

A

Fluids

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9
Q

In ARDS exudative phase occurs in the first __ days after exposure to ARDS risk factors

A

7

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10
Q

What causes tachypnea and increased work of breathing

A

Respiratory muscle fatigue and respiratory failure

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11
Q

Dyspnea is due _____

A

Increased work of breathing

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12
Q

Proliferative phase starts from day __-__

A

7-21

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13
Q

During what stage patients recover rapidly and liberated from mech vents

A

Proliferative

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14
Q

In proliferative phase even patient may improve they can still experience the following symtoms

A

Tachypnea, dyspnea, hypoxemia

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15
Q

What type of cell proliferates in the alveolar basement membrane to synthesise pulmonary surfactant to prevent alveolar collapse before converting to type 1 which lines the alveolar wall

A

Type 2 pneumocyte

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16
Q

Patients with ards recover lung function within how many weeks

A

3-4 weeks

17
Q

During this phase, patient may need for long term mechanical ventilation support and supplemental oxygen

A

Fibrotic phase

18
Q

In fibrotic phase there is what lung damage

A

Extensive alveolar duct and interstitial fibrosis, mark disruption of acinar architecture leading to emphysema like changes with large bullae

19
Q

What caused progressive vascular occlusion and pulmonary HTN

A

Intimal fibroproliferation in the pulmonary microcirculation

20
Q

Pulmonary HTN and vascular occlusion can cause the following:

A

Pneumothorax, decreased lung compliance and increased pulmonary dead space