Arrhythmias Flashcards

(34 cards)

1
Q

What are the sinus node rhythm disturbances?

A

Sinus Arrhythmia
Sinus Pause/Arrest
Sinus Bradycardia
Sinus Tachycardia

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2
Q

What is sinus arrhythmia? Tx

A

normal P, PR, QRS. rhythm sometimes appears irregular but originating from the sinus node.
-cyclic variation with respirations.

Tx: benign, not tx needed.

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3
Q

What is sinus pause/arrest? Tx

A

a missed beat, typically lasts 2 seconds to 2minutes.
-normal and fixed PR and RR intervals, lack p waves

Tx: -underlying cause

  • pacemaker
  • atropine IV if hemodynamic instability
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4
Q

Sinus Bradycarida what is this? Tx

A

heart rate less than 60BPM, may be caused by beta blockers and digoxin

Tx: 
SHORT TERM: 
-sx & hemodynamically unstable:  atropine &/or temporary pacemaker 
-asymptomatic: 
monitor and educate

LONG TERM:

  • no tx for long term if pt is asymptomatic
  • will need permanent pacemaker if sx
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5
Q

Sinus Tachycardia

  • what is this?
  • causes
  • tx
A

What: heart rate greater than 100BPM

Causes:
fever, pain, exercise, anemia, hypotension, thyrotoxicosis, anxiety

Tx:

  • treat underlying cause
  • calcium channel blockers (cardizem and verapamil)
  • beta blockers
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6
Q

What are the supraventricular arrhythmias

A
  • Supraventricular tachycardia
  • -WPW
  • -AV node re-entrant tachy (shortcut)
  • -atrial tachy
  • afib
  • aflutter
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7
Q

PSVT

  • heart rate
  • ekg findings
  • sx
  • tx
  • medications to prevent recurrence
A

HR 140-240

  • P wave buried in QRS
  • QRS is narrow** and normal morphology

Sx:

  • palpitations
  • dizziness
  • SOB
  • anxiety
  • chest pain

Tx:

  • Vagal maneuvers (hold breath, face in cold water, cough, carotid massage)
  • adenosine IV (blocks conduction at the AV Node)
  • cardioversion if adenosine doesnt work OR IV beta/Ca2+ blocker
Prevention: 
beta blockers 
ca2+ blocker
digoxin 
SVT ablation
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8
Q

WPW

  • HR
  • what causes this
  • what is it
  • sx
  • EKG findings
  • tx
A

HR: greater than 200
What causes this: congenital defect, most common causes of fast arrhythmias in infants and children.

-what is this? form of SVT that uses the normal plus accessory pathways to conduct impulses faster and in both directions .

-Sx:
palpitations, tachycardia, dizzines, dyspnea, anxiety, syncope, cardiac arrest.

EKG findings:
-PR interval is short, less than 0.12ms, Delta wave*

Tx:

  • radiofrequency ablation
  • beta blockers
  • calcium blockers
  • flecainide
  • vagal maneuvers
  • IV adenosine OR IV cardizem or verapamil if adenosine doesnt work.
  • if hemodynamically unstable cardioversion
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9
Q

Paroxysmal Atrial Tachycardia (PAT)

  • rate
  • what is this?
  • ekg findings
  • Tx
A

Rate: 150-250BPM
EKG findings:
-may conduct to ventricles but the AV node will try to block impulses
-P wave morphology varies from sinus*

Tx:

  • Treated with Vagal Maneuvers
  • if Vagal maneuvers fail…
  • -adenosine**
  • -cardioversion
  • -digoxin
  • -beta blockers
  • -Calcium channel blockers to prevent recurrence
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10
Q

Premature Atrial Contractions (PACs)

  • what causes this?
  • EKG findings
  • Tx
A

Cause: discharge from a non-sinus atrial pacemaker

EKG;
-p-wave preceding may not look like the p waves that originate from sinus node, may get lost in T wave.

Tx:
-non-life threatening, only treat if symptomatic w/ beta blockers.

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11
Q

Wandering Atrial Pacemaker

  • Rate
  • EKG findings
  • Tx
A

Rate: 45-100BPM

EKG findings
-P wave; needs to have 3 distinctly different P waves

Tx: no tx required

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12
Q

Multifocal Atrial Tachycardia

  • rate
  • ekg findings
  • Tx
A

Rate: greater than 100

EKG:
-3 different P waves in a given lead

Tx: treat the underlying medical condition

  • suppress rate with AV nodal blocking agents:
  • -Calcium channel and beta blockers
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13
Q

A-fib

  • cause
  • describe this rhythm
  • Rate
  • EKG
  • common with what underlying cardiac and pulmonary dz?
A

cause: multiple re-entrant loops generate chaotic atrial depolarization, AV node bombarded with rates greater than 400BPM from atrial foci..AV works hard to block impulses.

Rhythm: “irregularly irrregular”

Rate: 110-170BPM

EKG:
-no distinguishable P waves

Common w/ vavlular dz, heart failure, HTN, and sleep apnea.

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14
Q

What is the most common encountered arrhythmia in clinical practice?

A

Afib

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15
Q

Which lab always needs to be checked with new onset of Afib dx?

A

TSH!

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16
Q

Afib Tx

A

Stasis of blood in atria Tx: warfarin/pradaxa

Rate Control: 60-110
-Diltiazem/Beta blocker/ Digoxin

Rhythm Control if necessary:
Class 1A: Pronestyl(Procainimide), Quinidine(Cardioquin)
Class III. Satalol(Betapace), Ibutilide(Covert), amiodarone*
Class IC: propafenone(Rhythmol), Flecainide(Tambocor) *used only in pt with structurally normal heart.

Cardioversion:

  • safe to cardiovert if less than 48-72hrs of afib
  • make sure to get TEE prior to cardioversion to see if thrombus present in atria.

….if duration unknown:

  • rate control
  • anticoagulate 4-6wks then cardiovert
  • anticoagulate x6wks after successful cardioversion or indefinately if pt unaware they were in afib.

…..if failure of CV and medical therapy:
atrial ablation, AV ablation in extreme cases which would require pacemaker placement.

17
Q

What is the CHADS2 calculator? what is it used for?

A

CHADS2 is used to estimate embolic risk, does not apply to those with valve dz.

C=CHF 1pt
H= HTN 1pt
A=Age greater than 75 1pt
D=DM 1pt
S=stroke 2pts 

*if equal to or greater than 2pts anticoagulation unless CI.

18
Q

Evaluation of new onset afib pts

A

-echo: evaluate presence of valvular heart disease

Nuclear stress test: evaluate presence of ischemic heart dz

SLeep study: r/o sleep apnea

Thyroid function test*****

19
Q

Atrial Flutter

  • rate
  • rhythm description
  • ekg findings
  • tx
A

Rate: atria=250-350BPM
ventricles=150

Rhythm: regularly irregular, AV node blocks at 2:1, 3:1. 4:1

EKG:
-sawtooth pattern

Tx: 
ASA 325mg/day
-ablation if failed cardioversion and medical therapy 
-Class 1A antiarrhythmics to convert back to sinus: Pronestyle (Procainmide)
-Ventricular rate controlled with: 
--beta blocker
--calcium channel blocker
--digoxin
20
Q

What are the AV node disturbances

A
  • Junctional Escape
  • Accelerated Junctional
  • Junctional Tachy
21
Q

Junctional Escape &Accelerated Junctional

  • rate
  • EKG findings
  • tx
A

Rate:
-Junctional escape: 40-60BPM
-Accelerated Junctional:
60-100BPM

EKG findings both:

  • Narrow QRS complex
  • Retrograde P wave (inverted with short PR interval), P wave immediately after QRS, or no P wave

Tx:
no tx required

22
Q

Junctional Tachycardia

  • rate
  • tx
A

Rate: 150-250BPM

Tx: Acute:

  • vagal maneuvers
  • adenosine (DOC)*

Long Term:

  • beta blockers
  • calcium channel blockers
  • Class 1A, 1C, and III antiarrhythmics for resistant cases
23
Q

What are the types of AV Blocks?

A
  • 1st degree
  • 2nd degree Wenkebach
  • 2nd degree Mobitz
  • 3rd degree heart block
24
Q

1st degree AV block

  • tx
  • ekg findings
A

tx: treat the underlying cause (such as inferior MI, digitalis tox, hyperkalemia, myocarditis, rheumatic fever)

-ekg findings:
consistently prolonged PR interval greater than 0.20

25
2nd Degree Wenckebach - tx - where does this block occur? - ekg findings
Tx: no tx, since generally no sx -Block occurs in AV node and bundle of HIS EKG: PR interval gets progressively longer until a QRS is dropped
26
2nd degree AV block Mobitz Type II - where does this block occur? - EKG findings - Tx
- This block occurs below the bundle of his - EKG findings: "out of the blue drop a Q", usually widened QRS Tx: atrial pacing, permanent pacemaker
27
3rd Degree AV heart block - ekg findings - rate - tx
EKG: atrial rate is normal, normal P-P intervals, but not married to QRS intervals. -QRS may be normal or widened Rate: -atria faster than ventricles, ventricles around 70BPM - Tx: - external pacing and atropine for acute symptomatic episodes - permanent pacing for chronic complete heart block.
28
What are the ventricular dysrthmyias
- Premature ventricular contraction - Ventricular Tachycardia - Ventricular Fibrillation - Asystole - Idioventricular rhythm - PEA
29
Premature Ventricular Contractions - causes - EKG findings - Sustained VT is how long? Nonsustained? - rhthym - tx
causes: KNOW THESE, Jen said so... - Hypokalemia, low magnesium level, increasing catecholamines, coronary ischemia EKG: - P wave usually obscured by QRS - QRS is wide, morphology is bizarre -Sustained VT if greater than 30seconds or nonsustained if less than 30seconds. Rhythm: -PVCs may occur in singles, couplets(2 in a row), or triplets, or in bigeminy(every other is PVC), trigeminy(every 3rd is PVC), or quadrigeminy ``` Tx: -Lidocaine Class 1B antiarrhythmic -Rocainamide Class 1A -Amiodarone Class III -replace magnesium IF PULSE: -cardioversion & amiodorone ``` IF PULSELESS -defibrillation & amiodarone ****LIFE THREATENING ARRHYTHMIA!!!***
30
Torsades de Pointes - caused by - Tx
Caused by: hypokalemia and hypomagnesemia Tx: - Cardioversion - IV magnesium - IV potassium - Overdrive pacing
31
Ventricular Fibrillation - aka - results in ____ CO. - tx
aka: sudden cardiac death - results in absence of cardiac output. Tx: - Immediate defibrillation and ACLS protocol - Identify and tx underlying cause - ICD
32
Idioventricular Rhythm - EKG findings - Tx - aka
EKG: absent P wave, Wide QRS, Tx: improve CO and establish normal rhythm & rate. - pacemaker - atropine aka: the dying heart
33
Asystole - what is this? - Tx
-complete cessation of any electrical or mechanical activity Tx: - CPR - 100% O2 - IV - Intubation - transcutaneous pacing - epinephrine - atropine
34
Pulseless Electrical Activity (PEA) - aka - what happens? - EKG rhythm - underlying causes: 6H and 6T - Tx
aka: electromechanical dissociation Happens: electrical activity but no mechanical response -normal sinus rhythm BUT no pulse ``` Causes: 6H: -hypoxia -hypovolemia -hypoglycemia -hydrogen ion -hypothermia -hypo/hyperkalemia ``` 6T: - Toxins - Tamponade - Trauma - Tension pneumothorax - Thrombosis-cardiac - Thrombosis-pulmonary Tx: - correct underlying cause - epinephrine - atropine - CPR