arrhythmias Flashcards
(42 cards)
supraventricular tachycardia
atrial fibrillation
atrial flutter
ectopic atrial tachycardia
supraventricular bradycardia
sinus bradycardia
sinus pause
ventricular arrhythmias
ventricular ectopics or premature ventricular complexes PVC
ventricular tachycardia VT
ventricular fibrillation VF
asystole
atrio-ventricular node arrhythmias
AVN re-entry tachycardia AVNRT
AV reciprocating or AV re-entrant tachycardia AVRT
AV block - 1st, 2nd, 3rd degree
clinical causes
abnormal anatomy autonomic nervous system metabolic inflammation drugs genetic
ectopic beats
beats or rhythms that originate in places other than SA node
re-entry
requires more than one conduction pathway with different speed of conduction and recovery of excitability
symptoms
palpitations SOB dizziness syncope presyncope sudden cardiac death angina heart failure
investigations
ECG CXR echocardiogram stress ECG 24 hour ECG event recorder electrophysiological study
normal sinus arrhythmia
variation in heart rate, due to reflex changes in vagal tone during the resp cycle
inspiration reduces vagal tone and increases heart rate
sinus bradycardia
<60 beats/min
physiological athlete
drugs - beta blocker
ischaemia - common in inferior STEMI
sinus bradycardia treatment
atropine
pacing if haemodynamic compromise
sinus tachycardia
> 100 beats/min
physiological anxiety, fear, hypotension, anaemia
inappropriate drugs
sinus tachycardia treatment
underlying cause
beta adrenergic blockers
atrial ectopic beats
asymptomatic, palpitation
no treatment
beta adrenergic blockers may help
avoid stimulants
regular supraventricular tachycardia
AVN re-entrant tachycardia
AV reciprocating tachycardia/ AV re-entrant tachycardia
ectopic atrial tachycardia
regular supraventricular management
acute - increased vagal tone (valsalva, carotid massage), slow conduction in the AVN
chronic - avoid stimulants, electrophysiologic study and radiofrequency ablation, beta blocker, antiarrhythmic drugs
causing of heart block
age acute MI myocarditis infiltrative disease - amyloid beta adrenergic blockers, CCB calcific aortic valve surgery genetic - Lenergre's disease, myotonic dystrophy
first degree AV block
conduction after P wave takes longer
PR interval longer than normal
no treatment
second degree block Mobitz 1
progressive lengthening of PR interval
usually vagal in origin
second degree block
Mobitz 2
pathological, my progress to third degree
permanent pacemaker indicated
some action potentials fail to make it through the AV node
third degree block
complete heart block
no action fro the SA node gets through the AV node
ventricular pacing
ventricular ectopics
causes - structural, metabolic, may be marker for inherited cardiac conditions
beta blocker, ablation of focus
ventricular tachycardia
life threatening coronary artery disease previous MI cardiomyopathy inherited/familial arrhythmia syndromes