arrhythmias Flashcards

(42 cards)

1
Q

supraventricular tachycardia

A

atrial fibrillation
atrial flutter
ectopic atrial tachycardia

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2
Q

supraventricular bradycardia

A

sinus bradycardia

sinus pause

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3
Q

ventricular arrhythmias

A

ventricular ectopics or premature ventricular complexes PVC
ventricular tachycardia VT
ventricular fibrillation VF
asystole

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4
Q

atrio-ventricular node arrhythmias

A

AVN re-entry tachycardia AVNRT
AV reciprocating or AV re-entrant tachycardia AVRT
AV block - 1st, 2nd, 3rd degree

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5
Q

clinical causes

A
abnormal anatomy 
autonomic nervous system 
metabolic 
inflammation 
drugs 
genetic
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6
Q

ectopic beats

A

beats or rhythms that originate in places other than SA node

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7
Q

re-entry

A

requires more than one conduction pathway with different speed of conduction and recovery of excitability

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8
Q

symptoms

A
palpitations 
SOB
dizziness 
syncope 
presyncope 
sudden cardiac death 
angina 
heart failure
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9
Q

investigations

A
ECG 
CXR
echocardiogram 
stress ECG 
24 hour ECG 
event recorder 
electrophysiological study
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10
Q

normal sinus arrhythmia

A

variation in heart rate, due to reflex changes in vagal tone during the resp cycle
inspiration reduces vagal tone and increases heart rate

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11
Q

sinus bradycardia

A

<60 beats/min
physiological athlete
drugs - beta blocker
ischaemia - common in inferior STEMI

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12
Q

sinus bradycardia treatment

A

atropine

pacing if haemodynamic compromise

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13
Q

sinus tachycardia

A

> 100 beats/min
physiological anxiety, fear, hypotension, anaemia
inappropriate drugs

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14
Q

sinus tachycardia treatment

A

underlying cause

beta adrenergic blockers

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15
Q

atrial ectopic beats

A

asymptomatic, palpitation
no treatment
beta adrenergic blockers may help
avoid stimulants

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16
Q

regular supraventricular tachycardia

A

AVN re-entrant tachycardia
AV reciprocating tachycardia/ AV re-entrant tachycardia
ectopic atrial tachycardia

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17
Q

regular supraventricular management

A

acute - increased vagal tone (valsalva, carotid massage), slow conduction in the AVN
chronic - avoid stimulants, electrophysiologic study and radiofrequency ablation, beta blocker, antiarrhythmic drugs

18
Q

causing of heart block

A
age
acute MI
myocarditis 
infiltrative disease - amyloid
beta adrenergic blockers, CCB
calcific aortic valve surgery 
genetic - Lenergre's disease, myotonic dystrophy
19
Q

first degree AV block

A

conduction after P wave takes longer
PR interval longer than normal
no treatment

20
Q

second degree block Mobitz 1

A

progressive lengthening of PR interval

usually vagal in origin

21
Q

second degree block

Mobitz 2

A

pathological, my progress to third degree
permanent pacemaker indicated
some action potentials fail to make it through the AV node

22
Q

third degree block

A

complete heart block
no action fro the SA node gets through the AV node
ventricular pacing

23
Q

ventricular ectopics

A

causes - structural, metabolic, may be marker for inherited cardiac conditions
beta blocker, ablation of focus

24
Q

ventricular tachycardia

A
life threatening 
coronary artery disease 
previous MI
cardiomyopathy 
inherited/familial arrhythmia syndromes
25
ventricular fibrillation
chaotic ventricular electrical activity which causes the heart to lose the ability to function as a pump defibrillation, cardiopulmonary resuscitation
26
atrial fibrillation
chaotic and disorganised atrial activity irregularly irregular symptomatic or assymptomatic incidence increases with age
27
mechanisms of AF
ectopic foci muscle sleeves in the ostia of the pulmonary veins
28
AF termination
pharmacologic cardioversion with anti-arrhythmic drugs electrical cardioversion by direct current DCCV spontaneous reversion to sinus rhythm
29
paroxysmal AF
lasting less than 48 hours | often recurrent
30
persistent AF
episode of AF longer than 48 hours which can still be cardioverted to NSR unlikely to spontaneously revert to NSR
31
permanent AF
inability of pharmacologic or non-pharmacologic methods to restore NSR
32
AF associated disease/cause
``` hypertension congestive heart failure sick sinus syndrome coronary heart disease obesity thyroid disease familial cardiac valve disease alcohol abuse congenital heart disease cardiac surgery COPD, pneumonia septicaemia pericarditis, tumours vagal cause - high endurance atheletes ```
33
lone (idiopathic) AF
absence of any heart disease and no evidence of ventricular dysfunction could be genetic symptoms - palpitations, pre-syncope, syncope, chest pain, dyspnoea, sweatiness, fatigue
34
AF with slow ventricular rate
may co-exist with periods of fast VR | a pacemaker may be required
35
rate control of AF
digoxin beta blocker verapamil diltiazem
36
rhythm control of AF
``` pharmacological cardioversion direct current cardioversion anti-arrhythmic drugs catheter ablation of atrial focus/pulmonary veins maze procedure ```
37
``` anti-arrhythmic drugs class 1 ```
reducing Na channel current | lignocaine, quinidine, flecainide, propafenone
38
``` anti-arrhythmic drugs class 2 ```
beta adrenergic antagonists | verapamil
39
``` anti-arrhythmic drugs class 3 ```
action potential prolongation | amiodarone, sotalol, dronedarone
40
``` anti-arrhythmic drugs class 4 ```
Ca channel antagonists | verapamil
41
CHA2DS2VASc score
``` congestive heart failure hypertension >75 diabetes mellitus stroke valscular disease 65-74 sex - female ```
42
atrial flutter
``` rapid and regular form of atrial tachycardia usually paroxysmal macro-reentrant circuit may result in thromboembolism saw tooth f wave ```