Arrythmia Flashcards

(31 cards)

1
Q

AV block poem

A

R is far from P- first degree

Longer longerr longerrr drop- wenkebac

Some Ps dont get through- Mobitz 2

P and Q dont agree- third degree

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2
Q

QRS morphology in AV2

A

Mobitz 1= normal

Mobitz 2 = usually abnormal

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3
Q

Cause of mobitz 1

A

DIGOXIN

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4
Q

Cause of Mobitz 2

A

AVN ischemia

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5
Q

HR in 3°AV block

A

30-60

WIDE QRS COMPLEXES

AV dissociation

QRS and T in opposite direction

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6
Q

Total arrythmia types (14)

A
Sinus Bradycardia
Sinus arrest
1AVB
Mobitz1
Mobitz2
3AVB
Sinus tachycardia 
Unifocal atrial tachycardia
Multifocal atrial tachycardia 
Atrial flutter
Afib
PSVT
Vtach
Vfib
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7
Q

Max conduction rate of AVN

A

250

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8
Q

Atrial ectopic cause and rate

A

Hypoxia in COPD

100-250

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9
Q

Atrial flutter presentation and speed

A

Palpitations

250-350

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10
Q

Afib cause

A

Stretching of atrial fibres

LAE
CMP
ASD

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11
Q

DOC stable Afib and further mx

A

Beta blockers

(If not allowed then CCB/Digoxin)

MOA: block avn further so as to stop arrythmia from reaching ventricles

F/b convert afib into sinus rhythm

By either 1) Ibutilide 2)DC shock

Maintenance of sinus rhythm DOC= AMIODARONE

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12
Q

AFib + WPW syndrome C/I and doc

A

C/I CCB/BB — accentuated bundle of k

DOc- PROCAINAMIDE IBUTILIDE AMIODARONE

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13
Q

Afib most risky factor

A

Thrombo-embolism

Therefore always do TEE BEFORE CARDIOVERSION

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14
Q

PSVT ecg characteristic

A

P waves superimposed by QRS
(So usually no P)

If P wave present: in reverse direction (CIRCUS WAVE) d/t retrograde stimulation through bundle

HR regular and fast

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15
Q

Frog sign is positive in

A

PSVT

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16
Q

PSVT Mx

A

Carotid massage—6mg adenosine—12mg adenosise—DC shock

Doc maintainence: VERAPAMIL

Doc prophylaxis : class 2 and 4 antiarr

17
Q

V tach ecg characteristics

A

> 3 consecutive ectopics

HR: 100-250

Wide QRS

Qrs and T in opposite direction

18
Q

Rx all Ventricular Tachycardia

Except Vtach<30s = AMIODARONE

A

IMMEDIATE DC shock 350J

DOC: AMIODARONE»LIGNOCAINE

19
Q

TDP RX

20
Q

Causes of 1AVB

A

Drugs (mc)
K+ / Mg++
Rheumatic Fever

21
Q

Causes of 3AVB

A

Degenerative HD (mc)
Post sx
Congenital SLE

22
Q

Pathology of tachyarrythmia

A

Defect in impulse production
(Enhanced automaticity of pacemaker cells-IHD/deg/electrolyte)

Defect in impulse propagation

a) Re-entry
b) After depolarisation

23
Q

Differentiate Atrial and Ventricular tach

A

QRS normal. QRS wide
Carotid massage. -
- Variab H. Sounds

24
Q

Doc AFib + HF

A

DIGOXIN

Only antiarrythmic with inotropic action

25
Common in elderly in ICU?
MAT (Sepsis—-cytokines—pacemaker ++) MAT NEVER CAUSES HYPOTENSION
26
M.c tachyarrythmia in ICU Pt.
Afib
27
Causes of AFib
Young: -Valvular HD Holiday heart syndrome DCMP Old: Degenerative HD Thyrotoxicosis
28
Capture beats and Fusion beats
Both give evidence of VTACH CB- Normal beat in VTach (p followed by qrs) - that is impulse from atria has finally reached the ventricle, which was constantly getting depolarised by ectopics FB- complexes formed due to fusion of atrial impulses and ventricular ectopics
29
Sustained VT timing
30 seconds
30
Reperfusion Arrythmia treatment
Atropine & lignocaine (Aka AIVR- accelerated idioventricular rhythm) D/t sudden reperfusion of blocked coronary
31
AF thrombus risk (CHA2DS2 Vasc) If > 2 increased risk
``` CHF HTn Age>75 (2points) 64-75= 1 point DM Stroke H/o (2 points) Sex: F Vascular D/o ```