Bariatics And Robotics Flashcards

1
Q

At what point does BMI become a problem?

A

Morbid obesity

> 40 is automatically ASA III

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2
Q

Endocrine function of adipose tissue

A

• A reserve energy source & insulates vital organs
• Produces cytokines, chemokines and steroids

-result is chronic inflammation

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3
Q

Two types of adipose distribution

A

Android- central/trunkal-associated with cardiovascular risk

Gynecoid- all in the legs
NO increased CV risk

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4
Q

Leptin

A

Hormone signaling to CNS/hypothalamus

Produced in adipose cells

-signals fullness-but you can become resistant with the high circulating levels found in obesity

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5
Q

Adiponectin

A

Hormone produced in adipose

Signals CNS

Modulates insulin sensitivity- levels are low in obesity

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6
Q

Ghrelin

A

Hunger hormone of the stomach

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7
Q

Arcuate nucleus

A

In the hypothalamus

Receives and transmits signals based on information received from adipocytes

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8
Q

3 components of the pathophysiology of obesity

A

-leptin/adiponectin/ghrelin

-arcuate nucleus

-efferent system

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9
Q

What do we do with extra calories?

A

Converted to triglycerides and stored in adiopocytes

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10
Q

Explain adipocytes accommodation of extra triglycerides in obesity

A

They EXPAND until a BMI of 40

Then the PROLIFERATE to accept more energy

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11
Q

Percentage of energy needed for resting metabolic rate

A

60%

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12
Q

Define hypopnea

A

50% reduction in airflow for >10 seconds

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13
Q

Differentiate central sleep apnea and obstructive sleep apnea

A

-lack of brain signaling to respiratory muscles-usually in presence of severe systemic illness

-partial or complete periodic obstruction of the upper airway

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14
Q

3 criteria in obesity hypoventilation syndrome

A

BMI >30

PaCO2 over 45 mmhg

No alternative explanation

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15
Q

How is OSA classified?

A

Number of hypopneic events in an hour

Mild- 5-14

Moderate- 15-29

Severe > 30

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16
Q

How common is OHS in the morbidly obese?

A

Only 5-10%

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17
Q

Main difference between OHS and OSA

A

OSA is transient at night and OHS results in chronic changes in PaCO2 and PAO2

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18
Q

Obesity airway concerns

A

DECREASED pharyngeal area

Excess hypopharyngeal tissue

Posteriorly displaced hyoid bone

Difficulty laying flat

High incidence of GERD/OSA

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19
Q

Pulmonary compliance in obesity

A

Decreases exponentially with increasing BMI

chest wall lung compliance DECREASES to 35% of predicted values

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20
Q

Most commonly reported respiratory issues in obese patients

A

Decreased FRC and ERV

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21
Q

Most sensitive indicator of obesity affects on pulmonary function

22
Q

Change in cardiac output with obesity

A

Increases buy 20-30ml/kg of excess fat

Ex. 1kg of fat increases CO by 100ml/min

SO-increased blood volume and increased LV workload

23
Q

Impact of obesity on cardiovascular plumbing

A

Accelerated atherosclerosis
-fat can infiltrate cardiac tissue and the conduction system

24
Q

Fluid administration in obesity

A

Easy to overload with rapid administration
-can cause intraop ventricular failure

25
DVT risk in obesity
2 fold greater risk -immobility and inflammation
26
If someone is chair bound what are we worried about?
Unidentified cardiac disease -if they don’t exert themselves how do we know they don’t have exertional Dyspnea?
27
Hypertension in obesity
Renin-angiotensin system • ↑ levels of angiotensin, aldosterone, angiotensin converting enzyme: Hypertension • 5% reduction in body weight leads to significant reduction in levels
28
GI highlights in obesity
Give bicitra! -delayed gastric emptying, increased gastric volume and more acidic Drug metabolism can be adversely affected
29
Liver in obesity
Elevated LFTs Fatty infiltration (NAFLD) Inflammation Focal necrosis Cirrhosis
30
Metabolic syndrome
Cluster of metabolic abnormalities- Diagnosis-central obesity + four factors • Glucose intolerance • HTN • Dyslipidemia (raised triglycerides, HDL) • Associated with increased risk of CV disease
31
Renal and obesity
Increased Renal blood flow and GFR
32
Pharmacology of obesity
Increased Vd- consider with loading dose -don’t overload but be prepared with more
33
Good indicator of systemic changes with obesity
Do they have systemic HTN?
34
OSA implications
May require inpatient management for a “day surgery” Neck circumference > 40cm is strong predictor of OSA RAPID desaturation
35
Best predictor of difficult DL in obese patients
Neck > 40cm
36
Impact of BMI on DL view
Minimal, where is the fat?
37
Most common neuropathy after bariatric surgery
Carpal tunnel Watch positioning!!
38
Positioning in obesity
Rhabdo! Check weight guideline on table Careful in supine positioning
39
Stats on preoxygenation in obese patient
GA in an obese patient reduces FRC by 50% (compared to 20% in non-obese) Decreased FRC and increased oxygen need**
40
Only proven vent change to help with hypoventilation in obesity
Peep
41
Postop care of the obese patient
Ensure reversal Extubate upright CPAP NO intrathecal narcotics
42
Extubation criteria obesity
• 5-second head lift: following commands • Hemodynamically stable • Normothermic • TOF >0.9 • 10>RR>30 • SpO2 >95% • Tidal volume >5mL/kg (IBW) • Acceptable pain control
43
Consideration in sleeve gastrectomy
Make sure anything in the esophagus is not stapled… dilators/bougies/ogts
44
Two types of bariatric surgery
Restrictive vs malabsorptive
45
Roux-en-y
Restrictive and malabsorptive Most effective Worst complications- low rate
46
Most common cause of mortality in bariatric surgery
Anastomosis leak
47
Bariatric surgery complications
venous thromboembolism: PE Bowel obstruction Vitamin deficiency Intestinal stenosis Anastomosis leak* most common
48
What to never fucking do during robotic surgery
Move the patient with the robot is in the patient -always communicate with the surgeon
49
Robotic surgery and patient safety
Place a table over the patients face! Robots don’t feel pressure
50