Biopsychology Flashcards

(50 cards)

1
Q

The Biological Approach to Understanding Behaviour AO1+2

A

AO1:
• The biological approach explains behavior in terms of genetics, neurochemistry, and brain structure.
• It assumes behavior is influenced by biological processes such as evolution, genes, and neurotransmitters.

AO2:
• For example, schizophrenia is linked to an imbalance of dopamine, a neurotransmitter that affects brain function.

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2
Q

The Biological Approach to Understanding Behaviour AO3

A

AO3:
✔ Strength: Scientific methods (e.g., brain scans, twin studies) provide objective, empirical evidence.
✔ Strength: Led to practical applications, such as drug therapies for mental disorders.
✘ Weakness: Reductionist—ignores environmental and cognitive factors in behavior.
✘ Weakness: Deterministic—implies little free will in behavior.

Synoptic Link: Links to abnormal psychology, where biological explanations are used to understand disorders.
Mnemonic: BIO = Behavior Inherited & Observed

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3
Q

The Nervous System and the Endocrine System AO1+2

A

AO1:
• The nervous system transmits messages through neurons, while the endocrine system releases hormones to regulate body functions.
• The nervous system is divided into the central nervous system (CNS) and peripheral nervous system (PNS).

AO2:
• The fight or flight response involves the endocrine system releasing adrenaline to prepare the body for action.

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4
Q

The Nervous System and the Endocrine System AO3

A

AO3:
✔ Strength: Both systems work together to maintain homeostasis.
✔ Strength: Research on the endocrine system has led to hormone treatments (e.g., insulin for diabetes).
✘ Weakness: The interaction between the two systems is complex and difficult to isolate in studies.
✘ Weakness: Animal studies may not always generalize to humans.

Synoptic Link: Links to stress psychology, showing how biological systems contribute to the stress response.
Mnemonic: Nervous = Fast, Endocrine = Enduring

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5
Q

The Structure and Function of Neurons AO1+2

A

AO1:
• Neurons transmit electrical and chemical signals. The three types are sensory, motor, and relay neurons.
• Neurons consist of dendrites, axons, and synapses.

AO2:
• The action potential travels down the axon to release neurotransmitters at the synapse.

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6
Q

The Structure and Function of Neurons AO3

A

AO3:
✔ Strength: Understanding neurons has led to treatments for neurological disorders (e.g., Parkinson’s disease).
✔ Strength: Brain scans allow us to study neuron function in detail.
✘ Weakness: Research on neurons is complex and difficult to study in humans.
✘ Weakness: Some findings rely on animal studies, which may lack human generalizability.

Synoptic Link: Links to biological psychology, exploring how neuron function affects behavior.
Mnemonic: DAX = Dendrite, Axon, Synapse

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7
Q

Synaptic Transmission AO1+2

A

AO1:
• Synaptic transmission is the process of one neuron communicating with another via neurotransmitters.
• Excitatory neurotransmitters (e.g., dopamine) increase neural activity; inhibitory neurotransmitters (e.g., GABA) reduce it.

AO2:
• Low levels of serotonin are linked to depression, influencing antidepressant medication development.

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8
Q

Synaptic Transmission AO3

A

AO3:
✔ Strength: Neurotransmitter research has led to successful drug treatments (e.g., SSRIs for depression).
✔ Strength: Scientific methods (e.g., fMRI scans) provide empirical evidence.
✘ Weakness: Reductionist—focuses only on neurotransmitters, ignoring environment and cognition.
✘ Weakness: Causation issues—correlation does not prove that neurotransmitters cause mental disorders.

Synoptic Link: Links to abnormal psychology (e.g., depression and schizophrenia).
Mnemonic: SYN = Signal Yielded Neuron

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9
Q

The Autonomic Nervous System (ANS) AO1+2

A

AO1:
• The ANS controls involuntary bodily functions (e.g., breathing, digestion).
• It has two branches: sympathetic (fight or flight) and parasympathetic (rest and digest).

AO2:
• When faced with danger, the sympathetic nervous system increases heart rate and dilates pupils.

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10
Q

The Autonomic Nervous System (ANS) AO3

A

AO3:
✔ Strength: Explains physiological reactions to stress and survival instincts.
✔ Strength: Research has led to treatments for anxiety (e.g., beta-blockers to reduce heart rate).
✘ Weakness: Overemphasis on biological mechanisms—ignores psychological factors in stress response.
✘ Weakness: Not all stress responses fit the fight or flight model (e.g., some people freeze).

Synoptic Link: Links to stress and anxiety disorders, explaining physiological symptoms.
Mnemonic: ANS = Automatic Nervous System

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11
Q

The Central Nervous System (CNS) AO1+2

A

AO1:
• The CNS consists of the brain and spinal cord, responsible for processing information and controlling behavior.
• The brain is divided into four lobes: frontal, parietal, occipital, and temporal.

AO2:
• Damage to the frontal lobe (e.g., Phineas Gage) leads to personality changes.

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12
Q

The Central Nervous System (CNS) AO3

A

AO3:
✔ Strength: Brain scanning techniques (e.g., fMRI, PET scans) provide objective data.
✔ Strength: Localized brain damage studies support CNS function.
✘ Weakness: Brain research is often correlational—causation is difficult to determine.
✘ Weakness: Ethical issues in brain damage studies limit research possibilities.

Synoptic Link: Links to neuropsychology and brain disorders.

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13
Q

The Brain and Its Structures AO1+2

A

AO1:
• The brain is divided into four lobes:
• Frontal lobe (decision-making, movement, speech).
• Parietal lobe (processing sensory info).
• Occipital lobe (vision).
• Temporal lobe (memory, emotion, and hearing).
• The limbic system includes the amygdala (emotion) and hippocampus (memory formation).

AO2:
• Damage to Broca’s area (left frontal lobe) leads to expressive aphasia, impairing speech production.

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14
Q

The Brain and Its Structures AO3

A

AO3:
✔ Strength: Brain scans provide scientific evidence for brain structures.
✔ Strength: Real-world applications—neurosurgery (e.g., for epilepsy) based on brain function research.
✘ Weakness: Research on brain damage is often based on case studies, limiting generalizability.
✘ Weakness: The brain is highly interconnected, making it difficult to study regions in isolation.

Synoptic Link: Links to cognitive neuroscience, integrating psychology and biology.
Mnemonic: F-POT = Frontal, Parietal, Occipital, Temporal

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15
Q

Plasticity and Functional Recovery AO1+2

A

AO1:
• Plasticity: The brain’s ability to adapt by forming new neural connections.
• Functional recovery: The brain rewires itself after injury by recruiting undamaged neurons.

AO2:
• Maguire et al. (2000) found that London taxi drivers had more developed hippocampi due to navigation training.

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16
Q

Plasticity and Functional Recovery AO3

A

AO3:
✔ Strength: Supported by brain imaging studies showing changes after learning.
✔ Strength: Used in neurorehabilitation (e.g., stroke recovery therapy).
✘ Weakness: Plasticity declines with age, limiting its effectiveness in adults.
✘ Weakness: Negative plasticity can occur, e.g., phantom limb syndrome after amputation.

Synoptic Link: Links to learning theories, showing how experience shapes the brain.
Mnemonic: Plastic Brain = Flexible Brain

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17
Q

The Role of Hormones in Behavior AO1+2

A

AO1:
• Hormones are chemical messengers released by the endocrine system, influencing behavior.
• Examples:
• Testosterone (aggression).
• Oxytocin (bonding & trust).

AO2:
• Dabbs et al. (1995) found high testosterone levels in violent criminals.

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18
Q

The Role of Hormones in Behavior AO3

A

AO3:
✔ Strength: Research has practical applications (e.g., hormone therapy).
✔ Strength: Scientific methods (e.g., blood tests) provide reliable hormone measurements.
✘ Weakness: Reductionist—ignores environmental and social influences on behavior.
✘ Weakness: Correlation does not prove causation (e.g., testosterone and aggression).

Synoptic Link: Links to aggression studies in social psychology.
Mnemonic: HORMone = HORMonal Messages

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19
Q

The Fight or Flight Response AO1+2

A

AO1:
• Triggered by acute stress, activating the sympathetic nervous system.
• Adrenaline increases heart rate, dilates pupils, and boosts oxygen to muscles.

AO2:
• Taylor et al. (2000) found that females often use a tend-and-befriend response instead.

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20
Q

The Fight or Flight Response AO3

A

AO3:
✔ Strength: Explains stress-related health conditions (e.g., high blood pressure).
✔ Strength: Evolutionary advantages—helped early humans survive threats.
✘ Weakness: Not universal—some people show a freeze response instead.
✘ Weakness: Chronic activation can lead to heart disease.

Synoptic Link: Links to stress psychology, explaining physiological symptoms of anxiety.
Mnemonic: FFF = Fight, Flight, or Freeze

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21
Q

Localisation of Function in the Brain AO1+2

A

AO1:
• Different brain areas control specific functions:
• Motor cortex (voluntary movements).
• Somatosensory cortex (touch & pain).
• Visual cortex (sight).
• Auditory cortex (hearing).

AO2:
• Damage to Wernicke’s area (left temporal lobe) causes Wernicke’s aphasia, affecting language comprehension.

22
Q

Localisation of Function in the Brain AO3

A

AO3:
✔ Strength: Brain scans confirm function-specific activation.
✔ Strength: Neurosurgical evidence supports brain region specialisation.
✘ Weakness: The brain is highly interconnected, challenging strict localisation theories.
✘ Weakness: Plasticity suggests functions can shift after injury.

Synoptic Link: Links to neuroscience and neurorehabilitation.
Mnemonic: M-S-V-A = Motor, Sensory, Visual, Auditory

23
Q

Biological Rhythms: Circadian Rhythms AO1+2

A

AO1:
• Circadian rhythms follow a 24-hour cycle, e.g., the sleep-wake cycle.
• Controlled by the suprachiasmatic nucleus (SCN) in the hypothalamus.

AO2:
• Siffre’s cave study (1975) showed that without external cues, circadian rhythms lengthened to ~25 hours.

24
Q

Biological Rhythms: Circadian Rhythms AO3

A

AO3:
✔ Strength: Practical applications (e.g., shift work research on health risks).
✔ Strength: Supported by strong experimental evidence.
✘ Weakness: Individual differences in circadian cycles.
✘ Weakness: Lab studies lack ecological validity.

Synoptic Link: Links to sleep psychology and melatonin research.
Mnemonic: CIRCADIAn = CIRCle Around the Day

25
Biological Rhythms: Ultradian and Infradian Rhythms AO1+2
AO1: • Ultradian rhythms are less than 24 hours (e.g., sleep stages). • Infradian rhythms last longer than 24 hours (e.g., menstrual cycle).   AO2: • Dement & Kleitman (1957) found that REM sleep is linked to dreaming.
26
Biological Rhythms: Ultradian and Infradian Rhythms AO3
AO3: ✔ Strength: EEG studies support sleep cycle stages. ✔ Strength: Menstrual cycle research has medical applications (e.g., fertility treatments). ✘ Weakness: Individual differences in sleep cycles. ✘ Weakness: Menstrual studies often lack control over external influences.   Synoptic Link: Links to biological rhythms in health (e.g., sleep disorders).
27
The Endocrine System and Glands AO1+2
AO1: • The endocrine system regulates body functions via hormones. • Major glands: • Pituitary gland: Master gland, controls other glands. • Adrenal gland: Releases adrenaline (fight or flight). • Ovaries/Testes: Control reproductive hormones.   AO2: • Melatonin from the pineal gland regulates sleep-wake cycles.
28
The Endocrine System and Glands AO3
AO3: ✔ Strength: Scientific evidence from blood tests confirms hormone function. ✔ Strength: Practical applications (e.g., hormone therapy). ✘ Weakness: Reductionist—ignores social/environmental influences. ✘ Weakness: Ethical concerns over hormonal manipulation (e.g., testosterone therapy).   Synoptic Link: Links to stress research (e.g., cortisol in chronic stress). Mnemonic: P-A-O = Pituitary, Adrenal, Ovaries/Testes
29
The Suprachiasmatic Nucleus (SCN) and Sleep-Wake Cycle AO1+2
AO1: • The SCN, located in the hypothalamus, regulates circadian rhythms. • Responds to light cues via the optic nerve.   AO2: • Morgan (1995) removed the SCN in hamsters, disrupting sleep-wake cycles.
30
The Suprachiasmatic Nucleus (SCN) and Sleep-Wake Cycle AO3
AO3: ✔ Strength: Real-world applications (e.g., light therapy for sleep disorders). ✔ Strength: Animal research supports SCN’s role. ✘ Weakness: Animal studies may not generalise to humans. ✘ Weakness: External cues (e.g., social factors) also influence sleep-wake cycles.   Synoptic Link: Links to biological rhythms and shift work research. Mnemonic: SCN = Sleep Control Neuron
31
Exogenous Zeitgebers and Endogenous Pacemakers AO1+2
AO1: • Endogenous pacemakers: Internal biological clocks (e.g., SCN). • Exogenous zeitgebers: External cues affecting rhythms (e.g., light, temperature).   AO2: • Campbell & Murphy (1998) found that light exposure to the skin influenced sleep-wake cycles.
32
Exogenous Zeitgebers and Endogenous Pacemakers AO3
AO3: ✔ Strength: Supports nature-nurture interaction in biological rhythms. ✔ Strength: Practical applications in jet lag treatments. ✘ Weakness: Studies often lack control over other influences (e.g., social factors). ✘ Weakness: Individual differences in responses to zeitgebers.   Synoptic Link: Links to circadian rhythm disruptions in modern society (e.g., blue light from screens). Mnemonic: EXogenous = External, ENdogenous = INternal
33
Hemispheric Lateralisation AO1+2
AO1: • The left hemisphere controls language and logic. • The right hemisphere controls spatial ability and creativity.   AO2: • Sperry (1968) studied split-brain patients—left hemisphere processed language, right recognised faces.
34
Hemispheric Lateralisation AO3
AO3: ✔ Strength: Supported by brain scans showing functional differences. ✔ Strength: Helps understand brain damage effects (e.g., strokes). ✘ Weakness: Overly simplistic—hemispheres work together. ✘ Weakness: Individual variation in lateralisation.   Synoptic Link: Links to brain plasticity—functions can shift after damage. Mnemonic: L = Language, R = Recognition
35
Split-Brain Research (Sperry’s Study) AO1+2  
AO1: • Split-brain surgery (corpus callosum severed) used to treat epilepsy. • Sperry’s tests showed language dominance in the left hemisphere.   AO2: • Patients shown images in the right visual field could describe them; those in the left visual field could not.
36
Split-Brain Research (Sperry’s Study) AO3
AO3: ✔ Strength: Strong experimental control. ✔ Strength: Supports lateralisation theory. ✘ Weakness: Small sample—only severe epilepsy patients. ✘ Weakness: Some functions not strictly lateralised.   Synoptic Link: Links to brain function specialisation research. Mnemonic: SPLIT = Sperry’s Patients Lacked Inter-Hemisphere Transfer
37
Brain Scanning Techniques AO1+2
AO1: • fMRI: Measures blood flow changes. • EEG: Detects electrical activity. • ERPs: Measures brain response to stimuli. • Post-mortem: Examines brain after death.   AO2: • fMRI scans reveal active areas during tasks (e.g., memory tests).
38
Brain Scanning Techniques AO3
AO3: ✔ Strength: fMRI and EEG provide non-invasive brain data. ✔ Strength: Post-mortems offer detailed analysis. ✘ Weakness: fMRI is expensive and indirect. ✘ Weakness: EEG lacks spatial resolution.   Synoptic Link: Links to cognitive neuroscience developments. Mnemonic: fMRI = Flow, EEG = Electrical, ERP = Event-Related, PM = Post-Mortem
39
Recovery After Brain Trauma AO1+2
AO1: • Functional recovery: Brain reorganises itself after injury. • Axonal sprouting: New neural connections form.   AO2: • Danelli (2013) found a child’s right hemisphere took over left hemisphere functions after damage.
40
Recovery After Brain Trauma AO3
AO3: ✔ Strength: Supported by stroke recovery research. ✔ Strength: Real-world applications in neurorehabilitation. ✘ Weakness: Recovery varies—age and therapy matter. ✘ Weakness: Can cause maladaptive plasticity (e.g., phantom limb pain).   Synoptic Link: Links to plasticity research.
41
Synaptic Transmission AO1+2
AO1: • Synaptic transmission is how neurons communicate. • Process: 1. Action potential arrives at the presynaptic neuron. 2. Vesicles release neurotransmitters into the synaptic cleft. 3. Neurotransmitters bind to receptors on the postsynaptic neuron. 4. Excitatory or inhibitory response occurs.   AO2: • SSRIs (antidepressants) work by increasing serotonin levels in the synapse.
42
Synaptic Transmission AO3
AO3: ✔ Strength: Scientific support (brain imaging confirms synaptic activity). ✔ Strength: Explains effects of psychoactive drugs (e.g., dopamine in schizophrenia). ✘ Weakness: Reductionist—ignores wider brain networks. ✘ Weakness: Lab research may lack real-world validity.   Synoptic Link: Links to mental disorders (e.g., depression and schizophrenia). Mnemonic: “A Very New Robot Eats” → Action potential, Vesicles, Neurotransmitters, Receptors, Excitatory/Inhibitory
43
Role of Neurotransmitters AO1+2
AO1: • Neurotransmitters = chemical messengers in the brain. • Excitatory: Increase activity (e.g., dopamine). • Inhibitory: Decrease activity (e.g., serotonin).   AO2: • Low serotonin linked to depression (explains use of SSRIs). • High dopamine linked to schizophrenia (explains use of antipsychotics).  
44
Role of Neurotransmitters AO3
AO3: ✔ Strength: Drug treatments based on neurotransmitter theory (e.g., SSRIs). ✔ Strength: fMRI scans support neurotransmitter effects on brain function. ✘ Weakness: Correlational—low serotonin may be a symptom, not a cause. ✘ Weakness: Ignores psychological/social influences on mental health.   Synoptic Link: Links to biological explanations of mental illness. Mnemonic: “Sad People Dance Slowly” → Serotonin = Depression, Dopamine = Schizophrenia
45
The Central Nervous System (CNS) AO1+2
AO1: • Made up of the brain and spinal cord. • Brain: Controls thoughts, emotions, and actions. • Spinal cord: Relays messages between the brain and body.   AO2: • Damage to the spinal cord can cause paralysis, showing its importance in movement.  
46
The Central Nervous System (CNS) AO3
AO3: ✔ Strength: Scientific research (brain scans confirm CNS function). ✔ Strength: Explains effects of injury on behaviour. ✘ Weakness: Reductionist—ignores role of environment. ✘ Weakness: Studies often on brain-damaged patients, limiting generalisability.   Synoptic Link: Links to localisation of function in the brain. Mnemonic: CNS = Central for Nervous System Communication
47
The Peripheral Nervous System (PNS) AO1+2
AO1: • The PNS connects the CNS to the rest of the body. • Two branches: 1. Somatic NS (voluntary movement). 2. Autonomic NS (automatic functions like heart rate).   AO2: • Autonomic NS split into: • Sympathetic (fight or flight). • Parasympathetic (rest and digest).  
48
The Peripheral Nervous System (PNS) AO3
AO3: ✔ Strength: Explains stress responses (e.g., increased heart rate in danger). ✔ Strength: Practical applications (e.g., treating PTSD by controlling autonomic response). ✘ Weakness: Ignores psychological factors in stress. ✘ Weakness: Lab studies on autonomic NS lack ecological validity.   Synoptic Link: Links to fight or flight response. Mnemonic: S for Stress (Sympathetic), P for Peace (Parasympathetic)
49
Neuroplasticity Over the Lifespan AO1+2
AO1: • Neuroplasticity: The brain’s ability to change and adapt. • New neural connections form through learning and experience. • Greater plasticity in childhood, but still occurs in adults.   AO2: • Maguire et al. (2000): London taxi drivers had larger hippocampi due to navigation training.
50
Neuroplasticity Over the Lifespan AO3
AO3: ✔ Strength: Real-world applications (stroke recovery, rehabilitation). ✔ Strength: Supported by research on learning effects. ✘ Weakness: Not all brain functions can recover fully. ✘ Weakness: Age limits plasticity—more difficult in older adults.   Synoptic Link: Links to functional recovery after trauma.