Biopsychology Flashcards

(61 cards)

1
Q

Central Nervous System

A

-2 main functions; control behaviour and regulate physiological processes

-receives messages from sensory receptors, and sends messages to muscles and glands

-2 parts; brain and spinal cord

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2
Q

Brain

A

-centre of all conscious awareness
-outer layer=cerebral cortex ; where our higher mental functions occur,

-divided into 2 hemispheres and 4 key lobes. Each lobe spans both hemispheres;

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3
Q

Spinal Cord

A

-allows brain to monitor/regulate bodily processes, and to coordinate voluntary movements

-connected to parts of body by pairs of spinal nerves, which carry messages

-also contains circuits of nerve cells which perform simple reflexes without using the brain

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4
Q

Peripheral Nervous System

A

-all nerves outside the CNS
-relays nerve impulses from CNS to rest of body and back again
-divided into somatic NS and autonomous NS

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5
Q

Somatic Nervous System

A

-both sensory and motor neurons; receives information from sensor receptors and controls muscles
-control voluntary movements
-involved in reflex actions without CNS involvement

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6
Q

Autonomic Nervous System

A

-regulate involuntary actions (e.g heartbeat, digestion etc)
-transmits information to and from organs
-made up of 2 parts; sympathetic NS and parasympathetic NS

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7
Q

Sympathetic Nervous System

A

-helps deal with emergencies
-e.g increasing HR, BP etc
-neurons from SNS travel to virtually every organ/ gland in the body, in case of rapid action being needed when under threat
-fight or flight

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8
Q

Parasympathetic Nervous System

A

-relaxes bodily systems again once danger has passed
-slows HR down, decreased BP, restarts digestion again
-‘rest and digest’

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9
Q

Neurons consist of

A

Dendrites, Cell body, Axon, Myelin Sheath

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10
Q

Dendrites

A

receive signals from other neurons+ sensory reception

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11
Q

Axon

A

transmit electrical impulses, or action potentials, away from a neuron’s cell body

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12
Q

Myelin Sheath

A

improves speed of transmission

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13
Q

Sensory Neuron

A

-carry signals from receptors to spinal cord + brain
-found in places such as eyes, ears, tongue, and skin
-convert sensory info received into nerve impulses; these turn into sensations when they reach the brain

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14
Q

Relay Neuron

A

-carry messages from one part of CNS
-situated in spinal cord+brain

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15
Q

Motor Neurons

A

-carry signals from CNS to effectors
-connected to an effector (muscles/gland), stimulating it when an impulse us passed on
-release neurotransmitters when released, which, bind to receptors on muscle and trigger a response, leading to muscle movement

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16
Q

Outline the process of synaptic transmition

A

Action potential reaches axon terminal where it stimulates vesicles to release neurotransmitters. These diffuse across the synapse- activate receptor sites on the dendrite f the next neuron, unused neurotransmitters return to vesicle in a reuptake

2 types of neurotransmitters

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17
Q

Endocrine System

A

-network of glands, manufacturing and secreting hormones into bloodstream
-regulated by negative feedback

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18
Q

Negative Feedback

A

-signal sent to gland in the form of a ‘releasing hormone’
-this then either signals another gland to release its hormone, or effects a change itself
-as bloodstream levels of this hormone rise, this is detected by hypothalamus, which shuts down secretion of the releasing hormone, stopping the system until levels drop again

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19
Q

Pituitary Gland

A

-controlled by hypothalamus; secretes many hormones, some of which affect other glands
-controls other glands

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20
Q

Adrenal Gland

A

-helps trigger fight or flight
-Adrenal Medulla -> increases adrenaline
-Adrenal Cortex -> releases cortisol

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21
Q

Amygdala

A

responsible for emotional responses

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22
Q

Fight or Fight response process

A

stressor detected
assessed for threat by amygdala= responsible for emotional responses
If threat -> sends distress signal to hypothalamus
Hypothalamus will activate sympathetic branch of nervous system
SNS sends signal to Adrenal Medulla which releases adrenaline

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23
Q

Effects of adrenaline

A

pupils dilate -> improved vision of surroundings

shows digestion -> wasted energy

increased HR/ respiration/ blood pressure -> get more O2 to muscles

hairs to stand on end/ sweat -> cool you down

reduce blood flow to skin -> unimportant

reduce glycogen - energy boost

after c.20mins
body reverts to parasympathetic branch, everything reverses

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24
Q

Visual cortex

A

in occipital lobe
spans both hemispheres
right hemispheres receives visual info from left side of visual field and vice versa
different parts are responsible for colour, shape, movement etc

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25
Visual cortex processes
begins in retina at back of eye Light enters -> strikes photoreceptors -> causing nerve impulses to be transmitted to the brain via the optic nerve Terminate in the thalamus; relay station which pauses info onto visual cortex
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Auditory Areas
Mainly within temporal lobes on both sides of the brain, where we find auditory cortex
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Auditory Areas processes
Pathway begins in cochlea (inner ear) -> sound waves converted to nerve impulses -> impulses travel via auditory nerve -> passes through brain stem which does basic decoding such as duration and intensity of sound -> thalamus -> relay station and further processing -> auditory cortex
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Motor Cortex
Voluntary motor movements Frontal lobe of brain, along the precentral gyrus One in each hemisphere of brain, each controlling muscles on the other side of the body Different parts of it exert control over different body parts; arranged logically
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Somatosensory Cortex
Detects sensory events from different parts of the body Located in parietal lobe, found in the postcentral gyrus, where touch information is processed One somatosensory cortex in each hemisphere, each receiving sensory information from the opposite half of the body Somatosensory cortex uses sensory info from the skin to produce sensations of touch, pain, pressure, temp which it localises to specific body regions
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Broca's Area
named after Tan who was named this because that was the only syllable the patient could express He was able to understand spoken language but was unable to speak or express his thoughts in writing Broca studied 8 other patients who had the same language deficit and had lesions in their left frontal hemisphere. Those with lesions in right hemisphere did not have same problems Therefore there is a language centre in left hemisphere that is critical for speech production
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Fedorenko et al (Broca)
found 2 regions in Broca Area, one for language and the other for demanding cognitive tasks.
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Wernicke's Area
Wernicke found that patients with a lesion in the Wernicke's area could speak but were unable to understand lang The sensory regions located in Wernicke's area is close to regions of the brain responsible
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Language Centres AO3
✔ Support from Aphasia (blindness) studies -research into patients with Broca's Aphasia (inability to produce language) and Wernicke's aphasia (inability to understand) links them to varying levels of damage in the corresponding areas each time -this supports the idea that those specific brain areas at responsible ✘ Subsequent research suggests Broca's research into Tan may have been too vague -Dronkers re-examined the preserved brain of Tan, and found areas around Broca's area were also damaged -This is important as damage solely to Broca's areas rarely causes permanent loss of language production Therefore the founding research into localisation of language may be flawed ✘ Opposed by Equipotentiality Theory -Lashley argued that higher functions such as language and memory were relatively new to the human brain, and that other areas could take over these skills should the original areas become damaged -Support for this comes from JW; Broca's area badly damaged, and lost the ability to speak. Within a year he had regained his language skills; developed a new Broca's area in right hemisphere Therefore many functions may not be localised in the brain at all ✘ Overemphasises localisation, ignoring the importance of communication -Dejerine: had a patient with Wernicke's aphasia - Wernicke's area was undamaged -Auditory cortex was undamaged -but neural pathway connecting the two was damaged Therefore suggests that connectivity between areas of the brain is equally important, opposing localisation
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Hemispheric Lateralisation
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Corpus Callosum
bundle of nerve fibres that connects the two hemispheres
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Lateralisation AO3
✔ Increases Neural Processing (capacity) -if one hemisphere is responsible for one task, the other hemisphere can focus on other things e.g Rogers' Chickens -Lateralisation in their brains allows them to simultaneously search for food and watch for predators Therefore suggests a potential evolutionary advantage to hemispheric lateralisation ✘ Morfit and Weekes - Appears to have a negative payoff in other area -Left handers (high RH functioning) had higher incidences of immune disorders within their immediate families Therefore suggests HL may not be purely advantageous - there could be downsides ✘ Lateralisation changes with age -Szaflashi et al; Split Brain research -There are occasions when it is possible to observe the effects of severing the Corpus Callosum e.g epileptic patients to prevent seizures crossing to the other hemisphere
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Split Brain Research
-Sperry and Gazzaniga -Brain is cross-wired but SBP cannot experience this; info processed in RH cannot be passed to the left -SBP shown image to right visual field would correctly report seeing anything; the info was processed in the RH, where there is no language centre -SBPs shown image to left visual field would not report seeing anything - info was processed by in the RH, where there is no language centre and the CC is severed so info cannot pass to the LH - However, they could respond non verbally by pointing If 2 symbols presented simultaneously on either side of the visual field; -if they had to draw what they had seen with left hand, they would draw the left visual field symbol This matches hemispheric lateralisation; LH is where language centre is and RH is where visual-motor cortex is Therefore LH is responsible for speech and lang, RH for visual However does not conclude that there are specific regions for specific tasks; rather that there are general regions It also stresses the importance of connectivity between regions, as much as the operation of individual parts
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Split Brain Research AO3
✘ Research suggests language may not be restricted to LH after all -JW; LH was severely damaged, demonstrates the ability for RH to develop language functioning to the extent he can now speak about info presented to the left or right hemisphere -Some studies appear to support Equipotentiality Theory Therefore research may be limited and cannot be applied to all ✘ Limitation in the validity of split brain research -Split brain procedure is so rare Therefore Sperry and Gazzaniga's research involved a sample of just 11 -As a result, there is no way to ensure the sample being used are not anomalies with confounding disorders that make the result invalid Therefore such research provides interesting insights, the numbers involved are too small to be taken seriously ✘ Forms a theory of healthy brains based on research surrounding epileptic brains.
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Plasticity
the brain's ability to modify its own structure and functioning through experience
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What affects plasticity
Life experiences -new experiences strengthen the neural pathways being used, while rarely used pathways die out Therefore brain constantly adapts to a person's environment by strengthening the most often used pathways and neglecting the unimportant ones -Cognitive functioning naturally declines with age
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Davidson et al - Tibetan Monks
-compared 8 practitioners of Tibetan meditation with 10 student volunteers with no previous meditation experience -both groups fitted with electrical sensors and asked to mediate for short periods -the electrodes picked up much greater activation of gamma waves in the monks compared to only a slight increase in the students Therefore meditation not only changes the workings of the brain in the short term but may also produce permanent changes as the monks had increased gamma wave activity even before they started
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Boyke - Juggling
-taught 60yr olds how to juggle and saw development of brain plasticity -grey matter in visual cortex increased though this change reversed when practice stopped
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Kuhn et al - video games
-video games require complex cognitive and motor skills -trained a group 30mins/day over 2 months on super Mario -Found significant increase in grey matter in areas such as the cortex, hippocampus and cerebellum compared to a control group Therefore concluded that such charges involved areass concerning spatial navigation, strategic plannng, working memory and motor performance; all needed for the game
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Maguire et al
found taxi drivers had significantly larger posterior hippocampus than control group members, and volume of the P.H was positively correlated with the amount of experience each taxi driver had.
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Plasticity AO3
✔Support from Kemperman's animal studies -Kemperman et al found rats housed in complex environment had increased number of new neurons in brains compared to controls in cages -The biggest development occurred in the hippocampus; neccessary for navigation and forming new memories Therefore suggests support for brain plasticity from multiple species, not just humans, strengthening its claim
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Functional Recovery
the brain's ability to recover abilities and mental processes damaged by brain injury or disease
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Three main mechanisms of functional recovery
Neuronal Unmaksing Axonal Sprouting Stem Cells
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Neuronal Unmasking
-Wall discovered 'dormant synapses'. They normal have too low a neural input to accurate, but when an area of brain nearby is damaged it can increase input to them; they become unmasked - This allows opening of connections to regions of the brain not normally activated, creating a lateral spread of activation - This, in time, can result in development of new structures
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Axonal Sprouting
In areas where links between neurons have been damaged, undamaged axons can grow ('sprouts') new axons and nerve endings to reconnect the neurons, thus regaining neural activity
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Stem Cells
unspecialised cells, with the potential to become many different types of cells with various functions, including nerve cells Therefore might help provide treatments for brain damage.
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3 Methods of stem cell use
Replace; they could be transplanted in, and directly replace dead/dying cells Repair; it is possible that these stem cells might secrete growth factors that somehow repair injured cells Reroute; some argue the transplanted cells form a 'neural network', linking an uninjured part of the brain (where the stem cells are made) to the damaged section
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Functional Recovery AO3
✔ Support from animal studies Tajiri et al -2 groups of rats with brain injuries. 1 group received transplant of stem cells into the affected area of the brain; the other received a placebo solution -3 months later, the stem cell group showed clear signs of developing new neuron-like cells in the area, along with a solid stream of stem cells migrating to the affecting area. Not the case in the control Therefore suggests the brain has the potential to recover from trauma with the use of stem cells ✘ Age differences Huttenlocher -fucntional plasticity reduces with age -however, studies have suggested that even abilities commonly thought to be fixed in childhood can still be modified in adults -yet the capacity for functional recovery is much greater in children than in adults as their brains are more plastic ✘ Educational attainment and fucntional recovery Schnieder et al -Found that patients with college education are 7x more likely than those who didn't finish high school to recover after a moderate to severe brain injury Therefore cognitive reserve could be a factor in neural adapation during recovery from brain injury X2 It may be that those who have spent more time in school have better paying jobs Therefore they can afford better quality healthcare to aid their recovery
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Postmortem
studying the brain after death -if patient displays abnormal behaviour, could help to identify physical damage
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Postmortem AO3
✔ Allows for more detailed examinations than simple scans ✔ Results in more impact on our understanding and future treatment ✘ The brain is dead at the time of study Therefore cannot observe how it functions when active ✘ Cause of death can impact on the brain and variables such as treatments, age and length of time between death and post-mortem also have an effect ✘ Retrospective as you cannot help the patient with what you find
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fMRI scans
-measures change of blood flow in particular areas of the brain -as oxygen is carried through the bloodstream, an active part of the brain will have increased blood flow -researchers can therefore set specific tasks and observe where what parts of the brain are used for that particular task
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fMRI AO3
✔ Non-invasive; no physical violation or exposure to radiation, and you don't have to die first ✔ Allows study of brian activity, rather than just structures ✘ Not directly measuring the brain - only blood flow to areas ✘ Only looks at lcoations in the brain, not communcation bewteen areas
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EEG
-measures electrical activity in the brain -electrodes placed on the scalp measure tiny electrical chages and these signals form an EEG -can be used to detect brain disorders like epilepsy, as well as disorders which affect brain functions like Alzheimer's 4 EEG wave patterns -Alpha; awake but relaxed, rythmical waves -Beta; arousal, low amplitude, fast frequently -Theta; first stages of sleep, slow and low frequency -Delta; deeper sleep, slower and lower
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EEG AO3
✔ Shows real-time brain activity, not a still image ✔ Useful for clinical diagnosis - records associated with neural activity ✘Only focuses on superficial regions of the brain- can't see what's going on in deeper areas such as hippocampus ✘ Electrical activity is picked up from neighouring electrodes - EEG's can't pinpoint activity precisely
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Event Related Potential (ERPS)
-makes use of EEG technology -tiny voltage changes in the brain, triggered by events -hard to pick out from all other electrical activity in brain; we have to repeatedly present the target stimuli and average out a response; the related activity will occur each time, unrelated will not
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Event Related Potential (ERPS) AO3
✔ Allows us to see how brain processing is affected by specific manipulations ✔ Directly measures brain activity ✔ Shows live brain activity ✘ Needs a lot of trials before useful data can be extracted ✘ Electrical activity deep in the brain is not recored - only strong surface level activity
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