Block 3 Flashcards

(171 cards)

1
Q

Definitive Host (DH)

A

a host in which the parasite develops to mature stage

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2
Q

Intermediate Host (IH)

A

a host in which a parasite develops to the infective stage of the DH

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3
Q

Paratenic Host (PH)

A

a host not necessary for the development of the parasite (enhances the chance that the parasite life cycle will be complete)

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4
Q

• biological vector

A

o host in which a protozoan parasite develops through either asexual or sexual multiplication
o transmits parasite via
mouth parts after biting a DH
feces deposited on DH as it is feeding

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5
Q

Infection

A

when endoparasites infect the host INTERNALLY

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6
Q

infestation

A

when ectoparasites attach to OR occupy the host

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7
Q

Self-limiting Infections

A

Development of species or strain specific immunity

  • Parasite is eliminated
  • Protection from subsequent infection
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8
Q

Prepatent Period (PPP)

A

Time from infection to when eggs, cysts, and larvae appear in body fluids or excretions from the DH.

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9
Q

Spirocerca lupi

A

o esophageal worm
o FH: dogs, wolves, lions, (L3); IH: dung beetles (egg with L1) ; PH: chickens, birds and lizards
o Site: stomach to esophagus, migrating though the thoracic aorta possibly leaving lesions.
o Size: adults, pink/red 8cm. eggs small+elongated 30-35 um

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10
Q

Toxocara canis

A

o common: roundworm, ascarid
o FH: dogs ZOONOSIS ; PH: rodents
o Modes of infection: per os (eggs L2), transmammary (L3), transplacental (prenatal)(L3)
o Site: Small intestine
o Size: large white 10-15cm. eggs dark brown, round 85x75 um

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11
Q

Toxocara cati

A

o common name: common round worm/ascarid
o FH: cats PH: rodents
o Modes of infection: transmammary, per os (egg L2), paretenic hosts.
o Site: small intestine
o Size: large, white 4-10cm. eggs 60x80um round and thick shelled

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12
Q

Toxascaris leonia

A

). Eggs reach infective stage in about a week. Eggs hatch in the stomach and L enter the mucosa of small intestine (develop and molt then return to lumen of the intestine to mature). If eggs are ingested by rodents, they hatch and encyst as infective arrested larva (no placental or mammary gland transmission. Prepatent period is 2-21/2 months!

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13
Q

Ancylostoma caninum

A

o common name: hookworm
o FH: dogs, foxes; PH:rodents
o modes of infection: percutaneous, per os, paratenic hosts, transplacental, transmammary. (L3)
o Site: small intestine

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14
Q

Diphyllobothrium and Spirpmetra (Diphyllobthriidae)

A
  1. Copepods as first intermediate host
  2. Dog eats fish, amphibian, or reptile as a second intermediate host
  3. Eggs contain first stage larva = Oncosphere
  4. Second larva = Proceroid, in the body cavity or tissue of the first intermediate host.
  5. Third larva = Plerocercoid, in the second intermediate host
  6. Definitive Host = ingested the plerocercoids (aquatic food chain)
    - - Most digested but left the scolex and the neck

Treatment: Praziquantel

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15
Q

Taenia Identification

A
  • 10-100 cm in length
  • Non-retractable rostellum with two rows of hooks (hooks use for firm attachment in theDigestive Tract
  • Rectangular segments
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16
Q

Taenia Life Cycle

A
  • Definitive Host = Gravid segments
  • Eggs = Onchospheres,
  • Ingestion of the intermediate host, the eggs hatch to give hexacanth embryo (spines on the surface)
  • Hexacanth embryo migrates to the liver and both of the skeletal and cardiac muscle
  • Second-stage larva infective to the definitive host (must eat the immediate host/rabbits)
  • Scolex attaches to the small intestine and begins to bud off segments
  • Second stage larva (found in the intermediate host) can include:
    1. Cysticerus (Single bladder with one scolices, T. hydratigena)
    2. Strobilocercus (elongated segment in the intermediate host, infection of a long time, T. taeniaformis = feline)
    3. Coenurus (Single bladder with many scolices, T. serialis)
    4. Hydatid (Formed by Echinococcus it can be either unilocular or alveolar)
  • Cysts (juvenile) is more infective stage than adults
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17
Q

Taenia hydratigena

A

Mammal host needed for the intermediate host of Cysticercosis. Taenia hydratigena (canine), the infective stage is the cysticercus migrates through the liver and encysts

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18
Q

Traumatic hepatitis

A

(condemnation of infected livers by meat inspectors)

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19
Q

Taenia ovis

A

Canine, Cysticerus infects the cardiac and skeletal muscles of sheep

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20
Q

Taenia pisiformis

A

Cysticerus is found in the liver and the peritoneal cavity of rabbits

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21
Q

Taenia saginata

A

Human, cysticerus found in the striated muscles of cattle, no hooks Eating beef, because it has no hooks, the human being cannot act like intermediate host)

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22
Q

Taenia solium

A

Human, people become infected by eating undercooked pork containing cysticerci (containing ones’ fingers with the infected feces)
 Person feces containing eggs that can reach the mouth (lapse in personal hygiene)

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23
Q

Taenia taeniaformis

A

Strobilocercus larva of the cat (definitive host)

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24
Q

Taenia multiceps:

A

Sheep, goats, cattle causes coenurosis (intermediate host because of present of conurosis)
 Cysts invade cranial cavity
 Neurological Signs (Blindness, incoordination, walking in circles)
 Many canids (definitive host)

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25
Taenia serialis:
Dogs and Foxes (intermediate host)
26
Echinococcus Granulosus | Identification
Identification: - 45 to 65 testes and genital pore is located at or posterior to the middle of the segment - Parasite of the Dog, coyot, wolf and dingo
27
Echinococcus Granulosus | Life Cycle
Larva = hydatid Cyst (Sheep, swine, cattle, humans, moose, caribou, kangaroos) - Hydatid = Sheep, fertile (intermediate host) - Hydatid = Cattle, Sterile (intermediate host) - Echinococcus granulosus granulosus: Sheep and human (most effected) - Echinococcus granulosus equinus: Horses, Donkeys, and Mules (intermediate host
28
Echinococcus Granulosus | Distribution
Endemic in North and South America, England, Africa, and Middle East - Non-infiltrative, non-fatal, small in cattle, massive in humans, Unilocular or alveolar
29
Echonococcus multilocularis | Identification
17-26 testes, | - Endemic north central Europe, Alaska, Canada, and central United States
30
Echonococcus multilocularis | Disease
Pressure atrophy of surrounding organs - Allergic reactions to fluid leaks - Hydratidosis - Unilocular hydrated cysts is the second stage larva of Echinococcus granulosus and is infective to dogs and other canids (definitive). Starting as an onchosphere, the larva grows slowly. As humans live longer, fertile hydatid may grow very large
31
Echonococcus multilocularis | Life Cycle
Alveolar Hydatid Cysts: - Second stage larva of E. Multilocularis (infective to dogs, foxes, and cats, which serve as a definitive host) - Develop in voles, lemmings, cattle, horses, swine, and humans - Fatal and Infiltrative - Contamination of pastures results in domestic ruminants and swine infections - Pastoral Cycle results from feeding uncooked meat to dogs and cats
32
Pastoral and Sylvatic
Pastoral and Sylvatic of E. granulosus and E. Multilocularis: Sylvatic cycles (Predator-Prey), reaches human through their domesticated animals. E. granulosus cycles are maintained in wild ruminants and wolves (Canada); Wallaboes and dingoes
33
Echonococcus | Control
Rid of stray Dogs - Anthelminitic Medication - Prohibition against feeding uncooked OFFAL to dogs and cats
34
Echonococcus | Treatment
- Benzimidazole carbamate derivatives - Mebendazole and albendazole are currently used for chemotherapeutic treatment of Alveolar Echinococcus, as well as of cystic hydatid disease - The only curative treatment of AE is still radical surgical resection of the parasite tumor, supported by pre and post-operative chemotherapy
35
Moniezia | Identifcation
- Looks like broken glass, if in dog don’t treat, he ate poo - Unarmed Scolices - Four Suckers - Very wide segments with bilateral genitalia - Characteristic egg shaped (triangle in shaped)
36
Moniezia | Transmission
Oribatid Mites
37
Moniezia | Life Cycle
In small intestine of cattle, sheep, and goats - Involve arthropod (oribatid mites and Psocopterans lice) as intermediate host (cysticercoid). Ingestion of infected arthropods results in infection of grazing animals
38
Dipylidium caninum, Diplopylidum spp., Joyeuxiella | Identification
4 Suckers - Retractable rostellum (thorn-like hooks) - Segment looks like cucumber seeds - Bilateral genital pores
39
Dipylidium caninum, Diplopylidum spp., Joyeuxiella | Life Cycle
Cysticercoids (infective stage) develop in fleas (Ctenocephalides spp.) and biting lice (Trichodectes canis) - Onchospheres (fully developed embryo) are ingested by the flea larva - After emergence of adult flea, hexacanth develops into cysticeroid - Onchospheres (fully developed embryo) are ingested by self grooming - Adult worms develops in the small intestine - Diplopylidium and Joyexiella develops in the Beetle, Reptiles and small mammals serve as a second intermediate host (paratentic host)
40
Hymenolepis diminuta | Identification
In Rodents and Sometime in dogs or humans - No hooks - Three testes and a single ovary
41
Vampirolepis nana | Identification
Rodents and Humans - Fleas and flour beets are intermediate host (cysticercoid) - Single circle of hooks - The cysticercoid can be found in the intestinal mucosa of the definitive host - Three testes and a single ovary - Interal-organisms infection (copies itself with the host)
42
Mesocestoididae corti
Identification: - 4 suckers - No hooks - Mediodorsal genital pore - Eggs accumulate in paruterine organ Life Cycle: - Infective stage for definitive host is tetrathyridium (Peritoneal cavity) - Arthropods intermediate host (cysticercoid)
43
Nematoda | Identification
Constant body form - Large body cavity (pseudocoelom) - Inelastic cuticle and pseudocoelom fluid enable the worm to maintain constant body diameter - Special way of movement (no circular muscle layer) - Cuticle is permeable to both water and gases (respiration) - Mouth with 3 lips (may have teeth)  Pharynx  Intestine - Feces expelled under pressure - No circulatory or respiratory organs - Muscular esophagus - Males are smaller than the female - Female reproductive system can be monodelphic, didelphic (two branches) , or multidelphic
44
Nematoda | Life History
Morula (one cell) and vermiform (i.e. microfilaria) embryo are important in diagnosis
45
Order Strongylida Superfamilies
1. Strongloidea 2. Trychostrongyloidea 3. Ancylostomatoidea 4. Metestronglyloida
46
Strongyloidea | Identification
- Well-developed buccal capsules (may or may not have teeth) - Stoma - Male nematodes of the order Strongylida have a caudal copulatory bursa, dorsal, lateral and ventral expansions of the cuticle (lobes), supported by muscular processes (rays) - The disposition and configuration of these rays are used in classification and identifcation of Strongyloids - Have dorsal and lateral lobes of equal size, thin, and flexible spicules
47
Strongyloidea | Life Cycle
- Free-living microbivorous first and second larval stages - Eggs contain morula stage when deposited and passed put with feces - Morula develops into first stage larva which gives second stage larva (still in feces; feed on bacteria) - Second stage larva undergo second molt to give a third-stage larva that continue to keep the cuticle of the second larva until entering a suitable - Third Stage larva begin to mirgrate out the fecal mass to be ingested by the grazing animal
48
Strongyloidea | Control and Treatment
Separate ill animals (anemia, diarrhea, weakness) |  Anthelmintic may fasten the death of very sick animals
49
Trychostrongyloidea | Identification
Small buccal capsule which may be equipped with a tooth or lancet in blood sucking species  Common and pathogenic in grazing ruminants (abomasum and small intestine)  Trichostrongylus: Hairlike, no capsule, short spicules
50
Trychostrongyloidea | Life Cycle
 Free-living microbivorous first and second larval stages.  Eggs contain morula stage when deposited and passed out with feces  Morula develops into first stage larva which gives second stage larva (still in feces; feed on bacteria)  Second stage larva undergo second molt to give a third-stage larva that continue to keep the cuticle of the second larva until entering a suitable  Third Stage larva begin to mirgrate out the fecal mass to be ingested by the grazing animal
51
Trychostrongyloidea | Disease
1. Infective third stage larva survive the winter on pasture 2. Ruminants are exposed to infection in spring 3. All larvae die off in summer 4. Eggs laying continue over the fall to give the new winter generation of larvae
52
Trychostrongyloidea | Specie
 Trichostrongylus axei: parasite of stomach (abomasum) or ruminants , horses, and leporids  Amidpstromum: parasite of geese and ducks (but not mammals) has large toothed buccal capsule) Ostertagia ostertagi 1. Cattle (Floatation Diagnosis) 2. Chronic Abomasitis 3. Most serious helminth parasite of cattle in the USA  Teladorsagia cirumcincta 1. Sheep and goat
53
Haeminchus | Identification
 Buccal cavity armed with lancet |  Asymmetrical dorsal ray and short, wedge-shaped spicules
54
Haemonchus Disease
Haemonchus contortus 1. Removal of the circulating erythrocytes volume from lambs  Anemia  High egg counts (10,000/gram)
55
Mecistocirrus | Identification
 Parasites of abomasum of ruminants and stomach of pigs in Central America  Suck blood and produces anemia and diarrhea
56
Cooperia
 Buccal cavity is very small, the spicules are short and blunted at their tops  Confused with Trichostrongylus  Accumulation of feces result in foul condition that attract blowflies (resulting in myiasis) on sheep sm intestine
57
Nematodirus | Identification
Triangular tooth  Female has a spine at the tip of tail  Infect domestic ruminants
58
Nematodirus | Life Cycle
 Larva develops to the infective third stage within the egg shell  Hatching depends on extrinsic stimuli (weather change)
59
Nematodirus | Disease
Neatodirus battus causes severe and debilitating diarrhea in lamb
60
Hystrongylus rubidus
Stomach of swine  Larva invade the gastric glands  Causes gastritis
61
Ollulamus trcuspos
Ovoviviparous  larvae mature in the stomach (single host, very rare)  Transmitted by ingestion of vomitus from an infected host  Causes chronic gastritis in cats (fatal)
62
``` Dictyocaulus viviparous (Lung worms) Identification ```
Found in respiratory passages of ruminants and horses  Lumen of the bronchial tree  Cause chronic bronchitis  Only nematode that reaches maturity in the ling of the cattle  Free living stages depends on stored energy  When ingested larva migrates through mesenteric lymph nodes
63
``` Dictyocaulus viviparous (Lung worms) Treatment ```
Rotate pasture
64
Dictyocaulus arnfieldi:
Pathogenic in horses but tolerated in donkeys
65
TREATMENT of Trychostrongyloidea
 Ruminants: 1. Thiabendazole, 2. Fenbendazole 3. Albendazole 4. Ivermectin 5. Dormactin 6. Livamisole 7. Mornatel
66
Stronglyoidea | Identification
Large buccal cavity surrounded by a buccal capsule  Well developed copulatory bursa  Prodelphic vulva (caudal end) is common
67
Strongylinae "Large stronglyes" | Identification
 The most destructive parasites of the horse are: (all three are blood suckers as adult worms in the cecum and colon) (larvae needed to differenciate between the three species) 1. Strongylus vulgaris 2. Strongylus edentates 3. Strongulus equinus
68
Strongylus vulgaris | Life Cycle
 Larvae undergo migrations that cause great damage  Infective larvae can resist cold weather and desiccation  The third stage infective larvae enter the wall of the cecum and ventral colon upon ingested a horse (MORE MIGRATION Larvae)  Fourth larvae penetrate the colic and cranial mesenteric arteries
69
Strongylus vulgaris | Pathogenicity
Larval migrations damage the cranial mesenteric artery and its branches
70
Strongylus Treatment
Ivermectin and Albendazole
71
Strongylus edentates and Strongylus equinus
Adults are twice as large as S. vulgaris  Adults are more pathogenic and more difficult to treat  Third stage larvae of S. edentates reach the liver through the portal veins, molt in the hepatic parenchyma  The third stage larvae S. equinus molt in the wall of the cecum  Fourth stage larvae reaches the right half of the liver, enter the pancreas or abdominal cavity, adults enter intestinal lumen
72
Cyanthostominae | Identification
 Small strongyles  Small buccal cavity  Distinct inner and outer lead crowns  Common in horses
73
Cyanthostominae | Disease
Arrested larvae in large number cause disease = Cylicocyclus insigne
74
Cyanthostominae | Treatment
Not affected by treatment (arrested larvae); dexamethasome (adults)
75
Treatment Cyanthostominae
Let horses exercise in no grass areas (no contamination) 2. Periodic anthelmintic 3 Benzimidazole 4. Piperazines 5. Febantel 6. Ivermentin 7. Fenbendazole 8. Selective Treatment: method for avoiding the development of resistance
76
Oesophagostominae | Identification
Parasites of large intestine of ruminants, wine and primates  Known as NODULAR WORMS  Calcified nodules interfere with the intestine function
77
Oesophagostominae | Diseases
Oesophagostomum columbianum 2. Chabetria ovina 3. Oseophagostomum radiatum
78
Stephanures dentatus | Identification
 The Kideny worm of swine (females form cysts in the kidney fat)  Parasite of hepatic, renal, and perirenal tissue  Cup shaped buccal cavity  Stephanures dentatus and Cyathostoma: Earthworm serve as intermediate host (facultative become a must)
79
Stephanures dentatus | Infection
 Direct Ingestion  Indirect Ingestion  Penetration of third-stage larvae
80
Stephanures Dentatus | Disease
Liver Damage
81
Staphanus Dentatus | Treatment
Fenbendazole |  Ivermectin
82
Parasites of upper respiratory tract. EW serves as IH
Syngamus and Cyathostoma = birds | mammonogamus - mammals
83
Ancylostomatidae (Hookworms) | Identification
```  Parasite of the small intestine  Anterior end of the worm is “hooked”  Ancylostomatinae parasitize carnivorous hosts  Bumostominae parasitize herbivorous  Omnivorous are parasitized by both ```
84
Ancylostomatidae (Hookworms) | Life Cycle
 Free living microbivorous first and second larval stages  Eggs contain morula when deposited and passed out with feces  Third stage larva begin to migrate out the fecal mass to be ingested by the grazing animals
85
Ancylostomatidae (Hookworms) | Species
 Ancylostoma braziliense: one pair of teeth (Dogs)  Ancylostoma duodenal: two pairs of teeth (Human)  A. tubaeforme: three pairs of teeth (cat)  A Caninum: three pairs of teeth (Dogs)  Globocephalus urosubulatis: No teeth, no plates (Swine)  Placoonus lotoris: Five articulating plate (Raccoons)  Bunostomum: Ruminants  Bathmostomum: Elephants  Grammocephalus: Elephants and rhinoceroses  Necator: Human
86
A. Caninum Characteristics
(sharp teeth and more pathogenic)
87
A. Caninum Life Cycle
 Infection occurs through ingestion or penetration  Arrested larvae later becomes active to either mature into adults (intestine) or shed from mammary glands (pups infection) and placenta
88
A. Caninum Diseased
 A. caninum is more pathogenic Kennel and Pet shops accumulation of feces  Peracute hookworm disease: larvae passed through milk to pups  Acute hookworm disease: 1. Exposure of older pups to large numbers of infective larvae  Chronic hookworm: Asymptomatic (cont. transmission)
89
A. Caninum Treatment
Creeping eruption  Caused by migrating L in human  Blood Transfusion
90
Metastrongyloidea | Identification
Parasites of the respiratory, vascular, and nervous system of mammals  Required snail or slug intermediate host  Infective stage: develops in the earthworm
91
Metastrongyloidea | Life Cycle
Metastrongyloidea eggs laid can contain from a single cell embryo  Molluscan or annelid (earthworm) intermediate hose (first to third infective)  Infection occurs through ingestion of the intermediate host  EXCEPTION TO THOS ARE THE FILAROIDES OSLERI AND FILAROIDES HIRTHI (both can directly infect the dogs in the first larval stage)
92
Metastrongylidae
 Metastrongylus (only genus) 1. Bronchi and bronchioles of swine 2. Trilobed lips flanking the mouth 3. Earthworm (paratenic host) 4. Not very pathogenic, oviparous
93
Metastrongylidae | Treatment
Fenbendazole | 2. Ivermectin
94
Protrongylidae: | Identification
Parasite of sheep and goats |  Eggs deposited in lung, vascular or neural tissues, egg develop into L1 before feces
95
Protostrongylus rufescens (Sheep)
Live in the smaller bronchioles  Comb like spicules  Treatment: Fenbendazole
96
Muellerius Capillaris
Tiny, deeply embedded in lung tissue or reactive nodules  Moxidatin (Sheeps): heavy infection)  Eprinomectin (Goats): (Heavy Infection)
97
Parelaphostrongulus tenuis
 Parasite of the meninges of the white-tailed deer (Odocoileus virginianus)  Abnormal hosts (sheep, goats, camel, moose) tends to invade nervous system
98
Crenosoma vulpis
In the bronchi and bronchioles of foxes, raccoons, and dogs
99
Troglostrongylus
Felidae
100
Aelurostrongylus abstrusus | Identification
Lung parenchyma in cats  Eggs are deposited in “nests” (oviparous) that appears as nodules  Symptoms: Coughing, anorexia, dyspnea, and polynea, many remain free of clinical signs
101
Aelurostrongylus abstrusus | Treatment
Ivermectin 2. Selamectin 3. Fenbendazole: Paralysis, treatment can kill host
102
Angiostrongylous Vasorum:
Pulmonary arterial tree of dogs |  Disease: Pulmonary Thrombosis and interference with clotting leading to multiple subcutaneous hemorrhages
103
: Angiostrongylus cantonensis
Lingworm of the rat |  Causes eosinophilic meningitis and encephalomyelitis in humans (eating infected raw snails, slugs)
104
Filaroides osleri and F. hirthi
 Occur in nodules within the epithelium of the trachea and bronchi or w/n the lung parenchyma  Ovoviviparous directly infectiveaL1 larva  No intermediate host
105
Filaroides osleri and F. hirthi | Signs and Treatment
▪ F hirthi: normally no clinical signs; fatal in severely stressed and immunodeficient animals ▪ F. osleri: Dry cough, chronic disease ▪ F. hirthi :Albendazole, ivermectin ▪ F. osleri: Thiacetarsemide injection, Thiabendzole, fenbendazole, ivermectin; no satisfactory treatment ▪ Filaroides hirthi (reaction to dead and dying worms); Filaroides osleri (right) nodules near tracheal bifurcation
106
Rhabditida | Identification
Small nematodes  Parasites of lower vertebrates or invertebrates  Rhabditis, Halicephalobus, and Strongyloides parasitize domestic animals
107
Rhabditis (Pelodera) strongyloides: | Identification
Produce a pruritic, hyperemic dermatitis in cattle, swine, dog, horses and rodents  (Damp straw bedding causes dermatitis in canine that caused by this parasite)
108
Halicephalobus (Micronema) Deletrix: | Identification
 Rhabtitoid esophagus ; only one egg in uterus  Highly pathogenic parasite of horse and man  Equine  Fatal in humans
109
Strongyloides | Identification
Name mean round and compact |  Female lies deep in the mucosal crypts of the alimentary tract (sm. Intestine)
110
Strongyloides stercoralis
Humans dogs and cats
111
S. papillosus
Ruminates
112
Life Cycle of Strongyloides:
 Parasitic male do not exist  Parasitic females have no male gonads  Homogonic Rhabditiform: larvae in the external environment develop through two molts into infective filariform larvae or through four molts into free-living males and females  Filariform larvae penetrate the skin of suitable host, molt twice to give parasitic female
113
S. ransomi
Swine
114
S. westeri
horses
115
S. fuelleborni
African primates and humans
116
S. cebus
American primates
117
S. ratti and S. venezuelensis
Rats
118
Diseased caused by Stronglyloides
 Asymptomatic in most domestic specie  Anemia in Piglets, Diarrhea in fouls  Bronchoneumonia, diarrhea, hemorrhage
119
Control of Strongyloides
Cages and pens must be cleaned  Separate pups immediately after birth  Suitable anthelminitic
120
Oxyuris equi
Pinworm | Highly specific parasite of the larger intestines
121
Oxyuris Equi | Diagnosis
 Cellophane tape against the anus and then stick it to a slide  Disposable cloth to clean perineum of the horse
122
Oxyuris Equi | Treatment
Ivermectin |  Pyrantl tartrate
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Ascaridida | Identification
 Intestinal tract of domestic animals  Most common of all nematodes  Infective stage of development take place inside the egg  Parasitize aquatic vertebrates (have free-swimming larval stages and intermediate host)
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Ascaridida | Two types
Terrestrial | Aquatic
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Ascaris Suum | Identification
 Eggs hatch in the stomach and small intestine to give infective L3 which enter the wall of the cecum and colon and proceeds to the liver through the portal vein  Larvae out into the alveoli, reach the pharynx to be swallowed (mature in the intestine) how pigs gat a. sum
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Ascaris suum | Disease
Migration cause mechanical damage and allergic inflammation (eosinophilic inflammation)  Milk spot (fibrosis of the liver)  Condemnation  Pigs: Server respiratory problems
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Ascaris Suum | Treatment
Pyrantel tartrate can kill infective larva  Fenbendazole  Ivermectin  Piperazines
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Parscaries equorom | Identification
Found in horse |  Liver and lung damage results in chronic reduction
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Parascaries equorom | Results
Malnourished
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Parascaries equorom | Treatment
Paralysis the worms may cause obstruction of the bowel  Difficult to destroy eggs  Pyrantel tartrate  Somatic migration
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Toxascaris leonine | Identification
 Parasite of dogs and cats (cool climate) |  No placental or mammary gland transmission
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Toxocara canis | Identification
Canine (CNS invasion)(intermediate host)
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Toxocara canis | Life Cycle
 Larvae that undergo tracheal (newborn puppy) migration develop to sexual maturity  Somatic migration results in arrested lavae  Placenta and mammary glands transmissions  Tracheal ends in mature worms  Pups can be born with infection  Arrested larvae “encysted” in paratenic host  More pathogenic than Leonia in definitive host
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Toxocara canis | Control of Canine and Feline Ascarid Infection
 Sanitation  Clean all surfaces  Keep in dogs and cats indoor
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Toxocara canis | Treatment
Piperazine  Fenbendazole  Cesarean section of cats  No placental transmission
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Human Toxocarosis (Visceral larva migrans) (T. Canis):
Nodules in liver, lungs, kidneys, and brain  Larvae may cause granulomatous retinitis  Eating contaminated Soil
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T. Cati
less important as a cause of visceral larva migrans
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Baylisascaris spp | Identification
 Common in North American wildlife |  CNS invasion
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: Baylisascaris procynoins of the raccon | B. Columnaris of the skunk
Raccoons can be treated using anthelmintics active against T. Canis: 5 eggs cause death in humans
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Baylisascaris procyonois
Causes human visceral larva migrans |  Larvae grow as they migrate
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Camallanina (Dracunculus) and Spirurina | Identification
Require either an insect or crustacean intermediate host to develop into infective stage  Definitive host become infected through ingestion of intermediate host or paratentic host
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Dracunculus | Identification
Guinea worm |  Parasite of subcutaneous tissue of carnivorans and man
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Dracunculus | Life Cycle
Indirect  Larvae discharged into the water and when ingested by Cyclops (copepod) develop into infective stage  Definitive host infective by drinking water
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Dracunculus insignis
raccoons, dogs, cats in North America  Frogs as paratenic host  TX: winding the worm up on a stick
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Dracunculus medinensis
Man in the Middle east
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Spirurina | Identification
Six superfamilies which are parasite of domestic animals | 2. Characterisitcs stoma and surrounding structures
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Gnathostomatoidea
Doughnut-shaped collar of spines around the oral openings  Adults are found in cystic nodules  Amphibins, snakes, and fishes as parantenic host  Cystic nodules may break open into peritoneal cavity (severe inflammation called peritonitis, fatal)  May migrate to the brain  Tx: unknown
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Physalopteroida
 Parasite of the stomach of carnivorans (stomach worm)  Adults live in the stomach with the anterior end embedded in the muscosa  Eggs are passed in the feces. The eggs, which contain larvae, are ingested in the beetles  Cause bleeding in the stomach
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Thelaziidae
Parasites of conjunctival and lacrimal sacs of domestic animals (Eye worm) - - North American specie: Thelazia caiforniensis - - These larvae develop into infective larvae in the face fly (Musca autumnalis)
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Spiruroidea: Gongylonema
 Mucous membrane of the host esophagus (G. pulchrum) or rumen (G. verrucosum)  Infective: in dung beetle or cockroach
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Spirocera lupi
 Found in nodules in the esophagus wall or stomach  Eggs contin vermiform larvae/embryo Disease:  Dysphagia and vomiting
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Habronematoidea
Parasite of the equine stomach  If deposited in wounds will not complete the life cycle: Results in cutaneous granulomas
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``` Dirofilaria immittis (dog heartworm) Life Cycle ```
-- Transmitted by blood sucking insects where microfilaria develop into infective L3 -- Microfilaria circulate in the blood -- Adults are found in the pulmonary arteris -- Dead worms are carried deeper into the lungs -- Mosquitoes as vector
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``` Dirofilaria immittis (dog heartworm) Disease ```
- - Illness start with the appearance of microf. In blood with physical obstruction of vessels, heart chambres, and valves - - Pulmonary hypertensions and right heart failure - - Dogs appears fatigue, cough,
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``` Dirofilaria immittis (dog heartworm) Diagnosis ```
Microfilariae in blood (Dipetalonema reconditum)
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``` Dirofilaria immittis (dog heartworm) Control and Treatment ```
- - Ivermection and Milbemycin oxime - - Prevention of Macrocylic lactone - - Heartworm disease in cats. When dealing with cat infection, be aware that cats carry very few worms amicrofilaramic, no enough circulating antigens.
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Setaria labiatoppilloa and S. equine
 parasites of the serous membranes of cattle and horses  Migrating L may invade CNS causing neurological disease  Adults found moving in the horse eye anterior chamber
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Onchocerca spp
Black Flies | TX: Ivermectin
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Onchocerca cervicalis
Found in the nuchal ligament of the horse
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Onchocera. gutterosa
in cattle I nuchal ligament (connective tissue)
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Onchocera. linalis
connective tissue between the spleen and the rumen of cattle
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Parafilaria multipapillosa and Parafilaria bovicola
 Horses and cattle respectively  Subcutaneous and inter-muscular connective tissues causing bleeding from nodule through a tiny pore (Summer Bleeding)  Horses bleed when under sunlight
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Dipetalonema reconditum
 Canine  Hooked on end  arised from Drofilarie immitis (HW)
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Elaophara schneideri
 Deer, elk, and sheep  Carotid, iliac, and mesenteric arteries  Tabanids
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Elaeophara schneideri
Cattle |  Horn fly (Haemotobia irritans) ventral midline dermatitis
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Dioctophyme renale
Giant Kidney worm”  Min are principle definitive host  Brownish, thick-shelled eggs,  Bell shapes, eggs passes in urine  Eggs are infective to obligovhate annelid worms  Oligochates are ingested by fish or frogs
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Stichosome esophagus
Capillary tube surrounded by glands (Stichocyte)
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Trichinella spiralis:
 embedded in the mucosa of the small intestine of swine, carnivorans, and man  Found in the cysts in the striated muscle  Both sexes reach maturity within two days of eating infected meat  Host digestive enzymes liberate L from cysts  Raw or undercooked pork: Human infection  Treatment: Albendazole
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Trichuris (Whip-shaped)
embedded in the large intestine  lemon shaped eggs  free in the lumen
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Trichuris suis
in young cause diarrhea, anorexia and retardation of growth Control: Separating animals from eggs
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Trichuris vulpis
Treated with Fenbendazole (Panacur)