CAD/ACS Flashcards

(36 cards)

1
Q

CAD

A

Atherosclerosis of coronary arteries
“Hardening of the arteries”

Due to damage to vessel

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2
Q

Risk factors of CAD
Non modifiable

A

Age: 55+ women 45+ men
FH

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3
Q

Risk factors of CAD
Modifiable risks

A

Tabacco use
Sedentary lifestyle
stress
HTN
High cholesterol: total:200+ LDL:130+ triglycerides 150+
Obesity

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4
Q

Cholesterol

A

Total cholesterol normal: <200

HDL:
Females: >50+
Males: >40+

LDL: <100

Triglycerides: <150

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5
Q

Dietary interventions for hyperlipidemia
LDL

A

activity
Avoid trans/saturated fats and cholesterol in foods

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6
Q

Dietary interventions for hyperlipidemia
Increase HDL

A

Physical activity
Healthy fats
Modify ETOH intake
Smoking cessation
Weight loss

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7
Q

Dietary interventions for hyperlipidemia
Lower triglycerides

A

Control blood sugar (if DM)

Avoid excessive ETOH and refined sugars

Physical activity

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8
Q

Tx for hyperlipidemia: drug therapy

A

On these meds for lifetime

Types:
Statins (lower LDL raise HDL)
Niacin
Fibrates
Bile acid sequestrants
PCSK9 inhibitors

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9
Q

Statins

A

First line tx (best chance of reducing risk)

Lower LDL and triglycerides
Small increase in HDL

AE:
-Muscle aches
-Rhabdomylosis (DARK URINE, muscle pain, high CK) STOP STATIN
-Increase in AST/ALT

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10
Q

Niacin

A

B vit
Lower LDL and triglycerides
Increases HDLs

AE: flushin, pruritus

Taken if statin doesnt help enough
(For cholesterol)

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11
Q

Fibric acid derivates (gemfibrozil)
And
Bile acid sequestrants (cholestyramine)

A

Both work in the gut

Also given if statin doesnt help enough

(For cholesterol)

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12
Q

PCSK inhibitors (evolocumab)
And
Ezetimibe

A

Just know their also for cholesterol

Added to statins

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13
Q

Angina

A

Chest pain from myocardial ischemia
(Increase o2 demand or decrease o2 supply)

Blockage causes symptoms

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14
Q

Precipitating factors of angina

A

Physical exertion
Temp
Emotions
Consuming heavy meal
Tabacco
Sex
Stimulants

(Any thing to increase demand of O2)

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15
Q

Chronic stable angina

A

Episodic pain

Pattern: provoked by stress and activity

Improves with rwst or NTG

No longer than 15 mins

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16
Q

Unstable angina

A

New or change pattern

Occurs at rest
Does not imporve with NTG or rest

Emergency!

17
Q

Prinzmetals angina

A

Caused by coronary vasospasm

Comorbid with migraines, raynauds, heavy smoking

18
Q

Diagnostic studies for CAD

A

12 lead ECG

Labs: cardiac enzymes

Stress test: see if activity causes symptoms

Cardiac catheterization

19
Q

Cardiac enzymes

A

Markers of injury

CK total: nonspecific, any muscle injury

Specific:
CKMB: ⬆️ 4-8 hours, peals 12-24 hours
Troponin: ⬆️ 3-4 hours peaks 4-24 hours

Get 3 sets of cardiac enzymes 3-6 hours apart

20
Q

Cardiac catheterization
Gold standard
PCI
Complications

A

Gold standard to localized CAD for pt with worsening angina symptoms

PCI (percutaneous coronary intervention) can be done if a block is found
(stent placement)

Complication’s: infection, poke hole in heart, bleeding

21
Q

Cardiac cath nursing care
Pre and post procedure

A

Pre procedure:
Allergy: shell fish (contrast dye)
Lab studies: bleeding, kidney function

Post procedure:
Monitor: bleeding, dysrhythmia, chest pain

22
Q

Nitroglycerin (short acting nitrate)
Why important
MOA
Route
Other use
Cause

A

First line for acute angina

MOA: vasodilations
Route: SL 1 tab every 5 mins, mac 3 then call EMS
Spray can be on or under tongue

Used Prophlactically in stable angina before activity

HA common (Tylenol), watch BP

23
Q

Topical NTG

A

Nitro ointment (nitro-paste)
Ordered by inch
Wear gloves

Transdermal patch
Apply over non-hairy area, occlusive dressing

24
Q

Long acting nitrates

A

Used to reduce frequency
Tx for prinzmetals angina
Orthostatic hypotension

Ex: isosorbide

25
Antiplatelet therapy Med and dose Why given Risk Use What else we can give if not tolerant
Low dose ASA (81mg) daily Decrease platelet to prevent clots Bleeding risk Used for primary prevention Or can give clopidogrel (plavix) if ASa intolerant
26
MI/ACS
Prolonged ischemia Non-ST elevation (NSTEMI): Not as much of a emergency ST-segment-elevation MI (STEMI): emergency right now Need pci within 90 mins
27
ACS clinical manifestations: MI
Severe chest pain not relieved by rest , NTG: Heaviness, pressure, crushing Radiate (jaw,neck,shoulder) Diaphoresis Tachycardia Palor Low grade fever, N/V
28
ACS clinical manifestations: MI As it progresses
Low BP LOC weak pulse, oliguria
29
MI diagnositic studies Test Gold standard to localize the lesion
ECG: show ST elevation Cardiac biomarkers: increased CKMB, Troponin Gold standard to localize the lesion: cardiac catheterization
30
MI: intitial management Goal MONA
Goal: decrease O2 demand/increase O2 supply Semi fowler Bed rest Cardiac monitoring MONA: morphine, oxygen, nitroglycerin, ASA Asses pain/VS Plan for cardiac catheterization
31
MI med management
Antiplatelet therapay (ASA, clopidogrel) Anticoagulation (Heparin/LMWH) Cardiac cath/PCI May require CABG Rural hospital may use thrombolytic meds to break up clot due to lack of resources
32
CABG Coronary artery bypass graft
Placement of arterial or venous grafts to bypass coronary blockage Usually: internal mammary, saphenous, or radial artery Requires STERNOTOMY
33
CABG postop care
Assess: bleeding, fluid status, dysrhymthmias Sternotomy site and harvest site would care Pain management Prevent VTE Resp tx: splinting, IS
34
Post MI nursing management
Cardiac monitoring VS Labs/ECG Bedrest 12-24 hours Heparin drip Pain tx Oxygen
35
MI complications
Dysrhythmias HF: pulmonary congestion, dyspnea, edema Cardiogenic shock: decompensated Increased HR Decreased BP ***New murmur is emergency post-op*** Will need surgery
36
Discharge teaching
Lifestyle mods Cardiac rehab Likely to have another