Cardiac. 42, 43, 44 Flashcards

(40 cards)

1
Q

Cardiac Glycoside

A

Digoxin

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Anti-Anginal

A

Nitroglycerin

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Thiazide Diuretic

A

Hydrochlorothiazide

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Loop Diuretic

A

Furosemide

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Potassium-Sparing Diuretic

A

Spironolactone

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Beta,1 Blocker: Antihypertensive

A

Metoprolol

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Alpha-Adrenergic Blocker: Antihypertensive

A

Prazosin HCI

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Alpha, 2 Agonist: Antihypertensive (centrally acting)

A

Clonidine HCI

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Angiotensin Antagonist (ACE Inhibitor): Antihypertensive

A

Lisinopril

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Angiotensin II Receptor Blocker (ARB): Antihypertensive

A

Losartan Potassium

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Calcium Channel Blocker. Benzothiazepine: Antihypertensive

A

Diltiazem HCI

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Calcium Channel Blocker. Dihydropyridines: Antihypertensive

A

Amlodipine

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Tx: HF, Atrial Fibrillation.

A

Digoxin

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Control Angina Pectoris (Anginal Pain)

A

Nitroglycerin

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Increase Urine Output. Tx: Hypertension, Edema from HF, Hepatic Cirrhosis, Renal Dysfunction.

A

Hydrochlorothiazide

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Increase Urine Output. Tx: Fluid Retention/ Fluid Overload caused by HF, Renal Dysfunction, Cirrhosis.

A

Furosemide

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

Increase Urine Output. Tx: Hypertension, Acute Pulmonary Edema.

18
Q

Increase Urine Output. Tx: Fluid Retention/ Overload associated w/ HF, Hepatic Cirrhosis, or Nephrotic syndrome.

A

Spironolactone

19
Q

Control Hypertension

20
Q

Control Hypertension, Refractory HF

21
Q

Tx: Benign Prostatic Hypertrophy

22
Q

for Hypertension

A

Clonidine HCI

23
Q

for Hypertension & HF

A

Lisinopril HCI

24
Q

for Hypertension

A

Losartan Potassium

25
for Hypertension (SR form) & Angina Pectoris
Diltiazem HCI
26
IV form for Cardiac Dysrhythmias (Atrial Fibrillation)
Diltiazem HCI
27
for Mild-Moderate Hypertension & Angina Pectoris
Amlodipine
28
Inhibits Sodium-Potassium ATPase, promoting increased force of Cardiac Contraction, Cardiac Output, & Tissue Perfusion. Decreases Ventricular Rate.
Digoxin (MOA)
29
Decreases Myocardial Demand for Oxygen. Decreases Preload by Dilating Veins, indirectly decreasing Afterload.
Nitroglycerin (MOA)
30
Action is on the Renal Distal Tubules, promoting Sodium. Potassium, & Water Excretion & Decreasing Preload & Cardiac Output.
Hydrochlorothiazide (MOA)
31
also Decreases Edema. Acts on Arterioles, causing Vasodilation, thus decreasing Blood Pressure
Hydrochlorothiazide (MOA)
32
Inhibition of Sodium & Water Reabsorption from Loop of Henle & Distal Renal Tubules. Potassium, Magnesium, & Calcium also may be Excreted.
Furosemide (MOA)
33
Acts on distal Renal tubules to promote Sodium & Water Excretion & Potassium Retention.
Spironolactone (MOA)
34
Promotes Blood Pressure Reduction via Beta1-Blocking effect.
Metoprolol (MOA)
35
Dilates Peripheral Blood Vessels by Blocking Alpha-Adrenergic Receptors.
Prazosin HCI (MOA)
36
Alpha2 Agonists Decrease Sympathetic Response from Brainstem to Peripheral Vessels. They stimulate the Alpha2 Receptors, decreasing sympathetic Activity; Increases Vagus Activity; Decreases CO; & Decreases Epi., Norepinephrine, & Renin Release. Resulting in Reduced Peripheral Vascular Resistance & Increased Vasodilation.
Clonidine HCI (MOA)
37
Ace Inhibitors inhibit ACE, which in turn inhibits the formation of Angiotensin II (Vasoconstrictor) & Blocks release of Aldosterone (promotes Na+ retention & K+ excretion). When aldosterone is blocked, Na+ is excreted along w/ Water & K+ is Retained.
Lisinopril (MOA)
38
Prevent release of Aldosterone (sodium-retaining hormone); act on renin-angiotensin-aldosterone system (RAAS). ARBs block angiotensin I receptors found in many tissues, causing Vasodilation & decrease Peripheral Resistance.
Losartan (MOA)
39
Slow calcium channels are found in Myocardium & Vascular Smooth Muscle. Free calcium increases Muscle Contractility, Peripheral Resistance, & BP. Calcium Channel Blockers block the calcium channel in the VSM, promoting Vasodilation.
Diltiazem Amlodipine (MOA)
40
Decreases peripheral vascular resistance (vasodilation).
Diltiazem Amlodipine (MOA)