Cardiac Rhythm Disturbances - Johnston Flashcards

(52 cards)

1
Q

Sinus arrhythmia

A

Rate changes based on respiration

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2
Q

Wandering pacemaker

A

Multiple atrial foci, different P wave morphologies

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3
Q

Atrial escape beat

A

Pause of unhealthy SA node –> ectopic focus fires (different shape P wave)

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4
Q

Junctional escape beat

A

Pause of unhealthy SA node –> ectopic AV junction focus fires (giant QRS, no P wave)

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5
Q

Atrial escape rhythm

A

Sinus arrest –> 60-80 beats/min, different P wave morphology than normal

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6
Q

Junctional escape rhythm

A

Sinus arrest –> 40-60 beats/min, no P waves or inverted P waves AFTER QRS

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7
Q

Ventricular escape rhythm

A

Sinus arrest –> 20-40 beats/min, no P waves

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8
Q

A ventricular pacemaker focus may ultimately cause what phenomenon?

A

Stokes-Adams syndrome (transient syncope via insufficient cerebral perfusion)

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9
Q

Premature atrial beat

Treatment?

A

EARLY, unique P wave –> normal, aberrant, or no QRS (depends on how early)

  • IF symptomatic – Beta blocker (Metoprolol)
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10
Q

How does the heart respond to a single premature atrial beat?

A

The SA node is depolarized, then starts again at the same rate as before (RE-SETS)

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11
Q

Explain possible QRS’s after a premature atrial beat (3)

A
  • TOO early = refractory ventricles = no QRS
  • EARLY = 1 bundle branch refractory = ABERRANT QRS
  • OK = normal QRS
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12
Q

Ventricular beats appearing in groups of 2 or 3, with the last one early and a different P wave

A

Atrial Bigeminy or Trigeminy (ectopic focus tied to SA rhythm somehow)

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13
Q

EKG sign of significant myocardial hypoxia/ischemia

A

Premature/Ectopic ventricular beats

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14
Q

Premature ventricular contraction

Treatment?

A

WIDE QRS w/o a P wave, inverted T wave, pause afterward

IF symptomatic…Metoprolol (Amiodarone/Lidocaine if really unstable)

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15
Q

Why is an ectopic ventricular QRS wider than a normal one?

A

Conduction in myocardium is slower than in the conduction system

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16
Q

What to avoid administering w/ ectopic ventricular beats/rhythms?

A

SNS stimulants or K+ wasting drugs

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17
Q

Ventricular beats appearing in groups of 2 or 3, with the last one wide, early, and without a P wave

A

Ventricular Bigeminy or Trigeminy

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18
Q

Ventricular Parasystole

A

Dual QRS rhythms at once via 2 independent pacemakers (SA and ectopic)

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19
Q

PVC falling immediately after a normal QRS (where T wave would be)

Significance?

A

“R on T” - HIGH RISK for ventricular tachy-arrhythmia

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20
Q

Multiple different QRSs of different morphologies and varying rates

Risk?

A

Multifocal PVCs

RISK of V. FIB.

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21
Q

Barlow Syndrome

A

Multifocal PVCs caused by MITRAL VALVE PROLAPSE

22
Q

Tachycardia, rate 150-250, same PQRS every time

Treatment?

A

Paroxysmal Atrial Tachycardia (PAT) - ectopic focus overrides SA node

  • Magnesium sulfate + CCB (Verapamil), then Amiodarone/Adenosine instead of Magnesium
23
Q

Tachycardia (150-250), 2 P’s for every 1 QRS

A

PAT w/ AV Block - DIGITALIS TOXICITY + HYPO-K+

24
Q

Tachycardia (150-250), inverted Ps (or none), normal QRSs

A

Paroxysmal Junctional Tachycardia (PJT) - ectopic focus overrides SA node

25
Tachycardia (150-250), no Ps, abnormal QRSs
Ventricular tachycardia
26
What can often be seen occasionally w/ V. tach?
Capture (normal QRS btwn VT) or Fusion beats (1/2 and 1/2)
27
What is Supraventricular Tachycardia? Treatment?
Any tachycardia arrhythmia (not normal) that originates above the ventricular tissue (PAT or PJT or AVNRT) -- Tx = Adenosine
28
When suspecting VT, what MUST you rule out?
SVT + BBB/aberrant conduction --> wide QRS's like VT | - NOT TREATED THE SAME AS VT
29
How to distinguish VT from Wide QRS SVT?
Wide SVT = no RAD or coronary disease | VT = coronary disease, captures and fusions, EXTREME RAD
30
Cause of Torsades de Pointes
- Low K+, K+ blockers, and/or long QT (syndrome or drug)
31
Good Dx tool for atrial flutter Best leads to see it?
Vagal Maneuver (help slow AV node to show more flutter waves) 2, 3, AVF
32
Ventricular flutter
Smooth, sine-wave appearance (as opposed to wide QRSs), same size every time
33
Problem with V. flutter if not treated
PROGRESS to V. FIB due to coronary blood insufficiency
34
Flutter rate
250-350 beats/min
35
Fibrillation appearance
Random, super-fast, jagged beats w/ changing amplitudes and no recognizable pattern
36
What will be seen on long-term EKG w/ untreated V. FIB?
Diminished amplitude over time as heart dies
37
Immediate treatment for V. FIB
Defibrillation + CPR
38
Upward-curving R waves ("delta waves")
Wolff-Parkinson-White Syndrome (via Bundle of Kent)
39
V1-V3...RBBB (RSR') w/ ST elevation Treatment?
Brugada Syndrome (congenital) ICD placement
40
T wave inversion in V2 and V3 (upward) Cause? Treatment
Wellens Syndrome (congenital) LAD stenosis (stent or bypass)
41
QT interval greater than 1/2 the length of the cycle
Long QT syndrome (congenital)
42
COPD - EKG findings
- Low voltage - RAD - MAT (multiple p wave morphologies, abnormal rates)
43
Pulmonary embolus - EKG findings
``` S1 Q3 T3 - Large S in lead 1 - Large Q in lead 3 - Inverted T in lead 3 RAD ST depression in lead 2 T wave inversion in leads V1 - V4 ```
44
Flatter, wider P wave Wide QRS Peaked T waves
Hyperkalemia
45
``` Flat or inverted T waves U waves (if severe) ```
Hypokalemia
46
Hypercalcemia
Short QT
47
Hypocalcemia
Long QT
48
Sinus tachycardia Causes? Treatment?
Elevated HR, normal PQRST - Emotion, anxiety, fear, drugs, hyperthyroid, pregnancy, anemia, CHF, hypovolemia - Treat underlying cause
49
Sinus bradycardia
Slow HR, normal PQRST (
50
Tachycardia, followed by bradycardia (cycle)
Sick Sinus Syndrome
51
When to treat sinus bradycardia? How to treat? (preferred)
HR
52
SVT vs. VT
SVT - normal narrow QRSs | VT - wide QRSs