Cardio, Resp, Neuro Flashcards
(111 cards)
Marburg heart score
Rule out ACS
1 point each:
- Woman >64 or man >54
- Known CAD, cerebrovascular disease, peripheral vascular disease
- Pain worse on exertion
- Pain not reproducible on palpation
- Patient assumes cardiac cause
Score 2 or less - 98% non cardiac
Risk factors for ACS
Maori/Pacific
Male>female
Increasing age
First degree relative with cardiac/vascular event age <60
Smoker
Diabetes
HTN
Obesity
Hyperlipidaemia
ECG findings for STEMI
STE 1mm in 2+ limb leads
STE 2mm in 2+ precordial leads
STE >0.5mm in posterior leads
New LBBB
Sgarbossa criteria:
Positive QRS, concordant ST elevation 1mm
Negative QRS, discordant ST elevation 5mm
V1-V3, concordant ST depression 1mm
Discordant ST elevation at least 25% amplitude of preceding S wave
Types of ACS
STEMI - STE ECG changes, positive troponin
NSTEMI - other/no ECG changes, positive troponin
Unstable angina - new onset severe angina, prolonged angina at rest, increasing frequency/duration/lower threshold angina, angina after recent MI
Management of ACS + contraindications
Acute referral cardiology
Aspirin 300mg stat
IV morphine - if required for pain
O2 - if sats <93%, cardiogenic shock, respiratory distress
GTN - contraindicated in right ventricular, inferior MI, recent use of PDE5-i (sildenafil), hypotension, severe AS
Antiemetic if nausea/vomiting
Risks for aortic dissection
STEMI
Smoking
HTN
Connective tissue disorder (Marfan, Turner, Ehlers-Danlos)
Atherosclerosis
Iatrogenic injury, blunt trauma
80% mortality - 1/5 prehospital, 1/3 peri/post-op
Symptoms and signs of aortic dissection
Tearing thoracic pain radiating between scapula
SOB, palpitations, syncope, collapse
Hyper/hypotension, pulse delay, tachycardia, diastolic murmur, asymmetrical BP
Horner’s syndrome possible
Management of aortic dissection
No anticoagulants
BP control - aim 100-110 systolic
O2 if hypoxic
Urgent referral to vascular
Risk factors for infective endocarditis
Prosthetic valves
Rheumatic heart disease
Prev endocarditis
Immunocompromised
Unrepaired cyanotic congenital heart disease
Repair of congenital heart disease within 6 months
IVDU - suspect in right heart endocarditis
Antibiotic prophylaxis for endocarditis for at risk patients
Amoxicillin/clindamycin/clarithromycin 60mins PO/30 mins IM/ immediately IV prior to tonsillectomy or any dental procedure
Duke’s criteria for endocarditis diagnosis
Pathological criteria:
Microorganism isolated from specimen vegetation/abscess
OR specimen of vegetation/abscess showing active endocarditis
Clinical criteria:
2 major
OR 1 major + 3 minor
OR 5 minor
Major:
- 2 positive blood cultures with typical organisms, 12 hours apart/1 positive culture with Coxiella buretii
- Evidence of endocardial involvement (new murmur, cardiac mass, abscess, valve dehiscence on echo)
Minor:
- Fever >38 deg
- Vascular phenomena (Janeway lesions, splinter haemorrhage, petechiae, systemic emboli)
- Immunologic phenomena (Osler’s nodes, Roth spots)
- Prev endocarditis or IVDU
- Microbiological evidence not meeting major criteria
Causes of myocarditis
Viral infection
Autoimmune disease
Drug hypersensitivity/toxic reaction
ETOH
Thyrotoxicosis
ECG changes in myocarditis
Diffuse ST concave elevation without reciprocal change
Mild left ventricular dilatation + mild AV block
Long QRS
Diagnostic criteria for pericarditis
At least 2:
- Chest pain (sharp, retrosternal, pleuritic, better leaning forward)
- Pericardial rub
- Pericardial effusion
- ECG - widespread ST elevation or PR depression
Causes of pericarditis
Idiopathic
Infective - viral/bacterial/TB
Non-infective
- rheumatological disorder - autoimmune/vasculitis/SLE/rheumatic fever
- cancer, paraneoplastic, radiotherapy
- trauma
- medications - hydralazine, procainamide, isoniazid, phenytoin
- recent MI (Dressler’s syndrome)
Signs of poor prognosis in pericarditis
Temp >38
Subacute course
TB or cancer
Large pericardial effusion
Failure to resolve after 7 days NSAIDs
ECG progression of pericarditis
- Diffuse ST elevation, PR depression, aVR reciprocal changes (2 weeks)
- Normalisation, T wave flattening (1-3 weeks)
- Deep, symmetric, diffuse T wave inversion (3- several weeks)
- Normalisation (several weeks +)
Common organisms in endocarditis
Staph aureus, Strep bovis, Strep viridans, Enterococci
HACEK - Haemophilus sp, Aggregatibacter sp, Cardiobacterium hominis, Eikenella corrodens, Kingella sp
Management of pericarditis
Discuss with cardiology
NSAIDs
Colchicine 0.5mg OD/BD to reduce recurrence rate (normally 20-50%)
NZ criteria for diagnosis of rheumatic fever
Definite ARF:
- GAS infection + 2 major criteria
- GAS infection + 1 major + 2 minor criteria
Probable ARF:
- GAS infection + 1 major + 1 minor criteria
Possible ARF:
- Strong clinical suspicion
Recurrent ARF:
- Prev hx ARF + GAS infection + 2 major criteria OR 1 major, 2+ minor
Major manifestations:
- Carditis - subclinical rheumatic valve disease on echo
- Polyarthritis or aseptic monoarthritis
- Chorea
- Erythema marginatum
- Subcutaneous nodules
Minor criteria:
- Fever
- Raised ESR/CRP
- Polyarthralgia (present in 75% cases, can be masked by NSAID use)
- ECG - PR prolongation
Risk factors for rheumatic fever
Maori (x20) /Pacific (x40)
Crowded household
Age 3-35
Low SES
Prev rheumatic fever
FHx ARF/RHD
Causes of heart failure -
Cardiac
Systemic
Cardiac:
MI, arrhythmia, valvular disease, cardiomyopathy, conduction disorders
Systemic:
Sepsis, hyperthyroidism, hypertension
Heart failure severity - New York Heart Association classification
Class I - no restriction to physical activity
Class II - Slight limitation to physical activities, ordinary activity results in some symptoms
Class III - Marked limitation to physical activities, less than ordinary activities cause symptoms
Class IV - No physical activity, symptoms at rest
Management of heart failure
IV access
O2
ECG
Fluid restriction
Inpatient management - diuresis